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apoptosis
相关语句
  凋亡
    Experimental studies of proliferation apoptosis and gene therapy with telomerase anti-sense oligonucleotides of glioma
    脑胶质瘤增殖、凋亡及端粒酶反义基因治疗的实验研究
短句来源
    THE STUDY ON AUTOPHAGIC APOPTOSIS INDUCED BY VINCRISTINE IN HUMAN LIVER CELL LINE L02
    长春新碱诱导人肝细胞系L02细胞自噬性凋亡的研究
短句来源
    Regulation of apoptosis in hepatoma cells by bcl-2 and bax and primary study of hepatoma apoptosis-related monoclonal antibody
    bcl-2和bax对肝癌细胞凋亡的调控及肝癌凋亡相关单克隆抗体初步研究
短句来源
    Effects of Sodium Orthovanadate on and Its Mechanisms in the Regulation of Proliferation, Cell Cycle Status and Apoptosis in Raji Cells
    酪氨酸磷酸酶抑制剂正矾酸钠对Raji淋巴瘤细胞增殖、细胞周期分布及凋亡的影响及机制探讨的实验研究
短句来源
    Effects of BCL-6 on Pathogenesis of Non-Hodgkin's Lymphoma and the Differentiation and Apoptosis of K562 Cells of Leukemia
    BCL-6与非霍奇金淋巴瘤的发生及白血病细胞K562分化和凋亡的关系
短句来源
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  细胞凋亡
    EFFECT OF HOMOHARRINGTONINE ON INDUCTION OF APOPTOSIS IN T-CELL LEUKEMIA WITH INVOLVEMENT OF GENE REGULATION AND ITS SIGNALING PATHWAY
    高三尖杉酯碱通过影响信号传导途径和基因调控作用诱导T-淋巴细胞白血病细胞凋亡
短句来源
    Effect of homoharringtonine on apoptosis in leukemic cells,related mechanism and clinical significance
    高三尖杉酯碱诱导原代白血病细胞凋亡、相关机理及临床意义
短句来源
    Regulation of apoptosis in hepatoma cells by bcl-2 and bax and primary study of hepatoma apoptosis-related monoclonal antibody
    bcl-2和bax对肝癌细胞凋亡的调控及肝癌凋亡相关单克隆抗体初步研究
短句来源
    DcR3 Expression in HCC and Its Value in Suppressing Fas-induced Apoptosis and Chemotaxis
    DcR3基因扩增和表达与肝癌细胞凋亡、化疗抗性关系的实验研究
短句来源
    Lack of expression of hMSH2 correlated with methylation of the hMSH2 promoter and apoptosis in cervical adenocarcinoma
    子宫颈腺癌组织基因hMSH2的表达、启动子甲基化及细胞凋亡研究
短句来源
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  “apoptosis”译为未确定词的双语例句
    Effects and Mechanisms of β1 Integrin Overexpression on Cell Cycle and Apoptosis in Human Hepatocellular Carcinoma Cells
    β1族整合蛋白过表达对人肝癌细胞细胞周期的调控及其机制研究
短句来源
    Generation of the Recombinant Adenovirus Expressing IκBαM Activated by a Tumor-Specific Promoter and the IκBαM Gene Expression Induces Apoptosis of HCC Cells
    靶向表达IκBαM重组腺病毒的构建及抑瘤作用的体外实验研究
短句来源
    The Expression of Genes Related to Gallbiadder Carcinoma and the Apoptosis Induced by Antisense Oligonucleotides Targeting Survivin and Bcl-2 on Gallbladder Carcinoma Cell
    胆囊癌相关基因表达及联合转染Survivin和Bcl-2反义寡核苷酸对胆囊癌细胞调亡的影响
短句来源
    Effects of SAO on Apoptosis Expression of Bcl-2、Bax in Glioma
    Survivin反义寡核苷酸(SAO)对Bcl-2、Bax表达的影响及相关性
短句来源
    Effect of Hepatocyte Growth Factor Gene Transfection on Cell Apoptosis and Invasion in Gastric Cancer Cell Line, MKN-45
    肝细胞生长因子基因转染对胃癌MKN-45细胞的影响研究
短句来源
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  apoptosis
Prodigiosin also could induce apoptosis of pancreatic cancer cells at low concentration and results in the fragmentation pattern of DNA.
      
All these results demonstrate that prodigiosin can obviously inhibit the proliferation of pancreatic cancer cells H8898 by arresting the cell cycle and inducing apoptosis.
      
Detection of the apoptosis of Jurkat cell using an electrorotation chip
      
The apoptosis of cells is one of the fields that attract increasing attention in biology today.
      
Usually, the cells are treated with chemicals when detecting apoptosis.
      
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The ultrastructural changes of the leukemia L-1210 cells treated by harringtonine are described in this paper. Under the action of harringtonine the degeneration process of injured L-1210 leukemic cells exhibited a form of coagulation necrosis... "apoptosis". The mitotic figures of L-1210 cells were decreased and the nuclei of interphase cells in L-1210 leukemia were disintegreted by the drug into several masses.

