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fibrinogen
相关语句
  纤维蛋白原
    The -148C/T Polymorphism of β-Fibrinogen Gene and Plasma Fibrinogen Levels in Young Adults with Ischemic Stroke
    青年缺血性卒中患者纤维蛋白原β-148C/T基因多态性和血浆纤维蛋白原水平研究
短句来源
    Effect of Cigaiette Skoking on Plasma Fibrinogen Level: An Analysis of 444 Cases
    吸烟对血浆纤维蛋白原水平的影响(附444例分析)
短句来源
    The thrombin time (TT), prothrombin time (PT) and fibrinogen were detected before and after thrombolysis 6 h,3 d and 7 d.
    分别于溶栓前和溶栓后6h、3d、7d测定凝血酶(TT)、凝血酶原时间(PT)、纤维蛋白原(FIB)。
短句来源
    Methods Endothelin-1(ET-1),platelet α-granule membrane glycoproein(GMP)-140,11-dehydrothromboxane B_2(11-DH-TXB_2),fibrinogen(FIB),tissue-type plasminogen activator(t-PA) and plasminogen activator inhibitor-1(PAI-1) were measured in 100 hypertensive patients,100 hypertensive patients complicated with cerebral infarction and 100 normal controls.
    方法检测100例高血压病患者、100例高血压病合并急性脑梗死患者及100名健康对照者血浆内皮素-1(ET-1)、血小板α-颗粒膜蛋白(GMP-140)、11-去氢血栓烷B2(11-DH-TXB2)、纤维蛋白原(FIB)、组织型纤溶酶原激活剂(t-PA)及纤溶酶原激活剂抑制物-1(PAI-1)含量,并进行分析与评价。
短句来源
    Results: Polysaccharide sulfate might reduce TC,TG,LDL,blood viscosity,fibrinogen and thrombocyte aggregation rate before and after treatment(P<0.01 or P<0.05).
    结果:藻酸双脂钠可降低全血粘度、血浆粘度、血小板聚集率、纤维蛋白原及血清TC、TG、LDL,与治疗前比较有显著性改变(P<0.01或P<0.05);
短句来源
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  血浆纤维蛋白原
    The -148C/T Polymorphism of β-Fibrinogen Gene and Plasma Fibrinogen Levels in Young Adults with Ischemic Stroke
    青年缺血性卒中患者纤维蛋白原β-148C/T基因多态性和血浆纤维蛋白原水平研究
短句来源
    Effect of Cigaiette Skoking on Plasma Fibrinogen Level: An Analysis of 444 Cases
    吸烟对血浆纤维蛋白原水平的影响(附444例分析)
短句来源
    Correlativity of cerebral ischemia types with external thrombosis model to the fibrinogen molecular reactivity in plasma
    血浆纤维蛋白原分子活性与体外血栓模型和脑缺血类型的相关性分析
短句来源
    Effect of β-Fibrinogen-455 Gene Polymorphism on Plasma Fibrinogen Levels in Patients with Ischemic Stroke
    β-纤维蛋白原-455 G/A基因多态性对缺血性脑卒中患者血浆纤维蛋白原水平的影响
短句来源
    Changes of Plasma Fibrinogen Function in Acute Cerebra Infarction During Thrombolytic Therapy
    脑梗死溶栓治疗中的血浆纤维蛋白原功能的变化
短句来源
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  血纤维蛋白原
    Results:Compared with the control group,defibrasum decreased fibrinogen and hematocrit significantly (P<0 01).
    结果 :降纤酶治疗组与对照组比较 ,治疗后具有明显降低血纤维蛋白原、降低红细胞压积的作用 ,具有显著差异 (P <0 0 1)。
短句来源
    Fibrinogen increased significantly from H12h to H72h(P<0.05).
    血纤维蛋白原在H12h-72h显著升高。
短句来源
    The major controllable risk factors to recurrent cerebral infarction are hypertension, the increased value of blood fibrinogen, TC, plasma and whole blood viscosity and decreased HDL-C.
    复发性脑梗死的可控危险因素主要是高血压病,血纤维蛋白原、TC水平、血浆粘度和全血高切还原粘度升高及HDL-C降低;
短句来源
    We analyzed the correlation between middle cerebral artery blood velocity and both hematocrit and fibrinogen concentation in 51 healthy subjects in transcrantial doppler.
    采用经颅多普勒监测分析51名健康受试者大脑中动脉血流速度与空腹血纤维蛋白原及红细胞压积的关系。
短句来源
    All results suggested that focal cerebral infarction was treated early with langshuanmei that would be improve significantly drain electrical activity,Plasma fibrinogen and morphological changes
    结果提示:于脑梗塞后,早期用抗栓酶,对自发性脑电活动,血纤维蛋白原及形态学改变均有明显改善作用。
短句来源
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  “fibrinogen”译为未确定词的双语例句
    Fibrinogenβ-148 gene polymorphism associated with plasma fibrinogen levels in ischemic stroke
    Fgβ-148 C/T基因多态性对缺血性脑卒中患者血浆Fg水平的影响
短句来源
    Clinical significance of fibrinogen receptor expression on circulating activated platelets in patients with cerebral infarction
    脑梗死患者循环活化血小板FIB-R的检测及其临床意义
短句来源
    Clinical study of target fibrinogen level treated by Batroxobin in patients with acute cerebral infarction
    巴曲酶治疗急性脑梗死患者降纤靶水平的临床研究
短句来源
    The Association of PON2 C311S and Fibrinogen β-455G/A Genetic Polymorphism with Cerebral Infarction in Chinese
    PON2 C311S和Fgβ-455G/A基因多态性与脑梗死的相关性研究
短句来源
    Patients with BD had significantly elevated levels of cross-linked D-dimer and fibrinogen (P<0.01) compared with the controlled group.
    即组的D-dimer、Fbg水平升高,两组相比差异显著(-P均<0.01)。
短句来源
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  fibrinogen
The Mechanism of Cross-Linking of Fibrinogen and Its Early Structural Homolog-XFragment
      
