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hyperglycemia
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  高血糖
     Basic and Clinical Study of Acute Spinal Cord Injuries and Hyperglycemia
     急性脊髓损伤与高血糖的基础与临床研究
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     Relationship between Serum Paraoxonase 1 Activities, Gene L55M Polymorphism and the Angiographic Characteristics of Coronary Arteries in Chinese Patients with Coronary Heart Disease Complicated with Hyperglycemia.
     中国人PON1活性及其L/M55基因多态性和oxLDL水平与伴高血糖的冠心病患者冠脉病变关系的研究
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     Effect of experimental hyperglycemia on acute cerebral ischemia in rats
     实验性高血糖对大鼠急性脑缺血的影响
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     Relation between hyperglycemia and acute cerebral infarction
     高血糖与急性脑梗塞的关系
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     CAUSE AND TREATMENT OF HYPERGLYCEMIA IN BILIARY LITHIASIS——AN ANALYSIS OF 803 CASES
     胆石症时高血糖原因和处理——附803例例分析
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  高血糖症
     Hyperglycemia was defined as whole blood glucose ≥7.8 mmol/L (140 mg/dL).
     血糖≥7·8mmol/L(140mg/dL)定义为高血糖症
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     The morbidity of newly-identified hyperglycemia was significantly lower among heavy-labor population than among light-labor population(3.0% vs 5.0%; OR=0.44,95%CI=0.32~0.62).
     经过对一系列可能的混杂因素进行统计控制后,从事重度职业体力活动的人群中,新发现高血糖症的罹患率显著低于从事轻度职业体力活动人群中的患病率(3.0%vs 5.0%;OR=0.44,95%CI=0.32~0.62);
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     Results Only 18 patients (15%) had TIMI 3 flow before primary PCI and hyperglycemia was observed in 87 patients (72.5%).
     结果在接受急诊PCI术前仅有18例(15%)患者冠状动脉造影血流达到TIMI3级,高血糖症患者87例(72·5%)。
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     Results:The levels of glucagon and insulin antibody were higher in hyperglycemia patients than the control group,the values were 369.22±281.05ng/l VS 256.36±92.15ng/L and 1.71±1.39% VS 0.58±0.71% respectively. The insulin and peptide C were similar in two groups.
     结果 :高血糖症组的胰高血糖素水平高于正常对照组 (36 9.2 2±173.5 6ng/L VS 2 5 6 .36± 92 .15ng/L ,P <0 .0 5 ) ,胰岛素抗体也高于正常对照组 (1.71± 1.39%VS0 .5 8± 0 .71% ,P <0 .0 1) ,而两组胰岛素和C肽水平无统计学差异。
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     Analysis of 113 Cases of Burn Complicated by Hyperglycemia
     烧伤并发高血糖症113例分析
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  “hyperglycemia”译为未确定词的双语例句
     The percentage of time spent in hyperglycemia above 7.8 mmol/L, 8.9 mmol/L and 10.0 mmol/L accounted for 11 (0~28)%, 4 (0~16)% and 0 (0~11)% respectively.
     血糖>7.8、8.9和10.0mmol/L所占的时间百分比分别为11(0~28)%、4(0~16)%和0(0~11)%。
短句来源
     The percentage of time spent in hyperglycemia above 7.8 mmol/L and 11.1 mmol/L accounted for 95%(28%to 100%) and 62%(6%to 100%) respectively in type 2 diabetic patients.
     而2型糖尿病组血糖>7.8mmol/L及11.1mmol/L的时间百分比分别为95%(28%~100%)和62%(6%~100%)。
短句来源
     arrhythmia correlates with left ventricle function failure and hyperglycemia independently(OR: 14.71,CI:4.57-47.62,P<0.001;P<0.001);
     心律失常增加与左室功能减退、血糖浓度独立相关(OR值:14.71,CI:4.57-47.62,P<0.001;P<0.001);
短句来源
     Compared with the C and T groups, the S group exhibited hyperglycemia( F=3.86, q=3.83,4.36, P <0.05).
     S组与 C和 T组相比 ,FBG水平明显增高 (F=3.86 ,q=3.83,4.36 ,P<0 .0 5 )。
短句来源
     Influence of TGF-β1 antioligodeoxynucleotide on MMP-9, TGF-β1, coll Ⅳ excretion by Mesangial cell under hyperglycemia
     TGF-β1反义寡核苷酸对高糖下肾小球系膜细胞分泌MMP-9、TGF-β1、collⅣ的影响
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  hyperglycemia
The components of metabolic syndrome such as obesity, hyperglycemia, hypertension and dyslipidemia had a close correlation with SUA.
      
