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cerebral ischemic reperfusion     
相关语句
  脑缺血再灌注
     Study of the Changes in NF-κ Bp65, I-κ B_α and COX-2 Following Focal Cerebral Ischemic Reperfusion in Rats
     NF-κ Bp65,I-κ B_α及COX-2在大鼠脑缺血再灌注损伤中的作用
短句来源
     [Results] On the first day and second day after cerebral ischemic reperfusion expression of glutamate in CA1 neuron of hippocampus were 60.67 ±3.18 and 81.22±7.12; respectively both of which were significantly higher than that of control group being 42.25±2.79,P<0.01,the expression on the second day was significantly higher than that on the first day,P <0.01;
     [结果]脑缺血再灌注后第1和第2天,海马CA1神经元谷氨酸表达的辉度值分别为60.67±3.18和81.22±7.12,明显高于对照组的42.25±2.79(P<0.01),且第2天的表达明显高于第1天(P<0.01);
短句来源
     expression of NGF were 34.95±2.80 and 28.83±1.82 respectively both of which were significantly lower than that of control group being 49±2.38,P<0.01,expression on the second day was significantly lower than that on the first day,P<0.05. Neuronal disappearance was only observed in CA1 of hippocampus on the seventh day after cerebral ischemic reperfusion.
     神经生长因子表达的辉度值分别为34.95±2.80和28.83±1.82,明显低于对照组的49±2.38(P<0.01),且第2天表达明显低于第1天(P<0.05)。 脑缺血再灌注后第7天神经元脱失仅见于海马CA1区。
短句来源
     Effects of phycocyanin on the expressions of NF-κB,IL-6 and neuronal apoptosis after cerebral ischemic reperfusion in rats
     藻蓝蛋白对脑缺血再灌注后NF-κB和IL-6表达及神经细胞凋亡的影响
短句来源
     The Effect and Significance of Inosine on Expression of COX-2, CaN and VEGF Following Cerebral Ischemic Reperfusion in Rats
     肌苷对脑缺血再灌注大鼠脑组织中COX-2、CaN、VEGF表达的影响及意义
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  缺血再灌注
     CHANGES OF LEARNING AND MEMORY ABILITIES AND EXPRESSION OF HIPPOCAMPUS Glu,NR_1 AND NR_(2B) ON CEREBRAL ISCHEMIC REPERFUSION INJURY IN RATS
     缺血再灌注对大鼠学习记忆及海马神经元Glu、NR_1和NR_(2B)表达的影响
短句来源
     Relationship between AQP_4 expression and brain edema after cerebral ischemic reperfusion
     AQP_4基因表达与缺血再灌注后脑水肿的关系
短句来源
     Objective To observe the expression of N-methyl-D-aspartate receptor 1(NMDAR1) mRNA following cerebral ischemic reperfusion in rat brain,and to investigate neuroprotective effects of picrorhizae on neurons.
     目的观察缺血再灌注模型大鼠脑内N-甲基-D-天门冬氨酸受体1(NMDAR1)mRNA的变化,探讨胡黄连对神经元的保护作用。
短句来源
     EFFECTS OF PICRORHIZAE ON EXPRESSION OF NMDAR1 mRNA FOLLOWING CEREBRAL ISCHEMIC REPERFUSION IN RAT BRAIN
     胡黄连对缺血再灌注大鼠脑内NMDAR1 mRNA表达的影响
短句来源
     The lesion area appeared the budding sign of neurons axon in cerebral ischemic reperfusion from 48h to 7d,and showed synaptic fiber.
     缺血再灌注48h~7d损伤区域神经元轴突呈出芽征,发出突触纤维。
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  脑缺血再灌流
     Objective To investigate the relationship between the expression of Cyclin Dl and CDK4mRNA and neuronal apoptosis or gliosis after focal cerebral ischemic reperfusion in rats and to explore the intervention of Inosine.
