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avirulence
相关语句
  无毒性
    The results show that avirulence matching Pm3d is controlled by three recessive avirulence genes.
    结果表明:对应Pm3d的无毒性由3对隐性基因控制;
短句来源
    The results show that avirulence matching Pm3b and Pm3f is controlled by one gene respectively.
    结果表明:对应的抗病基因Pm3b、Pm3f无毒性和毒性分离比为1:1,表明无毒性受1对基因控制;
短句来源
    Each of avirulences matching Pml3, Pm4b, Pml3 and Pm22 is controlled by two dominant avirulence genes. Avirulence matching Pm (4a) is controlled by three dominant genes.
    对应的抗病基因Pm13、Pm4b、Pm22的无毒性和毒性呈现3:1的分离比例,表明无毒性受2对显性基因控制,对应的Pm4a的无毒性和毒性呈15:1的分离比例,表明无毒性受3对显性基因控制。
短句来源
    tritici isolates CPW-1 and E17. The result shows that the avirulence matching resistance genes Pm3b and Pm3f is controlled by one gene.
    1:对小麦白粉菌CPW-1与E17杂交所得的95个后代的无毒性进行了遗传分析。 结果表明:小麦白粉菌杂交群体对应于抗病基因Pm3b.
短句来源
    The avirulence matching Pml3 is controlled by two avirulence genes. Each of avirulence matching Pm2, PmXBD, Pm4a, and Pm4b is controlled by three genes.
    Pm3f的无毒性/毒性分离比为1:1,表明无毒性受1对基因控制:对应Pm13的无毒性/毒性呈现3:1的分离比例,表明无毒性受2对基因控制:对应Pm2、PmXBD、Pm4a、Pm4b的无毒性分离比为15:1,表明无毒性受3对基因控制。
短句来源
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  无毒
    Primary Study of Avirulence Gene of Xanthomonas oryzae pv.oryzae
    水稻白叶枯病菌无毒基因的研究初报
短句来源
    Through the cDNA-AFLP analysis of 4 pools constructed by different phenotypes of 6 avirulence genes in Phytophthora infestans,total 85 candidate transcript derived fragments (TDFs) were obtained,which were 20 for Awl,28 for Avr2,16 for Avr3-Avr10-Avr11,21 for Avr4,respectively.
    通过对由具有6个无毒基因不同表现型的马铃薯晚疫病菌构建的4个混合池进行cDNA-AFLP分析,共获得85个与无毒基因相关的差异表达片段,其中与无毒基因Avr1、Avr2、Avr3-Avr10-Avr11、Avr4相关的差异表达片段分别为20, 28, 16和21个。
    The fact of A24T194vr4S125 with the same sequence of EST PH051G10.XT7 demonstrated that selecting germinating cyst for identification of avirulence genes of P. infestans is available,and it could settle the base for quickly obtaining full length and the function analysis of Avr4 gene in the future.
    未发现与A24T24Avr4S144同源的序列,说明此序列可能为一新的马铃薯晚疫病菌EST. A24T19Avr4S125与EST PH051G10.XT7的序列一致性表明选择正在萌发的孢囊时期鉴定无毒基因的可行性,也为进一步快速获得全长无毒基因Avr4及进行功能验证奠定了基础.
    Development of a SCAR marker linked to avirulence gene AVR-Pik~m in rice blast fungus Magnaporthe grisea
    一个与稻瘟病菌无毒基因AVR-Pik~m连锁的SCAR标记的分离
短句来源
    A novel SCAR marker closely linked to the avirulence gene AVR-Pik~m in rice blast fungus Magnaporthe grisea
    一个新的与稻瘟病菌无毒基因AVR-Pik~m紧密连锁的SCAR标记
短句来源
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  “avirulence”译为未确定词的双语例句
    The special band of TN1 population maybe link with avirulence, it was quality character confirmed by genetics experiment and tested by X2-test, followed 3:1 segregation ratio.
    TN1种群的特异片段,可能与弱致害连锁,经遗传学实验卡方验证,为质量性状基因,遵循3:1的分离比例。
短句来源
    30 of transgenic lines expressing antisense mRNA (1A) lines were inoculated blast race 131 (an avirulence race to transgnic donor Aichiasahi). Results showed there were 14 of 1A lines altered in rice blast resistance from R (resistance) to S (susceptible) in T_1 and T_2 generation, there were 7 of 1A lines altered only in T_1 generation and 1 of 1A line altered only in T_2 generation.
    1A和1H结构转基因水稻接种非亲和性稻瘟菌131小种,由抗病性转变为感病性:其中1A的30个转基因水稻株系中,有14个株系的T_1、T_2后代抗感病发生了变化,另外有7个株系只在T_1代发生了抗感病变化和1个株系只在T_2代发生了抗感病变化;
短句来源
    The presence of Lr26 on chromosome 1B and Lr3a on chromosome 6B was also postulated by use of 8 P. recondita cultures previously characterized for the different avirulence and virulence gene combination.
    位于1B染色体上的基因可能是Lr26,位于6B上的可能是Lr3a,二者可抵抗我国小麦叶锈菌群体中的部分生理小种(或毒性基因组合)。
短句来源
    tritici which had previously been characterized for avirulence/virulence gene combiniation and 30 known Lr genes were used for postulation . Seven Lr genes,named Lr1, Lr3a,Lr3ka,Lr16,Lr26,Lr30 and Lr32 were detected in 14 wheat cultivars,either alone or in combination.
    在供试的30个已知抗叶锈病基因中,推导出了7个抗性基因:Lr1、Lr3a、Lr3ka、Lr16、Lr26、Lr30和Lr32。
短句来源
    78.6% of their progenies produced perithecia, 22 progenies were MAT1 1, and 11 progenies were MAT1 2. Tests on the Progenies for avirulence/virulence on 14 rice cultivars( lines) showed that avirulence on Chunjing 17, Xiu 4 10, Yunjing 135 and Chunjiang 06 was controlled by one gene from 94 84c, respectively;
    致病性分离的结果则初步表明:94-84c菌株对楚粳17号、岫4-10、云粳135和春江06分别持有1个非致病性基因,对福锦、春江11、云粳34和腾糯2号则分别持有2个非致病性基因。
短句来源
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  avirulence
Agroinfiltration-mediated transient expression assays showed that CRP neither acts as an avirulence factor in N.
      
