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   leukoplakia 在 口腔科学 分类中 的翻译结果: 查询用时:0.202秒
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leukoplakia
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  白斑
    C-Ha-ras Oncogene in Oral leukoplakia Tissues
    口胶粘膜白斑组织中C-Ha-ras癌基因的研究
短句来源
    Expression and Impact Factor of Proliferating Cell Nuclear Antigen in Oral Leukoplakia
    PCNA在口腔白斑的表达及影响因素
短句来源
    Study of DNA Ploidy in Oral Leukoplakia and Squamous Cell Carinomas
    口腔白斑及鳞癌组织中DNA倍体值的研究
短句来源
    The expression of epidermal growth factor receptor of oral leukoplakia tissue
    口腔白斑组织中表皮生长因子受体的表达
短句来源
    Relation Between Infection of HPV (Human Papilloma viruses) and Oral Leukoplakia
    口腔白斑与人乳头状瘤病毒的感染关系
短句来源
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  粘膜白斑
    Results The positive rate of PTEN protein was 72.2%(52/72)、93.3%(14/15)、100%(10/10) and 100%(10/10) in OSCC,oral leukoplakia,oral epithelial hyperplasia and normal oral mucosa,there was significant correlation among them(P<0.05).
    结果PTEN基因在口腔鳞癌、口腔粘膜白斑、口腔上皮单纯增生和正常口腔粘膜组织中的阳性率分别为72.2%(52/72)、93.3%(14/15)、100%(10/10)和100%(10/10),口腔鳞癌组与其他各组间有显著性差异(P<0.05);
短句来源
    Method:Livin genes in 48 cases of oral leukoplakia(the case included 12 mild hyperlasis,18 medium atypical hyperplasis,18 severe atypical hyperplasis) and 32 cases of oral carcinoma were detected by immunohistochemical staining(S-P).
    方法:应用免疫组化(S-P)法检测Livin基因在48例口腔粘膜白斑(轻度增生l2例,中度增生l8例,重度增生l8例)、口腔癌32例中的表达。
短句来源
    Alterations in expession of basement membrane protein in oral leukoplakia and squamous cell carcinoma
    口腔粘膜白斑、鳞癌基底膜蛋白的表达异常
短句来源
    Expression of bcl-2、p53 protein in oral leukoplakia and squamous cell carcinoma
    口腔粘膜白斑和鳞癌组织中p53和bcl-2蛋白的表达
短句来源
    Expression of bcl-2 protein and P~(21 waf1) protein during the occurrence and development of oral mucosa leukoplakia.
    口腔粘膜白斑发生发展中bcl-2、P~(21 wafl)蛋白的表达
短句来源
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  黏膜白斑
    METHODS: Expressions of Survivin were detected in normal oral mucosa (10 cases), oral leukoplakia (34 cases) and oral squamous cell carcinoma (OSCC) (27 cases) by immunohistochemical techniques using anti-Survivin monoclonal antibody.
    方法:采用免疫组织化学方法检测口腔正常黏膜(10例),口腔黏膜白斑(34例)和鳞状细胞癌(27例)中Survivin的表达。
    EXPRESSION OF MATRIX METALLOPROTEINASES IN ORAL LEUKOPLAKIA AND ORAL CANCERATION TISSUE
    基质金属蛋白酶在口腔黏膜白斑及其癌变组织中的表达
短句来源
    Expression of telomerase genes in oral leukoplakia and oral carcinoma
    端粒酶基因在口腔黏膜白斑与口腔癌中的表达
短句来源
    The retrospective analysis of oral leukoplakia in old people
    中老年人口腔黏膜白斑的回顾性分析
短句来源
    Research on Expression of Survivin in Oral Leukoplakia and Oral Squamous Cell Carcinoma
    Survivin在人口腔黏膜白斑和鳞状细胞癌中表达的研究
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  “leukoplakia”译为未确定词的双语例句
    PURPOSE: To evaluate the relationship of mRNA and protein level of p16CDKN2A during the experimental rat tongue leukoplakia.
    目的:探讨p16CDKN2A在实验性口腔白斑病损发生发展过程中的转录与翻译水平的改变,分析两者的关系。
    CONCLUSION: p16CDKN2A may play role(s) in 4NQO-induced rat tongue leukoplakia by posttranscriptional mechanism.
    结论:p16CDKN2A可能以转录后调控机制参与4NQO诱导的实验性大鼠舌部癌前损害的发生发展。
    The Age-related Changes of P53 Protein Expression in Oral Mucosa and Their Effects on Oral Leukoplakia
    P53蛋白在正常口腔粘膜和口腔白斑病中的增龄性变化
短句来源
    Posttranscriptional mechanism of p16~(CDKN2A) in the experimental rat tongue leukoplakia
    p16~(CDKN2A)在大鼠白斑病损发生发展中的转录后调控机制
    1. The three different types of fibroblasts of normal oral mucosa, oral leukoplakia and OSCC were cultured in vitro.
    1.对口腔正常粘膜、OLK及OSCC三种不同组织的成纤维细胞进行体外培养;
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  leukoplakia
These results and the established activity of retinoids in oral leukoplakia (a precursor of head and neck cancer) indicate the need for further study of this class of drugs in head and neck cancer.
      
There is little evidence that leukoplakia of the anal canal is premalignant but patients should be followed carefully, since the natural history of this rare lesion is unknown.
      
