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organophosphorus insecticides     
相关语句
  有机磷杀虫剂
     The model was established about quantitative structure-toxicity relationships (QSTR) of a series of organophosphorus insecticides: logLD_(50)=-10.229 0-17.047 2 W_(0)+ 2.066 4W_(i)(n=47, r=0.944 4, s=0.262 8, F=182).
     通过多元回归分析,得到有机磷杀虫剂的定量结构-毒性相关模型:logLD50=-10.2290-17.0472W0+2.0664ΣWi(n=47,r=0.9444,s=0.2628,F=182).
短句来源
     Resistance Mechanisms to Organophosphorus Insecticides in Pardosa Pseudoannulata
     拟环纹豹蛛(Pardosa pseudoannulata)对有机磷杀虫剂的抗性机制
短句来源
     RESEARCHES ON ORGANOPHOSPHORUS INSECTICIDES. Ⅱ. O,O-DIALKYL S-ALKYL (SUBSTITUTED ALKYL) THIO METHYL PHOSPHORODITHIOATE
     有机磷杀虫剂的研究Ⅱ. O,O-二烷基S-烴基(取代烴基)硫甲基二硫代磷酸酯的合成
短句来源
     Gas Chromatography of 44 Organophosphorus Insecticides
     44种有机磷杀虫剂的气相色谱分析
短句来源
     SELECTIVE ORGANOPHOSPHORUS INSECTICIDES SYNTHESIS OF O,O-DIALKYL S-CARBOALKOXYALKYL PHOSPHORODITHIOATES
     选择性有机磷杀虫剂 O,O-二烷基 S-烷基酯基烷基二硫代磷酸酯的合成
短句来源
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  有机磷农药
     SYNTHESIS OF ANTIDOTES FOR ORGANOPHOSPHORUS INSECTICIDES——4-(t-BUTYL)-1-[(3-SUBSTITUTED ACYL) PYRIDINO]-METHYOXYMETHYLENE-PYRIDINIUM DIIODIDE
     有机磷农药解毒剂——4-叔丁基-1-[(3-取代酰基)吡啶基]甲氧甲基-吡啶季铵碘盐的合成
短句来源
     ISOLATION OF FLAVOBACTERIUM SP. P3-2 AND ITS PROPERTIES RELEVANT TO DEGRADATION OF ORGANOPHOSPHORUS INSECTICIDES
     黄杆菌P3-2的分离及降解有机磷农药的某些性质
短句来源
     Results: High recoveries of 8 organophosphorus insecticides fortified at 0. 01 ,0. 1 ,and 1 mg· kg levels were obtained. The limit of detection ranged from 0. 002 0 to 0. 016 5 ng.
     结果:8种有机磷农药(氧乐果、甲胺磷、敌百虫、乐果、甲基对硫磷、马拉硫磷、对硫磷、亚胺硫磷)在天花粉中的回收率添加浓度分别为0.01,0.1,1.0 mg·kg~(-1),最低检测量为0.0020~0.0165 ng。
短句来源
     Lightaddressable potentiometric sensor (LAPS) can be used to determine organophosphorus insecticides by immobilizing acetyl cholinesterase (AchE) on its Si3N4 film (H+ selective).
     通过将乙酰胆碱酯酶用戊二醛交联法固定在光寻址电位式传感器的H+敏Si3N4膜上,即可用于检测有机磷农药
短句来源
     In the mode of differential-signal determination, use of two biosensors makes it possible todetect organophosphorus insecticides such as dichlorvos at the level of 10-7 mol/L, with a linear range of 5 × 10-7~8 × 10-6 mol/L.
     采用差动检测方式,用两只传感器可测出10-7mol/L的敌敌畏等有机磷农药
短句来源
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  有机磷中毒
     A Clinical Analysis of 31 Cases With Severe Acute Organophosphorus Insecticides Poisoning
     急性重度有机磷中毒31例临床分析
短句来源
     Objective To explore the relationship between P-selectin and SAPS Ⅱ in aute organophosphorus insecticides poisoning(AOPP).
