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    The application of intensive chemotherapy regimens in the treatment of children's B cell non Hodgkin's lymphoma:26 cases analysis
    强烈化疗方案在儿童B-细胞淋巴瘤中的治疗应用(附26例病例分析)
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    These percentages and 4G/4G genotype /non 4G/4G genotype in female subgroup do not significantly differ from the male subgroup, 4G allele frequency in female subgroup significantly differ from the male subgroup.
    ③女性肺心病患者PAI-1基因型分布为4G/4G型25.9%,4G/5G型55.6%,5G/5G型18.5%; 等位基因频率4G型为53.7%,5G型为46.3%;
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    We observed that new chromosome abnormalitises occured in part of our patients when transformed from one stage to another. 3 cases of RAEB—t were found to have t(9;11)(p~(21-22);q~(23-24))by using synchronization technique it was suggested that the chromosome rearrangement involving Ⅱq~(23-24)might be a non—random chromosome aberration in childhood myelodysplastic syndrome.
    亚型转化和转向白血病时有新的染色体异常出现,18例中在三例 RAEB—t 检出 t(9;11)(16%),提示 t(9;11)(p~(21-22);q~(23-24))或涉及11q~(23-24)的染色体重排在小儿 MDS 中存在一定的特异性。
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    in female patients with dipper and non dipper hypertension (115 0±4 7 g/m 2 vs.138 0±5 6 g/m 2, p <0 01).
    女性分别为138.0±5.6g/m2与115.0±4.7g/m2(P<0.01)。
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    There were significant differences of left atrial diastolic internal diameter (LADd) in male patients with dipper and non dipper hypertension (31 2±2 7 mm vs.35 8±2 9 mm, p <0 01);
    女性分别为32.4±2.5mm与29.4±1.8mm(P<0.05)。
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The method applies to the standard arithmetic subgroups ofSO(n,1) (a case which was proved previously by Millson [Mi]), to the non-arithmetic lattices inSO(n,1) constructed by Gromov and Piatetski-Shapiro [GPS] and to groups generated by reflections.
      
We also show how to distinguish examples of open subsets with a good quotient coming from Mumford's theory and give examples of open subsets with non-quasi-projective quotients.
      
An algebraicG-varietyX is called "wonderful", if the following conditions are satisfied:X is (connected) smooth and complete;X containsr irreducible smoothG-invariant divisors having a non void transversal intersection;G has 2r orbits inX.
      
Lichtenstein in the caseu =(n, ?) or(n?), we prove that ?(q).ζ1/2 is non zero for all harmonic polynomialsq ∈S() \ {0}.
      
In [Ka1] its geometric counterpart was applied to construct contractible smooth affine varieties non-isomorphic to Euclidean spaces.
      
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The clinical features of 415 attacks of rheumatic fever observed in 365 adult patients are described, and some of the diagnostic problems are discussed, 102 of the attacks were considered to be initial and 313 recurrent attacks. Pre-existing chronic valvular disease was diagnosed in 184 cases. The attack rate of rheumatic fever by season was found to be highest in late Winter and Spring, with the peak incidence in the months of March and April. A history of preceding sore throat was elicited in 41% of the rheumatic...