Two cases of leukoplakia of the anal canal are presented, with illustrations of the typical gross and microscopic appearance.
      
Retinoids are the most studied chemopreventive agents for the treatment of oral leukoplakia.
      
Successful culture of premalignant lesions (dysplastic mucosa, leukoplakia, erythroplakia) has resulted in establishment of a limited number of premalignant cell lines and cell cultures.
      
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Abstract The surface ultrastructure of experimental leukoplakic patches and human oral leukoplakia was studied by scanning electron microscopy.Especially the microplicae of surface cells was quantitatively analysed.The result showed that the simplex hyperplasia leukoplakic patches of hamster cheek pouch extremely coincided with that of human in the changes of ultrastracture,that is, the furrows between two adjacent microplicae were widened;The microplicae of dysplastic leukoplakic patches of hamster cheek...

Abstract The surface ultrastructure of experimental leukoplakic patches and human oral leukoplakia was studied by scanning electron microscopy.Especially the microplicae of surface cells was quantitatively analysed.The result showed that the simplex hyperplasia leukoplakic patches of hamster cheek pouch extremely coincided with that of human in the changes of ultrastracture,that is, the furrows between two adjacent microplicae were widened;The microplicae of dysplastic leukoplakic patches of hamster cheek pouch and human lingua dorsum was progressivery changed.The widths of microplicae af the former was widened and the furrows of between two adjacent microplicae of the latter was mainly widened,These results suggested that the changes of the microplicae progress with histologic changes and that SEM may detect early cancerous transformation of Oral leukoplakia.

用SEM技术,对仓鼠颊囊的实验性白斑样损害和人口腔白斑的表面超微结构进行了比较研究,重点定量观察了上皮细胞表面的微皱襞的改变情况,结果显示正常的仓鼠颊囊粘膜与正常人的舌背及颊囊粘膜均有相似之处;仓鼠单纯增生性白斑和人舌背的单纯增生性白斑的表面超微结构极其一致,都以微皱襞间宽增宽为特征;仓鼠颊囊不典型增生性白斑的表面细胞微皱襞和人舌背不典型增生性白斑的表面细胞微皱襞进一步发生改变,前者以微皱襞宽增宽为主,后者以微皱襞间宽增宽更为显著。这些改变说明仓鼠颊囊白斑模型的可用性,也说明上皮细胞微皱襞会随着组织学上的改变而改变,暗示SEM可用于口腔白斑癌变的监测。

Abstract Leukoplakia, lichen planus and hemangioma are common diseased in the oral mucous membrane.In this paper,27 cases were reported and analysed by microwave tissue thermo-coagulation method. There were 15 cases of hemangioma, 7 cases of lichen planus,2 cases of leukoplakia and 3 cases of the others. Among them 13 were male and 14 female. The lesions in the oral mucous membrane were located. 8 cases were in the cheek, 7 cases in the lip, 5 cases in the tongue, 4 cases in the palate and 3 cases in...

Abstract Leukoplakia, lichen planus and hemangioma are common diseased in the oral mucous membrane.In this paper,27 cases were reported and analysed by microwave tissue thermo-coagulation method. There were 15 cases of hemangioma, 7 cases of lichen planus,2 cases of leukoplakia and 3 cases of the others. Among them 13 were male and 14 female. The lesions in the oral mucous membrane were located. 8 cases were in the cheek, 7 cases in the lip, 5 cases in the tongue, 4 cases in the palate and 3 cases in the commissura labiorum. The mechanism and therapeutics of microwave tissue thermo-coagulation have been discussed.

自1993年6~12月,应用微波热凝治疗口腔常见病,计血管瘤15例,扁平苔藓7例,白斑2例,天疱疮1例,慢性唇炎1例,粘液腺囊肿1例,总计27例。其中痊愈21例,好转6例,无恶化病例,未发生严重并发症。对微波热凝的作用机制、治疗方法、疗效及注意事项进行了讨论。

Abstract Established a sensitive and specific method which can amplify multiple types HPVs DNA by degenerate primers PCR technique. The technique specifically amplify five types HPV (6, 11, 16, 18, 33)at least, without no-specific amplification of human DNA. Amplified HPVs DNA in formalin-fixed paraffinembedded tissues of oral squamous cell carcinoma and oral leukoplakia with carcinomatous transformation by this technique, the positive cases in these two groups are 22/30 and 23/30 respectively. The results...

Abstract Established a sensitive and specific method which can amplify multiple types HPVs DNA by degenerate primers PCR technique. The technique specifically amplify five types HPV (6, 11, 16, 18, 33)at least, without no-specific amplification of human DNA. Amplified HPVs DNA in formalin-fixed paraffinembedded tissues of oral squamous cell carcinoma and oral leukoplakia with carcinomatous transformation by this technique, the positive cases in these two groups are 22/30 and 23/30 respectively. The results indicated that HPVs might play an important role in the carcinogenesis of oral squamous cell carcinoma.

建立了一种简并引物PCR技术扩增多型人乳头瘤病毒(HPV)DNA的方法,本方法至少可以特异地扩增HPV6,11,16,18及33。将其用于扩增福尔马林固定石蜡包埋口腔鳞状细胞癌及癌变白斑组织中的HPVsDNA,其阳性例数分别为22/30,23/30例。本研究表明HPV极可能参与了口腔鳞状细胞癌和癌变白斑的癌发生。

 
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