We studied the mechanism of the cross-linking of fibrinogen, as well as its closest structural homolog Xfragment, under the influence of a fibronectin-stabilizing factor (factor XIIIa).
      
We propose that enzymatic cross-linking proceeds only with the gradual accumulation of structurally imperfect molecules of fibrinogen and fragment Xthat are prone to intermolecular D-Dend-to-end contacts.
      
The Influence of the End Products of Plasmin-Mediated Hydrolysis of Fibrinogen and Fibrin (EF and Ef Fragments) on Fibrinogen Cr
      
We studied the influence of the end products of plasmin-mediated hydrolysis of fibrinogen and nonstabilized fibrin (EF and Ef fragments) on covalent cross-linking of fibrinogen molecules catalyzed by a fibrin-stabilizing factor (factor XIIIa).
      
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In this report it's shown that this defibrase had thrombin-like effects but didn't actuate factor ⅫⅠ. Therefore, the formed clot is unstable, being easily dissolved without causing thrombosis.After treatment all the indices of coagulation were remarkably, prolonged. The defibrase also activated plasminogen to form plasmin, accelerating fibcinolysis, and increase of fibrinogen/fibrin degradation products also possess anticoagulationaction.Defibrase had no apparent effects on platelets and most of the plasma...

In this report it's shown that this defibrase had thrombin-like effects but didn't actuate factor ⅫⅠ. Therefore, the formed clot is unstable, being easily dissolved without causing thrombosis.After treatment all the indices of coagulation were remarkably, prolonged. The defibrase also activated plasminogen to form plasmin, accelerating fibcinolysis, and increase of fibrinogen/fibrin degradation products also possess anticoagulationaction.Defibrase had no apparent effects on platelets and most of the plasma coagulation factors, so it was effective, convenient and relatively safe to use clinically.

