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toxin
相关语句
  毒素
    The Experimental Study on Engineering Antibody of Anti-EHEC O157:H7 Shiga Like Toxin
    抗肠出血性大肠杆菌O157:H7志贺样毒素Ⅱ基因工程抗体的实验研究
短句来源
    EARLY TOXIC EFFECT OF T-2 TOXIN ON MURINE SMALL INTESTINE
    T-2毒素对小鼠小肠的早期毒性作用
短句来源
    Cloning and Expression of the Cholera Toxin B Subunit Gene
    霍乱毒素B亚单位基因的克隆和表达
短句来源
    Effect of inhibiting intestinal microflora and nonspecific carboxylesterase on the metabolism of T2 toxin in the in situ perfused rat small intestinal loop
    抑制肠道细菌丛和非特异性羧酸酯酶对T2毒素在原位灌流大鼠小肠袢中代谢的影响
短句来源
    CONSTRUCTION OF SHIGELLA SONNEI VACCINE STRAIN EXPRESSING VIBRIO CHOLERAE O ANTIGEN AND CHOLERA TOXIN B SUBUNIT WITH AN ATTENUATED STRAIN OF SHIGELLA SONNEI(S7)
    宋内痢疾菌无毒株S7表达霍乱弧菌O抗原及霍乱毒素B亚单位工程菌的构建
短句来源
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  “toxin”译为未确定词的双语例句
    Studies on the expression of cholera toxin B subunit oligerma in transgenic tomato plant
    霍乱肠毒素B亚单位在转基因番茄植物中表达的研究
短句来源
    USING TETANUS TOXIN IMMUNOFLUORESCENCE TO IDENTIFY NERVE CELLS IN CULTURE
    用破伤风毒素免疫荧光法显示体外培养的神经细胞
短句来源
    Gene Fusion and Expression of Cholera Toxin B Subunit with HCV Epitopes
    丙肝抗原决定簇与CTB的融合、表达及融合蛋白的纯化
短句来源
    Rapid detectiou of cbolera toxin gene of Vibrio cholerae strain O1 by polymerase chain reaction
    聚合酶链反应检测O1群霍乱弧菌肠毒素A亚单位基因
短句来源
    To construct a recombinant plasmid carrying the cholera toxin B subunit(CTB) gene and observe its expression in E.
    目的构建霍乱弧菌肠毒素B亚单位(Cholera toxin B subunit,CTB)基因的大肠杆菌表达重组质粒,并观察其在大肠杆菌和双歧杆菌中的表达。
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  toxin
Ochratoxin A was quantitatively monitored in grain extracts by indirect solid-phase enzyme immunoassay with the use of an immobilized conjugate of the toxin with gelatin and polyclonal rabbit antibodies raised against the ochratoxin A-BSA conjugate.
      
This monitoring found that 1.7 to 18.5% of the samples were contaminated with the toxin at a concentration of 25.9-291.7 μg/kg.
      
It has been demonstrated in model experiments that the decrease in the content of BET in the culture fluid is accounted for by the toxin interaction with an attendant product, the exogenous metabolite (EM).
      
Primary Structure of a Short Toxin from the Venom of a Vietnamese Scorpion (Buthus sp.)
      
The complete amino acid sequence of an important toxin (toxin 14) from the venom of a Vietnamese scorpion (Buthus occitanus sp.) has been determined, which includes 35 amino acid residues and three disulfide bridges (molecular weight, 3843 Da).
      
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ETEG, EIEC and EPEC are im-portant pathogens responsible for humanbacterial diarrhea. In recent years,the investigation of ETEC pathogenesishas advanced to a level of molecularbiology, and the study of EIEC hasalso made significant development.However, little is known about thepathogenesis of EPEC. Therefore, thereis some doubt about the value of clinicalapplication of classical EPEC diagno-stic sera to identify the cause ofdiarrhea in infants. The authors made a preliminarystudy of the pathogenesis of EPECdiarrhea....

ETEG, EIEC and EPEC are im-portant pathogens responsible for humanbacterial diarrhea. In recent years,the investigation of ETEC pathogenesishas advanced to a level of molecularbiology, and the study of EIEC hasalso made significant development.However, little is known about thepathogenesis of EPEC. Therefore, thereis some doubt about the value of clinicalapplication of classical EPEC diagno-stic sera to identify the cause ofdiarrhea in infants. The authors made a preliminarystudy of the pathogenesis of EPECdiarrhea. Enterotoxin and enteroinva-siveness tests were carried out in 5strains of EPEC. Only one of the fivestrains produced LT enterotoxin whilethe other four showed neither entero-toxin nor enteroinvasiveness. Then,these four strains were fed to domesticinfant rabbits in order to find outwhether they can induce diarrhea. Twostrains caused diarrhea in infant rabbitsand bacteria were found in the fecesof these rabbits much longer than thosethat were free from diarrhea. Further-more, they were the dominant strainsin the excrement. Histological sectionof rabbits intestine reyealed somepathological changes. These findingsindicate that the toxin of EPEC differsfrom LT and ST enterotoxin becauseit damages the structure of intestinaltissue. At the same time, the chiefcondition for EPEC to cause diarrheais that EPEC can colonize in the wallof small intestine and propagate on alarge scale. Possibly, these have some-thing to do with a colonization factor.A determination of the hemagglutina-tion patterns of seven strains of EPECrevealed that the four strains causedmannose-resistant hemagglutination ofguineapig, human and cow erythrocytes,which is different from the hemagglu-tination patterns produced by type Ⅰpili and CFA/Ⅰ, CFA/Ⅱ. Only one ofthirteen strains of common Escherichiacoli caused this hemagglutination pat-tern. Through statistic treatment, P<0.05, it shows clearly that this kindof hemagglutination is chiefly producedby strains of EPEC rather than bycommon Escherichia coli. Owing to the difference of pathoge-nesis between EPEC and ETEC and EIEC,it is still necessary to classify theserotype of classical EPEC in searchingfor the cause of diarrhea in infants.

