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subacute poisoning
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  中毒
     Subacute poisoning study indicated that, there was no obvious difference in Ca2++ Mg2+-ATPase, Na+ + K+-ATPase and ecto-Ca2+-ATPase activities between brain cells of the controlled and poisoned rats.
     在对大鼠进行的亚急性实验中,发现溴氰菊酯中毒组与对照组大鼠的Ca~(2+)+Mg~(2+)-ATP酶、Na~++K~+-ATP酶和ecto-Ca~(2+)-ATP酶的活性均无显著性差异。
短句来源
     283 fertilizer workers with working years varied form 1 to 35 years under the CO concentration below 91. 2mg/m~3 were examined. No case of acute or subacute poisoning was shown.
     我们调查了某化肥厂接触CO浓度在91.2mg/m~3以下,工龄为1~35年间的283名职工,未发现急性和亚急性中毒病例。
短句来源
     The results suggest that one of mechanisms of liver injury in the rats poisoned subacutly by CCl_4 was lipid peroxidation and the injury effects of lipid peroxidation in the hepatic mitochondria and microsome of rats were induced by subacute poisoning of CCl_4.
     CCl_4亚急性中毒引起肝细胞中的线粒体和微粒体发生LPO损害作用。
短句来源
     This paper reports the observations of pathological changes in the experimental animals by acute and subacute poisoning of Russell's Viper Venom and the studies of the causes of death.
     本文讨论,主要通过动物实验进行急性及亚急性中毒组来观察蝰蛇蛇毒中毒后所引起的病理变化及致死的原因。
短句来源
     As the ratio of inhibition is 40—70%, it will lead fish to be subacute poisoning The result shows phosphor is not only a strong toxicant which can lead fish to be acute toxicity, but also a stable accumulatable toxicant which will destroy fish liver ceils, inhibit fish normal metabolism, poison fish nerve and thus leading fish to die by subacute toxicity and chronic toxicity.
     当抑制率为40%~70%时,即可导致鱼类亚急性中毒死亡。 结果表明:元素磷不但是一种剧毒物质,能引起鱼类的急性中毒,而且是一种稳定的可积累的毒物,它能破坏鱼类的肝细胞,抑制正常代谢,造成神经毒性,以致引起鱼类的亚急性和慢性中毒死亡。
短句来源
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  亚急性中毒
     283 fertilizer workers with working years varied form 1 to 35 years under the CO concentration below 91. 2mg/m~3 were examined. No case of acute or subacute poisoning was shown.
     我们调查了某化肥厂接触CO浓度在91.2mg/m~3以下,工龄为1~35年间的283名职工,未发现急性和亚急性中毒病例。
短句来源
     This paper reports the observations of pathological changes in the experimental animals by acute and subacute poisoning of Russell's Viper Venom and the studies of the causes of death.
     本文讨论,主要通过动物实验进行急性及亚急性中毒组来观察蝰蛇蛇毒中毒后所引起的病理变化及致死的原因。
短句来源
     As the ratio of inhibition is 40—70%, it will lead fish to be subacute poisoning The result shows phosphor is not only a strong toxicant which can lead fish to be acute toxicity, but also a stable accumulatable toxicant which will destroy fish liver ceils, inhibit fish normal metabolism, poison fish nerve and thus leading fish to die by subacute toxicity and chronic toxicity.
     当抑制率为40%~70%时,即可导致鱼类亚急性中毒死亡。 结果表明:元素磷不但是一种剧毒物质,能引起鱼类的急性中毒,而且是一种稳定的可积累的毒物,它能破坏鱼类的肝细胞,抑制正常代谢,造成神经毒性,以致引起鱼类的亚急性和慢性中毒死亡。
短句来源
     The results suggest that one of mechanisms of liver injury in the rats poisoned subacutly by CCl_4 was lipid peroxidation and the injury effects of lipid peroxidation in the hepatic mitochondria and microsome of rats were induced by subacute poisoning of CCl_4.
     CCl_4亚急性中毒引起肝细胞中的线粒体和微粒体发生LPO损害作用。
短句来源
     subacute poisoning causesKashin-Beck's disease and Keshan disease;
     亚急性中毒可能导致人的大骨节病和克山病;
短句来源
  “subacute poisoning”译为未确定词的双语例句
     The subacute poisoning test was conducted for 5 weeks, with concentrations of 0.03mg/L, 0.05mg/L, 0.10mg/L, 0.18mg/L, 0.32mg/L.
     以水中铜浓度分别为0.03mg/L、0.05mg/L、0.10mg/L、0.18mg/L、0.32mg/L对鲤鱼进行5周的亚急性毒性试验。
短句来源
     The subacute poisoning test was conducted for 48 days,with CuSO_4 concentrations of 0.5 mg/1, 1.0 mg/1, 1.5 mg/1, 2.0 mg/1,2.5 mg/1. The mortality were respectively 70%,80%,86.67%,93.33%,100%.
     以水中硫酸铜质量浓度为:0.5 mg/L、1.0 mg/L、1.5 mg/L、2.0 mg/L和2.5 mg/L对金鱼进行48天亚急性毒性试验,各组金鱼的死亡率分别为70%、80%、86.67%、93.33%和100%。
短句来源
     Comparative histological study of liver fibrosis during subacute poisoning of some hepatic toxicants.
     几种亲肝毒物亚慢性实验中毒时肝纤维化过程的组织学比较
短句来源
     Pathomorphological Studies on Experimental Olaquindox Acute and Subacute Poisoning in Broiler Chickens
     实验性肉用雏鸡喹乙醇急性、亚急性中毒的病理形态学研究
短句来源
     Thirty-six cases with subacute poisoning spinal and peripheral neuritis were diagnosed in 1973.The epidemiologic features of those patients were:the same time and place occurrence,as well as family aggregation.
     36例亚急性中毒性脊髓周围神经炎的流行特征是:发病集中,有明显的家庭聚集性;
短句来源
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  subacute poisoning
Cadmium effects on bone and dental tissues of rats in acute and subacute poisoning
      
Effect of subacute poisoning by cyolane on acetylcholine esterase and succinic dehydrogenase in the rat
      
The influence of vitaral on some mineral and enzyme disturbances in subacute poisoning with hydrogen sulfide
      
Subacute poisoning, resulting from exposure over a few days, has been reported to cause reactive airways dysfunction syndrome months later.
      


