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tracheal cannula
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  气管插管
     Method 20 general anesthesia patients of ASA1~2 were continually given 1.5% Iso volatilizal jar target concentration with 1L/min O2 after tracheal cannula, estimated FETIso by anesthesia gas monitor.
     方法 20例ASA1~2级全麻病人 ,气管插管后持续以1L/minO2 载入挥发罐1.5%Iso靶浓度 ,麻醉气体监测仪测定病人呼气末Iso浓度 (FETIso)。
短句来源
     The days in ICU ( P =0.000),usage of broad-spectrum antibiotic ( P =0.018),critical bacterial infection ( P =0.000),COPD ( P =0.014),and tracheal cannula ( P =0.011) were significant risk factors.
     在单因素分析中 ,入住 ICU时间 (P=0 .0 0 0 )、使用广谱抗生素种类数 (P=0 .0 18)、合并严重细菌感染 (P=0 .0 0 0 )、具有 COPD基础 (P=0 .0 14 )、气管插管 (P=0 .0 11)为发生深部真菌感染的危险因素 ;
短句来源
     The volume of secretion in airway in Ⅱgroup and Ⅲ group was significantly less than that in Ⅰgroup from before induction to after tracheal cannula insertion 2 h(P<0.05).
     诱导前至气管插管后2h,Ⅱ组和Ⅲ组患者的气道分泌物明显少于Ⅰ组(P<0.05)。
短句来源
     The exogenous PS was instilled via a tracheal cannula in a stogie dose of 150mg phospholipid/kg.
     PS剂量为150mg磷脂/kg,从气管插管一次滴入。
短句来源
     Methods:Before the operation, Anisodamine, Ketamine and Vecuronium bromide were injected intravenously with dosage of 0.06mg, 2mg·kg -1 and 0.08-0.1mg·kg -1, respectively. Tracheal cannula or topical anesthesia using 1% of lidocaine hydrochloride was performed.
     方法 :术前用山莨菪碱 0 .0 6mg ,诱导用氯胺酮 2mg·kg-1、维库溴铵 0 .0 8~ 0 .1mg·kg-1静脉注射后行气管插管 ;
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  气管套管
     Results Observation group showed significant difference compared to the control group on the respects of life quality total score, psychiatry evaluation, social communication,food take in, tracheal cannula care (P<0.05).
     结果:观察组病人在生活质量总评、精神心理状态、社会交流技术、进食、气管套管护理评分明显优于对照组(P<0.05)。
短句来源
     Results Demends of eating skill, selves nursing knowledge of tracheal cannula and stoma and announcements of daily life and activity were 100%.
     结果进食技巧、气管套管及造瘘口的自我护理知识、日常生活和活动注意事项的需求达100%;
短句来源
     Except for 3 cases with total laryngectomy, larynx preservation were done in 14 cases and tracheal cannula were pulled out in 8, blocked in 2 and kept carrying in 4 cases.
     除 3例全喉切除患者外 ,14例成功保留喉功能 ,其中拔除气管套管 8例 ,堵管 2例 ,继续带管 4例。
短句来源
     It summarized airway humidification,sputum aspiration,chest physiothetapy,complication prevention,management of tracheal cannula,and nutrition,orificium fistulae,and tube fallig off nursing care of patients underwent tracheotomy.
     综述了气道湿化、吸痰方法、胸部物理疗法、预防并发症、气管套管的管理以及营养、瘘口、脱管的护理。
短句来源
     The sputum was collected from the tracheal cannula for bacterial culture on the 3rd day and the 15th day after the tracheotomy, respectively.
     两组分别在气管切开后第3天、第15天,从气管套管内留取痰标本进行细菌培养。
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  “tracheal cannula”译为未确定词的双语例句
     1 case had two times tracheal cannula and received windpipe-cutdown ensuring ARDS;
     1例术后二次插管、随后气管切开 ,并发ARDS;
短句来源
     Asphyxial anoxia was induced by clamping the tracheal cannula in the anesthetized rat. GSL was given intravenously in dose of 100mg/kg.
     人参茎叶总甙(GSL)100mg/kg静脉注射,提高大白鼠抗窒息性脑缺氧能力,能促进皮层脑电与呼吸正常节律的恢复。
短句来源
     96 p atients were treated with tracheal cannula,continuous positive airway pressure(C PAP),phenobarbital,clonazepam,diazepam,chloral hydrate,mannitol,furosemide,album in and dexamethasone. Results Among the 96 patients,54 were cured(56 25%),31 improved(32 29%),9 auto-discharge (9 38%) and 2 died(2 08%).
     结果  96例中 ,治愈 5 4例 (5 6 2 5 % ) ,好转 31例 (32 2 9% ) ,自动出院 9例 (9 38% ) ,死亡 2例 (2 0 8% )。
短句来源
     The common induc-ing factors are the using of corticosteroids andchemotherapy (34 cases , 58%) . tracheotoniy and tracheal cannula (7 cases . 11. 6% ).
     常见的诱发因素为应用糖皮质激素和化疗34例(56%)、气管切开和插管7例(11.6%)。
短句来源
     2. Grouping: (1)Control group ( C ): ACAR: Tracheal cannula was shut at end - expiration, rats were given artificial ventilation, chest compression 180 - 2207min and iv adnephrine 0. 02mg/kg when heart arrested ( showed as the cardiac functional curve be a line).
     2.实验分组:①对照组*,n=8人于呼气末夹闭气管插管,心跳停止~心功能曲线为直线广刻恢复人工通气,静推肾上腺素0.02m才kg,胸外按压180-220 w分,根据心电图及心功能的变化判断自主循环是否恢复。
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  tracheal cannula
In the studies to investigate leukotriene antagonism; anesthetized, spontaneously breathing guinea pigs were pretreated with propranolol and were exposed via tracheal cannula to aerosols generated by a Monaghan nebulizer.
      
