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alveolar surfactant
相关语句
  肺泡表面活性物质
     The results suggest that the lung injury resulting from acute hemorrhagic pancreatitis was characterized by alveolar surfactant System damage.
     结果提示:并发于急性出血性胰腺炎的肺损伤以肺泡表面活性物质系统受损为一重要的表现形式。
短句来源
     Fragments of alveolar surfactant could be seen in the 3rd hour to the 3rd day after intoxication.
     肺泡表面活性物质碎片见于中毒后3h~3d。
短句来源
  表面活性物质
     Fragments of alveolar surfactant could be seen in the 3rd hour to the 3rd day after intoxication.
     肺泡表面活性物质碎片见于中毒后3h~3d。
短句来源
     Measurement of alveolar surfactant after bleomycin-induced pulmonary injuries
     博莱霉素损伤后肺表面活性物质测定的实验研究
短句来源
     The results suggest that the lung injury resulting from acute hemorrhagic pancreatitis was characterized by alveolar surfactant System damage.
     结果提示:并发于急性出血性胰腺炎的肺损伤以肺泡表面活性物质系统受损为一重要的表现形式。
短句来源
     Comparative Study of Alveolar Surfactant in Plateau Pikas and Emigrated Rats
     高原鼢鼠、鼠兔肺表面活性物质的研究
短句来源
     Measurement of Alveolar Surfactant After Anatoxins-induced Lung Injuries
     鹅膏毒肽染毒后大鼠肺表面活性物质的测定
短句来源
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  肺表面活性物质
     Measurement of alveolar surfactant after bleomycin-induced pulmonary injuries
     博莱霉素损伤后肺表面活性物质测定的实验研究
短句来源
     Comparative Study of Alveolar Surfactant in Plateau Pikas and Emigrated Rats
     高原鼢鼠、鼠兔肺表面活性物质的研究
短句来源
     Measurement of Alveolar Surfactant After Anatoxins-induced Lung Injuries
     鹅膏毒肽染毒后大鼠肺表面活性物质的测定
短句来源
     Morphological change of alveolar II cell was slightly. It suggests that quartz dust can activate alveolar surfactant system, phosphate piperaque may protect this system, and improve the lipids metabolism of silicotic rats.
     结果提示石英能激活肺表面活性物质系统,磷酸喹派可以保护该系统,改善大鼠肺脂的肺脂质代谢。
短句来源
  “alveolar surfactant”译为未确定词的双语例句
     Through the use of lavaging techniques , alveolar surfactant has been elevated post
     结果显示,一定剂量照射后(10Gy),肺泡Ⅱ型细胞分泌Surfactant,肺泡灌
短句来源
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  alveolar surfactant
Effect of colchicine on secretion of alveolar surfactant in the intact and regenerating rat lung
      
Effect of oxygen and hypergravitation on alveolar surfactant
      
Role of the lung macrophages in regulation of the quantity of alveolar surfactant
      
Effect of general anesthetics on surface activity of the lung alveolar surfactant
      
The disturbance in ventilatory function observed in prolonged shock appeared to be related to the impairment of pulmonary microcirculation caused by DIC and subsequent hypoxia of lung tissue which led to a loss of alveolar surfactant.
      
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A stereologic study of lamellar bodies within pulmonary alveolar type II cells in dogs with experimental acute hemorrhagic pancreatitis(group P)and in dogs inje- cted muscularly with choloroquine before inducing pancreatitis (group PI)was carried out.Our findings showed that volumerical density (Vv), surface density (Sv)and numerical density(Nv)of lamellar bodies were decreased by 29.06%,30.28% and 40.75% in the group P as compared with those in the control group (group N), respectively, and their Sv and Nv...

A stereologic study of lamellar bodies within pulmonary alveolar type II cells in dogs with experimental acute hemorrhagic pancreatitis(group P)and in dogs inje- cted muscularly with choloroquine before inducing pancreatitis (group PI)was carried out.Our findings showed that volumerical density (Vv), surface density (Sv)and numerical density(Nv)of lamellar bodies were decreased by 29.06%,30.28% and 40.75% in the group P as compared with those in the control group (group N), respectively, and their Sv and Nv were decreased by 23.8% and 30.26% in comparison with those in the group PI.The results suggest that the lung injury resulting from acute hemorrhagic pancreatitis was characterized by alveolar surfactant System damage.Dramatical decrease in the number of the lamellar bodies affected the surface tension of alveoli in a certain degree, leading to atelectasis.There was a relationship between lung injury and the effect of PLA2(phospholipidase A2).Choloroquine might play a role in maintaining the number of lamellar bodies.

