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arsenic toxin
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  砷中毒
     Study on the expression of Rb and p53 protein in lesions of patients with coal arsenic toxin
     Rb和p53蛋白在燃煤型砷中毒患者皮损中表达的研究
短句来源
     Study on frequency of variant erythrocytes at the glycophorin-A locus in patients with arsenic toxin
     砷中毒患者GPA突变频率研究
短句来源
     Objective To study the expression of p16? Rb and p53 protein in lesions of patients with coal arsenic toxin(CAT).
     目的 研究Rb、p5 3蛋白在燃煤型砷中毒患者皮损中的表达及其意义。
短句来源
     Objective Applying Glycophorin A (GPA) mutation assay in normal adults and patients with coal arsenic toxin , we may observe the frequency of variant erythrocytes at the glycophorin A locus and also the relation between the change of it and stage of disease.
     目的 检测燃煤性砷中毒患者外周血红细胞GPA的突变频率及其与病程的关系。 方法 分离、固定4 0例燃煤性砷中毒患者外周血红细胞 ,与荧光素标记的单抗结合后 ,采用流式细胞术进行GPA变异分析 ,计算GPA变异频率。
短句来源
  相似匹配句对
     Phytopathogenic Toxin
     植物病原毒素
短句来源
     Cholera toxin
     霍乱毒素
短句来源
     Study on the expression of Rb and p53 protein in lesions of patients with coal arsenic toxin
     Rb和p53蛋白在燃煤型砷中毒患者皮损中表达的研究
短句来源
     Study on frequency of variant erythrocytes at the glycophorin-A locus in patients with arsenic toxin
     砷中毒患者GPA突变频率研究
短句来源
     Arsenic in Coal of China
     中国煤中的砷
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Objective To study the expression of p16?Rb and p53 protein in lesions of patients with coal arsenic toxin(CAT).Methods Immunohistochemistry mothod was used to assess p16?Rb and p53 proteins expression in 46 cases of CAT.Results The expression rate of Rb protein was 60.9%.It did not correlate with the pathological grades(P>0.05), and was associated with clinical grates(P<0.05).The expression rate of p53 protein was 67.4% It was signficantly correlated with pathological grades and clinical types.(P<0.05).Conclusions...

Objective To study the expression of p16?Rb and p53 protein in lesions of patients with coal arsenic toxin(CAT).Methods Immunohistochemistry mothod was used to assess p16?Rb and p53 proteins expression in 46 cases of CAT.Results The expression rate of Rb protein was 60.9%.It did not correlate with the pathological grades(P>0.05), and was associated with clinical grates(P<0.05).The expression rate of p53 protein was 67.4% It was signficantly correlated with pathological grades and clinical types.(P<0.05).Conclusions The pathogenesis of CAT was influenced by mutifactors, p53 protein maybe involved in development of CAT.

目的 研究Rb、p5 3蛋白在燃煤型砷中毒患者皮损中的表达及其意义。 方法 采用免疫组化SP法检测 46例燃煤型砷中毒患者皮损中Rb、p5 3蛋白的表达。 结果Rb蛋白的阳性表达率为 6 0 .9% ,与病理分型无明显的相关性 (P >0 .0 5 )但与临床分级相关 (P <0 .0 5 )。p5 3蛋白的阳性表达率分别为 6 7.4% ,与临床分级和病理分型明显相关 (P <0 .0 5 )。结论 p5 3蛋白可能参与了其发生及发展。

Objective Applying Glycophorin A (GPA) mutation assay in normal adults and patients with coal arsenic toxin , we may observe the frequency of variant erythrocytes at the glycophorin A locus and also the relation between the change of it and stage of disease. Methods Combination of immunolabeling and flow cytometry (FCM),18 normal adults and 40 patients were examined. Results GPA NN/NO/MM/MO variant frequencies(Vfs) of CAT patients were(21.23±13.97)×10 -6 ,(33.13±25.72)×10 -6...

Objective Applying Glycophorin A (GPA) mutation assay in normal adults and patients with coal arsenic toxin , we may observe the frequency of variant erythrocytes at the glycophorin A locus and also the relation between the change of it and stage of disease. Methods Combination of immunolabeling and flow cytometry (FCM),18 normal adults and 40 patients were examined. Results GPA NN/NO/MM/MO variant frequencies(Vfs) of CAT patients were(21.23±13.97)×10 -6 ,(33.13±25.72)×10 -6 ,(110.90±63.58)×10 -6 ,(20.35±21.26)×10 -6 respectively ,significantly higher than that of normal subjects. No difference of variant frequendies was found in the patients with different courses(P<0.01). Conclusions The contact of arsenic may induce the mutation of erythrocytes at the glycophorin A locus ,suggesting that arsenic may be a kind of potential mutagen.[

目的 检测燃煤性砷中毒患者外周血红细胞GPA的突变频率及其与病程的关系。方法 分离、固定4 0例燃煤性砷中毒患者外周血红细胞 ,与荧光素标记的单抗结合后 ,采用流式细胞术进行GPA变异分析 ,计算GPA变异频率。结果  4 0例燃煤性砷中毒患者外周血GPANN变异频率为 (2 1.2 3± 13.97)× 10 -6,NO变异频率为 (33.13± 2 5 .72 )× 10 -6,MM变异频率为 (110 .90± 6 3.5 8)× 10 -6,MO变异频率为 (2 0 .35± 2 1.2 6 )× 10 -6,GPA变异频率明显高于正常人群 (P <0 .0 1) ,但不同病程的燃煤性砷中毒患者之间的GPA变异频率未见明显差异。结论 砷中毒可导致人类外周血红细胞GPA的突变。提示GPA的突变频率可作为一项检测有否砷中毒的指标

 
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