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osteogenesis imperfecta
相关语句
  成骨不全
    X-ray examination and family-line investigation in 32 cases of osteogenesis imperfecta
    32例成骨不全症X线检查及家系调察分析
短句来源
    Osteogenesis Imperfecta (Analysis of 25 Cases)
    成骨不全(附25例分析)
短句来源
    Mutation Detection of COL1A1 Gene in a Pedigree with Osteogenesis Imperfecta
    一成骨不全家系的COL1A1基因突变检测
短句来源
    A new mutation in COL1A1 gene in a family with osteogenesis imperfecta
    成骨不全家系一个新的Ⅰ型胶原α1链蛋白基因突变
短句来源
    Genetic Studies of Osteogenesis Imperfecta Type Ⅰ and Two Candidate Genes of Osteoporosis
    Ⅰ型成骨不全及骨质疏松症两个候选基因的遗传学研究
短句来源
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  成骨不全症
    X-ray examination and family-line investigation in 32 cases of osteogenesis imperfecta
    32例成骨不全症X线检查及家系调察分析
短句来源
    Clinical effect of ossotide infection on osteogenesis imperfecta
    骨肽治疗成骨不全症临床观察
短句来源
    Fracture and non-fracture pain in children with osteogenesis imperfecta
    成骨不全症患儿的骨折性和非骨折性疼痛
短句来源
  成骨不全
    X-ray examination and family-line investigation in 32 cases of osteogenesis imperfecta
    32例成骨不全症X线检查及家系调察分析
短句来源
    Osteogenesis Imperfecta (Analysis of 25 Cases)
    成骨不全(附25例分析)
短句来源
    Mutation Detection of COL1A1 Gene in a Pedigree with Osteogenesis Imperfecta
    一成骨不全家系的COL1A1基因突变检测
短句来源
    A new mutation in COL1A1 gene in a family with osteogenesis imperfecta
    成骨不全家系一个新的Ⅰ型胶原α1链蛋白基因突变
短句来源
    Genetic Studies of Osteogenesis Imperfecta Type Ⅰ and Two Candidate Genes of Osteoporosis
    Ⅰ型成骨不全及骨质疏松症两个候选基因的遗传学研究
短句来源
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  “osteogenesis imperfecta”译为未确定词的双语例句
    Over 90% of patients with osteogenesis imperfecta have mutations in the COL1A1 and COL1A2 genes which encode the chains of typeⅠprocollagen.
    90%以上病例的发生与形成Ⅰ型胶原的两个基因(COL1A1和COL1A2)突变有关。
短句来源
    Tumoral Callus in Osteogenesis Imperfecta
    成骨不全:肿瘤样骨痂
短句来源
    The diagnostic significance of clinical and radiological findings in osteogenesis imperfecta
    成骨不全的临床和X线表现的诊断价值
短句来源
    An investigation on clinical radiological diagnosis of hereditary osteogenesis imperfecta(a report of 42 patients in a family)
    遗传性成骨不全临床及X线诊断探讨(附一家系42例报告)
短句来源
    Special Findings in Osteogenesis Imperfecta
    成骨不全:特殊表现
短句来源
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  osteogenesis imperfecta
Surgical treatment of the femur in osteogenesis imperfecta with intramedullary elongating rods
      
Beitrag zur Lehre von der Osteogenesis imperfecta tarda
      
Femurosteosynthesen mit dem Teleskopnagel nach Bailey-Dubow bei Osteogenesis imperfecta
      
Simultaneous Bilateral Tibial Tubercle Avulsion Fractures in a Boy with Osteogenesis Imperfecta
      
Treatment of children with osteogenesis imperfecta
      
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X-ray examination and family-line investigation were performed in 32 cases of osteogenesis imperfecta, suggesting that the modes of inheritance of the disease include autosomal dominant inheritance, autosomal recessive inheritance and sporadic cases caused by gene mutation. The authors found that the long bone X-ray changes of the patients in this series, besides short-thick, thin-long and saccular types, included 3 cases that were excluded from the above three types, and considered it necessary to add...

X-ray examination and family-line investigation were performed in 32 cases of osteogenesis imperfecta, suggesting that the modes of inheritance of the disease include autosomal dominant inheritance, autosomal recessive inheritance and sporadic cases caused by gene mutation. The authors found that the long bone X-ray changes of the patients in this series, besides short-thick, thin-long and saccular types, included 3 cases that were excluded from the above three types, and considered it necessary to add another type, or thin-cortical type, to Cao'S classification.