     目的探讨急性有机磷中毒(AOPP)患者血浆P-选择素、简化急性生理学评分系统(SAPSⅡ)变化及二者间关系。
短句来源
     Moreover, environmental temperature, the dosage and action time of toxic agents, sex differences and organophosphorus insecticides tolerance also influence organophosphorus insecticides poisoning-induced hypothermic response.
     此外,环境温度、毒剂剂量和作用时间、性别差异和有机磷杀虫剂耐受性也可影响有机磷中毒引起低温反应。
短句来源
     Conclusion Mechanical ventilation is an effective method to cure severity organophosphorus insecticides intoxication with respiratory failure.
     结论:机械通气是急性有机磷中毒并发呼吸衰竭的有效治疗方法,在应用过程中及时调整呼吸机参数,合理应用阿托品,防止并发症十分重要。
短句来源
     Insecticides especially organophosphorus insecticides such as methamidophos,parathion,and omethoate comprised a higher proportion,accounting for 86.02% of the pesticides poisoning.
     杀虫剂是引起农药中毒的主要类别,占86.02%,且以甲胺磷、对硫磷和氧乐果3种高毒类有机磷中毒为主。
短句来源
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  有机磷类杀虫剂
     among organophosphorus insecticides, phoxim toxicity was the smallest with root length EC50 of 879.81 mg/L and fenamiphos showed greatest toxicity of root length EC50 of 242.75 mg/L.
     有机磷类杀虫剂中,辛硫磷抑制效应最小,其EC50值是879.81 mg/L,虫胺磷抑制效应最大,其EC50值是242.75 mg/L。
短句来源
     Effects of Organophosphorus Insecticides(OPs)on G-protein Coupled Receptor Kinase 2-mediated Phosphorylation of M_2 Muscarinic Acetylcholine Receptors
     有机磷类杀虫剂对毒蕈碱乙酰胆碱M_2受体磷酸化的影响
短句来源
     Objective To explore the effect of organophosphorus insecticides(OPs) on G protein-coupled receptor kinase 2 mediated phosphorylation of M2 muscarinic receptors in vitro and to understand an alternative target of the OPs for human and other animals.
     目的研究有机磷类杀虫剂(OPs)对G蛋白结合受体激酶-2(GRK-2)介导的毒蕈碱乙酰胆碱M2受体(mAChR2)磷酸化的影响,进一步揭示OPs可能存在的其他作用途径。
短句来源
     Objective:To explore the effect of organophosphorus insecticides(OPs)on G-protein coupled receptor kinase 2-mediated phosphorylation of M_2 muscarinic receptors in vitro and to understand alternative target of the OPs for human and other animals.
     目的:研究有机磷类杀虫剂(OPs)对G蛋白结合受体激酶-2(GRK2)介导的毒蕈碱乙酰胆碱M2受体磷酸化的影响,进一步揭示有机磷类杀虫剂可能存在的其他作用途径。
短句来源
     Objective To explore the effect of organophosphorus insecticides(OPs) on G-protein coupled receptor kinase 2-mediated phosphorylation of m2 muscarinic receptors in vitro and to understand alternative target of the OPs for human and other animals.
     目的研究有机磷类杀虫剂对G蛋白结合受体激酶2介导的毒蕈碱乙酰胆碱m2受体磷酸化的影响和可能存在的其他作用途径。
短句来源
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      organophosphorus insecticides
    Improving the Selectivity of the Determination of Organophosphorus Insecticides and Carbamates by Enzyme Assay
          
