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chronic     
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  慢性
     Experimental Model of Chronic Cervical Cord Compression and Primary Study on Pathomechanism of Chronic Compression
     慢性颈脊髓压迫症实验模型的建立及慢性压迫伤损伤机制的初步研究
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     Polarized Populations and Natural Killer Cells of T Helper Cells in Patients with Chronic Hepatitis B Virus Infection
     Th细胞极化群体及自然杀伤性T细胞在慢性乙型肝炎病毒感染中的作用
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     Research on cardiomyocyte apoptosis in acute and chronic myocardial ischemia
     急慢性心肌缺血心肌细胞凋亡的机制研究
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     Experimental Study on Chronic Myelogenous Leukemia Cells Vaccine Modified in vitro with Human Wild-Type p-53,GM-CSF and B7-1 Genes Mediated By Recombinant Adenovirus Vector
     重组腺病毒介导人野生型p53、GM-CSF和B7-1基因体外修饰慢性髓性白血病细胞瘤苗的实验研究
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     The Study of Cerebral Tissue Channel in Normal,Acute and Chronic Hypertensive Rats
     正常与急慢性高血压大鼠脑组织通道的研究
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     The study of topology of genetic material relative to chronic myeloid leukemia in interphase nucleous
     间期核内与粒相关的遗传物质拓扑结构的研究
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     The Experimental Research of P21, P27, P16, P15 Gene (Cyclin-denpendent Kinase Inhibitors) Clone and STI571 to Treat Chronic Myeloid Leukemia
     细胞周期抑癌基因p21、p27、p16、p15基因克隆联合STI571治疗粒的实验研究
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     Treatment of Chronic Keshan Disease with Coenzyme Q10——Ⅰ.A Clinical Study
     辅酶Q_(10)治疗型克山病的疗效观察 一、临床研究
短句来源
     STUDY ON CHROMOSOMES IN CHRONIC GRANULOCYTIC LEUKEMIA(CGL) Ⅲ.The Observation of Chromosomes in the Course of Ph~1 Positive CGL
     性粒细胞白血病(粒)染色体的研究——Ⅲ.Ph~1阳性粒病程中染色体的观察
短句来源
     Treatment of Chronic Keshan Disease with Coenzyme Q10 Ⅱ Evaluation of Cardiac Function by Mechanocardiography and Impedance Cardiography
     辅酶Q_(10)治疗型克山病的疗效观察 二 心脏功能研究
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  慢性病
     Cross Cultural Epidemiology and Health Related Quality of Life Measurement Among Primary Care Patients with Chronic Diseases
     跨文化社区慢性病流行病学及健康相关生命质量研究
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     The Effect of Health-Related Quality of Life (HRQoL) on Health Service Utilization of Patients with Chronic Disease
     健康相关生命质量(HRQoL)对慢性病患者卫生服务利用影响的研究
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     Studies on the Development, Application and Evaluation of Clinical Pathway for Four Kinds of Common Chronic Diseases and Two-way Referral System in Urban Communities
     城市社区四种常见慢性病临床路径和双向转诊机制的建立及应用评价研究
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     Social Nursing of Chronic Diseases: Analysis of 50 Cases
     50例老年慢性病的社会护理学分析
短句来源
     Application of Uniscale Method in Chronic Disease Studies:Iniroduction to a Quantitative Recording Method for Subjective Feeling or Symptoms
     单尺度方法在慢性病学研究中的应用——介绍一种主观感受的定量记录方法
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  慢性的
     73.8% of the injuries were chronic or acute to chronic injuries.
     73.8%的损伤是慢性损伤或急性转为慢性的损伤。
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     In the course of transformation from acute to chronic arthritis,the changes of c-fos expression in hippocampus were in accordance with that in spinal cord ,which indicated that pain relieved in the course.
     3、佐剂性关节炎由急性转为慢性的过程中,海马c-fos表达的变化与脊髓c-fos表达的变化趋势一致,说明从急性期到慢性期疼痛程度逐渐减轻;
短句来源
     Diabetic gastroparesis (DGP) is a common and chronic complication of diabetes.
     糖尿病胃轻瘫(DGP)是一种常见的、慢性的糖尿病合并症之一。
短句来源
     Considering insulin resistance is a chronic, slowly developmental process with the existence of insulin in vivo, 3T3-L1 adipocytes wore treated with dexamethasone and insulin, induced to form insulinresistance, then glucose in medium was measured with clinical assay in minima] way and the molecular mechanism of insulin resistance was studied.
     基于体内胰岛素抵抗是在胰岛素存在下的长期的、慢性的病理过程,因此,本实验采用地塞米松与胰岛素联合作用,体外诱导3T3-L1脂肪细胞产生胰岛素抵抗,并用经微量化后的临床糖检测试剂盒测量细胞对葡萄糖的摄取,并对胰岛素抵抗产生的分子机制进行研究。
短句来源
     Rheumatoid arthritis (RA) is a complex autoimmune disorder, characterized by a chronic T-cell response that has evaded normal control mechanisms.
     类风湿性关节炎(Rheumatoid arthritis,RA)是一种复杂的自身免疫性遗传疾病,其特征是慢性的针对正常机体组织的T淋巴细胞反应。
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      chronic
    To investigate the expression of JWA after hemin and (or) thermal stress exposure, we treated K562 (chronic myelogenous leukemia cells) cells with different doses of hemin and thermal stress using different exposure times.
          