对32例成骨不全患者进行了家系随访调察和X线检查,提出本病的遗传方式为常染色体显性遗传、常染色体隐性遗传,以及基因突变引起的散发病例。发现本组病例长骨的X线改变除粗短型、细长型及囊型外。有3例不能归属以上3型。认为应在曹氏分类法的基础上,再补充一型,即薄皮质型。

Objective: To introduce a new conception of arthropathy. A traditional conception of arthropathy is that the cause of joint disease are acquired or secondary.Materials and Methods:256 films of 66 patient with dysplasia of bone including: Osteogenesis imperfecta (n=6), Marfan syndrome (n=4), mucopolysaccharidoses (n=8), multiple epiphyseal dysplasia (n=22), pycnodysostosis (n=2), metaphyseal chondrodysplasia (n=4), osteopetrosis (n=3), hereditary multiple exostosis (n=6), enchondromatosis (n=4), maffucci...

Objective: To introduce a new conception of arthropathy. A traditional conception of arthropathy is that the cause of joint disease are acquired or secondary.Materials and Methods:256 films of 66 patient with dysplasia of bone including: Osteogenesis imperfecta (n=6), Marfan syndrome (n=4), mucopolysaccharidoses (n=8), multiple epiphyseal dysplasia (n=22), pycnodysostosis (n=2), metaphyseal chondrodysplasia (n=4), osteopetrosis (n=3), hereditary multiple exostosis (n=6), enchondromatosis (n=4), maffucci disease (n=2), systemic fibrous dysplasia (n=4), and myositis ossificans progressive (n=1) were studied and compared with their clinical appearances retrospectively.Results:The results show that these patients diagnosed as osteodysplasia also have the abnormal shapes of articular faces, disordered structures and functions of joints. Moreover, their clinical manifestations were appeared before 40 year old (30 years).Conclusion: The arthropathy ought to be divided into two types: Congenital and acquired.

目的:关节病的传统概念其病因是获得性的或由后天继发因素所致,本文提出先天发育性(骨)关节病的概念。材料与方法:本组通过对成骨不全(6例),Marfan综合征(4例),粘多糖病(8例),多发骨骺发育异常(22例),致密性骨发育不全(2例),干骺软骨发育异常(4例),石骨症(3例),遗传性多发外生骨疣(6例),内生软骨瘤病(4例),Mafucci病(2例),系统性骨纤维发育异常(4例)和进行性骨化性肌炎(1例)等66例骨发育异常患者的256份X线照片的回顾性复习,并与其临床表现作了对照分析。结果:发现这些患者在骨发育异常的同时,均伴有关节端的形态和关节的结构与功能异常,并且均在40岁前(平均30岁)出现关节退变表现。结论:关节病应包括先天性和获得性两大类

Objective:To introduce the clinical and radiological findings of the tumoral callus in osteogenesis imperfecta and to describe its probable formative mechanism.Methods:The clinical radiographic and CT features of 6 patients with tumoral callus of 68 osteogenesis imperfecta were analysed retrospectively.Results:Among 6 patients with tumoral callus in osteogenesis imperfecta all had the history of multiple fractures of the extremities in childhood and hypertrophic deformity increasingly with...

Objective:To introduce the clinical and radiological findings of the tumoral callus in osteogenesis imperfecta and to describe its probable formative mechanism.Methods:The clinical radiographic and CT features of 6 patients with tumoral callus of 68 osteogenesis imperfecta were analysed retrospectively.Results:Among 6 patients with tumoral callus in osteogenesis imperfecta all had the history of multiple fractures of the extremities in childhood and hypertrophic deformity increasingly with tenderness.X-ray and CT revealed a cystic expansible osseous mass,and CT showd the diffuse decrease in osseous density which indicated fatty tissue(-40 HU—-90 HU).Conclusion:The tumoral callus of osteogenesis imperfecta is an unusual but important finding owing to its ability to mimic a tumor.The formation of excessive callus,possibly because the periosteum is very loosely attached and easy stripped.Meanwhile,there are fatty marrows extensive infiltration around previous fractures.

目的:介绍成骨不全并肿瘤样骨痂形成的临床和放射学表现,并试图探讨其形成机制。方法:对一组68例成骨不全中6例并有肿瘤样骨痂的临床、X线和CT表现进行回顾性分析。结果:有成骨不全病史的6例肿瘤样骨痂形成者,均有儿童期多次、多处骨折和肢骨局部缓慢增大畸形并有触痛。X线和CT均可显示一囊样膨胀性骨块,并经CT测定低密度区为脂肪组织(CT值为-40~-90HU)。结论:肿瘤样骨痂属成骨不全少见而又很重要的临床和放射学表现,因其颇似肿瘤。大量骨痂增生可能与骨膜的附着松驰而易剥离,同时伴有脂髓组织大量浸润增生有关。

 
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