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surfactant
相关语句
  表面活性物质
    Effect of Surfactant on Pulonary Homeostasis in the Mice
    肺表面活性物质对小鼠肺稳态性影响的研究
短句来源
    THE PATHOLOGICAL OBSERVATION OF EXPERIMENTAL SURFACTANT REPLACEMENT THERAPY.
    肺表面活性物质实验治疗的病理观察
短句来源
    Study of changes of pulmonary surfactant in respiratory distress syndrome and their mechanisms in rabbits
    呼吸窘迫综合征肺表面活性物质变化的探讨
短句来源
    Experimental Study of Pulmonary Surfactant Replacement Therapy and PEEP for the Respiratory Distress Syndrome
    羊水肺泡表面活性物质和呼气末正压通气治疗呼吸窘迫综合征的实验研究
短句来源
    SURFACTANT REPLACEMENT THERAPY FOR OLEIC ACID-INDUCED LUNG INJURY IN RABBITS
    肺表面活性物质替代疗法对兔油酸性肺损伤的治疗作用
短句来源
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  肺表面活性物质
    Effect of Surfactant on Pulonary Homeostasis in the Mice
    肺表面活性物质对小鼠肺稳态性影响的研究
短句来源
    THE PATHOLOGICAL OBSERVATION OF EXPERIMENTAL SURFACTANT REPLACEMENT THERAPY.
    肺表面活性物质实验治疗的病理观察
短句来源
    Study of changes of pulmonary surfactant in respiratory distress syndrome and their mechanisms in rabbits
    呼吸窘迫综合征肺表面活性物质变化的探讨
短句来源
    SURFACTANT REPLACEMENT THERAPY FOR OLEIC ACID-INDUCED LUNG INJURY IN RABBITS
    肺表面活性物质替代疗法对兔油酸性肺损伤的治疗作用
短句来源
    Experimental study on the treatment of smoke inhalation injury with lung lavage and exogenous pulmonary surfactant
    肺灌洗及外源性肺表面活性物质治疗烟雾吸入性损伤的实验研究
短句来源
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  “surfactant”译为未确定词的双语例句
    Objective To explore the changes of Clam cell protein (CC16) and the surfactant protein-D (SP-D) in the serum of patients with silicosis.
    目的探讨矽肺患者血清克拉拉细胞蛋白(CC16)和表面活性蛋白D(SP-D)的改变。
短句来源
    Alterations of surfactant protein A and B in the early stage of acute lung injury in rats
    急性肺损伤早期肺表面活性蛋白A、B含量的变化
短句来源
    Surfactant Protein A and Phospholipid Levels in BAL Fluids of Silicosis Patients
    早期矽肺BAL液中肺表面活性蛋白A和磷脂含量研究
短句来源
    Changes of Surfactant Proteins A and B After Lipopolysaccharide-Induced Acute Lung Injury in Rats
    肺表面活性蛋白A、B在内毒素性急性肺损伤时变化的研究
短句来源
    Nonspecific interstitial pneumonia, alveolar proteinosis, and abnormal proprotein trafficking resulting from a spontaneous mutation in the surfactant protein C gene
    非特异性间质性肺炎、肺泡蛋白沉积症及蛋白原运输异常均由于表面活性蛋白C基因的自发性突变
短句来源
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查询“surfactant”译词为用户自定义的双语例句

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  surfactant
The optimal constituents of microemulsion were found from pseudoternary phase diagrams: the mass ratio of Span80 to PS was 4:1 and complex surfactant to cosurfactant was 1:1.
      
Synthesis and surface-active property of bis-quaternary ammonium-sodium sulfate Gemini surfactant
      
N,N-dimethyldodecylamine, hydrochloride and epichlorohydrin (molar ration is 2:1:1) were used to synthesize bis-quaternary ammonium Gemini surfactant with a hydroxyl in its spacer group by the one-pot method.
      
This hydroxyl was sulfonated by chlorosulfonic acid and then neutralized to bis-quaternary ammonium-sodium sulfate zwitterionic Gemini surfactant.
      
The critical micelle concentration (cmc) and surface tension of the novel zwitterionic Gemini surfactant in aqueous solution at 15°C are 7.2×10-5 mol/L and 34.5 mN/m, respectively.
      
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The surfactant-deficiency model of rabbits were treated by exogenous surfactant in this study. Compared with controls, the rabbits receiving surfactant had higher PaO_2 and lower PaCO_2. The values of phospholipid increased and surface tension decreased in the lung lavage fluid and the lung-throax compliance increased(?)in animals treated with surfactant. Accrding to the size and degree of pulmonary atelectasis, amount and area of hyaline membrane in the patholgical examintion and the...

The surfactant-deficiency model of rabbits were treated by exogenous surfactant in this study. Compared with controls, the rabbits receiving surfactant had higher PaO_2 and lower PaCO_2. The values of phospholipid increased and surface tension decreased in the lung lavage fluid and the lung-throax compliance increased(?)in animals treated with surfactant. Accrding to the size and degree of pulmonary atelectasis, amount and area of hyaline membrane in the patholgical examintion and the degree of degeneration or necrosis of the Type Ⅱ pneumocytes and the degree of destruction of respiratory membrane in the electronmicroscopic observa- tion, the experimental group had slighter than controlgroup, but didn't completely recover in the treating group. Based on pathological and pa- thophysiological changes of this study, we mainly discussed about the mechanism of experimental therapy.

