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fibrinogen
相关语句
  纤维蛋白原
    The thrombin time (TT), prothrombin time (PT) and fibrinogen were detected before and after thrombolysis 6 h,3 d and 7 d.
    分别于溶栓前和溶栓后6h、3d、7d测定凝血酶(TT)、凝血酶原时间(PT)、纤维蛋白原(FIB)。
短句来源
    Methods Endothelin-1(ET-1),platelet α-granule membrane glycoproein(GMP)-140,11-dehydrothromboxane B_2(11-DH-TXB_2),fibrinogen(FIB),tissue-type plasminogen activator(t-PA) and plasminogen activator inhibitor-1(PAI-1) were measured in 100 hypertensive patients,100 hypertensive patients complicated with cerebral infarction and 100 normal controls.
    方法检测100例高血压病患者、100例高血压病合并急性脑梗死患者及100名健康对照者血浆内皮素-1(ET-1)、血小板α-颗粒膜蛋白(GMP-140)、11-去氢血栓烷B2(11-DH-TXB2)、纤维蛋白原(FIB)、组织型纤溶酶原激活剂(t-PA)及纤溶酶原激活剂抑制物-1(PAI-1)含量,并进行分析与评价。
短句来源
    AIM To explore the relation between the severity of coronary artery disease,prognosis and fibrinogen in acute myocardial infarction(AMI).
    目的探讨纤维蛋白原(FIB)含量与急性心肌梗死(AMI)冠脉病变程度及预后的关系。
短句来源
    Results: Polysaccharide sulfate might reduce TC,TG,LDL,blood viscosity,fibrinogen and thrombocyte aggregation rate before and after treatment(P<0.01 or P<0.05).
    结果:藻酸双脂钠可降低全血粘度、血浆粘度、血小板聚集率、纤维蛋白原及血清TC、TG、LDL,与治疗前比较有显著性改变(P<0.01或P<0.05);
短句来源
    The changes in blood platelet (PLT) count, activated partial thromboplastin time (APTT), prothrombin time (PT). D-dimer (DD), AT-Ⅲactivity, fibrinogen (FI), alanine aminotransferase (ALT), aspartate aminotransferase (AST), alkaline phosphatase (ALP) and lactate dehydrogenase (LDH) were determined 3 hours later.
    3 h后取血并处死大鼠,观察各组血小板计数(PLT)、活化部分凝血活酶时间(APTT)、凝血酶原时间(PT)、D-二聚体、AT-Ⅲ活性、纤维蛋白原(FI)、丙氨酸转氨酶(ALT)、天冬氨酸转氨酶(AST)、碱性磷酸酶(ALP)和乳酸脱氢酶(LDH)的水平,取肺、肾、肝组织观察其病理学变化。
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  血浆纤维蛋白原
    The dynamic changes of plasma fibrinogen, antithrombin Ⅲ,D-dimer in patients with acute cerebral in farction
    急性脑梗死患者血浆纤维蛋白原、抗凝血酶Ⅲ及D-二聚体的研究
短句来源
    The relationship among helicobacter pylori infection、virulence of Hp and acute myocardial infarction,fibrinogen,thromboxane B_2
    幽门螺杆菌感染及其毒力与急性心肌梗死、血浆纤维蛋白原及血栓素B_2的关系
短句来源
    β-fibrinogen gene -455A/G polymorphism and plasma fibrinogen level in Chinese stroke patients
    急性脑梗死患者β纤维蛋白原-455G/A基因多态性和血浆纤维蛋白原水平研究(英文)
短句来源
    Study of β-fibrinogen gene -148C/T polymorphism and plasma fibrinogen levels in young patients with acute cerebral infarction
    青年急性脑梗死患者β-纤维蛋白原-148C/T基因多态性和血浆纤维蛋白原水平的研究
短句来源
    Study on blood fibrinogen and lipoprotein(a) in the elderly with non-acute phase cerebral infarction
    老年脑梗死非急性期血清脂蛋白(a)和血浆纤维蛋白原含量的研究
短句来源
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  血纤维蛋白原
