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toxin
相关语句
  毒素
     Biochemical Characters and Gene Cloning of Aminopeptidase N-Receptor for Bt Toxin in Cotton Bollworm and Its Function in Relation to Bt Resistance
     棉铃虫Bt毒素受体蛋白生化特性、基因克隆及其与抗性的关系
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     Preparation of Specific McAb-PE40 Chimeric Toxin and Its Kill Action to Cryptosporidium Parvum
     特异性单抗-PE40重组毒素的制备及其对隐孢子虫杀灭作用研究
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     Study on the Structure and Function of Actinobacillus Pleuropneumoniae ApxⅡ Toxin and Its Mutant
     胸膜肺炎放线杆菌ApxⅡ毒素的结构与功能分析及其突变体的研究
短句来源
     Experimental Study on Construction of Recombinant Toxin MSH-Ang and Its Anti-tumor Activity
     MSH-Ang重组毒素的构建及其抗肿瘤效应的实验性研究
短句来源
     Molecular Cloning, Expression, Structure-function Relationship of K~+ Channel Scorpion Toxin from Buthus Martensii Karsch
     东亚钳蝎K~+通道毒素的克隆、表达及其结构与功能关系研究
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     In order to select low toxin crownvetch,suckers of crownvetch was irradiated by~(60) Co gam- man rays ranging from 0,0.5、1.0、2.0、2.5、3.0、4.0、5.0、6.0KR at a rate of 183R/min.
     为了选育低小冠花新品种,我们用~(60)Co—γ射线照射小冠花根蘖芽,照射剂量分别为0、0.5、1.0、2.0、2.5、3.0、4.0、5.0、6.0千拉德,剂量率为183拉德/分。
短句来源
     Results The potency of diphtheria toxin was 180 Lf/ml, 210 Lf/ml and 155Lf/ml respectively.
     结果 产效价分别为 180 L f/ml、2 10 L f/ml和 15 5 Lf/ml。
短句来源
     Oxidation stress effects of SO_2 and its systemic toxin
     氧化应激效应与SO_2全身性作用研究
短句来源
     In RK-13 cells, 81.0 %(17/21) strains with s1a/m1b produced vacuolating toxin activity.
     所有s1a/m1b和s1a/m1b-m2型Hp菌株对三种细胞均有明显的空泡作用,以RK-13细胞计,81.0%(17/21)的s1a/m1b型Hp菌株和1株s1a/m1b-m2型有空泡性。
短句来源
     It was inactivated whole vibrio cells(WC) or B subunit of cholera toxin(BS) and inactivated whole cell vaccine of vibrio cholerae, recombinant B-subunit, inactivated whole cell vaccine of vibrio cholerae (rBS-WC) and CVD103-HgR.
     即灭活霍乱弧菌全菌体(WC)疫苗或霍乱素B亚单位-灭活霍乱弧菌全菌体(BS-WC)疫苗、重组霍乱素B亚单位-灭活霍乱弧菌全菌体(rBS-WC)疫苗、由活的基因修饰的O1群霍乱弧菌减株(CVD103-HgR)组成的减活菌苗。
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  毒素的
     Radioimmunoassay for T2 toxin
     T2毒素的放射免疫测定
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     Construction and Expression of Recombinant EGF-PE35KDEL Toxin Gene
     EGF-PE35KDEL重组毒素的基因构建及表达
短句来源
     In the range of 0.005~0.10 g/L,the standard toxin showed linear correlation and the coefficient and recovery rate of the standard toxin were 0.971 3 and 98.2% respectively.
     在0.005~0.10 g/L范围内,标准毒素的线性回归良好,相关系数和回收率分别为0.971 3和98.2%.
短句来源
     Characterization and Mechanism of BTI-Tn-5B1 Cells Resistant to CryIA(c) Toxin
     粉纹夜蛾BTI-Tn-5B1细胞对苏云金芽孢杆菌cryIA(c)毒素的抗性选择及抗性机制的初步研究
短句来源
     The major metabolite was HT2 toxin and its formation half-life was 12.9 min.
     主要代谢产物HT2毒素的生成半衰期为12.9 min.
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  “toxin”译为未确定词的双语例句
     Studies on the expression of cholera toxin B subunit oligerma in transgenic tomato plant
     霍乱肠毒素B亚单位在转基因番茄植物中表达的研究
短句来源
     Studies on the Fruit-specific Expression of Cholera Toxin B in Transgenic Tomato Plants
     霍乱肠毒素B亚单位在转基因番茄果实中特异性表达的研究
短句来源
     Bt Resistance Selection, Receptor Gene Cloning and Synergism of PTD to Cry1Ac Toxin in Plutella Xylostella
     抗Bt小菜蛾的选育、受体基因克隆及PTD对Cry1Ac的增效作用
短句来源
     The Clinical Study of Clearing Away Heat and Relieving the Toxin Therapy on TGF β and VEGF in Rheumatoid Arthritis
     从血清TGFβ1、VEGF角度探讨清热解毒法治疗活动性类风湿关节炎的临床研究
短句来源
     CHANGES IN ELECTROPHYSIOLOGICAL PROPERTIES OF SPINAL MOTONEURONES AFTER INTRAMUSCULAR INJECTION OF BOTULINUM TOXIN
     肌内注射肉毒素后脊髓运动神经元电生理特性的变化
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  toxin
Ochratoxin A was quantitatively monitored in grain extracts by indirect solid-phase enzyme immunoassay with the use of an immobilized conjugate of the toxin with gelatin and polyclonal rabbit antibodies raised against the ochratoxin A-BSA conjugate.
      