本实验用外源性肺表面活性物质治疗家兔肺表面活性物质缺乏模型。治疗组除较非治疗组的动脉O_2分压上升、CO_2分压下降;肺磷脂增高;肺表面张力降低及肺顺应性增加外,根据病理检查其肺萎陷的面积和程度,肺透明膜数量和范围以及在电镜观察中Ⅱ型肺泡上皮细胞的变性坏死和呼吸膜的破坏程度亦较非治疗组为轻,但与基本正常尚有一段差距。基于治疗前后病理及病理生理的变化,本文对实验治疗的机理等问题着重进行了讨论。

In the rabbit model of experimental respiratory distress syndrome(RDS) it was found that total phospholipids, phosphotidylcholine,and phosphotidylglycerol were decreased in the bronchoalveolar lavage fluid (BALF)and the amount of albumin and C-reactive protein was increased in BALF in the RDS rabbits,which was correlated with an elevation of the surface tension of BALF.With ruthenium red staining,the continuous and velvety structure of the alveolar surface active layer of RDS animals was seen being converted...

In the rabbit model of experimental respiratory distress syndrome(RDS) it was found that total phospholipids, phosphotidylcholine,and phosphotidylglycerol were decreased in the bronchoalveolar lavage fluid (BALF)and the amount of albumin and C-reactive protein was increased in BALF in the RDS rabbits,which was correlated with an elevation of the surface tension of BALF.With ruthenium red staining,the continuous and velvety structure of the alveolar surface active layer of RDS animals was seen being converted into aggregated or vacuolized forms scattered in the alveolar cavity.It was also found that pulmonary surfactant was prevented by fibrin from unfolding on the alveolar surface.These findings suggest that the abnormality of the quality,quantity and morphology of pulmonary surfactant plays a critical role in the pathogenesis of RDS and the proteinous exudate of the alveoli can destroy the structure and inhibit the function of pulmonary surfactant.

实验性呼吸窘迫综合征(RDS)兔支气管肺泡灌洗液(BALF)总磷脂,磷脂酰胆碱和磷脂酰甘油减少,白蛋白和C-反应蛋白升高,且与BALF表面张力增大相关。钌红染色见肺泡表面活性层失去连续、绒状结构,而以变性状态散落于肺泡腔。还见到纤维素阻拦肺表面活性物质(PS),使之不能向肺泡表面解聚的特殊现象。以上结果表明,PS质、量和形态结构的异常在实验性RDS发病中有重要作用;蛋白性肺泡渗出物能破坏或抑制PS的结构和功能。

cases of spontanous hemopneumothorax treated with operation are reported. One of them died of reexpansion pulmonary edema post-operativelly. Spontanoushemopneumothorax is usually resulted from the rupture of pulmonary bleb and lacerationof adhesive band. Two aspects should be emphasized in the treatment of spontanoushemopneumothorax. One is to secure life; the other is to reexpand the lung slowly andgradully in order to prevent reexpansion pulmonary edema.Reexpansion pulmonary edema after spontanous hemopneumothorax...

cases of spontanous hemopneumothorax treated with operation are reported. One of them died of reexpansion pulmonary edema post-operativelly. Spontanoushemopneumothorax is usually resulted from the rupture of pulmonary bleb and lacerationof adhesive band. Two aspects should be emphasized in the treatment of spontanoushemopneumothorax. One is to secure life; the other is to reexpand the lung slowly andgradully in order to prevent reexpansion pulmonary edema.Reexpansion pulmonary edema after spontanous hemopneumothorax is a rare butlife-threatenning complication. It was first described in 1875 by Foucart. The two majorfators causing reexpansion pulmonary edema are poulmonary atelectasis and the rapidreexpansion of the collapsed lung. The incidence of reexpansion pulmonary edema increases with the duration of lung collapse. Mime obseved an 85% incidence of reexpansionpulmonary edema after 7-8 days of lung collapse and 17% after 3 days in his experimentalstudies.Although the etiology of reexpansion pulm onary edema is unknown, several poten tialmechanism have been suggested, including increased permeability of pulmonarycapillaries. a decrease in periV'ascular pressure. application of a high negative.pressuresuction for thoracic cavity. decreased surfactant in the pulmonary atelectasis, or decreased lymph flow, etc.The course of reexpansion pulmonary edem a appears to be variable. In most instances,it has been described as a radiologic diagnosis without m ajor clinical conseq uences; however, it may be severe enough to result in a catastrophic respiratory failure that may prove fatal. More likely, tack of awar6ness of this complication may lead to a mistaken diagnosisand result in unnecessary and at times potentially dangerous diagnostic procedures andtherapeutic regimens.All above mentioned suggests that it is important to recognize spontanoushemopneumothorax and its corn plication-reexpan sion pulmonary edema and to master thediagnosis.cure and prevention of reexpansion pulmonary edema.

报告38例,均经外科治疗,37例痊愈,1例因术后发牛复张性肺水肿死亡.作者认为自发性血气胸的治疗以抢救生命为第一原则,有效止血、排气,维持生命体征平稳,在促使肺复张时应缓慢、逐步进行.以防发生复张性肺水肿.本文就自发性血气胸术后发生复张性肺水肿的发病机制以及对该并发症的诊治和预防方法作了初步分析.

 
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