The surfactant, obtained by centrifugation of lung wash from adult rabbits, was deposited in the tracheal cannula before the onset of ventilation.
      
The animal rebreathed through a tracheal cannula for a period of 4 min from an apparatus of 200-400 ml capacity, containing 5-6% CO2 in O2.
      
To permit studies at different states of lung inflation the tracheal cannula is connected to a spirometer circuit.
      
In experiments with rabbits animals inhaled gases through a tracheal cannula under urethane narcosis.
      
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Clinically most of the deaths from tartar emetic intoxication were due to cardiac impairment,and animal experiments also demonstrated depression of the heart.The present work investigated the hemodynamic effects of tartar eme- tic in order to elucidate the circulatory disturbance during its intoxication. In 26 dogs,anesthetized with chloraloso,cardiac output was estimated by Fick’s principle.Cardiac catheters were inserted under fluoroscopic guidance.Blood was drawn from pulmonary artery and aorta,and the oxygen...

Clinically most of the deaths from tartar emetic intoxication were due to cardiac impairment,and animal experiments also demonstrated depression of the heart.The present work investigated the hemodynamic effects of tartar eme- tic in order to elucidate the circulatory disturbance during its intoxication. In 26 dogs,anesthetized with chloraloso,cardiac output was estimated by Fick’s principle.Cardiac catheters were inserted under fluoroscopic guidance.Blood was drawn from pulmonary artery and aorta,and the oxygen content was analyzed by Roughton-Scholander's method.Electromanometer was used to measure the pressure in pulmonary artery and right atrium.Oxygen consumption was determined with a BMR apparatus which was connected to a tracheal cannula.The normal values (mean±s.d.)were:cardiac output 1.94±0.6 1/min,cardiac index 3.0±0.9 1/min/m~2,stroke volume 11±3 ml,aortic mean pressure 168±21 mm Hg,pulmo- nary arterial mean pressure 20±6 mm Hg,total peripheral resistance 8000±2800 dyne.sec.cm~(-5),work of left ventricle 7.1±2.4 kg.m/min/m~2,and work of right ventricle 0.65±0.31 kg.m/min/m~2. In the control group,there was no remarkable change in the cardiac hemody namics within 5 hours. An intravenous injection of a therapeutic dose of tartar emetic(2 mg/kg) did not produce significant effects on the cardiac hemodynamics. A toxic dose(15 mg/kg)induced a noticeable hypotension.Electrocardiogram revealed a flattening or inversion of T wave.The change of the total periphera resistance was,however,not great. When the dosage was increased above 20 mg/kg,the cardiac depression be- came marked,with gradual weakening of the left ventricle.The work of the right ventricle was first increased in compensation for the augmented pulmonary re- sistance,and then decreased.The total peripheral resistance rose tremendously,indi cating constriction of most arterioles.Thus the hypotensive effect of tartar emetic was attributed chiefly to cardiac depression.The dogs died in 4—5 hours.

