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   benzene exposure 的翻译结果: 查询用时:0.08秒
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benzene exposure
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  苯接触
     Results The rate of GSTμ gene deletion in patients with severe benzene intoxication, moderate benzene intoxication, mild intoxication and benzene exposure, was 69.84%, 56.00%, 53.75% and 51.79%, which was significantly higher than that of the controls, 46.88%, P<0.05. The rates of GSTM1 gene deletion in the groups were increased with the severity of poisoning.
     结果对照组、苯接触组、轻度苯中毒组、中度苯中毒组、重度苯中毒组GSTμ 基因缺失频率分别为46.88 %、51.79 %、53.75 %、56.00 %、69.84 %。
短句来源
     Benzene exposure and lymphocytic DNA damage
     苯接触与淋巴细胞DNA损伤
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     Effects of Benzene Exposure with Different Concentrations on Workers' Health
     不同浓度苯接触对作业工人健康的影响
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     Objective To investigate the effects of benzene exposure on the expression of heat shock proteins(HSPs) and the probability of HSPs in plasma of workers as biomarkers.
     目的 了解苯接触对热应激蛋白(HSPs) 表达的影响以及HSPs 在苯毒性机理中的作用, 探讨HSPs 作为接触苯工人分子生物标志物的可能性。
短句来源
     The results showed that GSH Px activity of benzene exposure workers in blood was significantly higher than that of the control.
     苯接触组工人全血中GSH-Px 活性增高;
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     Results The rate of GSTμ gene deletion in patients with severe benzene intoxication, moderate benzene intoxication, mild intoxication and benzene exposure, was 69.84%, 56.00%, 53.75% and 51.79%, which was significantly higher than that of the controls, 46.88%, P<0.05. The rates of GSTM1 gene deletion in the groups were increased with the severity of poisoning.
     结果对照组、接触组、轻度中毒组、中度中毒组、重度中毒组GSTμ 基因缺失频率分别为46.88 %、51.79 %、53.75 %、56.00 %、69.84 %。
短句来源
     The author reviewed the relationship between genetic polymorphism of Ⅰ metabolic enzymes(CYP2E1,NQO1,MPO) and Ⅱ metabolic enzymes(GST,PST) involving benzene metabolite and interindividual variation in their genetic susceptibility to hematotoxicity from benzene exposure in this paper.
     本文就参与代谢的Ⅰ相 (CYP2E1、NQO1、MPO)、Ⅱ相 (GST、PST)代谢酶基因多态性与个体对致血液毒性遗传易感性的关系进行了综述
短句来源
     The mean diagnostic age of BP,benzene exposure duration,BMI and the proportion of genotypes of CYP2E1,GSTM1,GSTT1,MPO,and NQO1 were compared between WBC<4×10 9/L group and WBC≥4×10 9/L group. Logistic regression was used to set up the risk model to predict the prognosis of BP workers.
     [方法 ]对 12 0例曾经中毒者 ,按外周血白细胞计数 ,分为 <4× 10 9/L和≥ 4× 10 9/L两组 ,比较两组之诊断年龄、接工龄、体质指数 (BMI)均数和CYP2E1、MPO、NQO1、GSTM1和GSTT1各基因型的构成比 ,并用Logistic回归构建中毒预后的风险模型。
短句来源
     Similar negative correlation was also found between length of benzene exposure and red blood cell counts(r=-0.29,P<0.05).
     接工龄与红细胞计数也存在负相关 (r =- 0 .2 9,P <0 .0 5 ) ;
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     Benzene exposure and lymphocytic DNA damage
     接触与淋巴细胞DNA损伤
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  “benzene exposure”译为未确定词的双语例句
     Results The frequency of non-null GSTT1 gene in benzene poisoning workers with moderate benzene exposure level was higher than that in cases with lower benzene exposure(68.63% vs 38.00%,OR adj =4.32,95%CI 1.75~10.66,P=0.002).
     结果 中等强度接触组慢性苯中毒工人GSTT1基因缺失突变的发生率明显高于低接触组 ,差异有显著性 (6 8.6 3%vs38.0 0 % ,ORadj=4 .32 ,95 %CI 1.75~ 10 .6 6 ,P =0 .0 0 2 ) ;
短句来源
     Conclusions The increase of HSP70 may provide cells with toxic tolerance or cyto protection within a narrow concentration range of benzene exposure.
     结论 HSP70的增加可以提供一定程度的毒物耐受能力或细胞保护功能。
短句来源
     the level of TAC and the activity of SOD in high does group were gradually decreased with the time of benzene exposure;
     高剂量组 ,随着染毒时间的延长 ,SOD活性及TAC含量均逐渐降低 ;
短句来源
     Results Only a part of the population with the same level of benzene exposure would suffer from the disease, CBP cases with the same benzene exposure level exhibited different extents of severity of the illness. 78(89.7%) of the cases recovered completely, 8(9.2%) cases improved obviously, one(1.1%) died.
     结果同样接触水平的人群中只有部分发病,而病情严重程度不一,78例(89.7%)治疗后完全康复,8例(9.2%)明显缓解,1例死亡。
短句来源
     Besides,for 2 and 10 mmol/L benzene exposure,percentages of apoptotic cells in HSP70 group were higher than those in regular group except for 0 mmol/L benzene exposure.
     热应激组细胞除 0剂量染毒组外 ,其他剂量组细胞发生凋亡百分比均高于普通组。
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  benzene exposure
In this article, the author summarized the reports on the spermatotoxicity due to benzene exposure as well as presented an experience on the usefulness of ttMA biomarker determination on detection of spermatotoxicity due to benzene exposure.
      