Expression of the avirulence genes recognized by two dominant resistance genes of Brassica,Rxc1 in plants with the BB genome, and Rxc3in theCC plants, was lost after bacterial mutation in planta.
      
Thus, it is the first description of theavrRxc1/3 avirulence gene conferring race-specific interaction betweenX.
      
One to four genes for avirulence toward various barley genotypes were found in the isolates under study.
      
It is suggested that dominant suppressor genes are involved in the genetic control of avirulence toward four barley genotypes.
      
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Since Flor's“gene for gene”theory was published,many pathologistsand breeders have proved that gene for gene relationships are operativein host-pathogen system of many crops.The genetic relationships of res-istance and susceptibility of many crops,for example,potatoes and flax,to pathogenic races are well understood.Each specific locus in the hostdetermining resistance and susceptibility corresponds to a specific locusdetermining virulence and avirulence in the pathogen.Therefore,eachhost locus governs...

Since Flor's“gene for gene”theory was published,many pathologistsand breeders have proved that gene for gene relationships are operativein host-pathogen system of many crops.The genetic relationships of res-istance and susceptibility of many crops,for example,potatoes and flax,to pathogenic races are well understood.Each specific locus in the hostdetermining resistance and susceptibility corresponds to a specific locusdetermining virulence and avirulence in the pathogen.Therefore,eachhost locus governs either a resistant or a susceptible reaction to the rel-ated locus in the pathogen,and each locus in the pathogen governs avirulent or avirulent reaction toward the related locus in the host.The varieties can be grouped according to number of differentialraces attacking them and pathogens can be grouped according to numberof differential hosts they attack.

