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   fulminant hepatitis 的翻译结果: 查询用时:0.188秒
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fulminant hepatitis     
相关语句
  重型肝炎
     Results Quantities of HBV DNA inchronic hepatitis, fulminant hepatitis and cirrhosis were 109.17±1.60, 109.44±1.98 and 108.14 ±2.27 (copy·L- 1 ) re-spectively.
     结果慢性肝炎、重型肝炎和肝硬变者HBVDVA分别为:109.17±1.60、109.44±1.98和108.14±2.27(copyL-1);
短句来源
     The elderly group had a higher fulminant hepatitis rate( 22.45%)and higher mortality ( 13.33%) than that in the control group( 9.18%)(P< 0.05)and ( 5.33%)(P< 0.01).
     老年组重型肝炎发生率(22.45%)及病死率(13.33%)明显高于非老年组(9.18%)(P<0.05)及(5.33%)(P<0.01)。
短句来源
     There was no significant difference in the serum levels of HBV DNA between the group of chronic fulminant hepatitis B(107.3731±1.4381copies/ml) and the non-fulminant group(106.6516±1.8046copies/ml).
     慢性重型肝炎组HBV DNA含量(107.3731±1.4381copies/ml)与非慢性重型肝炎组HBV DNA含量(106.6516±1.8046copies/ml)比较差异无统计学意义(P>0.05);
短句来源
     Results The positive rate of mutants in chronic fulminant hepatitis and in non-fulminant groups was 70.0%(15/25) and 33.6%(44/131),respectively(P=0.023).
     结果HBV BCP变异在慢性重型肝炎组的阳性率为60.0%(15/25)显著高于非重型肝炎组的33.6%(44/131)(P=0.023);
短句来源
     Expression of fgl2 Prothrombinase Gene in Patients with Fulminant Hepatitis
     重型肝炎患者fgl2凝血酶原酶基因的表达
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  重症肝炎
     Of the 32 cases,there were 31.8%(7/22) acute hepatitis,28.6%(2/7) chronic hepatitis and 66.7%(2/3) fulminant hepatitis.
     急性、慢性、重症肝炎的TTVDNA检出率分别为31.8%(7/22)、28.6%(2/7)、66.7%(2/3)。
短句来源
     The TNF level in subacute fulminant hepatitis (19.38±13.25u/ml) increased significantly than chsonic active hepatitis (2.68±0.86n/ml), acute hepatitis (2.17±0.05u/ml) and healthy donors (<2u/ml) P<0.05.
     结果表明,重症肝炎组TNF(19.38±13.25u╱ml)较慢性活动性肝炎组(2.68±0.86u/ml)、急性肝炎组(2.17±0.05u/ml)及正常对照组(<2u/ml)明显升高,P<0.05。
短句来源
     Thymosin α_(1)in the Treatment of 21 Cases of Fulminant Hepatitis Complicated by Spontaneous Bacterial Peritonitis
     胸腺素α_1治疗重症肝炎并发自发性细菌性腹膜炎21例
短句来源
     RESULTS SENV-DNA positive rates were 14.7%(28/191) totally, from which 36.7% in plasmapheresis patients, 33.3% in intravenous drug users, 11.1% in chronic hepatitis B patients, 9.4% in hepatitis non A-E patients, 8.3% in fulminant hepatitis patients, respectively, in comparison with 5.6% in normal populations.
     结果 SENV的感染率为 14 7% ,6种人群中SENV感染率的高低依次为血浆置换患者 (36 7% )、静脉吸毒者 (33 3% )、慢性乙型肝炎患者 (11 1% )、非甲~非戊型肝炎患者 (9 4 % )、重症肝炎患者 (8 3% )、正常人群 (5 6 % )。
短句来源
     CHANGES OF BLOOD CONCENTRATION OF INSULIN AND GLUCAGON IN FULMINANT HEPATITIS BEFORE AND AFTER GLUCAGON-INSULIN TREATMENT
     重症肝炎Glucagon-Insulin疗法滴注前后血中胰岛素、胰升血糖素等浓度变化
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  暴发性肝炎
     TRANSGENIC RESCUE FROM FULMINANT HEPATITIS IN LEC RAT BY INTRODUCING HUMAN ATP7B cDNA
     人ATP7B基因对Wilson病动物模型LEC大鼠暴发性肝炎的治疗
短句来源
