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mannitol treatment
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  渗透处理
     (3) 0.5M mannitol treatment from 6h before to 18h after bombardment could increase the transformation frequency.
     (3)在基因枪轰击前6小时和轰击后18小时,用0.5mol/l的甘露醇进行渗透处理,其转化频率会有明显的提高;
短句来源
     (3)0.5mol·L~(-1) mannitol treatment from 6h before to 18h after bombardment could increase the transformation frequency.
     基因枪轰击前 6h和轰击后 18h ,用 0 5mol·L-1的甘露醇进行渗透处理 ,可提高愈伤组织的再生频率
短句来源
  用药(甘露醇
     MethodsICPs of 37 patients with large area cerebral infarction and intracranial hypertension were measured before and at 10 min, 20 min, 30 min, 40 min, 50 min and 60 min after mannitol treatment.
     方法对37例大面积脑梗死高颅压患者在用药(甘露醇)前、后(每隔10min)测1次颅压值,选取60min分析。
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  “mannitol treatment”译为未确定词的双语例句
     Results The plasma osmolarity increased quickly at the stage of mannitol treatment, from (300.7±3.3)mOsm/kg·H2O to (323.1 ±5.2)mOsm/kg·H2O at 48h after mannitol administration,and then remained constant.
     结果:开始使用甘露醇时,血浆渗透压迅速上升,48h即从(300.7±3.3)mOsm/kg H2O上升至(323.1±5.2)mOsm/kg H2O,然后维持相对稳定;
短句来源
     Results G-6-P pAb administration significantly reduced the permeability of BBB as well as the water content in the white matter in comparison with mannitol treatment (P<0.01), but the two treatments showed no obvious difference in reducing the water content in the gray matter (P>0.05).
     结果G-6-PpAb组同甘露醇组相比对降低BBB通透性及大脑白质水分含量均有显著效果(P<0.01),对灰质的脱水作用无显著性差异(P>0.05)。
短句来源
     The expression of TSRF1 was induced by pathogen infection, ethylene, salicylic acid, ABA, NaCl or mannitol treatment etc.
     Northern Blot实验显示TSRF1的表达可被病害青枯和灰霉,植物激素乙烯、水杨酸和ABA,以及NaCl诱导;
短句来源
     Methods 60 Wistar rats were randomly divided into normal group,VBE group,edema with mannitol treatment group,and edema with AC pAb treatment group.
     方法:将60只Wistar大鼠随机分为正常组、脑水肿组、脑水肿甘露醇组、脑水肿ACpAb组共4组。
短句来源
     With -0.9 MPa D Mannitol treatment,the elongation growth (LER) and H + extrusion of mung bean seedling hypocotyl was significantly inhibited,and the inhibition was even more serious with treatment time.
     -0.9MPa甘露醇渗透胁迫处理,明显抑制绿豆幼苗下胚轴延伸生长和生长部位H+分泌,而且随胁迫时间延长抑制程度增大。
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  相似匹配句对
     The Treatment of Hemorrhagic Shock with Mannitol
     高渗甘露醇抢救失血性休克42例分析
短句来源
     L. P. treatment.
     L. P.
短句来源
     TREATMENT OF INTESITNAL OBSTRUCTION WITH ORAL MANNITOL
     口服甘露醇溶液治疗肠梗阻的疗效观察
短句来源
     TREATMENT OF DYSMENORRHOEA
     痛经的治疗
短句来源
     D-mannitol (C-14);
     虫草酸(C-14);
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  mannitol treatment
Mannitol treatment resulted in histologic and biochemical alterations (reduced lactase, sucrase, maltase) limited to the proximal small intestine compared to the control group.
      
In contrast, mannitol treatment resulted in a small, nonsignificant improvement in these parameters.
      
These results may provide a correlation between increased expression of several PKC isoforms and, in particular, increased phosphorylation levels of PKCα and δ and hypocontractility induced by high glucose and mannitol treatment.
      
Mannitol treatment increased DAO but not NADH-PODactivities in roots of rice seedlings, suggesting that DAO contributes to thegeneration of H2O2 in the cell walls of mannitol-treatedroots.
      
Mannitol treatment of cerebral edema in rats with galactosamine-induced severe hepatitis
      
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Excised roots were osmotically sho-cked by 0.3,0.5,0.8 M mannitol respectively followed by water at roomtemperature.This treatment reduceduptake of K~+,Cl~- in shocked roots.0.3 M mannitol treatment merely re-duced the ability of uptake,0.5 M and0.8 M treatment depressed uptakeseverely and even induced efflux ofions.Osmoticum of higher concen-tration may caused some damages toplasmalemma. Proteins were released from shock-ed roots and accompanied by a simul-taneous reduction of uptake activity.Recovery...

