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   abnormal immunity 在 心血管系统疾病 分类中 的翻译结果: 查询用时:0.195秒
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abnormal immunity
相关语句
  异常免疫
    EFFECTS OF ABNORMAL IMMUNITY IN ITP PATIENTS ON MEGAKARYOCYTOPOIESIS
    ITP患者的异常免疫对骨髓巨核祖细胞的影响
短句来源
    These results indicate that SAA is pathologically heterogenous and particularly related to abnormal immunity and that once the abnormal immunity suppressed, SAA patients may get complete or partial recovery of hematopoiesis.
    这表明SAA确是一组异质性疾病,其造血功能衰竭与异常免疫关系密切,若去除这种异常,造血功能可获得部分甚至完全重建。
短句来源
    Objective A report of a group of patients with pancytopenia which might be related to abnormal immunity.
    目的 报告一组与异常免疫相关的全血细胞减少症。
短句来源
    Conclusion Immunorelated pancytopenia might be caused by abnormal immunity (especially autoantibody) mediated destruction or abnormal function of blood cells. It is beneficial to differentiate immunorelated pancytopenia from aplastic anemia or myelodysplastic syndromes.
    对肾上腺皮质激素为主的试验性治疗反应良好。 结论 该组疾病与异常免疫 (特别是自身抗体 )介导的造血细胞破坏或功能异常有关 ,暂称为免疫相关性全血细胞减少症 (immunorelatedpancytopenia,IRP) ,将其从再生障碍性贫血 (AA)、骨髓增生异常综合征 (MDS)中区分出来 ,患者可得到及时、有效的治疗 ,并且可减少误诊的AA、MDS。
短句来源
  “abnormal immunity”译为未确定词的双语例句
    Although its etiology is not clear, it is suggested that the abnormal immunity might play an important role in the development of AA.
    尽管其病因并不十分清楚,但临床上免疫抑制治疗能使大部分患者康复的事实说明免疫机制在AA的发生和发展中起极其重要的作用。
短句来源
    The pathogenesis of Henoch-Schonlein Purpura(HSP) has not completely known currently. However,more researches were designed for abnormal immunity and heritage genes of its pathogenesis,mainly concentrating on immunity after infection,humoral immunity,cellular immunity,abnormal cytokine and individual heredity susceptibility,etc.
    过敏性紫癜(HSP)的发病机制尚不十分清楚,但对其发病机制中的免疫及遗传基因异常已有较多的研究,主要集中于感染后免疫、体液免疫、细胞免疫及细胞因子异常、个体的遗传易感性等。
短句来源
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  abnormal immunity
Thus, considerable work remains to be done in determining the nature and cause of abnormal immunity in MCNS prior to investigating its potential role in the pathogenesis of proteinuria.
      
In addition to abnormal immunity, oxidative imbalance possibly existed in MAIDS model, as lipid peroxide increased significantly in spleen and whole blood glutathione peroxidase (GSH-Px) activity decreased markedly.
      
Unusual organisms, or unusual presentations, may prompt the clinician to look for evidence of abnormal immunity.
      


To evaluate more precisely the degree of bone marrow hematopoietic function recovery in patients with severe aplastic anemia (SAA) after intensive immunosuppressive therapy (IST), changes of bone marrow hematopoietic progenitor cells, including late burst-forming unit-erythroid (mBFU-E) and colony forming unit-granulocyte/macrophage (CFU-GM), of patients with SAA after 1ST were observed by using the technique of semisolid culture in vitro. The results showed that the levels of mBFU-E and CFU-GM decreased significantly...

To evaluate more precisely the degree of bone marrow hematopoietic function recovery in patients with severe aplastic anemia (SAA) after intensive immunosuppressive therapy (IST), changes of bone marrow hematopoietic progenitor cells, including late burst-forming unit-erythroid (mBFU-E) and colony forming unit-granulocyte/macrophage (CFU-GM), of patients with SAA after 1ST were observed by using the technique of semisolid culture in vitro. The results showed that the levels of mBFU-E and CFU-GM decreased significantly for all patients before IST as compared with the normal controls (P<0.001); after IST, 29 responding patients showed significant enhancement of mBFU-E and CFU-GM, which were correlated well with their clinical responses; the mBFU-E levels in 12 patients and CFU-GM levels in 10 patients recovered to normal, and both mBFU-E and CFU-GM levels in 8 patients recovered to normal simultaneously. These results indicate that SAA is pathologically heterogenous and particularly related to abnormal immunity and that once the abnormal immunity suppressed, SAA patients may get complete or partial recovery of hematopoiesis.