    A method for the determination of some organophosphorus insecticides in human serum
          
    A simple and rapid method for the determination of organophosphorus insecticides in serum of poisoned patients is described.
          
    Determination of organophosphorus insecticides, their oxygen analogs and metabolites by high pressure liquid chromatography
          
    Several widely used organophosphorus insecticides, diazinon, methyl-parathion, fenitrothion, malathion, fenthion and methidathion were selected for inclusion in this work.
          
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    Dipterex is an organophosphorus insecticide, characterized by its anticholinesterase property. The present study is concerned with its toxicity and experimental therapy for schistosomiasis japonica, as well as the antidotal effects of atropine and PAM. The intragastric and subcutaneous LD_(50) of dipterex in mice were found to be 0.8 and 0.6 g/kg, respectively. In mice, concomitant injections of atropine and PAM 1/2 hour prior to dipterex yielded a better detoxicating effect than either antidote given...

    Dipterex is an organophosphorus insecticide, characterized by its anticholinesterase property. The present study is concerned with its toxicity and experimental therapy for schistosomiasis japonica, as well as the antidotal effects of atropine and PAM. The intragastric and subcutaneous LD_(50) of dipterex in mice were found to be 0.8 and 0.6 g/kg, respectively. In mice, concomitant injections of atropine and PAM 1/2 hour prior to dipterex yielded a better detoxicating effect than either antidote given alone. Rabbits receiving dipterex 30 or 60 mg/kg/day by stomach tube for 2 weeks showed a 70%-inhibition of plasma cholinesterase. Monkeys were daily fed dipterex, the dosage of which being started from 4 mg/kg and increased by 4 mg/kg successively each day. On the 6th day the monkeys refused food and the cholinesterase activity was depressed markedly. They collapsed on the 7th day, but recovered 5 days after discontinuance of dipterex. Mice infected with Schistosoma japonicum were fed dipterex 0.2 g/kg/day for 2 weeks. The average number of worms remained in each mouse of the treated group was found to be 13± 5, and that of the control group 24±6. The difference was highly significant. Infected rabbits receiving daily 30 mg/kg intragastrically or 40 mg/kg hypodermically for 2 weeks also showed a reduction of the number of helminths. Five infected dogs were daily fed dipterex for 2 weeks. All the fecal examinations for ova became no longer positive. After a holding period of 2 weeks, the average number of worms remained in each dog was reduced to 8±9, whereas that of the 6 control dogs was 47±29. The difference was significant. During the course of treatment in dogs, there appeared no conspicuous alterations in serum BSP retention and blood picture. The plasma cholinesterase activity fell to 25% of the original level, and restored to 75% subsequent to a 2-week holding period. The combined therapy with dipterex and tartar emetic resulted in a better effect than either drug alone. Atropine and PAM did not attenuate the therapeutic efficacy of dipterex. In conclusion, definite therapeutic action of dipterex for schistosomiasis japonica was revealed. It is cheap and can be taken by mouth. Despite certain toxic reactions, dipterex has surely opened up a new terrain for searching non-antimonial remedies.

    敌百虫是有机磷杀虫剂,又是胆碱酯酶抑制剂。本文試驗动物內用敌百虫的毒性和对日本血吸虫病的疗效,以及阿託品和PAM对敌百虫毒性与疗效的影响。小白鼠灌胃和皮下注射敌百虫1次的LD_(50)分別为0.8和0.6克/公斤。小白鼠在服敌百虫前30分钟注射阿託品和PAM的解毒效能比单用阿託品或单用PAM为佳。家兔每天灌胃30或60毫克/公斤共2周,抑制血浆胆碱酯酶活力70%左右。猴子灌胃剂量从4毫克/公斤开始,逐日递增4毫克/公斤,至第6天不食,胆碱酯酶活力也明显受到抑制,第7天躺臥不动,停药5天后恢复。小白鼠每天灌胃敌百虫200毫克/公斤,經2周后平均每鼠余存虫13±5条,和对照组24±6条相差非常显著.兔每天灌胃30毫克/公斤或皮下注射40毫克/公斤历2周后虫数也有减少.狗口服敌百虫2周后粪便转为阴性.然后停药2周解剖,平均每狗余存虫8±9条,比对照组6狗平均47±29条显著减少。在病狗治程中,血清磺溴酞钠存留率与血象无明显改变,血浆胆碱酯酶活力降为原来水平的25%左右.停药2周后恢复至原来水平的75%左右。敌百虫与吐酒石合并使用比单独应用一药治疗的效果要更好。阿品及PAM并不减弱敌百虫的疗效。敌百虫对动物的日本血吸...