A hypothesis is proposed whereby the processes responsible for destruction of the organism in the case of diabetes-long-term hyperglycemia and generation of methylglyoxal-may substantially impact tumor development.
      
The resultant effect is determined by the type of tumor and the duration and level of hyperglycemia.
      
Hyperglycemia resulted in significant increases in the antioxidative enzymes, Cu, Zn-SOD, CAT, GSH-Px, and GSSG-R after four and eight weeks, respectively.
      
In conclusion, some oxidative changes provoked in the testis of rabbits by hyperglycemia, were found to be reduced with repaglinide treatment at therapeutic dose.
      
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The structural formulae of the 4 benzodioxanes are: The intraperitoneal LD_(50)'s of BD-3, 933F, BD-1, and BD-5 in mice were 0.16,0.20, 0.26, and 0.22 g/kg respectively. Deaths of the animals occurred mostly in 1—3 hours for the BD-3 and 933F groups, while those for the BD-1 and BD-5 groups occurred mostly in 1—3 days. BD-5 markedly decreased the toxicity of intraperitoneal epinephrine in mice, whereas the other 3 compounds did not. Intravenous injections of these 4 drugs in anaesthetized rats and cats all produced...

The structural formulae of the 4 benzodioxanes are: The intraperitoneal LD_(50)'s of BD-3, 933F, BD-1, and BD-5 in mice were 0.16,0.20, 0.26, and 0.22 g/kg respectively. Deaths of the animals occurred mostly in 1—3 hours for the BD-3 and 933F groups, while those for the BD-1 and BD-5 groups occurred mostly in 1—3 days. BD-5 markedly decreased the toxicity of intraperitoneal epinephrine in mice, whereas the other 3 compounds did not. Intravenous injections of these 4 drugs in anaesthetized rats and cats all produced a fall of blood pressure, but the differences among the hypotensive potencies were nonsignificant. BD-3, 933F, and BD-1 resulted in a reversal of the epinephrine hypertension in anaesthetized cats, while BD-5 yielded merely an inhibition. All these 4 compounds inhibited the pressor effects of tyramine and norepinephrine. Intravenous administration of 10 mg/kg of 933F in anaesthetized cats raised the blood glucose level, but BD-5 did so only at the dosage of 20mg/kg. BD-5 and 933F did not significantly inhibit the epinephrine-induced hyperglycemia and hyperlactacidemia. Since BD-5 blocked both vasoconstrictor and dilator responses and did not cause epinephrine reversal, it might be better than 933F in treating vasospastic diseases. Clinical trial is recommended.

四种苯駢二氧六环化合物的結构式为: 小白鼠腹腔注射BD-3,933F,BD-1和BD-5的LD_(50)分別为0.1,0.20,0.26和0.22克/公斤,BD-3和933F組死亡多在1—3小时內,而BD-1和BD-5組死亡多在1—3天內。BD-5在小鼠能显著減低腎上腺素的毒性,而其他3药则不能。麻醉大白鼠和猫靜脉注射4药后均可引起降压,但4药间差异均不显著。BD-3,933F和BD-1使麻醉猫靜脉注射腎上腺素的升压引起反轉作用,而BD-5仅具抑制作用。对酪胺及去甲腎上腺素的升压,4药均可抑制。麻醉猫靜脉注射10毫克/公斤的933F有增血糖作用,BD-5注射20毫克/公斤才增高血糖。933F和BD-5对腎上腺素所引起的血糖及乳酸均无明显的抑制作用。因为BD-5同时阻断收縮和舒张血管的作用,但不引起腎上腺素的反轉作用,因此在治疗血管痙攣性疾患中可能比933F效果更好。

Acute experiments were performed on rabbits anesthetized with urethane. The change of concentration of plasma free fatty acid (FFA) was observed after electrical stimulation of the peripheral ends of left greater splanchnic nerves. The results were as follows: (1) In normal rabbits with intact adrenal glands, the effect of stimulation of splanchnie nerves upon the plasma FFA level was rather inconsistent, the result being either an increase, or a decrease, or no change. (2) After left adrenalectomy, however,...

