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   chronic inflammation 的翻译结果: 查询用时:0.184秒
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chronic inflammation     
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  慢性炎症
     In Hp infection group the scores of chronic inflammation were 1. 635± 0. 376 , 1.808 ±0. 301 (P> 0.05) and 1.661±0.398,2.232±0. 335 (P< 0.01 ) in 0,5 and 0,10 years respectively;
     Hp长期阳性者5年前和现在及10年前和现在慢性炎症严重程度积分分别为1.635±0.376与1.808±0.301(P>0.05)和1.661±0.398与2.232±0.335(P<0.01);
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     CD8~+CD28~-T cells are positively correlated with chronic inflammation;
     CD8+CD28-T细胞可能与慢性炎症变化成正相关。
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     [Methods] immunohistochemical SP method was used to detect the expression of HPV16-E6 and it′s positive rate in 23 cervical infiltrating carcinoma patients, 25 chronic inflammation patients and 26 normal control.
     方法采用免疫组化SP法分别检测宫颈癌浸润癌23例、慢性炎症25例和正常对照组26例宫颈组织临床样本HPV16E6的阳性率检测及宫颈癌组织中HPV16-E6的表达。
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     In Hp reversed group the scores of chronic inflammation were 1. 684 ±0.369,1. 182±0. 396(P<0.01) in 0,5 and 0,10 years;
     Hp转阴者5年前和现在及10年前和现在慢性炎症严重程度积分分别为1.684±0.369与1.369±0.426(P<0.05)和1.647±0.389与1.182±0.396(P<0.01);
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     Result:Colon chronic inflammation had 409cases(62.9%),and the age between 21~50 was the highest morbidity rate ;
     结果结肠慢性炎症409例(62.9%),年龄在21 ̄50岁发病率最高;
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  慢性炎
     【Results】 Positive rate of HTERT in NPC was 78.6%,significantly higher than 15.8% in chronic inflammation of nasopharyngeal epithelium(P<0.01).
     【结果】①鼻咽癌组织hTERT的阳性率为78.6%,与鼻咽慢性炎性黏膜组织hTERT的阳性率15.8%比较差异具有显著性(P<0.01);
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     Result:The expression of p53 protein was 9.5% in chronic inflammation of nasopharyngeal epithelium(CINE),which was significantly lower than it in NPCs with 76.3%(P<0.01).
     结果:鼻咽部慢性炎性组织中p53阳性率为9.5%,明显低于NPC组织中的阳性率(76.3%)(P<0.01)。
短句来源
     Results Positive rate of HTERT in NPC was 83 7%(70/84),while positive rate of HTERT in dysplasia of nasopharyngeal epithelium and chronic inflammation of nasopharyngeal epithelium both were 0 0%(0/8 and 0/16) (P<0 01),there are significant differences for HTERT in positive rate of clinical TNM stage group and for HTERT in positive intensity of lymph node metastasis group (P<0 05).
     结果 ⑴鼻咽癌组织HTERT的阳性率为 83 4% (70 /84) ,鼻咽粘膜上皮非典型增生组织及慢性炎性黏膜组织HTERT的阳性率均为 0 0 % (0 /8及 0 /16) (P <0 0 1) ; ⑵鼻咽癌临床TNM分期组间HTERT定性表达率存在显著差异 (P <0 0 5 ) ;
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     Results: The positive signal of HPV was mainly located in the nuclei of the cells. 48.81% (41/84)of squamous cell carcinoma(SQCC) and 6.12%(6/49) of chronic inflammation tissue were positive for HPV-DNA by in situ hybridization;
     结果HPV原位杂交阳性信号位于细胞核内,肺鳞癌HPV检出率为48.81%(41/84)、对照组黏膜慢性炎HPV检出率为6.12%(3/49),两组比较差异有统计学意义(P=0)。
短句来源
     Methods Immunohistochemical method was used to determine the expression of P63 and PTEN protein in 68 cases of NPC,25 cases of nearby mucosa of the carcinoma and 20 cases of chronic inflammation of nasopharyngeal mucosa. The relationship between the gene expression and clinicopathological features,metastasis was analyzed.
     方法应用免疫组织化学方法检测68例NPC、25例癌旁黏膜和20例鼻咽黏膜慢性炎组织中P63和PTEN蛋白表达,并分析其与临床分期、病理组织类型和转移的关系。
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  慢性炎症反应
     Conclusion:The production of the recombinant adenovirus vector encoding NF κB RNA aptamer provides a foundation for investigation of its activity and gene therapy for treatment of acute and chronic inflammation caused by NF κB over activity in clinic.