本文报道了对犬实验性急性出血性胰腺炎(P组)与加用氯喹(PI组)时Ⅱ型肺泡上皮细胞的板层小体体视学形态定量的研究。结果发现:P组板层小体的体积密度(Vv),表面积密度(Sv)及数密度(Nv)分别较对照组下降29.06%,30.28%,及40.75%;其Sv及Nv分别较PI组减少23.8%和30.26%。结果提示:并发于急性出血性胰腺炎的肺损伤以肺泡表面活性物质系统受损为一重要的表现形式。板层小体数目显著减少,一定程度影响到肺泡的表面张力,从而造成肺不张。肺损伤与磷脂酶A_2的作用有关;氯喹在维持板层小体的数量方面可能有一定的作用。

The pathological changes of the lungs after a single exposure to an atmosphere containing 100±15 ppm of hydrogen sulfide(H2S)for 3 hours were observed with optical and electron microscopy in rats.It was found that bronchial epitheliem,alveolar epithelium,pulmonary vessels and pulmonary interstitium were extensively involved.Pulmonary edema,focal pulmonary hemorrhage,exfoliation of the damaged epithelium,and infiltration of neutrophils occurred mainly in the 3rd hour to the 3rd day after H2S intoxication.Chronic...

The pathological changes of the lungs after a single exposure to an atmosphere containing 100±15 ppm of hydrogen sulfide(H2S)for 3 hours were observed with optical and electron microscopy in rats.It was found that bronchial epitheliem,alveolar epithelium,pulmonary vessels and pulmonary interstitium were extensively involved.Pulmonary edema,focal pulmonary hemorrhage,exfoliation of the damaged epithelium,and infiltration of neutrophils occurred mainly in the 3rd hour to the 3rd day after H2S intoxication.Chronic inflammatory response and proliferation of fibrous tissue occurred mainly from the 7th to the 15th day after intoxication.Ultrastruc-turally,there were marked changes of alveolar epithelium,phagocytes,vascular endothelium,fi-broblasts,and inflammatory cells.Initial pulmonary edema emerged in the pulmonary interstitium and gradually affected the alveoli.Fragments of alveolar surfactant could be seen in the 3rd hour to the 3rd day after intoxication.These findings indicate that H2S inhalation exerts extensive injurious effects on the lungs in the rats.

本实验应用光镜、电镜对大鼠吸入100±15ppm硫化氢(H_2S)3h后15d内肺的形态变化作了动态观察。结果发现病变广泛累及支气管上皮、肺泡上皮、肺小血管及间质。肺水肿、灶性肺出血、退变上皮的脱落及中性白细胞渗出见于中毒后3h~3d,慢性炎症及纤维组织增生见于中毒后7~15d。肺泡上皮、吞噬细胞、血管内皮、纤维母细胞及炎细胞的超微结构变化明显。肺水肿初为间质性并渐波及肺泡腔。肺泡表面活性物质碎片见于中毒后3h~3d。实验结果表明H_2S对肺具有广泛的损伤作用。

Effect of pathologic features of RDS in rats treated by Hui Sheng Yin was observed under microscope and electronic microscope. Result showed that Hui Sheng Yin could reduce pulmonary edema, atelectasis, and pneumonrrhagia caused by RDS; increase contents of alveolar surfactant factors; and also obveously steady and protect mul-tilamellar body of type Ⅱ alveolar cell and mitochomdria. So it provided scientific proof for the efficiency of Hui Sheng Yin "invigorating vital energy and benefit the lung....

Effect of pathologic features of RDS in rats treated by Hui Sheng Yin was observed under microscope and electronic microscope. Result showed that Hui Sheng Yin could reduce pulmonary edema, atelectasis, and pneumonrrhagia caused by RDS; increase contents of alveolar surfactant factors; and also obveously steady and protect mul-tilamellar body of type Ⅱ alveolar cell and mitochomdria. So it provided scientific proof for the efficiency of Hui Sheng Yin "invigorating vital energy and benefit the lung.

本实验应用光镜和电镜观察了回生饮对大鼠油酸型呼吸窘迫综合症的病理学的影响。结果表明,回生饮能减轻呼吸窘迫综合症所致的肺水肿、肺不张、肺出血;增加肺表面活性物质的含量,对Ⅱ型肺泡上皮细胞的板层体、线粒体均有显著的稳定和保护作用,从而为回生饮“益气、补肺”的功效提供了科学依据。

 
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