     目的:观察大鼠局灶性脑缺血再灌流后细胞周期蛋白D1(CyclinD1)mRNA和周期蛋白依赖蛋白激酶4(CDK4)mRNA的表达与神经细胞凋亡、神经胶质细胞增生的关系以及INOSINE的干预作用,从细胞增殖角度探讨INOSINE的神经保护作用机制。
短句来源
     METHODS A cerebral ischemic reperfusion injury model was established by 4 VO method to measure the numbers of peripheral blood white cell,numeral pathology of hippocampal CA 1 section, the content of MDA and the activity of Na +, K + ATP enzyme in hippocampus, were measured in rats.
     方法 采用 4- VO法制作大鼠脑缺血再灌流损伤模型 ,观察环磷酰胺预治疗大鼠外周血白细胞数 ,海马CA1 区的计量病理 ,海马区丙二醛含量和 Na+ ,K+ - ATP酶活性的变化 .
短句来源
     Results: Pretreatment with GHBA (3.6 ml·kg-1)significantly declined the con- tents of EAAs in the focal cerebral ischemic reperfusion after different durations of reperfusion and decreased the volumn of cerebral infarction.
     结果:腹腔注射GHBA3.6ml·kg-1可明显减少局部脑缺血再灌流后各时间点EAAs的含量,缩小再灌流后各时间点脑梗死的体积。
短句来源
     Objective To study the mechanism of neurocyte on inhibitor of nitric oxide synthase (NOS) in the focal cerebral ischemic reperfusion and the relationship between inhibitor of NOS and apoptosis especially.
     目的 研究一氧化氮合酶(NOS)抑制剂在局灶性脑缺血再灌流过程中对神经细胞的作用机理,特别是NOS抑制剂(NNLA)与细胞凋亡的关系。
短句来源
     Conclusion BDE may have protect effect on cerebral ischemic reperfusion damage by decreasing Nos activity.
     结论 BDE抑制神经元NOS活性 ,对脑缺血再灌流损伤具有脑保护作用。
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  脑缺血再灌注损伤
     Study of the Changes in NF-κ Bp65, I-κ B_α and COX-2 Following Focal Cerebral Ischemic Reperfusion in Rats
     NF-κ Bp65,I-κ B_α及COX-2在大鼠脑缺血再灌注损伤中的作用
短句来源
     Study on the influence of transforming growth factor-β_1 to expression of TNF-αafter cerebral ischemic reperfusion injury in rats
     转化生长因子β_1对大鼠脑缺血再灌注损伤后肿瘤坏死因子-α表达的影响
短句来源
     Methods We established the model of MCA focal cerebral ischemic reperfusion injury according to the longa method and measured the level of IL 1β by ELISA method.
     方法 根据Longa方法建立了MCA局部脑缺血再灌注损伤模型 ,并用ELISA方法检测IL 1β水平。
短句来源
     Objective To explore the role of interleukin 1β(IL 1β) in rat focal cerebral ischemic reperfusion injury.
     目的 探讨IL 1β在大鼠局部脑缺血再灌注损伤中的作用。
短句来源
     METHODS:The model of focal cerebral ischemic reperfusion was established with thread embolism method, and TGF β1 was injected into the stereotaxic lateral ventricle, so as to observed the effect of different doses of TGF β1 on focal cerebral ischemic reperfusive injury and cellular apoptosis.
     方法:用线栓法建立局灶脑缺血再灌注模型,立体定向侧脑室内注入转化生长因子β1,观察不同剂量TGF-β1对局灶脑缺血再灌注损伤及对细胞凋亡的影响。
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  cerebral ischemic reperfusion
Infarction in adult rat brain was induced by middle cerebral arterial occlusion (MCAO) followed by reperfusion to examine whether taxifolin could reduce cerebral ischemic reperfusion (CI/R) injury.
      