    敌百虫是有机磷杀虫剂,又是胆碱酯酶抑制剂。本文試驗动物內用敌百虫的毒性和对日本血吸虫病的疗效,以及阿託品和PAM对敌百虫毒性与疗效的影响。小白鼠灌胃和皮下注射敌百虫1次的LD_(50)分別为0.8和0.6克/公斤。小白鼠在服敌百虫前30分钟注射阿託品和PAM的解毒效能比单用阿託品或单用PAM为佳。家兔每天灌胃30或60毫克/公斤共2周,抑制血浆胆碱酯酶活力70%左右。猴子灌胃剂量从4毫克/公斤开始,逐日递增4毫克/公斤,至第6天不食,胆碱酯酶活力也明显受到抑制,第7天躺臥不动,停药5天后恢复。小白鼠每天灌胃敌百虫200毫克/公斤,經2周后平均每鼠余存虫13±5条,和对照组24±6条相差非常显著.兔每天灌胃30毫克/公斤或皮下注射40毫克/公斤历2周后虫数也有减少.狗口服敌百虫2周后粪便转为阴性.然后停药2周解剖,平均每狗余存虫8±9条,比对照组6狗平均47±29条显著减少。在病狗治程中,血清磺溴酞钠存留率与血象无明显改变,血浆胆碱酯酶活力降为原来水平的25%左右.停药2周后恢复至原来水平的75%左右。敌百虫与吐酒石合并使用比单独应用一药治疗的效果要更好。阿品及PAM并不减弱敌百虫的疗效。敌百虫对动物的日本血吸虫病确有疗效,价格低廉,且可口服,为找寻有效的非锑剂开辟了新的途径。

    The present paper is a study of the correlation between esterase inhibition andhouse-fly poisoning by Rogor, Dipterex and TOCP (tri-o-cresyl phosphate). One-day-oldhouse-flies were used as testing material. After treatment by topical application with theLD_(50) doses of these insecticides to the tip of the abdomen, the symptoms of the house-flies were observed and the in vivo inhibitions of cholinesterase and ali-esterase withinfly-heads and trunks were determined by Warburg method. The substrate protectiontechnique...

    The present paper is a study of the correlation between esterase inhibition andhouse-fly poisoning by Rogor, Dipterex and TOCP (tri-o-cresyl phosphate). One-day-oldhouse-flies were used as testing material. After treatment by topical application with theLD_(50) doses of these insecticides to the tip of the abdomen, the symptoms of the house-flies were observed and the in vivo inhibitions of cholinesterase and ali-esterase withinfly-heads and trunks were determined by Warburg method. The substrate protectiontechnique was employed. The results showed that the inhibition of cholinesterase, whether in fly-heads or intrunks, was closely related to the symptoms induced by these toxicants and the activity ofcholinesterase reached to the minimum after the flies were knocked down. The inhibi-tion of ali-esterase was also related to the symptoms but symptoms did not appear whenali-esterase alone was inhibited (even to the extent of 90%). For the present it seemsjustifiable to conclude that the major toxic action of organophosphorus insecticides is theinhibition of cholinesterase, though it is quite possible that the toxic action might be dueto joint inhibition of both cholinesterase and ali-esterase.

    本文研究了乐果、敌百虫和TOCP等三种药剂对家蝇胆碱酯酶与脂族酯酶的体内抑制与中毒症状之间的关系。试验以家蝇为材料,用上述药剂的LD_(50)剂量处理后观察其在不同时间内的中毒症状,并用Warburg测压法分别在不同时间内测定家蝇头部和胸腹部胆碱酯酶和脂族酯酶的抑制情况 所得结果表明:无论头部和胸腹部胆碱酯酶的抑制程度都与中毒症状密切相关。当家蝇中毒倒伏时胆碱酯酶的活性最低。脂族酯酶的抑制作用与中毒症状虽有一定的关系,但是单独抑制脂族酯酶不表现出任何症状。因此,可以认为有机磷杀虫剂的主要中毒作用是抑制胆碱酯酶,虽然也可能是两种酶同时受抑制的作用。

    Laboratory and field tests in 1963-1964 indicated that both the eggs and 1-3 instar larvae of either Agrotis ypsilon Rottemberg or Euxoa segetum Schiffmuler were killed by the organophosphorus insecticides such as E-605 and dipterex; the chlorinated hydrocarbon insecticides such as DDT and BHC killed the 1-3 instar larvae, but not the eggs. Application of a mixture with the above two kinds of toxicants increased the efficiency for killing the eggs.

    两年来室内和試驗場試驗証实:有机磷杀虫剂如1605及敌百虫等能兼治小地老虎和黄地老虎的卵和初龄幼虫;有机氯杀虫剂如DDT及666等对卵几乎无效,但对初龄幼虫效果頁好。两类药剂混用杀卵效果提高。大田試驗用50%敌百虫乳剂1份、25%DDT乳剂1份、水600份,在卵孵化率为28.6—95.0%时亩施混合液50公斤,防治效果良好;防治适期比药杀初龄幼虫可提早十天左右,大大緩和了时間紧张的矛盾。

     
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