    Objective To eveluate the efficacy of Defibrase in treating Acute Cerebral Infarction (ACI) and the variation of patients’plasma fibrinogen (FIB) Methods Thirty patients following ACl were given intravenous Defibrase,while another fifteen patients were given Low molecular dextran and Salvia miltiorrhiza Then we compare experimental test result,clinical efficiencfy and the side effects between the two groups.
    目的 评估蛇毒降纤酶治疗急性脑梗死的临床疗效 ,以及患者血纤维蛋白原 ( FIB)的变化 方法 对 30例急性脑梗死患者静脉注射降纤酶 ; 同时对 1 5例患者常规使用低分子右旋糖酐和丹参 ,对 2组临床疗效、 FIB改变、不良反应进行对照讨论。
短句来源
    Results: The neurological function of the patients treated with defibrase was significantly improved, the scores of daily living abilities increased, and the level of fibrinogen in blood decreased.
    结果 :去纤酶组显著改善神经功能缺失程度 ,提高日常生活能力 ,降低血纤维蛋白原水平 ;
短句来源
    Conclusion: Treating with Defirbin could rise the effective rate and decrease the blood viscosity, plasma fibrinogen concentration in acute cerebral infarction. The safety was high also.
    结论:急性脑梗塞使用巴曲酶治疗能提高好转率,降低患者血粘度和血纤维蛋白原浓度,安全性高。
短句来源
    The serum fibrinogen level was decreased significantly in the treatment group.
    治疗组血纤维蛋白原水平降低显著 ,不良反应轻微。
短句来源
    C group 60%,effect is 33.3%. The patients in A and B groul)'whose fibrinogen in blood is lowerblo viscoicy has been improved.
    A组、 B组治疗后血纤维蛋白原明显降低 ,血粘度得到改善 ,且 A组优于 B组。
短句来源
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  “fibrinogen”译为未确定词的双语例句
    METHODS Ninety patients with AMI were divided into two groups(group A: fibrinogen >4 g/L,group B: fibrinogen≤4 g/L) based on fibrinogen.
    方法根据AMI初期FIB含量将90例AMI分为两组,A组FIB值>4 g/L,B组FIB值≤4 g/L。
短句来源
    Result Partial correlation analysis showed that plasma fibrinogen was positively correlated with C reactive protein. (r =0.592, P<0.01).
    结果偏相关分析显示,急性心肌梗死患者FIB与CRP明显相关,相关系数r=0.592,P<0.01。
短句来源
    Clinical study of target fibrinogen level treated by Batroxobin in patients with acute cerebral infarction
    巴曲酶治疗急性脑梗死患者降纤靶水平的临床研究
短句来源
    Objective:to explore the levels of Leptin,interleukin-6(IL-6) in serum of acute cerebral infarction patient and the association of Leptin level with IL-6,blood pressure, blood fat and fibrinogen,and to investigate the role of the serum Leptin,IL-6 in patients with cerebral infarction(CI).
    目的:研究脑梗死急性期患者血清瘦素、IL-6(白细胞介素-6)水平以及两者之间的相关性,评价瘦素、IL-6 在脑梗死发病中的作用,为阐明脑梗死的发病机理以及有效防治提供理论依据。
短句来源
    Objective:To investigate the changes in plasma interleukin-6(IL-6)and fibrinogen(Fib)and the relationship between them in patients with AMI,26 patients with AMI were studied.
    目的:探讨AMI后血中IL-6.Fib浓度变化及二者之间的关系。
短句来源
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  fibrinogen
The Mechanism of Cross-Linking of Fibrinogen and Its Early Structural Homolog-XFragment
      
