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fibrinogen
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  纤维蛋白原
    A Study on Quantitation of human plasma Fibrinogen by Radial Immunodiffusion Assay
    圆周免疫扩散法检测人血浆纤维蛋白原的研究
短句来源
    COMPARISON OF FOUR METHODS DETERMINING PLASMA FIBRINOGEN
    四种血浆纤维蛋白原测定方法比较
短句来源
    A Batroxobin assay for plasma fibrinogen
    巴西洞蝮蛇毒法测定血浆纤维蛋白原
短句来源
    Clinical Application of Plasma Fibrinogen Determination
    血浆纤维蛋白原测定在临床中的应用
短句来源
    Results Patients with high CRP had a lower dialysis dose(P<0.01),higher troponin T(P<0.01),and higher fibrinogen(P<0.01).
    结果升高组较正常组透析剂量低(P<0.01)。 纤维蛋白原和TnT明显高于正常组(P均<0.01)。
短句来源
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  血浆纤维蛋白原
    A Study on Quantitation of human plasma Fibrinogen by Radial Immunodiffusion Assay
    圆周免疫扩散法检测人血浆纤维蛋白原的研究
短句来源
    COMPARISON OF FOUR METHODS DETERMINING PLASMA FIBRINOGEN
    四种血浆纤维蛋白原测定方法比较
短句来源
    A Batroxobin assay for plasma fibrinogen
    巴西洞蝮蛇毒法测定血浆纤维蛋白原
短句来源
    Clinical Application of Plasma Fibrinogen Determination
    血浆纤维蛋白原测定在临床中的应用
短句来源
    Results Compared with normal controls,activities of FⅤ:C(P<0.01),FⅧ:C(P<0.0001)and FⅡ:C(P<0.05),levels of plasma fibrinogen and d-dimer(P<0.0001)were significantly higher in the VTE group.
    发色法底物检测蛋白C活性(PC∶A)和抗凝血酶活性(AT∶A)。 结果与对照组相比,静脉血栓栓塞组血浆纤维蛋白原(Fg)含量、D-二聚体含量、FⅡ∶C、FⅤ∶C和FⅧ∶C水平均明显增高,FⅨ∶C与对照组比较差异无统计学意义(P>0.05);
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  “fibrinogen”译为未确定词的双语例句
    Objective: To establish the quantitative analytical method to determine Fibrinogen Degradation Products(FDP)'s concentration in plasma, and evaluate the method's analytical performance and clinical application value.
    目的:建立定量检测血浆纤维蛋白(原)降解产物(FDP)含量的实验方法,评价该方法实验性能与临床应用价值。
短句来源
    Methods Of 95 VTE patients and 95 normal controls,we used quantitative latex agglutination test to measure for plasma levels of D-dimer and fibrinogen,one-stage clotting method for activities of factors Ⅱ(FⅡ:C),Ⅴ(FⅤ:C),Ⅷ(FⅧ:C)and Ⅸ(FⅨ:C),and chromogenic substrate assay for activities of PC and AT.
    方法95例静脉血栓栓塞患者和95名正常对照,用定量乳胶凝集试验行D-二聚体含量检测; 一期法检测血浆凝血因子Ⅱ活性(FⅡ∶C)、凝血因子Ⅴ活性(FⅤ∶C)、凝血因子Ⅷ活性(FⅧ∶C)和凝血因子Ⅸ活性(FⅨ∶C);
短句来源
    Comparison of CO_2 laser and fibrinogen tissue adhesive on cranial nerve repair
    CO_2激光与纤维蛋白粘合剂修复颅神经的比较
短句来源
    Relation of nitric oxide in human semen with the contents of β_2-microglobulin fibrinogen and C-reactive protein
    人精液中NO与β_2-MG、FIB及CRP含量的关系
短句来源
    Establishment and Evaluation of Quantitative Analytical Method of Fibrinogen Degradation Products in plasma
    纤维蛋白(原)降解产物定量检测方法的建立与评价
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  fibrinogen
The Mechanism of Cross-Linking of Fibrinogen and Its Early Structural Homolog-XFragment
      
We studied the mechanism of the cross-linking of fibrinogen, as well as its closest structural homolog Xfragment, under the influence of a fibronectin-stabilizing factor (factor XIIIa).
      
We propose that enzymatic cross-linking proceeds only with the gradual accumulation of structurally imperfect molecules of fibrinogen and fragment Xthat are prone to intermolecular D-Dend-to-end contacts.
      
The Influence of the End Products of Plasmin-Mediated Hydrolysis of Fibrinogen and Fibrin (EF and Ef Fragments) on Fibrinogen Cr
      
We studied the influence of the end products of plasmin-mediated hydrolysis of fibrinogen and nonstabilized fibrin (EF and Ef fragments) on covalent cross-linking of fibrinogen molecules catalyzed by a fibrin-stabilizing factor (factor XIIIa).
      
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The electrophoretio behaviour for human platelets and erythrocytes in their own plasma were compared and a straight line correlativity was found between both conditions.The slowing factors of cell electrophoresis in plasma were also studied. It was found that the increase in blood lipids, including beta-lipoprotein, glyceride and cholesterol and globulin and fibrinogen and the increase of plasma viscosity all contribute to the slowing down in plasma. We have also found that the electrophoretic times for...

The electrophoretio behaviour for human platelets and erythrocytes in their own plasma were compared and a straight line correlativity was found between both conditions.The slowing factors of cell electrophoresis in plasma were also studied. It was found that the increase in blood lipids, including beta-lipoprotein, glyceride and cholesterol and globulin and fibrinogen and the increase of plasma viscosity all contribute to the slowing down in plasma. We have also found that the electrophoretic times for erythrocytes and platelets suspended in plasma and in saline also showed straight line correlativity, showing that the structural changes on the cell surface might be another cause of changes in electrophoretic behaviour.