This monitoring found that 1.7 to 18.5% of the samples were contaminated with the toxin at a concentration of 25.9-291.7 μg/kg.
      
It has been demonstrated in model experiments that the decrease in the content of BET in the culture fluid is accounted for by the toxin interaction with an attendant product, the exogenous metabolite (EM).
      
Primary Structure of a Short Toxin from the Venom of a Vietnamese Scorpion (Buthus sp.)
      
The complete amino acid sequence of an important toxin (toxin 14) from the venom of a Vietnamese scorpion (Buthus occitanus sp.) has been determined, which includes 35 amino acid residues and three disulfide bridges (molecular weight, 3843 Da).
      
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Thirteen adult guinea-pigs were divided into 3 groups:2 as normal controls;6 and 5 being treated with streptomycin sulphate 200 and 400mg per kilo body weight res- pectively for 21—60 days.The vestibular and auditory functions were tested.Their brains were prepared for Bielschowsky and Nissl sections.Microscopic examination re- vealed the following facts. Vascular disturbances prevail in the nervous system.The blood vessels are crumpled with concommittent perivascular edema.The adjacent nervous tissue shows...

Thirteen adult guinea-pigs were divided into 3 groups:2 as normal controls;6 and 5 being treated with streptomycin sulphate 200 and 400mg per kilo body weight res- pectively for 21—60 days.The vestibular and auditory functions were tested.Their brains were prepared for Bielschowsky and Nissl sections.Microscopic examination re- vealed the following facts. Vascular disturbances prevail in the nervous system.The blood vessels are crumpled with concommittent perivascular edema.The adjacent nervous tissue shows ischemic softening and the nerve fibres become demyelinated. The intoxicated nerve cells are generally dehydrated and shrunken often with eccen- tricity of nuclei and chromophilic lumps.Gliosis and neurophagosis are common. The vestibular and cochlear systems of the eighth or stato-acoustic nerve exhibit intoxicative changes in the 2 groups of animals.On the peripheral as well as the central side,the degeneration of the vestibular structures supercedes that of the cochlear system. The site and nature of the intoxication are much alike in the light and heavy-dose groups,yet the latter shows a higher severity. The degenerative alterations appear simultaneously in the perpheral and central struc- tures.A question of primacy in intoxication—peripheral or central—is not existent. There is a chain of structures on the peripheral and central sides of the stato-acoustic system;their degeneration does not proceed along the conduction path,nor the degree of intoxication varies with their relative positions on the conduction scale.The peri- pheral and central units get intoxicated independently. Besides the stato-acoustic structures streptomycin afflicts other sensory(e.g.trige- minal) and motor systems,the viscero-motor in particular.Streptomycin intoxication is general in nature,yet some structures are especially sensitive to it. The relative vulnerability to streptomycin of the nervous structures is apparently conditioned by their chemical constitution. Streptomycin injuries higher brain parts which are mainly motor,especially viscero- motor.The diencephalic viscero-motor nuclei,the striate complex and the visceral cor- tex are excessively disintegrated.The extensive intoxicative manifestations have a neurological basis. The toxin of streptomycin affects the entire nervous system and hence the whole bodily mechanisms.Its medical application calls for a meticulous consideration with respect to the patient's sensitivity to the antibiotic,the size of the dose and the length of the therapeutic course.