Excised roots were osmotically sho-cked by 0.3,0.5,0.8 M mannitol respectively followed by water at roomtemperature.This treatment reduceduptake of K~+,Cl~- in shocked roots.0.3 M mannitol treatment merely re-duced the ability of uptake,0.5 M and0.8 M treatment depressed uptakeseverely and even induced efflux ofions.Osmoticum of higher concen-tration may caused some damages toplasmalemma. Proteins were released from shock-ed roots and accompanied by a simul-taneous reduction of uptake activity.Recovery of uptake was observed during subsequent immersion of shockedroots in aerated 0.1 mM CaSO_4.After4 hr washing,0.3 M mannitol shockedroots completely recovered their uptakecapacity.But 0.5 M and 0.8 M mannitol shocked roots were capable ofrecovering only a small portion after24 hr washing.0.1mM cycloheximideand 2mM p-fluorophenylalanine inhibited the recovery of uptake capacity.These may be the evidences that someproteins released by osmotic shock wereinvolved in the ion transport system andde novo synthesis of proteins correlatedwith restoration of uptake capacity. Although osmotic shock inhibitedthe uptake of ions but had less effect onrespiration. Other finding in this investigationwas the less reduction of KCl uptakerate in twice shocked (Shock-Recovery-Shock) roots as comparing with that ofonce shocked roots.

0.3M甘露醇对小麦离体根作渗透震动处理,降低K~+、C1~-的吸收速率;0.5、0.8M甘露醇不仅严重地抑制吸收,有时还引起离子的外流。0.3M甘露醇处理后的小麦根在通气的0.1mM CaSO_4中浸洗,可以全部恢复离子吸收的能力;0.5M、0.8M甘露醇处理的只能部分恢复离子吸收能力。环已酰亚胺及对氟苯丙氨酸对吸收能力的恢复有抑制作用。小麦根在渗透震动之后,在吸收速率恢复后再进行一次渗透震动,仍能降低离子吸收速率。小麦根在渗透震动过程中释放的蛋白质中有一部分与离子吸收有关。渗透震动处理对小麦根呼吸的影响不大。

Effects of water stress due to 0.2 M mannitol treatment and of cotyledon excision on growth and osmotic adjustment in the epicotyls of mung bean seedlings were investigated.

在水分供应正常的情况下,切除1/4~1/2的绿豆子叶对上胚轴生长没有明显的抑制作用,但是经0.2M甘露醇处理的幼苗却随着切除的子叶量的增加,上胚轴的生长呈直线剧减,全部切除子叶的幼苗生长几乎完全停止。子叶在水分胁迫状况下对生长的这种作用与它在上胚轴生长区渗透调节中的作用密切相关。幼苗上胚轴的生长与细胞的有效渗透势之间的相关系数为-0.76,有较高的相关性,但是生长与细胞的总渗透势无相关性(相关系数为-0.29)。细胞的渗透势与细胞液中的糖、游离氨基酸和钾离子浓度之间有很高的相关性。表明水分胁迫下生长所需的低渗透势的维持依赖于来自子叶的渗透物质。

Three cases of cerebral vascular disease with nonketotic hyperglycemic hyperosmolar coma caused by the administration of mannitol are reported. There was no previous history of diabetes mellitus. Diagnosis was made by lumbar puncture and brain CT scanning. All three caseswere treated with intravenous drip of mannitol. During the course of mannitol treatment the blood sugar was elevated to levels 609 mg% to 840 mg% with glucosuria (++) and ketonuria (-). All three pataients died. We suggest that if there...

Three cases of cerebral vascular disease with nonketotic hyperglycemic hyperosmolar coma caused by the administration of mannitol are reported. There was no previous history of diabetes mellitus. Diagnosis was made by lumbar puncture and brain CT scanning. All three caseswere treated with intravenous drip of mannitol. During the course of mannitol treatment the blood sugar was elevated to levels 609 mg% to 840 mg% with glucosuria (++) and ketonuria (-). All three pataients died. We suggest that if there isfurther increase in blood sugar level, no improvement of consciousness or even further worsening following mannitol dehydration therapy for reducing the intracranial pressure, the possibility of NHHC should be considered. The possible mechanism Of NHHC caused by mannitol was discussed.

本文报告3例脑血管病病人,由于甘露醇脱水治疗所引起高渗性非酮症性糖尿病昏迷。均无糖尿病病史。入院后经腰穿和脑CT确诊。静脉输入甘露醇期间,血糖升高达609~840mg%,尿糖(++),尿酮体(-)。最终均死亡。我们认为在颅内病变脱水治疗时,若有血糖上升或病人意识障碍并未因脱水而好转,且逐渐加重时,应警惕本病可能。本文还讨论了甘露醇诱发本病可能机理。

 
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