1.本文描述了三尖杉酯碱对小鼠L_(1210)腹水瘤细胞超微结构的影响。瘤细胞主要是以“凋落”(Apoptosis)的形式裂解死亡。并观察和讨论了这种坏死细胞的形态变化过程。2.三尖杉酯碱引起瘤细胞有丝分裂减少,从超微结构的变化分析,可能是由于药物引起分裂间期细胞的损伤,从而使进入分裂期的细胞数减少。3.通过观察三尖杉酯碱对瘤细胞超微结构的作用,我们对药物作用的原理进行了讨论,初步认为它可能是直接对核酸的结构与代谢发生作用,但也不能排除对蛋白质代谢的干扰。4.实验组细胞中A型病毒颗粒的减少,可能主要是瘤细胞受到药物的破坏,而间接影响了病毒的正常复制。

The most outstanding pathological changesof perinatal babies and children, based onthe review of the autopsy files in the 1stAffiliated Hospital of West China Universi-ty of Medical Sciences, are lymphocytic de-pletion, and reticuloepithelial cell swellingand/or fusiform malformation in the cortex;while in the medulla apoptosis is more pro-minent. We suppose that these alterationsare due to immaturity of cortical thymocy-tes, and in the diseased condition, they areeasily affected by extraordinary factors,...

The most outstanding pathological changesof perinatal babies and children, based onthe review of the autopsy files in the 1stAffiliated Hospital of West China Universi-ty of Medical Sciences, are lymphocytic de-pletion, and reticuloepithelial cell swellingand/or fusiform malformation in the cortex;while in the medulla apoptosis is more pro-minent. We suppose that these alterationsare due to immaturity of cortical thymocy-tes, and in the diseased condition, they areeasily affected by extraordinary factors, es-pecially the influence of corticosterone in-ducing acute severe necrosis, so the numberof lymphocytes are obviously diminished.But, in the medulla, as intact mature lym-phocytes exist, physiological phenomenonsuch as apoptosis is rather prominent in it.While in the medulla, Hassall's corpuscleshave various characteristic changes, such ascornification, calcification, fusion, cysticchange and disintegration. Besides, we ob-served a new alteration in the thymus de-fined as vacuolization. All the above pathological changes reachedthe peak in the 28- day group, there after,they might become either worse or betteraccording to the condition of the disease andgrowth of the body. However, these are stillproblems to be further studied separately.

作者对1002例围产儿和儿童尸检的胸腺进行了研究,发现最突出的病理学改变是胸腺皮质淋巴细胞减少和网状上皮细胞肿胀或梭形变,雨淋巴细胞的凋谢则以髓质显著。所有改变在28天组明显加重,以后或继续加重,或逐渐减轻。此外,还首次观察到了皮质的空泡化。

This article studied the effects of triptolide on DNA and RNA sysntheses of FGC_(85) cells by means of scintillation counting as well as their ultrastructural changes with electron microscopy. The experimcnts indicated that after 12 h of treatment with triptolide(0.51μg/ml),both FGC_(85) cells and their mitotic figures were markediy decreased.Under light microscope the majority of cells contracted and rounded,and died at last.Electron microscopic studies have showed that the last ultrastructural changes of FGC_(85)...

This article studied the effects of triptolide on DNA and RNA sysntheses of FGC_(85) cells by means of scintillation counting as well as their ultrastructural changes with electron microscopy. The experimcnts indicated that after 12 h of treatment with triptolide(0.51μg/ml),both FGC_(85) cells and their mitotic figures were markediy decreased.Under light microscope the majority of cells contracted and rounded,and died at last.Electron microscopic studies have showed that the last ultrastructural changes of FGC_(85) were identical with those of“apoptosis”discribed by Kerr et al. Because the mitotic cells didn t show any abnormal change,it seems that triptolide only acts on the interphase cells. Since the drugs,which can induce apoptosis,are different in mechanism,we paid more attention on the early ultrastructural changes induced by triptolide,and its effects on DNA and RNA precursor incorporation of FGC_(85) cells.After 2 h of treatment with triptolide,nucleolar changes of FGC_(85) cells took place.After 4~8 h,nucleolar alterations got drastic.They consisted of nucleolar degranulation, appearence of perichromatin granules and nucleolar fragmentation.Surprisingly,at the same time triptolide had not any effect on the mitotic figures and 3~H-TdR,3~H-UR incorporation of FGC_(85) cell. While under the same experimental condition cytosine arabinoside and aetinomycin D inhibited the incorporation of 3~H-TdR and 3~H-UR into FGC_(85) cells respectively. Therefore the prmary target of action of triptolide on FGC_(85) cells does not lie in the disturbance of nuclcic acid synthesis.Effects of triptolide on cellular membrane and protein synthesis of FGC_(85) cell remain to be studied.

本文应用核苷掺入技术及电镜观察首次证实雷公藤内酯对人胃癌细胞株 FGC_(85)的杀伤作用。用药早期,细胞数,分裂指数及 DNA,RNA 合成无明显变化,但出现核仁脱粒及核仁破碎等变化;晚期,电镜观察发现细胞以凋落方式死亡,药物主要作用于间期细胞,其杀伤机构的始动环节可能与核酸代谢障碍无关。

 
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