十三个成长的豚鼠分为三粗,2个作正常对照,此外6个和5个每日分别注射链霉秦硫酸盐,一公斤体重200和400毫克,共经21至60天。未了,豚鼠都经过平衡和听觉的测验。各脑分别染制Nissl和Bielschowsky切片。显微镜的检查得到以下的结果。神经系中普见血管的病变。血管塌陷,管外间质水肿。邻近的神经组织失血软化。神经纤维表现髓鞘溶解。中毒的神经细胞脱水萎缩,常见胞核偏位,染色质偏聚、结块。也时有神经胶质增生和神经吞噬现象。二组豚鼠在位听神经的前庭系和耳蜗系都有中毒的征象。在周缘部和中枢部,前庭系各极结构的病变,与耳蜗系相较,更为严重。在注射轻剂和重剂的二组豚鼠,神经系中毒的部位和性质是相似的,但重剂组的病变程度较高。在位听系,中毒的病变同时见于周缘部和中枢部。中毒的第一性的问题——在周缘, 抑在中枢——是不存在的。周缘和中枢各有一系结构,病变的表现不沿神经传导的路线;病变的轻重也不随路线上地位的高低。周缘和中枢的各项结构是各自中毒的。链霉素不仅毒害位听神经一系,而更损伤其它感觉系(如三叉神经系)和运动系——特是内脏运动系——的结构。链霉素中毒是一般性的,但有些结构特易感受它的毒害。在各级结构中,神经细胞的...

十三个成长的豚鼠分为三粗,2个作正常对照,此外6个和5个每日分别注射链霉秦硫酸盐,一公斤体重200和400毫克,共经21至60天。未了,豚鼠都经过平衡和听觉的测验。各脑分别染制Nissl和Bielschowsky切片。显微镜的检查得到以下的结果。神经系中普见血管的病变。血管塌陷,管外间质水肿。邻近的神经组织失血软化。神经纤维表现髓鞘溶解。中毒的神经细胞脱水萎缩,常见胞核偏位,染色质偏聚、结块。也时有神经胶质增生和神经吞噬现象。二组豚鼠在位听神经的前庭系和耳蜗系都有中毒的征象。在周缘部和中枢部,前庭系各极结构的病变,与耳蜗系相较,更为严重。在注射轻剂和重剂的二组豚鼠,神经系中毒的部位和性质是相似的,但重剂组的病变程度较高。在位听系,中毒的病变同时见于周缘部和中枢部。中毒的第一性的问题——在周缘, 抑在中枢——是不存在的。周缘和中枢各有一系结构,病变的表现不沿神经传导的路线;病变的轻重也不随路线上地位的高低。周缘和中枢的各项结构是各自中毒的。链霉素不仅毒害位听神经一系,而更损伤其它感觉系(如三叉神经系)和运动系——特是内脏运动系——的结构。链霉素中毒是一般性的,但有些结构特易感受它的毒害。在各级结构中,神经细胞的化学合成决定它们反应链霉素的灵钝和轻重。链霉素伤害高级的脑部,这多属运动性,特是内脏运动性。间脑的内脏性核团、纹状体簇和内脏性皮质中毒特别剧烈。链霉素中毒症状广泛,这是有神经基础的。链霉素的毒质伤害全神经系,也影响全身;为医疗使用这个抗菌素,关于患者对此药的敏感,剂量和疗程是要慎重考虑的。