尖吻蝮蛇毒去纤酶有类凝血酶的作用,但不激活血浆ⅩⅢ因子,故形成的纤维蛋白不稳固,很易被溶解清除,不致造成栓塞。治疗30例缺血性脑血管病患者后,凝血指标明显延长。去纤酶还能激活胞浆素原变为胞浆素促使纤溶亢进。纤维蛋白原/纤维蛋白的降解产物增加又有抗凝血作用。去纤酶对血小板数及大多数凝血因子无明显影响。去纤酶具有疗效好,治疗方法简便,比较安全等优点。

Objective of this study was to find the rish factors in ischemic stroke. Hemorrheological study of 90 cases of acute ischemic stroke, including 46 cases of cerebral thrombosis diagnosed with CT, 44 cases of transient ischemic attack(some of them were confirmed by CT)and 80 normal subjects was reported. On the other hand, we have studied 50 patienta with ischemie attack for in vitro their platelet aggregation. In this article, formula of Grotta's CBF and YSS was used to find the relationship beween the whole...

Objective of this study was to find the rish factors in ischemic stroke. Hemorrheological study of 90 cases of acute ischemic stroke, including 46 cases of cerebral thrombosis diagnosed with CT, 44 cases of transient ischemic attack(some of them were confirmed by CT)and 80 normal subjects was reported. On the other hand, we have studied 50 patienta with ischemie attack for in vitro their platelet aggregation. In this article, formula of Grotta's CBF and YSS was used to find the relationship beween the whole blood viscosity parameters and cerebral blood flow, It was found that hemorrheological parameter in patients with ischemic stroke were higher than out of the normal groups respectively, excluding plasmic osmotic pressure (P>0.01); and the whole blood viscocity of cerebral thrombosis was higher than that of TIA. Significant negative correlation was present between CBF and hematocrit (P<0.01), wholc blood vascosity (P<0.01), reversal whole blood viscosity (P<0.01), plasmic viscosity (P<0.01), concentration of fibrinogen (P<0.01) and platelet aggregation (P<0.05). But when fibrinogen and hematocrit were considered together in the form of YSS, their correlation with CBF was much higher than that when these variables were considered individually. However, difference of platelet aggregation was not found between cerebral thrombosis and TIA. It suggested that the increased hemorrheological parameter might decrease CBF and had an important effect on ischemic stroke; YSS and CBF were valuable and simple index in medical practice, especially in basic level medical units; increased platelet aggregation might be a cause of ischemic stroke rather than its result.

对90例缺血性卒中患者做了多项血液流变学及血小板聚集性实验室研究,发现与对照组有明显的统计学差异,认为血液流变学的变化是导致脑血栓的重要因素,并认为血小板功能亢进应视为发病原因,而非结果。本文采用公式推算法认为红细胞压积及纤维蛋白元两个指标,有利于推算脑血流及血液的变应力,故这种推算有利于中风预报。

The effect of vinpocetine on 30 hypertensive patients with cerebral arteriosclerosis and cerebral thrombosis was studied.The drug was administered in dosage of 5 mg tid for 4 wesks.The subjective symptoms were improved in 93% and markedly improved in 25% of patients; psychic symptoms were improved in 84% and markedly improved in 11% of patients; neurologic symptoms were improved in 96% and markedly improved in 22%.The blood viscosity were improved,lowering down of plasma platelet-aggregative ratio and fibrinogen...

The effect of vinpocetine on 30 hypertensive patients with cerebral arteriosclerosis and cerebral thrombosis was studied.The drug was administered in dosage of 5 mg tid for 4 wesks.The subjective symptoms were improved in 93% and markedly improved in 25% of patients; psychic symptoms were improved in 84% and markedly improved in 11% of patients; neurologic symptoms were improved in 96% and markedly improved in 22%.The blood viscosity were improved,lowering down of plasma platelet-aggregative ratio and fibrinogen level. Vinpocetine properly increased the blood supply in the cerebral circulation, improved the cerebral ischemic condition and protected the brain tissues.The adverse effects were seldom.

本文报道长春西汀5mg,tid,治疗高血压伴脑动脉硬化和脑血栓形成后遗症患者3O例,4wk为一疗程。自觉症状总有效率为93%,显效率为25%;精神症状总有效率为84%,显效为11%;神经症状总有效率为96%,显效为22%。血液粘度明显降低,血小板聚集率及纤维蛋白原下降。并能选择性增加脑缺血部位的血供,改善脑循环和代谢,保护缺血脑组织。无明显不良反应。

 
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