A mouse learning and memory disfunction model was established by cerebral ischemic reperfusion. The protecting effects of Yinao granules on the model were studied by step down and step through tests. Results of the experiment showed that cerebral ischemic reperfusion could decrease the ability of learning and memory (P<0. 01) and Yinao granules could improve the learning and memory defect induced by cerebral ischemic reperfusion. The difference between tile results of Yinao granules groups...

A mouse learning and memory disfunction model was established by cerebral ischemic reperfusion. The protecting effects of Yinao granules on the model were studied by step down and step through tests. Results of the experiment showed that cerebral ischemic reperfusion could decrease the ability of learning and memory (P<0. 01) and Yinao granules could improve the learning and memory defect induced by cerebral ischemic reperfusion. The difference between tile results of Yinao granules groups and control groups is significant (P<0. 01).

采用脑缺血再灌致小鼠学习记忆损伤造模法,并通过小鼠一次性回避性条件反射方法,观察了益脑冲剂对脑功能的保护作用。结果显示,脑缺血再灌可使小鼠学习记忆能力降低(P<0.01)。中药益脑冲剂各组对脑缺血再灌所致学习记忆损伤有明显的改善作用,与脑代谢活化剂都可喜的作用相似,给药各组与模型组之间差异非常显著(P<0.01)。

In this study, 45 Wistar Rats were divided into three groups, sham - operated(n=3),control group(n = 21),Batroxobin group (n = 21). Batroxobin was examined for its effect on HSP 70 induction in rat cerebral cortex at various recir-culation levels after 90min ischemia using middle cerebral vessel thread occlusion rat model. In immunocytochemical analysis using anti-HSP70 antibody,in control group a little induction of HSP 70 appeared after 90min ischemia followed by Ih reperfusion and reached a maximum level...

In this study, 45 Wistar Rats were divided into three groups, sham - operated(n=3),control group(n = 21),Batroxobin group (n = 21). Batroxobin was examined for its effect on HSP 70 induction in rat cerebral cortex at various recir-culation levels after 90min ischemia using middle cerebral vessel thread occlusion rat model. In immunocytochemical analysis using anti-HSP70 antibody,in control group a little induction of HSP 70 appeared after 90min ischemia followed by Ih reperfusion and reached a maximum level at 48h. In atninals treated with batroxobin 90 min after ischemia,the time course of HSP 70 induction after recirculation was same as the control animals,The results suggest that HSP 70may play an important role in the neuroprotective mechanism of batroxobin in cerebral ischemic reperfusion injury.

本文研究目的为巴曲酶是否影响热休克蛋白起到神经保护作用。用中大脑动脉(MCA)线栓法缺血再灌注大鼠模型。Wistar大鼠共51只。发现:在再灌注1h、2h、3h对照组与巴曲酶组(8BU/kg ip)HSP70均呈轻度表达,从再灌注12h起表达显著,至再灌注后24h达高峰,至再灌注后6d仅见于坏死灶周围,至再灌注后14d恢复至假手术组水平。巴曲酶组的HSP70表达变化在时程上与对照组一致,但在再灌注12h起至6d较对照组显著,同时相应时间点的MCA血供区皮层神经细胞有缺血变性者巴曲酶组少而轻。本文结果提示巴曲酶的神经保护作用,可能与它能影响HSP70蛋白合成的调控机制有关。

The activity of interleukin-1(IL-1),Interleukin-6(IL-6)of peripheral blood mononuclear cells(PBMC)as well as the level of tumor necrosis factor(TNF)in serum were measured in 26 normal controls and 34 patients with acute and recovery cerebral infarction.Results showed that:the activity of IL-1,IL-6 of PBMC as well as the level of TNF in serum of the patients with acute and recovery cerebral infaction were significantly higher than those of normal controls(P<0.01);the high activity of IL-1,IL-6 of PBMC as well...

The activity of interleukin-1(IL-1),Interleukin-6(IL-6)of peripheral blood mononuclear cells(PBMC)as well as the level of tumor necrosis factor(TNF)in serum were measured in 26 normal controls and 34 patients with acute and recovery cerebral infarction.Results showed that:the activity of IL-1,IL-6 of PBMC as well as the level of TNF in serum of the patients with acute and recovery cerebral infaction were significantly higher than those of normal controls(P<0.01);the high activity of IL-1,IL-6 of PBMC as well as the high level of TNF in serum were related to volume of infarct.The results indicated that early inflammatory response,producing of cytokines may play a key role in cerebral ischemic reperfusion injury after cerebral infarction.

本文对26名健康对照者及34例脑梗塞患者急性期和恢复期外周血单个核细胞(PBMC)白细胞介素-1(IL-1)、白细胞介素-6(IL-6)活性及血清肿瘤坏死因子(TNF)水平进行检测。结果示:急性期和恢复期脑梗塞患者PBMCIL-1、IL-6活性及血清TNF水平较对照者明显增高,急性期脑梗塞患者又较恢复期脑梗塞患者高,均有显著性差异(P<0.01);增高的程度与梗塞灶大小密切相关。提示:脑梗塞后早期炎症反应、细胞因子的产生在脑缺血和(或)再灌注损伤中可能起着重要作用。

 
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