The experiments were made on guinea pigs. The results obtained demonstrate that after crushing the nerve the regeneration of the motor nerve endings in muscle poisoned by botulinum toxin can proceed with the same effectiveness as in unpoisoned muscle. At the same time they show that causing the poisoned nerve endings to degenerate and then regenerate may be employed as a means of accelerating the recovery of the poisoned muscle; for example, the very slow spontaneous recovery usually shown by a poisoned...

The experiments were made on guinea pigs. The results obtained demonstrate that after crushing the nerve the regeneration of the motor nerve endings in muscle poisoned by botulinum toxin can proceed with the same effectiveness as in unpoisoned muscle. At the same time they show that causing the poisoned nerve endings to degenerate and then regenerate may be employed as a means of accelerating the recovery of the poisoned muscle; for example, the very slow spontaneous recovery usually shown by a poisoned muscle requiring about a year for completion, can be shortened to about 11/2 months by crushing the nerve. The suggestion is made that the slow spontaneous recovery iust mentioned is probably the result of a process of slow spontaneous tissue renewal constantly occurring in the motor nerve endings. It is also pointed out that the experimental finding reported here constitutes a new piece of corroborative evidence for the conclusion that the site of action of botulinum toxin is confined to the motor nerve endings.

实驗在豚鼠进行,結果揭示,夹毁神经后运动神经末梢的再生在肉毒杆菌毒素中毒的肌肉与在正常肌肉完全一样地有效。用夹毁神经的办法,可以比较迅速地解除肉毒中毒肌肉的麻痹。本文结果,亦对肉毒的作用地点问题提供了新的佐証。

After botulinum poisoning, the "slow" and "fast" muscle fibres of the chick exhibit opposite trophic reactions. This was first demonstrated with the pure "slow" muscle, anterior latissimus dorsi (ALD), and the pure "fast" muscle, posterior latissimus dorsi (PLD). After botulinum injection, for periods from 1 to 8 weeks studied, ALD regularly showed hypertrophy, while PLD manifested the usual atrophy. That the hypertrophy of ALD was really brought about in some specific way by botulinum toxin and was not...

After botulinum poisoning, the "slow" and "fast" muscle fibres of the chick exhibit opposite trophic reactions. This was first demonstrated with the pure "slow" muscle, anterior latissimus dorsi (ALD), and the pure "fast" muscle, posterior latissimus dorsi (PLD). After botulinum injection, for periods from 1 to 8 weeks studied, ALD regularly showed hypertrophy, while PLD manifested the usual atrophy. That the hypertrophy of ALD was really brought about in some specific way by botulinum toxin and was not due to incidental stretching, was ascertained in experiments, in which any gross difference in the operation of mechanical factors on ALD on the poisoned and on the control side was excluded. Additional evidence for the contrasting trophicreac tions of the two types of muscles to botulinum toxin was provided by observations on the sartorius muscle. Histological studies of this muscle following botulinum poisoning revealed the presence of both hypertrophied and atrophied fibres, corresponding to the fact that the chick sartorius muscle contains both "slow" and "fast" fibres. The effects of botulinum toxin on the chick muscles described in this paper form an exact parallel to the effects of denervation described by us in a previous report. It is remarkable that the novel phenomenon of post-denervation muscular hypertrophy in chick could also be exactly reproduced by botulinum poisoning.

肉毒杆菌毒素象去神经一样,在鸡的“慢”肌和“快”肌引起形成鮮明对比的营养性变化。在肉毒中毒后1—8周純“慢”肌前背阔肌无例外地发生肥大,而純“快”肌后背阔肌則出现通常的萎縮。混合肌肉缝匠肌中毒后,其中“慢”和“快”两种纤维,相应地出现肥大和萎縮两种相反的营养性反应。

 
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