本文根据 Flor 基因对基因学说,及清泽茂久运用这一学说进行稻瘟病抗性基因分析,阐述这个学说的基本原理及适用范围。许多作物品种对病原菌表现抗病反应,是因为抗病品种中存在着抗病基因,而病原菌中也存在着与抗病基因相对应的非致病性基因。病原菌中存在着对作物品种具致病性的不同生理小种,而作物品种中存在着相应的不同抗病基因。要把病原菌小种区别开来,需要一套鉴别品种;要鉴定品种的抗性基因,需要一套致病性稳定的鉴别菌系。每个品种只包含一个互不相同的抗病基因具有最强的鉴别能力。充分理解基因对基因学说及清泽在水稻抗性遗传研究方面的理论和方法,似将有助于国内抗病育种工作的进一步开展。

This paper reviews some current results and progress in pathogenicity genes of Xanthomonas oryzae pv.oryzae,introduces some molecular genetic approach and strategies for analysis of pathogenicity genes fo Xoo, and describes some characteristics and functions of virulence genes,avirulence genes and host induced genes.It also elucidates the regulatory relation of pathogenicity genes to phenotype of pathogenicity and pathogenical biochemical factors and discusses the research status and perspective of this...

This paper reviews some current results and progress in pathogenicity genes of Xanthomonas oryzae pv.oryzae,introduces some molecular genetic approach and strategies for analysis of pathogenicity genes fo Xoo, and describes some characteristics and functions of virulence genes,avirulence genes and host induced genes.It also elucidates the regulatory relation of pathogenicity genes to phenotype of pathogenicity and pathogenical biochemical factors and discusses the research status and perspective of this field.

笔者综述了分子植物病理学科组对水稻白叶枯病菌致病基因的研究结果和进展,介绍了病菌致病基因研究中分子遗传学分析方法,报道了病菌毒性基因、无毒基因和受寄主诱导基因的特征和功能,阐述了致病基因对致病性、致病生化因子可能的调控关系,讨论了该领域的研究现状和前景。

During 1990-1993, a set of 21 monosomics of Chinese Spring and 8 near-isogenic Thatcher lines of wheat resistance to leaf rust was used to locate the leaf rust resistance genes of Beijing 837, a new wheat cultivar developed by CAAS. As the results indicated, the resistance of the cultivar to Puccinia recondita f. sp. tritici race CLR I was controlled by two complementary dominant genes located on chromosome 1B and 6B, respectively. The presence of Lr26 on chromosome 1B and Lr3a on chromosome 6B was also postulated...

During 1990-1993, a set of 21 monosomics of Chinese Spring and 8 near-isogenic Thatcher lines of wheat resistance to leaf rust was used to locate the leaf rust resistance genes of Beijing 837, a new wheat cultivar developed by CAAS. As the results indicated, the resistance of the cultivar to Puccinia recondita f. sp. tritici race CLR I was controlled by two complementary dominant genes located on chromosome 1B and 6B, respectively. The presence of Lr26 on chromosome 1B and Lr3a on chromosome 6B was also postulated by use of 8 P. recondita cultures previously characterized for the different avirulence and virulence gene combination.

1990~1993年间,引用中国春全套单体系列和抗叶锈病小麦近等基因系(或单基因系)为材料,采用单体遗传分析和基因推导相结合的方法,对冬小麦品种北京837抗叶锈病基因进行染色体定位研究,明确其对生理小种叶中1号的抗性系由分别位于染色体1B和6B上的两个显性互补基因所控制。位于1B染色体上的基因可能是Lr26,位于6B上的可能是Lr3a,二者可抵抗我国小麦叶锈菌群体中的部分生理小种(或毒性基因组合)。

 
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