Benzene exposure can result in chronic toxicity including spermatotoxicity.
      
A health hazard, specifically the leukaemia risk, is evaluatedfrom different sources of benzene exposure with relation to apopulation living in an urban area of Italy.
      
Benzene Exposure Assessment at Indoor, Outdoor and Personal Levels.
      
Lowest benzene exposure was found in the laboratory technicians, with a GM of 0.02 ppm.
      
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This paper describes a GC method developed for determining the content of benzene metabolite in human urine. 85% H_3 PO_4 solution was added to the Sample to hydrolyse the conjugated phenols, and the phenols freed were extracted by CS_2.The CS_2 extract was then subjected to GC assay of phenol and p-cresol. Experimental data show that long contact with benzene will cause a high phenol content in urine. The rate of extraction of phenols from aqueous layer to the CS_2 layer is found to be 28.85±1.07% and 66.68±0.92%...

This paper describes a GC method developed for determining the content of benzene metabolite in human urine. 85% H_3 PO_4 solution was added to the Sample to hydrolyse the conjugated phenols, and the phenols freed were extracted by CS_2.The CS_2 extract was then subjected to GC assay of phenol and p-cresol. Experimental data show that long contact with benzene will cause a high phenol content in urine. The rate of extraction of phenols from aqueous layer to the CS_2 layer is found to be 28.85±1.07% and 66.68±0.92% for phenol and p-cresol respectively.This method is recommended to be used in the measurement of phenol and p-cresol in urine, the result of which can be taken as an index of benzene exposure.

本文以气相色谱法测定人体中苯代谢物——尿酚,样品以85%H_3PO_4水解,然后游离酚萃取入CS_2层,取萃取液进样色谱仪作定量分析,测定数据表明,长时间与苯接触的人引起尿酚含量增加。从水层到CS_2层的萃取率:苯酚为28.85±1.07%,甲酚为66.68±0.92%。尿酚含量可作为与苯接触的指标,本文为调查苯污染情况提供了一简便可靠的方法。

The preventive effect of Photomagetic Quantum Bio-Information Therapy-Unit on animals experimentally exposed to benzene has been observed. The WBC counts of peripheral blood deceased dramatically in animals untreated with Therapy-Unit and remained unchanged in animals treated with Therapy-Unit. Experiment showed that photomagnetic Quantum Bio-Information Therapy-Unitmight have preventive effect on leukopenia induced by benzene exposure.

采用光—磁量子生物信息技术,预防苯染毒作用的动物实验结果表明:该项生物信息技术具有防止因苯染毒而导致的白细胞减少的作用。而未接受该项信息技术治疗的对照实验动物,则导致了白细胞减少。 我们曾用该项治疗技术,对21例因苯中毒而引起白细胞减少的患者进行了治疗。其治愈率为90.5%。 为了观察远期效应,我们于患者出院后280天进行了随访观察。结果表明,治愈后的患者白细胞总数仍维持在正常水平。可以认为,信息技术治疗效果是理想的。

Changes in micronuclei rates in peripheral blood lymphocytes of 63 workers exposed to benzene were observed and calculated, very markedly higher than those of 89 controls (P<0.0l); and, at similar concentration level of benzene exposure there, exist a sex difference, rates in female workers were very markedly higher than those in males (P<0.01).

报道63名苯作业工人外周血淋巴细胞微核率的变化,发现笨暴露者微核率非常显著地高于对照组(P<0.01),且接触相似浓度水平的笨作业者微核率变化存在着性别差异,女工徽核率高于男性工人(P<0.01),差异非常显著。

 
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