     结论 :产生具有生物学活性的重组腺病毒 Ad-aptamer,为下一步基因治疗因 NF- κB过度激活引起的急慢性炎症反应奠定基础
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     Conclusion Aspirin can change chronic inflammation in elderly patients with insulin resistance,and lower CRP,and improve insulin sensitivity.
     结论 阿司匹林可改变老年胰岛素抵抗患者慢性炎症反应 ,从而降低CRP水平 ,改善胰岛素抵抗
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     Conclusion:A low-grade chronic inflammation may be involved in the pathogenesis of ACIN.
     结论 :持续低程度的慢性炎症反应可能与ACIN的发病有关
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     Role of chronic inflammation in type 2 diabetes and obesity has been recognized in the past few years.
     慢性炎症反应在2型糖尿病和肥胖发生发展中的作用越来越受到人们的重视。
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     Results:Chronic inflammation occurred in liver of autoimmune mice and the serum titers of autoantibodies including anti dsDNA and antinuclear antibodies were significantly elevated.
     结果 :CJ致敏小鼠的肝脏组织发生了慢性炎症反应 ,血清自身抗体如抗核抗体和抗双链DNA抗体的水平明显升高 ;
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  慢性宫颈炎
     Results The positive rates of HPV16/18DNA and COX-2 protein in cervical adenocarcinoma were 65.1% and 86.0% respectively,which significantly higher than that in cervical chronic inflammation 8.3% and 33.3% (P< 0.01 ).
     结果HPV16/18DNA和COX-2蛋白在宫颈腺癌中的阳性表达率分别为65.1%与86.0%,均显著高于慢性宫颈炎组织8.3%与33.3%(P<0.01)。
短句来源
     The positive rate of HPV16/18-E6 protein in cervical adenocarcinoma was 65.1%,which significantly higher than that in cervical chronic inflammation 4.2%(P<0.01).
     HPV16/18-E6蛋白阳性表达率为65.1%,明显高于慢性宫颈炎组织4.2%(P<0.01)。
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     [Results] The positive rates of HPV16/18DNA and Skp2 protein in cervical adenocarcinoma were 65.1% and 68.6% respectively, which significantly higher than that in cervical chronic inflammation(P <0.01).
     结果宫颈腺癌组HPV16/18DNA与Skp2蛋白阳性表达率分别为65.1%和68.6%,均显著高于慢性宫颈炎组(P<0.01)。
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     The total coincidence rate of cervical pathological changes and chronic inflammation and early cervical carcinoma was 91.04%(305/335),97.84% and 75%,respectively.
     阴道镜诊断宫颈病变的总符合率为91.04%(305/335),诊断慢性宫颈炎的符合率为97.84%,诊断早期宫颈癌的符合率为75%。
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     The positive rate of p27(kip1) in cervical adenocarcinoma was 58.5%, which was markedly lower than that in cervical chronic inflammation(P<0.05).
     p27kip1阳性表达率为58.5%,明显低于慢性宫颈炎组织(P<0.05)。
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  chronic inflammation
In addition to having a key role in thrombogenesis, platelets are actively involved in acute and chronic inflammation: they induce the release of proinflammatory mediators, expose adhesion molecules, and recruit leukocytes.
      
Protease-antiprotease imbalance, generation of oxidative stress, and chronic inflammation are recognized as the principal mechanisms leading to irreversible airflow obstruction and parenchymal destruction in the lung.
      
Further characterizations of DN Treg cells are that infection indicates that MHV-3 could induce the chronic inflammation in livers of C3H/Hej mice.
      
When the endothelium is physically disrupted or functionally perturbed by postischemic reperfusion, acute and chronic inflammation, atherosclerosis, diabetes and chronic arterial hypertension, then completely opposing actions pertain.
      
Chronic inflammation or myocarditis and chronic alcohol abuse are considered two main etiologic factors in dilated cardiomyopathy.
      
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