This paper reports the observations of pathological changes in the experimental animals by acute and subacute poisoning of Russell's Viper Venom and the studies of the causes of death. The pathological changes by acute poisoning are mainly caused by a strong blood coagulation, fo-rming widespread thrombus in vessels, which can be seen with the naked eyes, in the venae cava superior, venae renales etc. Subsequently the acute circulatory failure results in death. In the pathological changes by subacute...

This paper reports the observations of pathological changes in the experimental animals by acute and subacute poisoning of Russell's Viper Venom and the studies of the causes of death. The pathological changes by acute poisoning are mainly caused by a strong blood coagulation, fo-rming widespread thrombus in vessels, which can be seen with the naked eyes, in the venae cava superior, venae renales etc. Subsequently the acute circulatory failure results in death. In the pathological changes by subacute poisoning,the forming of very small thrombus can only be seen under microscopes and some petechiae can be seen on the sufcace of some bowels. The causes of death of subacute poisoning can be considered as the disseminated intravascular coagulation and the circulatory failure caused by the vascular poisons.

本文讨论,主要通过动物实验进行急性及亚急性中毒组来观察蝰蛇蛇毒中毒后所引起的病理变化及致死的原因。观察结果:在急性中毒组病理变化的主要原因是蝰蛇毒的强促凝作用,引起上腔静脉、肾静脉……等血管肉眼可见的广泛血栓形成,最后由于急性循环衰竭而死亡。而亚急性中毒组病理变化仅在镜下可见有微血栓形成,且脏器已出现灶性出血,其死亡原因考虑为弥漫性血管内凝血及血管毒素作用导致循环衰竭而引起死亡。

Twenty-eight rats with experimental cadmium subacute poisoning were used to study the alteration of ALP activity in renal proximal convoluted tubules (PCT) histochemically. In cadmium group CdCL2 was administered intraperitoneally for 43-45 days. Rats which had received saline were used as controls. The ALP activity of PCT was quantified by S575 digital image processing system and classified into the four classes according to the intensity. Subcellular nepho-pathological changes of PCT in subacute poisoning...

Twenty-eight rats with experimental cadmium subacute poisoning were used to study the alteration of ALP activity in renal proximal convoluted tubules (PCT) histochemically. In cadmium group CdCL2 was administered intraperitoneally for 43-45 days. Rats which had received saline were used as controls. The ALP activity of PCT was quantified by S575 digital image processing system and classified into the four classes according to the intensity. Subcellular nepho-pathological changes of PCT in subacute poisoning rats corresponding to the alteration of ALP activity were also observed. The results showed that the average of the area of each PCT occupied by ALP product of the experimental animals became smaller than that of the control animals (p<0.01) and ALP activity of PCT decreased predominantly in rats injected with CdCl2 (p<0.01). In histochemical ultrathin sections the number, size and density of product granules of PCT decreased, too. The ultra-structural alteration of microvilli, basal foldings, mitochondria and lysosomes were present in ultrathin sections.

实验性大鼠亚急性镉中毒时,半定量法及S_(575)数字图象处理系统测量均表明其紧近曲管ALP 活性明显降低,反应物所占面积明显减小。常规电镜及电镜组化标本分别显示近曲管微绒毛、线粒体、基底褶和溶酶体均有明显改变,ALP 阳性反应颗粒减少。

The alterations of G6PD and GDH activity in proximal and distal tubules(PCT)of the kidney in 67 rats with acute and subacute Cd-induced poisoningwere investigated histochemically.The change of G6PD activity in the subacutepoisoning group was measured by TX-82 Computer Image Processing System qua-ntitatively.The histochemical quantifying method of Image Processing Systemused on photographic negative of automatic control photomicrograph was introd-uced and discussed.The results of semi-quantification and Image...

The alterations of G6PD and GDH activity in proximal and distal tubules(PCT)of the kidney in 67 rats with acute and subacute Cd-induced poisoningwere investigated histochemically.The change of G6PD activity in the subacutepoisoning group was measured by TX-82 Computer Image Processing System qua-ntitatively.The histochemical quantifying method of Image Processing Systemused on photographic negative of automatic control photomicrograph was introd-uced and discussed.The results of semi-quantification and Image Processing measurement revea-led that the activity of G6PD and GDH was decreased in PCT of the rats withacute poisoning.However,a significant increase of G6PD and GDH activity pres-ented in PCT of rats with subacute poisoning.The activity of G6PD and GDHappeared variedly in different segments of proximal and distal tubules.

67只急性和亚急性镉中毒大鼠肾组织化学检查,发现急性中毒大鼠近端小管曲部G6pD 和 GDH 活性均减弱,亚急性中毒组均增强。远端小管曲部 GDH 活性在亚急性申毒组增强。此外,还用 Tx—82计算机图象处理系统对部分结果进行定量测定,并进行了方法学探讨。

 
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