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   b type hepatitis 的翻译结果: 查询用时:0.009秒
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b type hepatitis
相关语句
  乙型肝炎
     How to cure chronic B type hepatitis is a whole world scholar a problem for paying attention to.
     如何治疗慢性乙型肝炎是全世界学者所关注的问题。
短句来源
     Conclusion:Ara Amp combined traditional Chinese medicine pharmaceutics Yigansan can obviously improve the liver function of patients with chronic B type hepatitis and promote their conversion of HbeAg from positive to negative.
     结论 :中药制剂乙肝散联合Ara Amp治疗慢性乙型肝炎 ,能明显改善肝功能 ,并提高HBeAg阴转率。
短句来源
     Methods: Acute jaundice B type hepatitis 20 cases, hepatic cirrhosis 10 cases, primary hep-atoma 15 cases, and control group 30 cases. The PT with one step method ,total prothrombin and after sbsorb prothrombin were deteminted with Ecarin-Chromogenic peptide substrate method.
     方法:急性黄疸型乙型肝炎20例,肝硬化10例,原发性肝癌15例,对照组30例,用Ecarin-发色底物法检测其血浆中全凝血酶原量与吸附后凝血酶原量,PT用一期法测定。
短句来源
     OBJECTIVE To inquire the influence of the capacity of B type hepatitis virus DNA (HBV DNA) carried by pregnant women on the result of passive immunity by injecting hepatitis B immunoglobulin (HBIG) for preventing intrauterine infection.
     目的 探讨孕妇携带乙型肝炎病毒DNA(HBVDNA)的载量 ,对注射乙型肝炎免疫球蛋白 (HBIG)预防宫内传播效果的影响。
短句来源
     We collected plasma, blood cells, sera, urine and tissues from patients of HCC (and their healthy control), B type hepatitis and liver cirrhosis.
     在此基础上,我们对HCC及其健康对照、乙型肝炎、肝硬化病人的各类临床样本进行了收集,包括血浆、血细胞、血清、尿液和组织。
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  “b type hepatitis”译为未确定词的双语例句
     The HBV DNA positive rate of B type hepatitis patients was 41%,the average HBV DNA concentration was 3.8×10 5copies/ml. The BCP gene mutation rate was 8%. The BCP gene mutation rate of hepatoma remarkably higher than of B type hepatitis patients.
     1 0 0例非肝癌乙肝患者HBV DNA阳性率4 1 % ,平均拷贝量 3.8× 1 0 5拷贝 /ml,BCP基因突变率为 8% ,肝癌患者的BCP基因突变率明显高于乙肝患者。
短句来源
     B type hepatitis virus( HBV) infection is the big dease to risk for the humanbing .
     乙型肝炎病毒(HBV)感染是危及全球人类健康的疾病。
短句来源
     MethodsThe trial group was 141 cases of chronic carriers with HBV. The matched control groups included: 138 healthy control, and 158 hospitalized cases with chronic B type hepatitis.
     方法观察组为来自门诊的慢性HBV携带者,共141人; 对照组包括:健康对照组,共138人;
短句来源
     Purpose To study the positive rates of HBsAg and anti-HBc in the new college students and the prevention effects of B type Hepatitis vaccine to HBV. Method Retrospectively compare the positive rates of HBsAg and anti-HBc in the new college students of Grade 94 and 95 and the new affection rates in five years after vaccinating to the negative students.
     目的 探讨大学入学新生HBsAg和抗—HBc阳性率及乙肝疫苗对乙肝的预防作用。 方法 回顾性地研究了 94级、95级新生HBsAg和抗—HBc阳性率及阴性者接种乙肝疫苗五年内的乙肝病毒新感染率。
短句来源
     Capacity of B Type Hepatitis Virus Influencing the Result of Passive Immunity in Pregnant Women for Preventing Intrauterine Infection
     孕妇携带乙型肝炎病毒载量影响被动免疫预防宫内感染效果的研究
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  相似匹配句对
     Type of Y.
     针对 Y.
短句来源
     Superinfection of Type A. B. C Hepatitis
     甲、乙、丙型肝炎病毒重叠感染
短句来源
     TTV is mixed with all type hepatitis.
     TTV与各型肝炎均有混合感染;
短句来源
     3 patients with severe type of B hepatitis;
     重症乙肝3例;
短句来源
     STUDY ON THE HEPATITIS B VIRUS TYPE 2
     乙型肝炎病毒2型的研究
短句来源
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  b type hepatitis
So it is easy to catch AIDS, B type hepatitis and so on when the handpieces are used again.
      
