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   膀胱癌细胞 的翻译结果: 查询用时:0.065秒
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膀胱癌细胞     
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  bladder cancer cell
     2. COX expression in two bladder cancer cell lines, T24 and 5637, was examined after cells were stimulated with recombinant human epidermal growth factor (rhEGF) alone and combined with selective COX-2 inhibitor SC-58125 or nuclear transcription factor kappaB (NF-κB) inhibitor PDTC.
     2. 检测人膀胱癌细胞T24和5637在不同浓度的重组人表皮生长因子(rhEGF)作用下COX的表达,以及选择性COX-2抑制剂SC-58125或核转录因子κB(NF-κB)抑制剂PDTC与rhEGF一起作用时细胞COX表达的变化。
短句来源
     The Role of DNA Damage Repair in the Apoptosis Induced by 4HPR in Bladder Cancer Cell T24
     DNA氧化损伤在4HPR诱导膀胱癌细胞T24凋亡过程中的作用
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     1. The expression of RASSF1A mRNA showed negative in T24 bladder cancer cell line.
     1. RASSF1A mRNA 在膀胱癌细胞株 T24 表达阴性。
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     Influence of P53 Gene Plasmid Combined with Antisense c - myc Gene Plasmid on Growth of Human bladder Cancer Cell T24
     P53基因质粒联合反义c-myc基因质粒对人膀胱癌细胞T24生长的影响
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     Expression and identify of CH50 polypeptide in bladder cancer cell line BIU-87
     CH50多肽在膀胱癌细胞系BIU-87中的表达和鉴定
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  bladder cancer cells
     Research of Survivin Expression by Interference RNA in 5637 Bladder Cancer Cells
     RNA干扰对5637膀胱癌细胞survivin基因表达的研究
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     Conclusions: As 2O 3 could significantly inhibit the growth of bladder cancer cells.
     结论 :As2 O3可显著抑制膀胱癌细胞生长 ;
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     Conclusion:It demonstrated that 131I-BDI-1-MT can effectively target to bladder cancer cells.
     结论:131I-BDI-1-MT有抗肿瘤活性,可有效地靶向膀胱癌细胞
短句来源
     Antisense Inhibition of COX-2 and NSAIDs Reduce Malignant Phenotype of Bladder Cancer Cells
     COX-2反义RNA与NSAIDs抑制膀胱癌细胞恶性表型的实验研究
短句来源
     Effect of CAR Expression on Antitumoral Activity of E1B-Deleted Adenovirus in Bladder Cancer Cells
     膀胱癌细胞CAR的表达对E1B缺失腺病毒抗瘤活性的影响
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  bladder carcinoma cells
     Effects of 8-Br-cAMP on the Expression of c-erbB-2 c-myc Gene in Bladder Carcinoma Cells
     8-Br-cAMP对膀胱癌细胞中c-erbB2c-myc表达的影响
短句来源
     Conclusion 8-Br-cAMP could inhibited the expression of c-erbB-2,c-myc and mRNA in bladder carcinoma cells, it indicated that 8-Br-cAMP was an effective inhibitor for mitotic and proliferation of tumor cells.
     结论8-Br-cAMP可抑制膀胱癌细胞中c-erbB-2、c-myc蛋白及mRNA表达,进而抑制肿瘤细胞的分裂和增殖。
短句来源
     Objective To study the effects of 8-Br-cAMP on the expression of c-erbB-2,c-myc gene and mRNA in bladder carcinoma cells.
     目的探讨环腺苷酸(cAMP)类似物8-溴-环腺苷酸(8-Br-cAMP)对人原代培养的膀胱癌细胞中c-erbB-2、c-myc蛋白及mRNA表达的影响。
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     The influence of transfection with E1AF on invasive ability of the bladder carcinoma cells
     转染E1AF对膀胱癌细胞侵袭能力的影响
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     Objective To study the inhibition effect of VEGF expression in human bladder carcinoma cells by ~125I-coupled antisense oligonucleotides (~125I-ASODN).
     目的观察125I偶联VEGF反义寡核苷酸(125I-ASODN)对膀胱癌细胞VEGF表达的抑制情况。
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  bladder carcinoma cell
     METHODS We constructed an inducible expression vector pMDNA3 p16 and transfected it into bladder carcinoma cell line T24 with inactivation of multiple tumor suppressor1 (MTS1).
     方法 通过可诱导的真核表达载体 p MDNA3- p16将野生型多肿瘤抑制基因(multiple tumor suppressor1,MTS1)转染至该基因表达功能丧失的人膀胱癌细胞 T2 4中 .
短句来源
     Study on the results of different quantities of expression of HER2 of bladder carcinoma cell line after treated by the inhibitor of protein tyrosine kinase, PD168393
     酪氨酸激酶抑制剂PD168393对HER2不同表达水平膀胱癌细胞株的作用研究
短句来源
     Bystander effects of HSV-TK/GCV system on murine bladder carcinoma cell line T739 in vitro
     HSV-TK/GCV系统治疗鼠膀胱癌细胞T739旁观者效应的实验研究
短句来源
     Methods Hunman bladder carcinoma cell line T-24 was transfected with adenovirus expression vector containing TK gene and green fluorescent protein (GFP) gene, and the transfection efficiency was observed and TK expression detected by PCR.
     方法使用含有HSV-TK基因、绿色荧光蛋白(GFP)基因的复制缺陷腺病毒转染人膀胱癌细胞T-24细胞,表达GFP为转染成功标志,同时观察转染率,PCR检测TK表达。
短句来源
     Objective To investigate the influence of over expression of p27 KIP1 on the malignant phenotype of EJ human bladder carcinoma cell line.
     目的 探讨p27KIP1 的过度表达对人膀胱癌细胞系EJ的影响。
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  bladder cancer cell
Study of recombinant human IFN-α-2b bacilli calmette-guerin activated killer cells and against bladder cancer cell in vitro
      