作者使用传统的肠道致病性大肠杆菌株进行了肠毒素、肠侵袭性及幼兔实验性腹泻等试验。实验结果表明,肠道致病性大肠杆菌是大肠杆菌性腹泻的重要病原,其致病机理既不同于产肠毒素性大肠杆菌,也不同于肠道侵袭性大肠杆菌。因此,临床实验室在检查婴幼儿腹泻的病原时,常规作传统的肠道致病性大肠杆菌血清分型仍属必要。

Plasma levels of PGE and PGF2α were determined with radioimm- unoassay in venous blood from anesthetized dogs, before and after in- travenous administration of E. coli endotoxin, and from children suff- ered from epidemic meningococcal meningitis. The administration of endo- toxin in the dogs induced an apparent increase in plasma levels of PGE and PGF2α with a negative linear correlation between the plasma levels of PGE and total systemic peripheral resistance(TSPR) at the early st age.Intravenous injection...

Plasma levels of PGE and PGF2α were determined with radioimm- unoassay in venous blood from anesthetized dogs, before and after in- travenous administration of E. coli endotoxin, and from children suff- ered from epidemic meningococcal meningitis. The administration of endo- toxin in the dogs induced an apparent increase in plasma levels of PGE and PGF2α with a negative linear correlation between the plasma levels of PGE and total systemic peripheral resistance(TSPR) at the early st age.Intravenous injection of anisodamine showed no influence on the pla sma levels of PGE and PGF2α in the endotoxin-shocked dogs. Plasma levels of PGE and PGF2α in the children suffered from epidemic flom meningitis were also determined which first markedly increased and then decreased to normal during convalescence. Results suggested that PGE might reduce TSPR.The relationship between the plasma PGF2α and pulmonary vascular resistance was also discussed.

大肠杆菌内毒素血症狗血浆PGE和PGF_2a含量显著增高。休克初期血浆PGE增高值与体循环总外周阻力的变化呈负相关。静脉注射654-2对血浆PGE和PGF_2a水平无明显影响。休克型流脑患儿治疗前血浆PGE和PGF_a含量显著增高,康复期降至正常水平。结果提示:PGE可能是引起休克时体循环外周阻力降低的因素之一。

Fluorocarbon emulsion, a substitute of blood, 20 ml/kg were injected once into dogs intravenously. As a result, part of the substitute deposited in the lympho-retic-ular system and was phagocytized by fixed and free histiocytes (rnonophagocytic system), and the histiocytes then converted into foaming cells.One month after injection, the fluorocarbon's deposits in the foaming cells remained at the peak level in the liver, spleen and other organs of the lympho-reti-cuJar system. After six months,no deposits could...

Fluorocarbon emulsion, a substitute of blood, 20 ml/kg were injected once into dogs intravenously. As a result, part of the substitute deposited in the lympho-retic-ular system and was phagocytized by fixed and free histiocytes (rnonophagocytic system), and the histiocytes then converted into foaming cells.One month after injection, the fluorocarbon's deposits in the foaming cells remained at the peak level in the liver, spleen and other organs of the lympho-reti-cuJar system. After six months,no deposits could be found in all viscera and no signs of any pathological changes, except the spleen under a light microscope.After twelve months, no foaming cells were found in the spleen, liver, and kidney under light cmicroscope,yet but foaming cells could be found in the spleen and liver with the help of an electron microscope and we believed that this sign had no pathological significance in clinic practice.The target cells only showed the action of phagocitizing and depositing.According to our observation, the fluorocarbon emulsion is a non cytoplasmic toxin and an inert biologic substance, so there are no secondary histo-pathologic changes caused by fluorocarbon deposition. It seems that 20 ml/kg of fluorocarbon emulsion (equal to 1200 ml for adult human)injected intravenously is rather safe.

本文用临床通用剂量(20ml/kg,相当于成人一次使用1200ml)的氟碳代血液作慢性毒性病理学研究。狗在一次静脉注入本品后主要沉积在单核巨噬系统,并被固定和游离的组织细胞所吞噬,吞噬后的组织细胞转变为泡沫细胞。 注入后1月仍属潴留高峰,6月后除脾脏外各脏器均已基本排空并不留任何病理痕迹,12月后所有被检脏器在光镜下均已不能查见泡沫细胞,但在电镜下肝脾仍可偶见个别部位留有少量小泡状物质,作者认为此种极微量的潴留缺乏临床意义。 作者也认为氟碳代血液对靶细胞只是被吞噬和潴留,未出现任何明显病理学改变。 根据研究结果,作者认为本品属非原浆毒并为生物惰性物质,亦不存在由于本品在单核巨噬系统潴留而引起的继发性病理损害问题,并认为本剂量的临床应用是安全的。

 
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