We studied the mechanism of the cross-linking of fibrinogen, as well as its closest structural homolog Xfragment, under the influence of a fibronectin-stabilizing factor (factor XIIIa).
      
We propose that enzymatic cross-linking proceeds only with the gradual accumulation of structurally imperfect molecules of fibrinogen and fragment Xthat are prone to intermolecular D-Dend-to-end contacts.
      
The Influence of the End Products of Plasmin-Mediated Hydrolysis of Fibrinogen and Fibrin (EF and Ef Fragments) on Fibrinogen Cr
      
We studied the influence of the end products of plasmin-mediated hydrolysis of fibrinogen and nonstabilized fibrin (EF and Ef fragments) on covalent cross-linking of fibrinogen molecules catalyzed by a fibrin-stabilizing factor (factor XIIIa).
      
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The dynamic changes of serum LP (a) Levels and the effects of thrombolysis were studied in 24 cases of acute myocardial infarction , including 15 treated by intravenous infusion of urokinase or difibrase and 9 with conventional therapy as control. It showed that LP (a) levels increased transiently, peaked during 7-14th day after onset and decreased toward baseline around 28th day in all cases. Comparing with baseline levels, the LP (a) levels increased by 74. 1 % on the 7th day in thrombolysis group and by 108%...

The dynamic changes of serum LP (a) Levels and the effects of thrombolysis were studied in 24 cases of acute myocardial infarction , including 15 treated by intravenous infusion of urokinase or difibrase and 9 with conventional therapy as control. It showed that LP (a) levels increased transiently, peaked during 7-14th day after onset and decreased toward baseline around 28th day in all cases. Comparing with baseline levels, the LP (a) levels increased by 74. 1 % on the 7th day in thrombolysis group and by 108% on the 14th day in controls, it decreased to baseline in thrombolysis group at 28th day, but not in controls. LP (a)j level was significantly higher in patients with unsuccessful thrombolysis (499. 5± 271. 0 mg/L n= 8 , M±S) than those with successful thrombolysis (259. 4 ± 142. 9 , n = 7), and also higher in dead cases (595. 8±284. 1, n= 6) than in the survivals (322. 8± 200. 0 ,n = 18). There was significant positive correlation between the size of infarct and LP (a) level (r= 0. 74, p<0. 001). It indicates that serum LP (a) level may be related to thrombolytic effectiveness or acute phase prognosis to some extent. In addition, the activities of t-PA and fibrinogenolysis declined, and that of PAI increased during early periods of infarction. After intravenous infusion of thrombolytic agents (urokinase or difibrase), the activities of t-PA and fibrinogenolysis increased, while the PAI activity and fibrinogen level decreased significantly in difibrase group from the lst to 3rd day after onset. Correlations between LP (a), lipids and fibrinolysis parameters, were found.

观察24例急性心肌梗塞(AMI)患者的血清脂蛋白(a)[Lp(a)]动态变化及其与静脉法溶栓疗效关系。结果表明,AMI患者的Lp(a)呈一过性升高,7~14天达峰值,溶栓组比对照组峰值较快降至基线水平。Lp(a)浓度在溶栓不成功、住院期死亡或梗塞范围大的患者中增高更为明显,提示Lp(a)水平变化与AMI溶栓疗效及急性期预后间可能有一定的联系。另外,还发现AMI发病后早期组织型纤溶酶原激活物(t-PA)活性及纤溶蛋白原溶解活性下降、纤溶酶原激活物抑制物(PAI)活性升高;静脉滴注尿激酶或去纤酶后均可使t-PA和纤溶活性升高,而且用药后第1~3天还使PAI活性及纤维蛋白原含量明显降低,Lp(a)、血脂及纤溶指标相互之间相关性不甚明显。

o evaluate the thrombolytic effect of Snake Venom Antithrombus Enzyme (SVATE) in the treat-ment of early acute myocardial infarction (AMI),2 cases with AMI were randomly allocated to three groups,control( 22 cases) ,SVATE (15 cases) ,and Urokinase(15 cases). The results show that SVATE can inhibit platelet aggregation,activate slightly fibrinolytic system and decrease markedly plasma fibrinogen level.However,the thrombolytic effect of SVATE in early treatment of AMI is not ideal,it can be used in combination...