The clinical features of 415 attacks of rheumatic fever observed in 365 adult patients are described, and some of the diagnostic problems are discussed, 102 of the attacks were considered to be initial and 313 recurrent attacks. Pre-existing chronic valvular disease was diagnosed in 184 cases. The attack rate of rheumatic fever by season was found to be highest in late Winter and Spring, with the peak incidence in the months of March and April. A history of preceding sore throat was elicited in 41% of the rheumatic attacks, and inflammatory signs in the pharynx and tonsils were observed in 58.6% of the attacks. The antistreptolysin O titer was above 250 units in 80% of the cases examined. Among the clinical manifestations arthritis and carditis were most common, occurring in 55.7% and 49.9% respectively, while arthralgia without frank inflammatory signs was noted in 30% of the attacks. The incidence of arthritis was lower in the cases with chronic valvular defects than in cases without. In 29% of the cases ECG abnormalities constituted the sole clinical evidence of carditis. Since prolongation of P-R interval or higher grades of atrioventricular block are relatively common in rheumatic fever and other manifestations of rheumatic carditis like murmurs, cardiac enlargement, pericarditis and heart failure are equally non-speclfic, it is suggested that a-v block be included as evidence of carditis rather than considered as a minor manifestation if one adopts Jones' criteria for the diagnosis of rheumatic fever. In the presence of chronic valvular disease frank arthritis was uncommon and signs of carditis were often masked by those of valvular defects so that in 38.5% of cases the diagnosis of rheumatic activity had to depend upon a combination of clinical and laboratory signs generally concidered as minor rheumatic manifestations according to Jones' criteria.

1.本文报告成人急性风湿热366例,經住院观察的急性发作共415次,其中102次診断为初发,313次为复发。发作見于已有慢性心瓣缺损者計184例(218次发作),无心瓣缺损証据者182例(197次发作)。对发病季节、与溶血性鏈球菌感染的关系、临床表現、实验室发見、病程及病死率等作了分析。重点討論了本病的診断问題。 2.风湿热在春冬二季的发病率較高,41%的风湿发作前有咽痛,58.6%的风湿发作时咽部尚有炎症表現,仅24.7%的风湿发作前后均无咽炎的証据。約有80%发作中抗鏈球菌溶血素“O”滴定度在250单位以上。以上数字有力地說明了风湿热和鏈球菌感染之间的密切关系。 3.临床表現中以关节炎最为常見,发生率为55.7%,仅有关节酸痛的有30%。已有慢性心瓣缺损的病例中仅37.6%有关节炎,其发生率远低于无慢性心瓣缺损的病例(75.6%)。 4.心炎見于49.9%的发作中,其临床表現有时颇不明显。仅在心电图檢查中发見者有41例,占所有經过心电图檢查的心炎病例的29%。由此可見心电图检查对診断心炎的独特价值。 5.在曾作心电图檢查的250次发作中有82次(32.8%)发見房窒傳导阻滞,其中68次为P-R间期延长。作...

1.本文报告成人急性风湿热366例,經住院观察的急性发作共415次,其中102次診断为初发,313次为复发。发作見于已有慢性心瓣缺损者計184例(218次发作),无心瓣缺损証据者182例(197次发作)。对发病季节、与溶血性鏈球菌感染的关系、临床表現、实验室发見、病程及病死率等作了分析。重点討論了本病的診断问題。 2.风湿热在春冬二季的发病率較高,41%的风湿发作前有咽痛,58.6%的风湿发作时咽部尚有炎症表現,仅24.7%的风湿发作前后均无咽炎的証据。約有80%发作中抗鏈球菌溶血素“O”滴定度在250单位以上。以上数字有力地說明了风湿热和鏈球菌感染之间的密切关系。 3.临床表現中以关节炎最为常見,发生率为55.7%,仅有关节酸痛的有30%。已有慢性心瓣缺损的病例中仅37.6%有关节炎,其发生率远低于无慢性心瓣缺损的病例(75.6%)。 4.心炎見于49.9%的发作中,其临床表現有时颇不明显。仅在心电图檢查中发見者有41例,占所有經过心电图檢查的心炎病例的29%。由此可見心电图检查对診断心炎的独特价值。 5.在曾作心电图檢查的250次发作中有82次(32.8%)发見房窒傳导阻滞,其中68次为P-R间期延长。作者等认为,P-R间期延长或更高度的房室傳导阻滞,若发見于有鏈球菌感染之后的年青患者,应作为急性风湿性心炎的診断依据之一。 6.在已有慢性心瓣病的病例中,有38.5%得根据Jones氏診断标准中的几項次要表現而診断为风湿活动。 7.366例中58.5%有风湿热的复发,其中半数发生子初发以后的第一年內。住院期的当时病死率为6.5%。

Cardiac output was determined according to Fick's principle, while renaI blood flow (RBF) and glomerular filtration rate (GFR) were measured from the clearances of PAH and creatinine. One hour after an i.v. injection of guanethidine 5 mg/kg, normal dogs showed a very slight hypotension, which was relatively more noticeable in renal hypertensive dogs. The cardiac output, stroke volume, and ventricular work tended to decrease tn normotensive dogs, but tended to augment in hypertensive dogs. These alterations,...