    Clinically, 36.9% (15/41) were categorized as first onset type, 36.9% (15/41) were chronic persistent and 26.8% (11/41) were chronic recurrent.
          
    Effect of renal function and hemodialysis on the serum tumor markers in patients with chronic kidney disease
          
    It has been recently shown that some tumor markers are higher in patients with chronic kidney disease (CKD) than in the normal population.
          
    The 232 non-dialysis patients with CKD and 37 chronic uremic patients treated with maintenance hemodialysis were enrolled in this study.
          
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    Observations were made to compare the effect of HCl introduced into the small intestine of dogs on pancreatic secretion in the following three conditions: (1) Acute experiments with barbiturates(pentothal sodium or pentobarbital sodium) as an(?)sthesia; (2) Chronic experimental dogs with Thomas pancreatic fistula; (3) Chronic dogs in(2) an(?)sthetized by means of the same barbiturates as in the acute experiments. The results indicated that in the acute experiments, the latent period of pancreatic secretion was...

    Observations were made to compare the effect of HCl introduced into the small intestine of dogs on pancreatic secretion in the following three conditions: (1) Acute experiments with barbiturates(pentothal sodium or pentobarbital sodium) as an(?)sthesia; (2) Chronic experimental dogs with Thomas pancreatic fistula; (3) Chronic dogs in(2) an(?)sthetized by means of the same barbiturates as in the acute experiments. The results indicated that in the acute experiments, the latent period of pancreatic secretion was about 3-5 minutes and the amount secreted was usually below 20 drops in the first 30 minutes after the introduction of the acid. But in the case of chronic experiments, the results were quite different. The latent period was shorter, usually less than 2 minutes and the amount of pancreatic secretion was usually 10 times as much as in the acute experiments. Moreover, the pancreatic secretion of the chronic fistula dogs was not changed either in latent period or in amount when under the barbiturate an(?)sthesia. From the above results, it is quite natural to assume that the influence of the barbiturate an(?)sthetics, pentothal sodium and pentobarbital sodium on pancreatic secretion induced by HCl in the intestine of dogs is negligible. The great difference in the pancreatic secretion between the acute and the chronic experiments might, therefore, be due to the operative trauma which is unavoidable in acute experiments. Another observation was made to determine the effect of atropine on pancreatic secretion induced by HCl in chronic fistula dogs. The result showed that atropine distinctly reduced the response of pancreas to the acid. The latent period was increased and the amount of pancreatic secretion was decreased as compared with the result obtained in normal chronic fistula dogs. Hence it is quite suggestive that there might exist a nervous component in the mechanism of pancreatic secretion induced by HCl in the small intestine. We wish to express our gratitude to Comrade for his valuable advice and to professors T. C. Shen and T. F. Liu for their encouragement throughout this work.

    本實驗比較急性實驗狗、慢性胰瘻狗和經過麻醉的慢性胰屢狗對於鹽酸注入小腸所引起的胰液分泌量和潛伏期,結果證明: (1)在急性實驗情况下,狗胰腺對鹽酸刺激小腸所引起的胰液分泌量遠較在慢性實驗時為少,且潛伏期較長。 (2)巴比妥類麻醉劑:硫賁妥鈉(sodium pentothal)和戊烷巴比妥鈉(sodiumpentobarbital)對鹽酸所引起的胰液分泌量及潛伏期影響極微。 (3)在急性實驗情况下,由鹽酸所引起的胰液分泌量的減少和潛伏期的加長,似乎不是由於巴比妥類麻醉劑的作用,而可能是由於手術創傷的影響。 (4)注射阿托平後,胰腺對於鹽酸刺激小腸所引起的反應顯著减小,故推测在鹽酸引起胰液分泌的機制中可能有神經反射作用的參與。本工作在进行過程中,承蘇聯專家同志親切地給予指導,并承沈(?)淇、劉曾復二教授关懷和支持,(?)此誌謝。

    It has been shown in our previous paper that intravenous injection of adrenalin and stimulation of the splanchnic nerve in the dog produced an inhibition of gastric secretion induced by histamine. Baxter, working on cats under experimental conditions comparable to ours, however, reported that intravenous injection of adrenalin either had no marked effect or a slightly augmentative effect on the histamine-induced secretion, and that stimulation of the splanchnic nerve yielded similar results. The question thus...