本文报道人血小板和红细胞在自身血浆中电泳时间互呈线性相关。本文还对血浆中某些红细胞电泳减缓因子进行了探讨,结果表明血浆中血脂(包括β脂蛋白、甘油三酯和胆固醇)、球蛋白和纤维蛋白原以及血浆粘度的增加是血浆中红细胞电泳减缓的原因之一。本文还发现红细胞和血小板分别在血浆中和生理盐水中的电泳时间也呈线性相关,这表明细胞膜表面结构的改变也是变化原因之一。

During the period from December 1979 to April 1981, serial observation of hemorheological indices was made in 31 inpatients with acute myooardial infarction. 18 blood samples were taken from each, of them at intervals during the first 2 months of their illness. For each sample, 8 hemorheologioal indices, including fibrinogen, relative viscosity o?whole blood and plasma, erythrocyte sedimentation rate and its K value, erythrooyte eleotrophoretic time, whole blood reduced viscosity and hematocrit, were determined....

During the period from December 1979 to April 1981, serial observation of hemorheological indices was made in 31 inpatients with acute myooardial infarction. 18 blood samples were taken from each, of them at intervals during the first 2 months of their illness. For each sample, 8 hemorheologioal indices, including fibrinogen, relative viscosity o?whole blood and plasma, erythrocyte sedimentation rate and its K value, erythrooyte eleotrophoretic time, whole blood reduced viscosity and hematocrit, were determined. The JB value which reflects the quantitative changes of the hemorheological indices was calculated from the data. In 18 survivals, their hemorheological indices were followed-up for 1.5 to 2 years after onset of the disease.The results showed that the increases in fibrinogen, relative viscosity of whole blood and plasma, whole blood reduced viscosity and erythrocyte electrophoretio time on the first day of the disease were highly significant as compared with normal (P<0.001), the increases in erythrocyte sedimentation rate and its K value were also significant(P<0.05). Their peak levels were reached during the first 2 weeks, then decreased gradually to the initial level by the second month. The fibrinogen, erythrocyte electrophoretic time and whole blood viscosity remained higher than normal, while erythrooyte sedimentation rate and its K value returned to normal after 1.5 to 2 years. While the packed cell volume showed an insignificant decrease throughout the period of this study. The JB value increassed markedly on the first day of infarction, and no significant decrease was observed until the third week. It remained higher than normal after 1.5 to 2 years.In contradiction with those reported in the literature, our results showed that the severity of the illness and patient's chance of survival could not be inferred from the hemorheological determinations at the time of admission. Factors which might be related with the discrepancy were discussed.

我们在31例急性心肌梗塞病人中通过定期检测血液流变学的各项指标,发现在梗塞后均呈现特征性的变化过程。于起病后第1天纤维蛋白原、血浆比粘度、红细胞电泳时间、全血还原粘度、全血比粘度、血沉及血沉方程K值等均高于正常值,于第1~2周升至高峰,此后渐降,至第2月回复起病初的水平,在1.5~2年时,血沉与血沉方程K值回复正常,但纤维蛋白原、红细胞电泳时间及全血粘度仍高于正常。

In this study,determinations of plasma viscosity(Y)and its 6 chemical factors --x1 albumin, x2 globulin, x3 fibrinogen, x4 cholesterol, x5 triglyceride and x6 beta-lipo-protein were carried out in 39 subjects. Linear regression equation aswas made. The contributions of these factors to viscosity were foundin order of x2→x3→x6→x4→x5→x1. Then the progressive regressions were done at different levels of F, the main factors being chosen and the minor ones d iscarded. On verification by the coefficient of multiple...

In this study,determinations of plasma viscosity(Y)and its 6 chemical factors --x1 albumin, x2 globulin, x3 fibrinogen, x4 cholesterol, x5 triglyceride and x6 beta-lipo-protein were carried out in 39 subjects. Linear regression equation aswas made. The contributions of these factors to viscosity were foundin order of x2→x3→x6→x4→x5→x1. Then the progressive regressions were done at different levels of F, the main factors being chosen and the minor ones d iscarded. On verification by the coefficient of multiple correlation, standard error of estimate, analysis of variance and its application, to practice, the equation was proved to be satisfactory, for it was found to coincide with actual condition as Y=0.59453+3.23404·10-4 x2+ 5.15788·10-4 x3 + 5.14465·10-4 x6. Theoretically, the above equation showed the intrinsic relation between viscosity and the main factors, and clinically it seemed to be of great help to the diagnosis and treatment of high plasma viscosity syndrome.

本文对39例对象的血浆粘度(Y)及其六个血浆成分——白蛋白(x_1)、球蛋白(x_2),纤维蛋白原(x_3)、胆固醇(x_4)、甘油三酯(x_5)和β脂蛋白(x_6)进行了测定,并对Y和x_1~x_6进行了六元回归,还对这六个因子产生血浆粘度的贡献作了排列,依次为x_2→x_3→x_6→x_4→x_5→x_1。继之以不同的F值的界限,进行了逐步回归,选取其主要因子和剔除其次要因子,从其复相关系数、标准估计误差、方差分析并在实际应用中加以检验,最后获得的三元方程式:Y=0.59453+3.23404·10~(-4)x_2+5.15788·10~(-4)x_3+5.14465.10~(-4)x_6是与实际相符合的较为理想的方程式。这一方程在理论上反映了与血浆粘度形成有关的主要化学成分,以及粘度与其主要化学成分间的定量关系;在临床上对于诊治高粘血浆症有一定的指导意义。

 
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