吐酒石中毒死亡病人大多死于心臟因素,动物实驗也表現有抑制心脏作用,本文从心臟血流动力作用的变化来說明吐酒石中毒时的循环障碍。狗26只,三氯醛糖麻醉,根据Fick氏原理测定心輸出量。在X光透視下插心脏导管取肺动脉及主动脉血,用Roughton-Scholander氏法测定血氧含量。基底代谢测驗器測定氧消耗量,电測压计測定右心房及肺动脉平均压。給藥前各項正常数值(平均±标准差)为:心輸出量1.9±0.6升分,心臟指数3.O±0.9升/分/米~2,心搏量11±3毫升,主动脉平均压168±21毫米汞柱,肺动脉平均压20±6毫米汞柱,总外周阻力8000±2800达因·秒·厘米~(-5),左心室作功量7.1±2.4千克·米/分/米~2,右心室作功量0.65±0.31千克·米/分/米~2。对照組狗在5小时内心輸出量及心搏量基本稳定,主动脉、肺动脉平均压及血管阻力均无大变化,心电圖正常,心室作功量无抑制現象。吐酒石的一次給藥量(2毫克/千克)未引起心臟血流动力作用的显著变化。吐酒石的中毒剂量在15毫克/千克时引起血压显著下降,心輸出量在2小时后似有减少傾向,心电圖T波变平或倒置,总外周阻力变化則不大。剂量在20毫克/千克以上...

吐酒石中毒死亡病人大多死于心臟因素,动物实驗也表現有抑制心脏作用,本文从心臟血流动力作用的变化来說明吐酒石中毒时的循环障碍。狗26只,三氯醛糖麻醉,根据Fick氏原理测定心輸出量。在X光透視下插心脏导管取肺动脉及主动脉血,用Roughton-Scholander氏法测定血氧含量。基底代谢测驗器測定氧消耗量,电測压计測定右心房及肺动脉平均压。給藥前各項正常数值(平均±标准差)为:心輸出量1.9±0.6升分,心臟指数3.O±0.9升/分/米~2,心搏量11±3毫升,主动脉平均压168±21毫米汞柱,肺动脉平均压20±6毫米汞柱,总外周阻力8000±2800达因·秒·厘米~(-5),左心室作功量7.1±2.4千克·米/分/米~2,右心室作功量0.65±0.31千克·米/分/米~2。对照組狗在5小时内心輸出量及心搏量基本稳定,主动脉、肺动脉平均压及血管阻力均无大变化,心电圖正常,心室作功量无抑制現象。吐酒石的一次給藥量(2毫克/千克)未引起心臟血流动力作用的显著变化。吐酒石的中毒剂量在15毫克/千克时引起血压显著下降,心輸出量在2小时后似有减少傾向,心电圖T波变平或倒置,总外周阻力变化則不大。剂量在20毫克/千克以上,心臟抑制更为明显,左心室作功量逐漸减弱,右心室作功量初加强以代偿增高之肺血管阻力,当中毒程度加深后亦趋衰竭。中毒时总外周阻力增大,小动脉大多收縮,指示吐酒石之降压作用主要由于抑制心臟,狗在給藥后4—5小时死亡。