    It has been shown in our previous paper that intravenous injection of adrenalin and stimulation of the splanchnic nerve in the dog produced an inhibition of gastric secretion induced by histamine. Baxter, working on cats under experimental conditions comparable to ours, however, reported that intravenous injection of adrenalin either had no marked effect or a slightly augmentative effect on the histamine-induced secretion, and that stimulation of the splanchnic nerve yielded similar results. The question thus arose as to whether the discrepancy between Baxter's and our reports was due to the use of different experimental animals. Experiments were therefore carried out on cats in our laboratory in an attempt to throw some light on the question. It was observed that, in both acute and chronic experiments, intraveous injection of adrenalin in most cases produced a marked diphasic effect on the secretion induced by histamine. The effect consisted of an initial phase of inhibition followed by one of augmentation, the two phases being usually about equal in size, sometimes the second phase somewhat larger than the first. When a dose of 0.02-0.1mg of adrenalin was administered in a single injection intravenously, the total duration of the diphasic response lasted 10-15min. It would be evident that if rather long intervals, e.g. 10-30min. were chosen for the collection of gastric juice, the diphasic feature of the response would be missed, and one might easily come to the conclusion that in the cat adrenalin either had no marked effect or an augmentative effect on the histamine-induced secretion, as Baxter did. In acute experiments, the stimulation of the splanchnic nerve showed an inhibitory effect on the secretion, disregarding whether the adrenal veins were ligated or not. In contrast with the adrenalin effect, that of splanchnic stimulation was rarely diphasic. We wish to express our deep gratitude to Prof. T. P. Feng for his constant guidance throughout this work.

    (一)靜脈注射腎上腺素對組織胺引起的貓胃分泌不論在急性或慢性實驗,通常是双相的,開頭抑制分泌,接着增加分泌,兩相大小相似,有時第二相還稍為大些。在一次注射0.02—0.1毫克腎上腺素之後,整個效應過程歷時約10—15分钟。若用較長的間隔如每10—30分鐘收集一次胃分泌,則此双相效應就會被掩蓋,因而得出腎上腺素對貓胃分泌無明顯效應或有增加分泌的效應的結論,如Baxter等人所得到的一樣。 (二)在急性實驗中,刺激大内臟神經對组織胺引起的貓胃分泌有顯著的抑制效應。与腎上腺素的效應不同,刺激大內臟神經的效應通常是單相的。我們在工作中经常得到馮德培所长的指导。谨致谢意.

    The acute and chronic toxicities of parathion, O,O-diethyl O-p-nitrophenyl thiophosphate, in the white mice (Mm rmtseulus albus Bechstein) were investigated by oral administrations. The dosage rate was based on the weight of the mouse; and the calculated volumes of the diluted parathion emulsion in various concentrations were fed to the test animals by means of a needle of the micrometer syringe. The various dosage rates for five groups of mice were as follows:(1) A single oral dose of 20 mg. of parathion...