Preliminary study of the in vitro growth inhibition of human bladder cancer cell line BIU-87 by arsenic trioxide
      
The growth inhibition rates of human bladder cancer cell line BIU87 by various concentrations of As2O3 were detected by using MTT method.
      
Adenovirus-mediated transfer of p53 and p16 inhibiting proliferating activity of human bladder cancer cell EJin vitro andin vivo
      
Under the induction of Lipofectamine 2000, the recombinant was transfected into Uroplakin II negative bladder cancer cell line EJ.
      
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  bladder cancer cells
Conclusion: The constructed chimeric antibody was expressed successfully in eukaryotic cells, and the chimeric antibody had desired affinity against human bladder cancer cells.
      
Effect of photodynamic therapy with BPD-MA on the proliferation and apoptosis of human bladder cancer cells
      
OBJECTIVE This study was conducted to explore the effect of Rg3 on inhibition of proliferation and induction of apoptosis in bladder cancer cells.
      
On Day 21 the tumors were resected and examined pathologically to see the immune response.Results: The observation of morphology of MBT-2 cells in vitro and MTT assay indicated that Allicin has apparent direct cytotoxicity to bladder cancer cells.
      
To study the effects of arsenic trioxide (As2O3) on the in vitro growth of human bladder cancer cells and the mechanisms.
      
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  bladder carcinoma cells
The in vitro growth of bladder carcinoma cells was analyzed by MTT colorimetry, and the apoptosis rate was assayed by TUNEL methods.
      
Using this in situ assay method, we detected the reduced permeability to HRP in aortic and brain microvascular endothelial cells after 4-5 days culture with conditioned media of human astrocytes or bladder carcinoma cells.
      
In line with these data, expression of either p63RhoGEF or GEFT in J82 human bladder carcinoma cells induced the formation of actin stress fibres.
      
Nucleic acid metabolism of bladder carcinoma cells in vitro
      
Nucleic acid metabolism was investigated to determine the metabolic activity of bladder carcinoma cells.
      
更多          
  bladder carcinoma cell
Expression of a mutant hTERT in human bladder carcinoma cell line T24 and its clinical significance
      
After transfecting the fusion gene into bladder carcinoma cell line T24 by calcium phosphate-DNA coprecipitation, the steady expression of GFP-hTERT fusion protein was tested by fluorescent light microscopy.
      
The proliferation changes of bladder carcinoma cell line T24 were detected by light microscopy and senescence correlated β-galactosidase staining.
      
Conclusion: The mutant-type hTERT gene suppresses the proliferation of bladder carcinoma cell line T24 by competitive effect on telomerase activity.
      
Apoptosis induced by ginsenoside Rg3 in a human bladder carcinoma cell line
      
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