Hepatitis B virus is a small hepatotropic DNA virus, causing acute and chronic B-type hepatitis in man.
      


Anagallidum,Yi gan ning and Arenaria,Chinese tibetan medicinal herbs all have shown apparent therapeutic effect on homunised hepatitis mice. The activation of sGPT was apparently(P<0. 05). Yi gan ning and Angallidium apparently reduced the activation of hepar P450 (P<0.002),but the effect of Arenaria was not apparent. Observation of these that they are all effective cure drugs on B type hepatitis but according to their effect they may be arranged in this order:Anagallidium>Yi gan ning>Arenaria.There...

Anagallidum,Yi gan ning and Arenaria,Chinese tibetan medicinal herbs all have shown apparent therapeutic effect on homunised hepatitis mice. The activation of sGPT was apparently(P<0. 05). Yi gan ning and Angallidium apparently reduced the activation of hepar P450 (P<0.002),but the effect of Arenaria was not apparent. Observation of these that they are all effective cure drugs on B type hepatitis but according to their effect they may be arranged in this order:Anagallidium>Yi gan ning>Arenaria.There is no significant difference microscoplcally on the hepatic tissue between the three groups of drugs taken,and that of the control group, That means the linprovement is not obscure.This may due to the time of drug administratinn is too short (5 day) Itherefore restoration of hepatic tissue impairment is hard to observe.

藏茵陈、乙肝宁和雪灵芝三种中藏药对小鼠免疫性肝炎均有明显的治疗作用,使sGPT活性明显降低(P<0.05),对肝P450的作用,乙肝宁和藏茵陈均能降低酶的活性(P<0.002),而雪灵芝的作用不明显。说明三药治疗乙型肝炎是有效的,疗效强弱顺序为藏茵陈>乙肝宁>雪灵芝。但三药对肝组织形态学镜检与对照组比较,改善不明显,可能与给药时间短(给药5天)有关,未见恢复肝组织的明显疗效。

Purpose: The present studies were first to determine the plasma levels of tissue factor path-way inhibitor (TFPI) in several type of hepatitis and decompenstion cirrhosis. Methods: TFPI antigen (TFPI:Ag) was determined with an enzyme linked immunity (ELISA). TFPI: active (TFPI:A) was de-termined with chromogenic peptide substrate method. Results: Acute toxin hepatitis (n=14), acute viral hepatitis (n=20), chronic B type hepatitis (n=48)and decompensation cirrhosis (n=20), TFPI:Ag/...

Purpose: The present studies were first to determine the plasma levels of tissue factor path-way inhibitor (TFPI) in several type of hepatitis and decompenstion cirrhosis. Methods: TFPI antigen (TFPI:Ag) was determined with an enzyme linked immunity (ELISA). TFPI: active (TFPI:A) was de-termined with chromogenic peptide substrate method. Results: Acute toxin hepatitis (n=14), acute viral hepatitis (n=20), chronic B type hepatitis (n=48)and decompensation cirrhosis (n=20), TFPI:Ag/ TFPI: A was: 160.3±26.1 ng/ml/515. 8±50.2 u/ml, 197.7±18.6ng/ml/532.1±98.7u/ml, 162.5±28.3 ng/ml/636.7±?54.8u/ml, 81.7±16.7ng/ml/747.2±81.2u/ml respectively. Except for group of cirrhosis, the other groups TFPI: Ag was increased than control(103. 2±?11.5 ng/ml) and TFPI: A were increased than control(112. 5±23.6 u/ml) in all groups. Conclusion: TFPI can over showdown on inflammation due to active of mono-macrophages and endothelial cell of vessel irritated by inflammatory fac-tor. But TFPI: Ag decreased to low levels of normal in group of cirrhosis, be concerned with thrombosis of portal vein system, The TFPI:Ag would decreased in a consumption coagulation.

目的:测定各类型肝炎及失代偿期肝硬化患者血浆中的组织因子途径抑制物(Tissue factor pathway in-hibitor,TFPI),并探索其临床意义。方法:TFPI抗原(TFPI:Ag)测定采用双夹心ELISA抗原测定法,TFPI活性(TFPI:A)测定采用发色底物法。结果:急性中毒性肝炎(n=14)TFPI:Ag为160.3±26.1ng/ml,TFPI:A为515.8±50.2 u/ml;急性病毒性肝炎(n=20)各为197.7±18.6 ng/ml与532.1±98.7 u/ml;慢性乙型肝炎(n=48)各为162.5±28.3ng/ml与636.7±54.8 u/ml;失代偿期肝硬化(n=20)各为81.7±16.7 ng/ml与747.2±81.2u/ml。与对照组相比,TF-PI:Ag(正常值103.2±11.5 ng/ml)除肝硬化组接近正常低值外,其它各组均有增高(P<0.05);TFPI:A(正常值112.5±23.6 u/ml)各组均增高(P<0.01)。结论:炎症时因单核-巨噬细胞活跃及炎症因子刺激血管内皮细胞,故使TFPI过度表达,但在肝硬化时TFPI:Ag已趋向正常低值...