Cardiac output was determined according to Fick's principle, while renaI blood flow (RBF) and glomerular filtration rate (GFR) were measured from the clearances of PAH and creatinine. One hour after an i.v. injection of guanethidine 5 mg/kg, normal dogs showed a very slight hypotension, which was relatively more noticeable in renal hypertensive dogs. The cardiac output, stroke volume, and ventricular work tended to decrease tn normotensive dogs, but tended to augment in hypertensive dogs. These alterations, however, were statistically insignificant. The total peripheral resistance was not much influenced by guanethidine in normal dogs, yet markedly diminished in hypertensive dogs. Diuresis was evident during the 1-hour postmedication period in both models. In hypertensive dogs, i.v. administrations of guanethidine caused a conspicuous rise of RBF and GFR, but a great reduction of the renal vascular resistance. In normal rats, following an i.p. injection of 20 mg/kg, the circulating blood volume (studied with I~(131)-labeled serum protein) exhibited a slight but non-significant increase within 3 hours, and returned to the original level at 24 hours. No significant change was found in DOCA-treated rats. Normal rats receiving guanethidine revealed an increase of blood volumes in all organs, especially spleen and kidney. These results demonstrate that the hypotensive action of guanethidine is not via the reduction of blood volume, but by way of a vasodilator action, which particularly amends the ischemic state of kidney and thus alleviates the pathologic process of renal hypertension.

本文用正常血压及腎型高血压狗研究胍乙啶对心和腎的血流动力作用,并用大白鼠試驗其对循环及器官血容量的影响。根据Fick氏原則測定心輸出量,同时从腎脏对于对氨基馬尿酸及肌酐的清除率計算腎血流量及腎小球滤过率。靜脉注射胍乙啶5毫克/公斤1小时后,正常狗血压略有降低,高血压狗降压則較为明显。胍乙啶对正常血压狗的心輸出量、心搏量及心室作功量有減 低的趋向,而对腎型高血压狗則略有增加,但均不显著。胍乙啶不影响正常狗的外周血管阻力,而能显著降低腎型高血压狗的总外周阻力,扩张其外周血管。注射胍乙啶后,正常血压及腎型高血压狗的尿量增加均非常显著。腎型高血压狗的腎小球滤过率及腎血流量都非常显著地低于正常狗,腎血管阻力則大大地高于正常狗,靜脉注射胍乙啶后,滤过率及血流量均明显增加,而血管阻力則大大降低。正常大白鼠腹腔注射胍乙啶20毫克/公斤后,3小时內循环血量略有增加,但不显著,6小时后开始恢复,2斗小时后回至原水平。对注射DOCA的大白鼠的循环血量則沒有明显影响。正常大白鼠注射胍乙啶后,各器官及組織內合血量普遍有增加趋势,其中以脾及腎的血容量增加最为明显,說明胍乙啶对这些部位的血管有扩张作用。上述資料証明:胍乙啶的降压作用并非通...

