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hepatic
相关语句
  肝脏
    Several index will be tested and evaluated: hepatic index , Alanine aminotransferase (ALT), Aspartate transaminase (AST), Hyaluronic acid(HA).
    实验结束后分别测定各组大鼠的肝脏指数和肝功能:ALT、AST; 肝纤维化指标:透明质酸(HA);
短句来源
    Feeding guinea pigs with lithogenous diet with low protein, BXC was administrated for 3 months at the same time. In the end, the gallstones formation rate, biochemical indies and hepatic morphology were observed.
    应用低蛋白致石饲料喂养豚鼠并同时给予保肝消石胶囊,3个月后观察胆囊成石率、胆汁、血清及肝脏中生化指标和肝脏病理学变化。
短句来源
    The lithogenous diet with low protein led to the hepatic morphologic injury firstly, such as the fatty change of liver, the enhancement of the activity of endogenous p-Glucuronidase, the total ATPase and Ca~(2+)Mg~(2+)-ATPase. The metabolism of bile acids was abnormal, such as the decrease of the concentration of glycocholic acid in bile.
    低蛋白饲料首先引起肝脏病理损害,如脂肪变性、内源性β-葡萄糖醛酸酶活性以及总ATP酶、Ca~(2+)Mg~(2+)-ATPase的活性增强,继而导致胆汁酸代谢异常,如胆汁中甘胆酸降低,另外胆汁中结合胆红素减少、游离胆红素百分比上升、[Ca~(2+)]和[H~+]升高,这些都促进胆色素结石的形成。
短句来源
    Objective hepatic fibrosis is the common pathological stage in the process of the chronic liver disease caused by variant etiology. It is associated with the production and deposition of extracellular matrix which is regulated by MMPs and TIMPs, especially by TIMP-1. TIMP-1 inhibiting MMPs is elevated while the liver is damaged, and leads to the decreasement of the ECM degradation and the increasement of the deposition which is directly associated with hepatic fibrosis .
    肝纤维化(hepatic fibrosis)是各种原因所致慢性肝病共同的病理变化,机制为细胞外基质(ECM)在肝脏中过多沉积,ECM代谢主要由基质金属蛋白酶(MMPs)及基质金属蛋白酶组织抑制因子(TIMPs)所调节,以TIMP—1为主。
短句来源
    Establishment of the Assay for Determining CYP3A Activity in Rat Murine Hepatic MicrosomeLiver tissue was surgically obtained from adult SD rats and homogenized, and murine hepatic microsome was prepared from homogenate by differential centrifugation. The microsome protein was diluted to a final concentration of 20mg/ml.
    取健康成年SD大鼠肝脏,差速离心法制备肝微粒体,微粒体蛋白定量至20mg/ml,用于建立鼠肝微粒体孵育反应体系。
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  肝组织
    Effect of Decoction for Warming the Kidney and Restoring Yang on Hepatic Ultrastructure of Rabbit Model with Yang Deficiency
    温肾助阳药对甲低阳虚模型兔肝组织超微结构的影响
短句来源
    The serum concentrations of ALT and AST were determined and the liver tissue were sampled to observe the expression of TNF-α and IL-1. Results After the ischemia/reperfusion the serum concentration of ALT and AST and expressions of TNF-α and IL-1 in the hepatic tissue in the NS group significantly increased (P<0.01),and the hepatocytic morphologic change was obvious compared with the SO group.
    结果:SO组与NS组血清ALT和AST含量,肝组织TNF-α和IL-1活性明显升高(P<0.01),肝细胞形态学发生异常变化。
短句来源
    The high(100mg·kg-1·d-1)and middle(50mg·kg-1·d-1)doses could significantly lower serum ALT and AST,hepatic SOD and MDA,and improve the liver histology. But it had no influence on the liver index of mice.
    MP-Ⅰ高(100mg.kg-1.d-1)、中(50mg.kg-1.d-1)剂量组均能显著降低CCl4所致小鼠急性肝损伤血清ALT、AST活性,提高肝组织匀浆SOD活性,降低MDA,明显改善肝组织损伤的程度,与模型组比较差异有统计学意义,但对肝指数无明显影响。
短句来源
    Methods The mice were orally given at the doses of GPC and killed after 30 days. The proliferation activity of the hepatic cells was determined by MTT assay, and the levels of PKC and PCNA protein expression in the cell were measured by immunohistochemistry method.
    方法每日给小鼠经口灌胃不同剂量的GPC,30天后取肝组织,用MTT法测各组小鼠肝细胞的增殖活性水平,免疫组化的方法检测各组PKC和PCNA的表达。
短句来源
    After hepatic ischemia for 15 minutes and after reperfusion for 1 hour, 3 hours and 24 hours respectively, the hepatic tissue were resected for determining the activity of ATPase and for enzyme-histochemistry dyeing.
    肝脏缺血15min,分别于再灌注后1、3、24h取肝组织测定Na+-K+-ATP酶、Ca2+-Mg2+-ATP酶水平,并行酶组织化学染色,观察Ca2+-ATP酶的活性变化。
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  “hepatic”译为未确定词的双语例句
    Influence of Radix Astragali on the Norse-Caused Change in Hepatic Glucogen Content of Rats
    黄芪对噪声所致大鼠肝糖原含量变化的影响
短句来源
    the content of hepatic glycogen was decreased (P<0.01) in DM mice and increased(P<0.01)after APS treatment;
    DM组肝糖原含量减少,且显著低于DA组(P<0.01);
短句来源
    Conclusions: Aikete injection could inhibit proliferation of SMMC-7721 cells in vitro and the growth of hepatic cancer H22 in vivo.
    结论:埃克特注射液对人肝癌细胞SMMC-7721细胞具有明显体外抗增殖作用,对小鼠移植性肿瘤H22有一定的抑制作用。
短句来源
    CONCLUSION Nobiletin could inhibit the proliferation of SMMC-7721 cells in vitro and the growth of hepatic cancer H22 in vivo.
    结论川陈皮素对人肝癌细胞SMMC-7721具有明显体外抗增殖作用,对小鼠移植性肿瘤H22有一定的抑制作用。
短句来源
    Method:By using the MTT method to study the effect of AP on the growth of primary hepatic carcinoma cell(PHCC)and peripheral blood monouclear cell(PBMC).
    方法:采用四氮唑盐法(MTT)法检测不同浓度的芦荟多糖对体外培养的原发性肝癌细胞(PHCC)与自体外周血单个核细胞(PBMC)生长的影响。
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  hepatic
Hepatic glutathione, lipid peroxides, glutathione peroxidase, alcohol dehydrogenase, aldehyde dehydrogenase, glycogen and total protein in liver were also significantly altered.
      