目的:测定各类型肝炎及失代偿期肝硬化患者血浆中的组织因子途径抑制物(Tissue factor pathway in-hibitor,TFPI),并探索其临床意义。方法:TFPI抗原(TFPI:Ag)测定采用双夹心ELISA抗原测定法,TFPI活性(TFPI:A)测定采用发色底物法。结果:急性中毒性肝炎(n=14)TFPI:Ag为160.3±26.1ng/ml,TFPI:A为515.8±50.2 u/ml;急性病毒性肝炎(n=20)各为197.7±18.6 ng/ml与532.1±98.7 u/ml;慢性乙型肝炎(n=48)各为162.5±28.3ng/ml与636.7±54.8 u/ml;失代偿期肝硬化(n=20)各为81.7±16.7 ng/ml与747.2±81.2u/ml。与对照组相比,TF-PI:Ag(正常值103.2±11.5 ng/ml)除肝硬化组接近正常低值外,其它各组均有增高(P<0.05);TFPI:A(正常值112.5±23.6 u/ml)各组均增高(P<0.01)。结论:炎症时因单核-巨噬细胞活跃及炎症因子刺激血管内皮细胞,故使TFPI过度表达,但在肝硬化时TFPI:Ag已趋向正常低值,可能与门静脉系血栓形成过程中TFPI不断大量消耗有关。

Objective:To investigate the relation between syndecan-1 protein and malignant phenotypes of hepatocellular carcinoma (HCC). Methods:Thirty-one formalin-fixed sections were obtained. The syndecan-1 was measured with immunohistochemistry assay (ABC method). Results:Among 31 HCC tissues, 18(58.1%) show negative staining; the syndecan-1 negative rate was higher in HCC with poor differentiation than in those well differentiated (80% vs 37.5%, P<0.05); in large HCC than in small HCC (85.9% vs 35.3, P<0.01); and in...

Objective:To investigate the relation between syndecan-1 protein and malignant phenotypes of hepatocellular carcinoma (HCC). Methods:Thirty-one formalin-fixed sections were obtained. The syndecan-1 was measured with immunohistochemistry assay (ABC method). Results:Among 31 HCC tissues, 18(58.1%) show negative staining; the syndecan-1 negative rate was higher in HCC with poor differentiation than in those well differentiated (80% vs 37.5%, P<0.05); in large HCC than in small HCC (85.9% vs 35.3, P<0.01); and in HCC with the presence of tumor-cells in blood than in those without. No correlation was found between syndecan-1 expression and serum AFP level, as well as chronic B-type hepatitis and liver cirrhosis. Conclusion:Our study indicated that syndecan-1 expression was correlated with the growth, differentiation, invasiveness, metastasis of HCC. It was possible that syndecan-1 was a negative regulator of these malignant phenotypes of HCC and might be regarded as suppressive gene for tumor.

目的 :探讨syndecan 1蛋白与肝细胞肝癌的关系。方法 :应用免疫组化技术 (ABC法 )检测syndecan 1在肝细胞肝癌中的表达。结果 :发现在 31例肝癌组织中的syndecan 1阴性表达 18例 ,分化不良的肝癌syndecan 1阴性表达率高于分化较好者 (80 .0 %比 37.5 % ,P <0 .0 5 ) ;直径 >5cm的肝癌syndecan 1阴性表达率高于直径≤ 5cm肝癌 (85 9%比 35 3% ,P <0 .0 1) ;大体标本或镜下癌栓syndecan 1阴性表达率高于无癌栓的肝癌 (91 7%比 6 8% ,P <0 .0 1)。syndecan 1的阴性表达与患者的性别、年龄、血清甲胎球蛋白水平、乙型肝炎病毒感染、肝硬化有无、包膜的有无等因素均无明显的相关性 (P >0 .0 5 )。结论 :syndecan 1的低表达与肝癌增大、分化程度低、侵袭转移发生等恶性表型相关 ,可能对这些表型起负调节作用 ,可视其为抑癌基因。

 
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