Acute experiments were performed on rabbits anesthetized with urethane. The change of concentration of plasma free fatty acid (FFA) was observed after electrical stimulation of the peripheral ends of left greater splanchnic nerves. The results were as follows: (1) In normal rabbits with intact adrenal glands, the effect of stimulation of splanchnie nerves upon the plasma FFA level was rather inconsistent, the result being either an increase, or a decrease, or no change. (2) After left adrenalectomy, however, a marked decrease in the plasma FFA level was obtained after splanchnic stimulation in all the rabbits observed. The effect was abolished When the α-cells of the pancreatic islets were selectively destroyed by the administration of cobalt chloride. This effect was repeated in acute experiments performed on dogs with blockade of left adrenal venous return. A marked decrease in the plasma FFA with concomitant hyperglycemia was observed after splanchnic stimulation. This effect was abolished after extirpation of the body and tail (where most of the α cells are concentrated) of the pancreas. (3) By using cortisone-pretreated rabbits for assaying, it was found that a substance with glucagon-like activity by which hyperglycemia and lowering of the plasma FFA level can be induced was present in the blood of the portal vein after splanchnic stimulation. It is concluded from the above results that the greater splanchnic nerves appear to innervate the α-cells of the pancreas from which a glucagon-like substance capable of lowering the plasma FFA level is liberated into the blood stream after splanchnic stimulation.

本工作主要用兎做急性实驗,研究直接刺激内脏大神經外周端对血浆自由脂肪酸濃度的影响。結果如下: (一)在正常的不切除腎上腺的兎体上,刺激內脏大神經外周端对血浆自由脂肪酸濃度有不同的影响:有的使血浆自由脂肪酸濃度升高;有的降低;有的則基本不变。 (二)切除左侧腎上腺,刺激左侧內脏大神經外周端后,血浆自由脂肪酸濃度表現明显降低。若預先注射氯化钴以破坏胰島α細胞后,再做此項观察,血浆自由脂肪酸濃度的降低反应卽消失。在急性实驗狗身上,阻断左侧腎上腺靜脉血回流,刺激左侧內脏大神經外周端,亦可同样引起血浆自由脂肪酸濃度的降低,以及血糖濃度显著升高。切除胰尾部和胰体部以消除大部分胰島α細胞的作用后,再刺激內脏大神經外周端,上述反应卽消失。 (三)血液鉴定实驗指出,对切除腎上腺兎刺激內脏大神經后,門靜脉血血浆中有升高血糖物质存在,后者具有使血糖濃度升高和血浆自由脂肪酸濃度降低的作用。总結以上結果,可以认为,内脏大神經除支配腎上腺體质影响血糖和血浆自由脂肪酸濃度外,还极可能支配胰島α細胞,釋放类胰高血糖素物质,使血糖濃度升高及血浆自由脂肪酸濃度降低。

The effects of hyperglycemia upon dog's salivary secretion were observed in chronic and acute experiments The main results were as follows: (1)Chronic experiments were conducted on 6 dogs. Intravenous infusion of hypertonic glucose solution(30—40%, 2—3 ml per minute for 40—45 minutes) caused great augmentation of the acid-induced parotid secretion, but infusion of 7% NaCl(iso-osmotic to 40% glucose solution)and of 5% glucose-saline was without effect on the volume of secretion. (2)In acute experiments...

The effects of hyperglycemia upon dog's salivary secretion were observed in chronic and acute experiments The main results were as follows: (1)Chronic experiments were conducted on 6 dogs. Intravenous infusion of hypertonic glucose solution(30—40%, 2—3 ml per minute for 40—45 minutes) caused great augmentation of the acid-induced parotid secretion, but infusion of 7% NaCl(iso-osmotic to 40% glucose solution)and of 5% glucose-saline was without effect on the volume of secretion. (2)In acute experiments infusion of hypertonic glucose solution and of normal saline neither induced spontaneous secretion nor affected the volume of the nerve- stimulated secretion of the parotid or submaxillary glands. (3)It is suggested that in the chronic experiments the effect of hyperglycemia was most probably produced via the parasympathetic center in the central nervous system.

本工作系用具有一侧腮腺瘘的清醒狗及麻醉狗,观察了高血糖对唾液分泌的潜伏期、分泌量等的影响,发现在慢性实验中,人工地提高血糖浓度(静脉灌注30—40%糖溶液),可使唾液分泌酸反射量有显著的增加。当由静脉内灌注与40%糖溶液渗透压相当的7%NaCl 溶液或同样量的5%葡萄糖生理盐水时,则酸反射量无大改变。在急性实验,高血糖不能诱发唾液腺的分泌,亦不能使刺激鼓索神经、耳颞神经引起的唾液分泌量增加。推测高血糖对清醒动物的上述作用可能是兴奋副交感神经中枢的结果。

 
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