Picroliv significantly reverted most of the above said altered blood and hepatic parameters in the alcohol-fed male and female rats to almost normal levels.
      
The results suggest that HCC patients with esophageal varices and hypersplenism should undergo hepatic resection plus esophageal devascularization or splenectomy if radical resection of HCC can be expected.
      
However, it has been realized that the IVC stent may compromise the hepatic veins and cause catastrophic consequences, thus the indication for IVC stenting should be reevaluated and especially not to be overused.
      
All patients underwent direct anastomoses of the RHV and the inferior vena cava (IVC), and in 23 cases, reconstruction of the right inferior hepatic vein was also done.
      
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Two alkaloids (Ⅰ and Ⅱ) were isolated from the folk medicine——Crotarlaia sessiliflora L. Alkaloid I was found to be monocrotalline, while alkaloid Ⅱ is a new pyrrolizidine alkaloid, from which trans-trans platynecic acid (trans, 2 S, 3 R) and retronecine were obtained after hydrolysis. Thus, it is proposed to have the structural formula (C). Alkaloid Ⅰ showed inhibitory activity against several rodent tumors such as sarcoma 180, leukemia 615 and walker carcinoma 256. Eight derivatives of monocrotalline were...

Two alkaloids (Ⅰ and Ⅱ) were isolated from the folk medicine——Crotarlaia sessiliflora L. Alkaloid I was found to be monocrotalline, while alkaloid Ⅱ is a new pyrrolizidine alkaloid, from which trans-trans platynecic acid (trans, 2 S, 3 R) and retronecine were obtained after hydrolysis. Thus, it is proposed to have the structural formula (C). Alkaloid Ⅰ showed inhibitory activity against several rodent tumors such as sarcoma 180, leukemia 615 and walker carcinoma 256. Eight derivatives of monocrotalline were prepared in an attempt to reduce its hepatic toxicity. Three (1, 7, 8)of them were shown to possess antitumor activity against walker carcinoma 256 in rats, but the toxicity showed in the preclinical studies kept them from entering clinical study.