     Methods Some common hepatic diseases and digestive cancers were investigated,including 13 cases of autoimmune hepatic diseases, 19 cases of fulminant hepatitis,22 cases of viral chronic hepatitis, 25 cases of hepatic fibrosis or hepatic cirrhosis, 18 cases of hepatocarcinoma and 9 cases of gastric cancer.
     方法各种肝病患者包括自身免疫性肝病13例,急性暴发性肝炎19例,慢性病毒性肝炎22例,肝纤维化或肝硬化25例,肝癌18例,胃癌9例。
短句来源
     These data showed that the LEC rats were successfully rescued from fulminant hepatitis after introducing of human ATP7B gene.
     说明人ATP7B的导入 ,成功地抑制了Wilson病动物模型LEC大鼠的暴发性肝炎的发生。
短句来源
     Relevance of Hepatitis B Variants for the Development of fulminant Hepatitis
     乙型肝炎病毒变异与暴发性肝炎
短句来源
     Results The level of IL-17 was significantly higher in the hepatic disease groups than that in the normal control(P<0.01). The level of IL-18 was markedly higher in the autoimmune hepatic disease groups and fulminant hepatitis. However, the level of IL-18 in viral chronic hepatitis,hepatic fibrosis and cancer groups was significantly lower than that of the healthy control(P<0.05).
     结果106例肝病患者及胃癌患者血清中IL-17的水平较正常对照组均有升高(P<0.01),IL-18水平除了在自身免疫性肝病和急性暴发性肝炎中有升高外,而在慢性病毒性肝炎、肝纤维化、肝癌和胃癌中较正常对照均有不同程度的降低(P<0.05)。
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  急性重型肝炎
     Methods The expression levels of IL-8 in PBMCs from 36 patients with chronic liver disease (CLD), 17 patients with fulminant hepatitis, 11 patients with primary carcinoma of liver, and 18 normal controls (NCs) after exposure to phytahematoagglutinin (PHA) were measured by reverse transcription polymerase chain reaction (RT-PCR).
     方法 采用半定量逆转录 聚合酶链反应 (RT PCR)检测 36例慢性肝病患者、17例急性重型肝炎患者、11例原发性肝癌患者和 18名正常对照者PBMCs中IL 8mRNA的表达水平。
短句来源
     Materials and methods:Patients: 25 cases of fulminant hepatitis (Numbered as FH) aged from 25 to 56, 21 cases were male and 4 were female. Among them, FH (1) and FH (S24) were without previous hepatitis history; the other 23 were with previous hepatitis history.
     病例选自1999~2002年我院收治的HBV DNA阳性的重型乙型肝炎患者25例(编号FH),男21例,女4例,年龄25~56岁,其中FH(1),FH(S24)为急性重型肝炎,既往无肝炎病史,其余23例为慢性重型肝炎,既往有肝炎病史;
短句来源
     HGV Detection in liver tissues of patients with acute fulminant hepatitis.
     急性重型肝炎患者肝组织中庚型肝炎病毒的检测
短句来源
     EXPRESSION AND SINGNIFICANCE OF HGV ANTIGEN IN LIVER TISSUE OF PATIENT WITH ACUTE FULMINANT HEPATITIS
     庚型肝炎病毒抗原在急性重型肝炎患者肝组织中的表达及意义
短句来源
     The result was six of the twenty—two patients were positive for HGV RNA. The infection rate was 27.3%,in which subacute fulminant hepatitis was 36.4%(4/11) for HGV RNA positive,chronic fulminant hepatitis was 20.0%(2/10) for HGV RNA positive.
     结果发现22例重肝患者血清标本中检出6例HGVRNA阳性,阳性率27.3%,其中亚急性重型肝炎11例,HGVRNA阳性4例,阳性率36.4%,慢性重型肝炎10例,HGVRNA阳性2例,阳性率20.0%,急性重型肝炎1例未检出。
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      fulminant hepatitis
    Fulminant hepatitis is fatal in most cases and timely liver transplantation is the only effective treatment.
          