A case of hyaline membrane disease was treated successfully with pulmonary surfactant (PS) we had isolated from human amniotio fluid. The exogenous PS was instilled via a tracheal cannula in a stogie dose of 150mg phospholipid/kg. 24 hours after the administration of PS, clinical symptoms, transoutaneous partial pressure of oxygen (ToPO2) and fractional inspired oxygen concentration (FiO2) were significantly improved. The leoithin/sphingomyelin (L/S) ratio, phosphotidylgly-cerol (PG) and minimal surface...

A case of hyaline membrane disease was treated successfully with pulmonary surfactant (PS) we had isolated from human amniotio fluid. The exogenous PS was instilled via a tracheal cannula in a stogie dose of 150mg phospholipid/kg. 24 hours after the administration of PS, clinical symptoms, transoutaneous partial pressure of oxygen (ToPO2) and fractional inspired oxygen concentration (FiO2) were significantly improved. The leoithin/sphingomyelin (L/S) ratio, phosphotidylgly-cerol (PG) and minimal surface tension (γmin) of traoheal aspirates gradually became normal. The radiologio finding "ground glass appearance" .in the lung fields showed striking improvement 3 days after PS treatment. The diseased condition was stabilized on the sixth day. She was eventually discharged with complete recovery.

作者用人羊水肺表面活性物质(PS)治疗一例肺透明膜病取得成功。PS剂量为150mg磷脂/kg,从气管插管一次滴入。临床症状,经皮测氧(TcPO_2)和吸入氧浓度(FiO_2)24h后明显好转,气道吸出物中的卵磷脂/鞘磷脂(L/S)比值、磷脂酰甘油(PG)和最低表面张力(γmin)逐渐恢复。肺部X线片原示毛玻璃样改变,PS治疗后3d明显改善,第6天病情基本恢复,最后痊愈出院。

Forty nine premature rabbits of gestational age of 27 days were used as models of hyaline membrane disease (HMD) in our experiments. They were divided into 4 groups. Group A and B were treated by human amniotio fluid PS and the mixed PS (a mixture of human amniotio fluid PS plus dipalmitoyl phosphatidyl choline DPPO, and phosphatidylglyoerol PG) respectively. Groups C1 and C2 were served as controls, C1 being treated by DPPO and PG at a ratio of 7:3 and C2 by saline alone. PS was instilled via a tracheal...

Forty nine premature rabbits of gestational age of 27 days were used as models of hyaline membrane disease (HMD) in our experiments. They were divided into 4 groups. Group A and B were treated by human amniotio fluid PS and the mixed PS (a mixture of human amniotio fluid PS plus dipalmitoyl phosphatidyl choline DPPO, and phosphatidylglyoerol PG) respectively. Groups C1 and C2 were served as controls, C1 being treated by DPPO and PG at a ratio of 7:3 and C2 by saline alone. PS was instilled via a tracheal cannula in a single dose of 100 mg (phosho-lipid)/kg. The amniotio PS and mixed PS could prolong the survival time of prema-ture rabbits greatly and improve the pressure-volume loops and pathologic changes of the lung significantly, but the efficacy of DPPC+PG and saline alone was not so obvious. Results indicated tha t the mixed PS was as efficacious as amniotic PS. The use of the mixed PS can reduce the need for human amniotio fluid. It may probably be valid for clinical use of PS treatment.

本实验用人羊水肺表面活性物质(PS)和混合PS治疗胎龄27d未成熟兔肺透明膜病(HMD),并作疗效比较。这两种PS都能明显延长未成熟兔的存活时间,改善肺压力-容量滞后环,减轻肺部病理变化,疗效均佳,而只用二棕榈酰卵磷脂(DPPC)加磷脂酰甘油(PG)和只用生理盐水治疗的两种对照组,疗效均差。

 
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