从民间治疗皮肤癌的一种野百合属植物农吉利(Orotalaria sessilufkira L.)中分离出两个生物碱(Ⅰ,Ⅱ)。结构测定表明碱Ⅰ为一野百合碱(monocrotaline);碱Ⅱ为下向千里光次碱(Retronecine)和阔叶千里光次酸(platynecic acid)组成的一个新的双稠吡咯啶生物碱。碱Ⅰ对小鼠肉瘤180,白血病615,瓦克癌256均有抑制作用。为了降低其对肝脏的毒性,合成了碱Ⅰ的八个衍生物(1~8),其中1,7,8对瓦克癌256有抑制作用,但都因毒性较大而未推荐临床试用。

40 rabbits were divided randomly into 4 groups: ①uninfected experimental group treated with different total doses in various courses,②infected experimental group,③infected control group, and④ normal control group. Pathomorphology, immunofluorescence specific lymphocytic transformation test and skin test were used w find that the allergic reactions both type Ⅲ and Ⅳ play an important role in the pathogenesis of nithiocyamine induced hepatic lesions, also find especially in the uninfected experimental group,Some...

40 rabbits were divided randomly into 4 groups: ①uninfected experimental group treated with different total doses in various courses,②infected experimental group,③infected control group, and④ normal control group. Pathomorphology, immunofluorescence specific lymphocytic transformation test and skin test were used w find that the allergic reactions both type Ⅲ and Ⅳ play an important role in the pathogenesis of nithiocyamine induced hepatic lesions, also find especially in the uninfected experimental group,Some important pathological features,sacb as: hepatic autoimmune granulom (4/22)and cholangiolar proliferation with pericholangiolitis(21/22)etc. No significant differences could be found statistically (P>0.05), there fore the occurrence and the degree of various pathological changes have no definite interrelactionship with the dose and the duration of the drug.The hepatic lesions of the experimental animals with the exception of one rabbit were rather mild.

实验采用家兔40只,随机分成4组:①单纯服药组,用不同疗程和剂量;②感染治疗组;③感染对照组;④正常对照组,我们应用病理形态学、免疫荧光、特异性淋巴细胞转化试验和皮肤试验等项进行药物性肝损害发病机制的研究。实验结果表明本药所致肝损害主要是变态反应性(兼有Ⅲ及Ⅳ型)机制。我们看到实验动物肝内有重要的病理形态学变化,以单纯服药组较为明显,主要有肝自身免疫性肉芽肿(4/22),细胆管增生及其周围炎(21/22)等,分别经显著性检验,无显著差异(P>0.05),表明各项病变的发生及其程度与药量和疗程无平行关系。本实验组家兔肝脏病变除1只兔外均较轻。

Qinghaosu (Artemisine) suspension in oil was injected im to 30 rhesus monkeys qd×14 d. Damages to several organs and tissues were noticed in 96 and 192 mg/kg/d groups on d 3 after the last injection. They were mainly erythroblastopenia, myeloblastope-nia and megakaryocytosis in bone marrow, degeneration and necrosis of myocardium, degeneration of hepatic cells and epithelia of proximal convoluted tubule of the kidney, and soft tissue injury at the injection site. Bone marrow and myocardial damages were...

Qinghaosu (Artemisine) suspension in oil was injected im to 30 rhesus monkeys qd×14 d. Damages to several organs and tissues were noticed in 96 and 192 mg/kg/d groups on d 3 after the last injection. They were mainly erythroblastopenia, myeloblastope-nia and megakaryocytosis in bone marrow, degeneration and necrosis of myocardium, degeneration of hepatic cells and epithelia of proximal convoluted tubule of the kidney, and soft tissue injury at the injection site. Bone marrow and myocardial damages were more prominent than those in other organs. Only mild lesions or no damage could be found on d 35 after the last injection. Therefor, it is concluded that qinghaosu suspension in oil in doses over 96 mg/kg/d might induce transient organ or tissue damages which disappear within 5 weeks. 3 of 4 rhesus monkeys in 192 mg/kg/d dose group died of severe myocardial damage within 1-3 days after the last injection.

恒河猴30只,经连续14天肌注青蒿素油悬剂,于停药后3天,主要在每天肌注96和192mg/kg组中引起多种脏器组织的损伤。表现为:骨髓红系和粒系细胞数减少,成熟发育障碍,巨核细胞增生,心肌细胞变性和灶性坏死(以超微结构病变为主);肝、肾营养不良性改变;淋巴组织萎缩;注射部位的损伤等,其中以骨髓和心肌损伤较为明显。于停药后35天,上述病变明显减轻或消失,表明青蒿素的毒副作用是可逆性的。每天肌注青蒿素24和48mg/kg为轻微中毒剂量;96mg/kg为严重中毒剂量;192mg/kg为致死剂量。每天肌注192mg/kg组的4只猴,有3只于末次给药后1~3天死于严重心肌损伤。

 
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