o evaluate the thrombolytic effect of Snake Venom Antithrombus Enzyme (SVATE) in the treat-ment of early acute myocardial infarction (AMI),2 cases with AMI were randomly allocated to three groups,control( 22 cases) ,SVATE (15 cases) ,and Urokinase(15 cases). The results show that SVATE can inhibit platelet aggregation,activate slightly fibrinolytic system and decrease markedly plasma fibrinogen level.However,the thrombolytic effect of SVATE in early treatment of AMI is not ideal,it can be used in combination with effective thrombolytic drugs to prevent reocclusion and reinfarction in AMI.

对52例急性心肌梗塞(AMI)患者应用蝮蛇抗栓酶(SVATE)及尿激酶(UK)静脉溶栓治疗进行比较研究,结果表明SVATE具有抑制血小板聚集、微弱的激活纤溶系统及明显的降低纤维蛋白原作用,而对AMI早期溶栓效果不理想。如能与有效的溶栓药物合并应用,可能防止再次堵塞或再次梗塞的发生,确切疗效有待进一步研究。

Abstract Plasma urokinase(UK)antigen,tissue plasminogen activator (t-PA) antigen, plasminogen activator(PA)activity, plasminogen activator inhibiter (PAI) activity, plasmin activity and fibrinogen antigen Were measured before and after thrombolytic treatment with intravenous UK in 44 patients suffering from their first attack of acute myocardial infarction(AMI). The results showed that:(1)The action of intravenous UK lasted for only a short period of time,as 53.2% and 93.7% of UK antigen were cleared...

Abstract Plasma urokinase(UK)antigen,tissue plasminogen activator (t-PA) antigen, plasminogen activator(PA)activity, plasminogen activator inhibiter (PAI) activity, plasmin activity and fibrinogen antigen Were measured before and after thrombolytic treatment with intravenous UK in 44 patients suffering from their first attack of acute myocardial infarction(AMI). The results showed that:(1)The action of intravenous UK lasted for only a short period of time,as 53.2% and 93.7% of UK antigen were cleared from the plasma 1 hour and 5 hours respectively after the completion of UK treatment.(2)Compared to pretreatment values,PA and plasmin activities increased; PAI activity decreased during treatment, indicating enhanced fibrinolytic activity following UK therapy. However,immediately after treatment PAI activity showed again an significant increase (PAI rebound phenomenon), suggesting the existence of an abnormal procoagulant state in the early stage of thrombolytic treatment.(3) The changes in PAI activity were correlated possibly with the degree of damage of the left ventricular function and the severity of AMI. it seems important that adjunctive anticoagulant therapy should be given in the early stage of thrombolytic therapy.

观察44例首次急性心肌梗塞(AMI)患者的尿激酶(UK)静脉溶栓治疗前后血浆UK抗原、组织纤溶酶原激活物(t-PA)抗原、纤溶酶原激活物(PA)活性、纤溶酶原激活物抑制物(PAI)活性、纤溶酶活性和纤维蛋白原抗原的变化。结果显示:(1)UK静脉注射的作用持续时间较短,注射后1小时和5小时,分别有53.2%和93.7%的UK抗原从血浆中清除。(2)与治疗前相比,UK静注后PA和纤溶酶活性升高、PAI活性降低,纠治了AMI时纤溶系统的功能紊乱状况。但是,停止UK注射后即出现PAI“反弹”现象,PA和纤溶酶活性随之降低,说明溶栓早期存在反常的高凝状态。(3)PAI活性升高程度与左心室功能受损及AMI病情程度有一定关系。以上各点均提示,溶栓早期辅助的抗凝治疗尤为重要。

 
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