为更好地评价重型再生障碍性贫血(SAA)患者免疫抑制治疗(IST)后骨髓造血功能恢复程度,采用造血祖细胞体外培养技术,动态观察了48例接受IBT的SAA患者及20例正常对照者骨髓晚期红系爆式集落形成单位(mBFU-E)及粒-巨噬系集落形成单位(CFU-GM)的水平变化。结果表明,IST前所有SAA患者骨髓mBFU-E及CFU-GM水平均显著低于正常对照组(P<0.001);IST后1年,29例有效者骨髓mBFU-E及CFU-GM水平显著增高,增高程度与其临床疗效相关;12例mBFU-E及10例CFU-GM水平恢复正常,其中8例患者mBFU-E及CFU-GM水平同时恢复正常。这表明SAA确是一组异质性疾病,其造血功能衰竭与异常免疫关系密切,若去除这种异常,造血功能可获得部分甚至完全重建。

Objective A report of a group of patients with pancytopenia which might be related to abnormal immunity. Methods The clinical and laboratory features of 29 patients with positive bone marrow mononuclear cell(BMMNC) Coombs tests were analyzed. Results Most of these cases were pancytopenia with normal or decreased bone marrow cellularities and increased normoblasts. They were all found to have negative results of conventional hemolysis tests and hematopoietic nutrient determination and had no evidence...

Objective A report of a group of patients with pancytopenia which might be related to abnormal immunity. Methods The clinical and laboratory features of 29 patients with positive bone marrow mononuclear cell(BMMNC) Coombs tests were analyzed. Results Most of these cases were pancytopenia with normal or decreased bone marrow cellularities and increased normoblasts. They were all found to have negative results of conventional hemolysis tests and hematopoietic nutrient determination and had no evidence of malignant clonal hematopoiesis. However, all of them showed positive BMMNC Coombs test and good response to corticosteroids. Conclusion Immunorelated pancytopenia might be caused by abnormal immunity (especially autoantibody) mediated destruction or abnormal function of blood cells. It is beneficial to differentiate immunorelated pancytopenia from aplastic anemia or myelodysplastic syndromes.

目的 报告一组与异常免疫相关的全血细胞减少症。方法 分析 2 9例骨髓单个核细胞Coombs试验阳性的血细胞减少症患者的临床及实验室检查特征。结果 该组患者大部分表现为全血细胞减少 ;绝大部分患者骨髓增生良好 ,少部分增生减低 ,但红系比例不低或增高 ;常规溶血试验阴性 ;无造血原料缺乏证据 ;无异常克隆造血证据 ;骨髓单个核细胞Coombs试验阳性 ;对肾上腺皮质激素为主的试验性治疗反应良好。结论 该组疾病与异常免疫 (特别是自身抗体 )介导的造血细胞破坏或功能异常有关 ,暂称为免疫相关性全血细胞减少症 (immunorelatedpancytopenia,IRP) ,将其从再生障碍性贫血 (AA)、骨髓增生异常综合征 (MDS)中区分出来 ,患者可得到及时、有效的治疗 ,并且可减少误诊的AA、MDS。

The pathogenesis of Henoch-Schonlein Purpura(HSP) has not completely known currently.However,more researches were designed for abnormal immunity and heritage genes of its pathogenesis,mainly concentrating on immunity after infection,humoral immunity,cellular immunity,abnormal cytokine and individual heredity susceptibility,etc.A deeper understanding and illustration of HSP's pathogenesis are very important for searching new immunology and future gene therapy.

过敏性紫癜(HSP)的发病机制尚不十分清楚,但对其发病机制中的免疫及遗传基因异常已有较多的研究,主要集中于感染后免疫、体液免疫、细胞免疫及细胞因子异常、个体的遗传易感性等。深入了解并阐明HSP的发病机制对寻求新的免疫学和未来的基因治疗十分重要。

 
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