本文用正常血压及腎型高血压狗研究胍乙啶对心和腎的血流动力作用,并用大白鼠試驗其对循环及器官血容量的影响。根据Fick氏原則測定心輸出量,同时从腎脏对于对氨基馬尿酸及肌酐的清除率計算腎血流量及腎小球滤过率。靜脉注射胍乙啶5毫克/公斤1小时后,正常狗血压略有降低,高血压狗降压則較为明显。胍乙啶对正常血压狗的心輸出量、心搏量及心室作功量有減 低的趋向,而对腎型高血压狗則略有增加,但均不显著。胍乙啶不影响正常狗的外周血管阻力,而能显著降低腎型高血压狗的总外周阻力,扩张其外周血管。注射胍乙啶后,正常血压及腎型高血压狗的尿量增加均非常显著。腎型高血压狗的腎小球滤过率及腎血流量都非常显著地低于正常狗,腎血管阻力則大大地高于正常狗,靜脉注射胍乙啶后,滤过率及血流量均明显增加,而血管阻力則大大降低。正常大白鼠腹腔注射胍乙啶20毫克/公斤后,3小时內循环血量略有增加,但不显著,6小时后开始恢复,2斗小时后回至原水平。对注射DOCA的大白鼠的循环血量則沒有明显影响。正常大白鼠注射胍乙啶后,各器官及組織內合血量普遍有增加趋势,其中以脾及腎的血容量增加最为明显,說明胍乙啶对这些部位的血管有扩张作用。上述資料証明:胍乙啶的降压作用并非通过循环血量的減少,而是由于外周血管的扩张所致。胍乙啶能扩张腎血管,改善腎缺血状态,从而緩解腎型高血压的病理生理过程。

(1)In the chloralose-urethanized or curarized rabbits,infusion of saline or blood into the right auricle in amount of 8—10 ml/kg at the rate of 0.5—1.0 ml/sec induced bradycardia and hypotension. (2)Section of sino-aortic nerves and bilateral vagotomy did not alter the negative chronotropic effect due to auricular infusion,thus indicating that a local mechanism was responsible for the effect. (3)Distension of the right auricle by inflating an indwelling balloon with various pressures(20—70 mm Hg)also evoked...

(1)In the chloralose-urethanized or curarized rabbits,infusion of saline or blood into the right auricle in amount of 8—10 ml/kg at the rate of 0.5—1.0 ml/sec induced bradycardia and hypotension. (2)Section of sino-aortic nerves and bilateral vagotomy did not alter the negative chronotropic effect due to auricular infusion,thus indicating that a local mechanism was responsible for the effect. (3)Distension of the right auricle by inflating an indwelling balloon with various pressures(20—70 mm Hg)also evoked bradyeardia which persisted after cutting the vagi.Plotting the change in heart rate against pressure within the balloon,a non- linear relationship was revealed.The bradycardia observed could not be attributed to anoxemia resulting from reduced venous return,since the temporary occlusion of superior and inferior venae cavae failed to evoke bradycardia. (4)The blockage of sino-auricular node by local novocaine infiltration as well as by local or general hypothermia(26—28℃)abolished the negative chronotropio res- ponse to the atrial infusion.Therefore it was suggested that such a response occurred as a result of alteration in the functional state of sino-auricular node. (5)Under our experimental conditions,the cardiac acceleration reflex originally found by Bainbridge could not be demonstrated in the rabbits.

(一)在氯醛糖和氨基甲酸乙酯麻醉的兔或未麻醉的箭毒化兔,右心房内输液(输入量为8—10毫升/公斤,输入速度为0.5—1.0毫升/秒),可规律地引起心率减慢和动脉血压下降。(二)右心房输液引起的心率变化,在切断两对缓冲神经和迷走神经后并不消失,说明此种输液效应并非反射作用。(三)经颈外静脉向右心房内引入一小气囊,以不同压力扩张后,也可导致心率减慢。此时气囊内压值与心率变化之间的关系不是完全呈直线式的。切断迷走神经后,效应仍存在;且此效应也不是由于气囊扩张阻断血液回流所致。(四)以奴佛卡因溶液阻断窦房结区后,右心房输液不再引起心率减慢。全身低温或选择性心脏低温(26—28℃)后,大多数实验中的心率在输液后不再变慢,由此说明右心房内输液对心率的影响是刺激直接作用于窦房结而改变其机能活动的结果。(五)在我们的实验条件下,从兔身上不能证示有 Bainbridge 反射的存在。

 
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