    The acute and chronic toxicities of parathion, O,O-diethyl O-p-nitrophenyl thiophosphate, in the white mice (Mm rmtseulus albus Bechstein) were investigated by oral administrations. The dosage rate was based on the weight of the mouse; and the calculated volumes of the diluted parathion emulsion in various concentrations were fed to the test animals by means of a needle of the micrometer syringe. The various dosage rates for five groups of mice were as follows:(1) A single oral dose of 20 mg. of parathion per kg. of body weight;(2) A daily dose of 5 mg. of parathion per kg. of body weight;(3) A daily dose of 10 mg. of parathion per kg. of body weight;(4) The mouse recieved a dose of 1 mg. of parathion per kg. of body weight at the 1st day and thereafter the dosage was increased by 1 mg. per kg. of body weight with the increase of days;(5) The mouse recieved 2 mg. of parathion per kg. of body weight at the 1st day and the dosage was increased by 2 mg. per kg. with the increase of days.In the above various daily dosage rates for the different groups of mice, parathion was fed continuously over a period of 12 days except in cases when all the test animals under experiments were dead before that period of time. The experimental results may be summarized as follows.1. The symptoms of parathion poison in the white mice were observed at follows: inaction, slight tremor, followed by omitting, tears, diarrhea, and the intensive convulsion. Finally the mouse with its erected and stiffened tail ran about for a few seconds and died.2. Either in treatments of a single acute oral dose or of daily oral dosages of parathion in succesion, the poisoned mice showed no apparent differences in susceptibilities between males and females or among animals with various body weights.3. It was found that a single dase acute oral LD50 value within 24 hours was between 10 mg. and 20 mg. of parathion per kg. of body weight.4. The average period of time required from the last administrations to the dealh of the animals was about 2 hours in the treatment (1), (3), (4), and (5). The treatment (2) took about 7 hours. In all the treatments, the required time varied from only 16 minutes to 11 hours.5. In the treatment of daily dosage of 5 mg./kg. 6 mice died within 4 days after the beginning of the administration, 9 mice survived to the end of the 12 days, having taken a total dosage of 60 mg./kg.The treatment with a daily dosage of 10 mg./kg. showed that 11 out of the 15 tested mice died before the 3rd day. Of the remaining 4 mice, 2 died suddenly at the 7th day, while the other 2 survived to the end of the 12th day, having taken a total dosage of 120 mg./kg.In those cases, when the mice recieved a dose of 1 mg./kg. at the 1st day, and then dosage increased by 1 mg./kg. at the successive days, some of the mice (so treated) died at the 4th day and the death of the poisoned mice continued to happen till the end of the 12th day. Only 3 out of the 15 mice survived after haying taken a last dose of 12 mg./kg. and total dosage of 78 mg./kg.In those cases, when the mice recieved a dose of 2 mg./kg. at the 1st day and continuously recieved a dosage which increased by 2 mg/kg. at the successive days, all the 15 test mice died before the 8th day, after having taken a total dosage of 72 mg./kg.The results of the four above dosage treatments showed that the daily administration of parathion at sublethal dosages to the mice did not apparently have any accumulative action, while the daily dosages near the acute lethal level did cause some susceptible mice to die. This may be due to the possibility that the poisoned mice could not recover their cholineste-rase level enough to resist the inhibiting effects of the next applications of parathion.

    1.小白鼠吞食“E.605”後的中毒症状为:痉挛、呕吐、流泪、不成次地排便,最後痉挛加剧,突然间尾竖直作惊慌状,爬行数秒钟,而倒伏气绝。 2.小白鼠吞食“E.605”致死50%剂量介於10—20毫克/千克。雌雄两性或不同体重的小白鼠对“E.605”的忍受力,未见有显明的差别。 3.小白鼠吞食“E.605”後,中毒死亡距各鼠最後一次吞食药剂的时间:吞食5毫克/千克/每日剂量的,为7小时,吞食10毫克/千克/每日,1毫克/千克/每日递增,或2毫克/千克/每日递增3种剂量的,平均各约为2小时,最快的仅为16分钟,最慢的不超过11小时。 4.小白鼠连日吞食“等剂量”的“E.605”,如吞食剂量远较各该鼠的一次吞食的致死剂量为小时,未见有因连日剂量积累而致中毒死亡的趋势,但个别对药剂忍受力小的鼠只在吞食“E.605”的剂量已接近於各该鼠的一次吞食致死剂量时,也能在再一次或多次接受一定量的“E.605”,而遭致中毒死亡。这点似可以解释为鼠体内胆硷酯酶受抑制後的恢复速度赶不上继续给药的被抑制量的缘故。小白鼠每日吞食的“E.605”剂量如果是逐日递增的,则在某一日或连续几日内的剂量已经增加到接近或达到各该鼠的致死剂量时,便中...

    1.小白鼠吞食“E.605”後的中毒症状为:痉挛、呕吐、流泪、不成次地排便,最後痉挛加剧,突然间尾竖直作惊慌状,爬行数秒钟,而倒伏气绝。 2.小白鼠吞食“E.605”致死50%剂量介於10—20毫克/千克。雌雄两性或不同体重的小白鼠对“E.605”的忍受力,未见有显明的差别。 3.小白鼠吞食“E.605”後,中毒死亡距各鼠最後一次吞食药剂的时间:吞食5毫克/千克/每日剂量的,为7小时,吞食10毫克/千克/每日,1毫克/千克/每日递增,或2毫克/千克/每日递增3种剂量的,平均各约为2小时,最快的仅为16分钟,最慢的不超过11小时。 4.小白鼠连日吞食“等剂量”的“E.605”,如吞食剂量远较各该鼠的一次吞食的致死剂量为小时,未见有因连日剂量积累而致中毒死亡的趋势,但个别对药剂忍受力小的鼠只在吞食“E.605”的剂量已接近於各该鼠的一次吞食致死剂量时,也能在再一次或多次接受一定量的“E.605”,而遭致中毒死亡。这点似可以解释为鼠体内胆硷酯酶受抑制後的恢复速度赶不上继续给药的被抑制量的缘故。小白鼠每日吞食的“E.605”剂量如果是逐日递增的,则在某一日或连续几日内的剂量已经增加到接近或达到各该鼠的致死剂量时,便中毒死亡。各该鼠中毒致死前的最後一次所吞食的“E.605”剂量,起了重要的致毒作用。

     
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