    In general, it is extremely difficult to treat fulminant hepatitis by conservative regimen, particularly, in cases with rapid progression.
          
    Emergency adult living-donor liver transplantation is an effective treatment for fulminant hepatitis patients and is relatively safe for donors.
          
    Construction of shRNA of fulminant hepatitis related gene mfgl2 and investigation of its biological effects in vitro
          
    He developed fulminant hepatitis with acute liver failure and an acute respiratory distress syndrom (ARDS).
          
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    Liver biopsies were performed in 110 cases of deep-jaundiced viral hepatitis of different types. Fulminant hepatitis showed two different types of liver cell damage. One was severe swelling of liver cells with foci of ceil necrosis of various extent, the mortality of this type was low (3/16). Theother type was characterized by massive cell necrosis, all of them died (3/3).The clinical diagnosis corresponded with the pathological findings in 66%. Thus liver biopsy might be used to correct the clinical...

    Liver biopsies were performed in 110 cases of deep-jaundiced viral hepatitis of different types. Fulminant hepatitis showed two different types of liver cell damage. One was severe swelling of liver cells with foci of ceil necrosis of various extent, the mortality of this type was low (3/16). Theother type was characterized by massive cell necrosis, all of them died (3/3).The clinical diagnosis corresponded with the pathological findings in 66%. Thus liver biopsy might be used to correct the clinical misdiagnosis, offer a histological basis for evaluating the treatment and prognosis, and as a possible way,for studying the machanism of viral hepatitis.

    110例黄疸深的各型病毒性肝炎作肝穿活组织检查,发现临床诊断为暴发型肝炎的病例有两种病理表现:一种是肝细胞严重肿胀伴有大小不等坏死灶病变,其病死率低(3/16)。另一种是肝细胞大块坏死病变,全部死亡(3/3)。本组临床诊断与病理相符者66%。因此,肝穿活检可纠正临床的误诊,并为临床评价疗效、判断预后提供病理依据,且将为研究其发病机制提供可能的途径。

    We attempted to find criteria of clinical diagnosis of chronic active hepatitis ( CAH ) and raise clinical diagnosis rate through analysing clinical data of 104 cases of patients with CAR confirmed by pathological examination of liver biopsy. But clinical features and laboratory findings of patients with CAH are extremely different. The mild patients may have-no symptom at all, but the severe patients may mimic the patients with acut fulminant hepatitis. Clinical diagnosis is still difficult without the...

    We attempted to find criteria of clinical diagnosis of chronic active hepatitis ( CAH ) and raise clinical diagnosis rate through analysing clinical data of 104 cases of patients with CAR confirmed by pathological examination of liver biopsy. But clinical features and laboratory findings of patients with CAH are extremely different. The mild patients may have-no symptom at all, but the severe patients may mimic the patients with acut fulminant hepatitis. Clinical diagnosis is still difficult without the pathological examination of liver biopsy. It is necessary to study how to-raise clirical diagnosis rate of CAH further.

    我们企图通过104例肝穿活检病理检查确诊为慢性活动性肝炎的临床分析,找出慢性活动性肝炎的临床诊断标准,以提高临床诊断率。但慢性活动性肝炎的临床表现和实验室所见极为悬殊,轻者可无明显症状,重者则酷似急性重症肝炎,如无肝穿活检病理检查,临床诊断仍然困难。怎样提高临床诊断率仍待进一步研究。

    Anti-HAV-IgM, anti-HBc-IgM, and three antibody systems of hepatitis B were tested in 48 patients with fulminant hepatitis to determine which of the hepatitis viruses A and B was the causative agent. HBV was found in 31 cases, HAV in one case, mixed infection of HBV and HAV in 15 cases (31.25%),and uncertain agent in 1 case.Ten cases out of the 15 with mixed infection were subacute, and the other 5 chronic. 13 patients were male and 2 female. The age of the patients ranged from 15 to 49 years with...

    Anti-HAV-IgM, anti-HBc-IgM, and three antibody systems of hepatitis B were tested in 48 patients with fulminant hepatitis to determine which of the hepatitis viruses A and B was the causative agent. HBV was found in 31 cases, HAV in one case, mixed infection of HBV and HAV in 15 cases (31.25%),and uncertain agent in 1 case.Ten cases out of the 15 with mixed infection were subacute, and the other 5 chronic. 13 patients were male and 2 female. The age of the patients ranged from 15 to 49 years with an average of 30.39. The clinical conditions of 10 cases became more serious and some of them even died during hospitalization. The clinical and biochemical parameters of the patients with mixed infection showed no statistical difference from those of the patients with single infection of either HBV or HAV of the same time period. 7 patients complained of sudden aggravation of their clinical conditions after they exhibited a group of symptoms and signs of "acute icteric hepatitis". One such case was found during his hospitalization and his anti-HAV-IgM turned positive at the same time. This patient died eventually.It is considered that aggravation of acute or chronic hepatitis can be resulted from superinfection and this problem deserves close attention.

    报告经血清免疫学抗HAVIgM、抗-HBcIgM、HBV-DNA、HBsAg/IgM复合物以及乙肝三种抗原抗体系统检测48例重型肝炎中甲、乙型肝炎病毒感染情况。结果:31例为HBV感染,1例为HAV感染,1例未定型,15例(31.25%)为甲、乙型合并感染(混合感染7例、重叠感染8例)。15例中亚急性10例、慢性5例;死亡或恶化10例,与同期“单纯”感染的重肝比较,包括主要临床生化指标均无显著差异。有7例患者病程中有近期出现“急黄肝”症状的历史,随后病情突然加重;1例发生在住院期间,且血清抗-HAVIgM转阳,最后死亡。认为合并感染可使部分急、慢性肝炎病情加重,甚至发展成重型。应注意发现。

     
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