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human protective
相关语句
  机体保护
     B - cell clonal dominance may be the cause of human protective immunodeficiency after HCV infection.
     B细胞克隆优势化可能是HCV感染后机体保护免疫缺陷的原因之一。
短句来源
  “human protective”译为未确定词的双语例句
     Study on the cause of human protective immunodeficiency after HCV infection
     HCV感染后机体保护性免疫缺陷原因探讨
短句来源
     B-cell clonal dominance may be the cause of human protective immunodeficiency after HCV infection.
     B细胞克隆优势化可能是HCV感染后机体保护性免疫缺陷的原因之一。
短句来源
     Results Human protective tests showed that 10% and 20% of compound 23 displayed the repellent activity of 2.5~3 h and 4.5~5 h to Aedes albopictus respectively, while 10% and 20% of compound 31 held repellent activity as long as 4~5 h and 5~6 h to Anopheles sinensis respectively.
     结果10%和20%的23号化合物对白纹伊蚊有2.5~3h和4.5~5h的驱避活性,10%和20%的31号化合物对中华按蚊则有4~5h和5~6h的驱避活性。
短句来源
     Results: Human protective tests showed that 10% and 20% of compound 23 displayed the repellent activity of 2.5 to 3 h and 4.5 to 5 h to Aedes albopictus,respectively. 10% and 20% of compound 31 held repellent activities as long as 4 to 5 h and 5 to 6 h to Anopheles sinensis,respectively.
     结果:10.0%和20.0%的23号化合物对白纹伊蚊有2.5~3 h和4.5~5 h的驱避活性,10.0%和20.0%的31号化合物对中华按蚊则有4~5 h和5~6 h的驱避活性。
短句来源
     Conclusion Human protective test was needed to find repellents from those eight compounds under the national standard.
     结论 进一步的人体保护试验有希望筛选出符合国家标准、具有开发前景的萜类驱避剂。
短句来源
  相似匹配句对
     human;
     human ;
短句来源
     Protective Effect of Ulinastatin on Human Body
     乌司他丁对机体的保护作用
短句来源
     Protective effect of selenium on human erythrocyte rheology
     硒对心肌缺血/再灌注期间冠状静脉窦血中红细胞流变性的保护作用
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     Human is Liberty
     人是自由
短句来源
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  human protective
Human protective protein/cathepsin A is a human CPY homolog protein, which is a dimeric serine carboxypeptidase.
      
Epidemiologic studies suggest presence of human protective immunity against Mtb.
      


AIM To study the cause of protective immunodeficiency of patients with hepatitis C.METHODS An antigen capture ELISA in which HCV synthetic peptides SP42, CP10 and CP9 were derived from HCV NS4 and core gene region, respectively, was used as solid-phase antigens to detect the differences in light chain isotype expression of anti-HCV antibodies. RESULTS Antibodies in 116 sera of HCV-infected patients against HCV SP42, CP10 and CP9 were characterized by a skewed light chain isotype expression. One hundred and thirteen...

AIM To study the cause of protective immunodeficiency of patients with hepatitis C.METHODS An antigen capture ELISA in which HCV synthetic peptides SP42, CP10 and CP9 were derived from HCV NS4 and core gene region, respectively, was used as solid-phase antigens to detect the differences in light chain isotype expression of anti-HCV antibodies. RESULTS Antibodies in 116 sera of HCV-infected patients against HCV SP42, CP10 and CP9 were characterized by a skewed light chain isotype expression. One hundred and thirteen out of 116 sera samples of HCV infection (97.41%) showed at least one of the three anti-HCV antibodies skewed from the normal ratio of light chain isotype kappa/lambda. The kappa/lambda ratios of anti-HCV in patients with hepatitis C were found to be unique and constant in all for one year follow-up. CONCLUSION Anti-HCV response was stable and clonally restricted in HCV infection. B-cell clonal dominance may be the cause of human protective immunodeficiency after HCV infection.

目的探讨丙型肝炎患者保护性免疫缺陷的原因。方法根据HCVC区和NS4区基因序列设计并人工合成3条合成肽,采用抗原捕捉酶联免疫吸附试验检测HCV感染者血清中抗-HCV IgG抗体轻链κ/λ比值,同时以正常人血清做对照。结果抗-HCV SP42,CP10和CP9抗体轻链的表达呈明显的偏斜;116例抗-HCV阳性者中113例(97.41%),抗-HCV IgG抗体轻链κ/λ比值偏高,所有病例追踪观察1a(其中11例抗-HCV阳性者随访2a);30例患者接受α-干扰素治疗,发现抗-HCV抗体κ/λ比值恒定不变。结论 HCV感染者抗-HCV抗体的产生不均匀性并呈稳定的克隆性。B细胞克隆优势化可能是HCV感染后机体保护性免疫缺陷的原因之一。

Objective To study the cause of protective immunodeficiency of patients with hepatitis C. Methods An antigen capture ELISA in which HCV synthetic peptides SP42, CP10 and CP9 derived from HCV NS4 and core gene region, respectively, were used as solid - phase antigens was developed to detect the differences in light chain isotype expression of anti - HCV antibodies. Results antibodies in 84 sera of HCV - infected patients against HCV SP42, CP10 and CP9 were characterized by a skewed light chain isotype expression....

Objective To study the cause of protective immunodeficiency of patients with hepatitis C. Methods An antigen capture ELISA in which HCV synthetic peptides SP42, CP10 and CP9 derived from HCV NS4 and core gene region, respectively, were used as solid - phase antigens was developed to detect the differences in light chain isotype expression of anti - HCV antibodies. Results antibodies in 84 sera of HCV - infected patients against HCV SP42, CP10 and CP9 were characterized by a skewed light chain isotype expression. Eighty - two out of 84 sera of HCV infection (97.62 %) showed at least one of the three anti - HCV antibodies skewed from the normal ratio of light chain isotype kappa/lambda.The kappa/lambda ratios of anti - HCV in patients with hepatitis C were found to be unique and constant in all for one year follow - up, 11 of them for two years follow - up. Conclusion Anti - HCV response was stable and clonally restricted in HCV infection.B - cell clonal dominance may be the cause of human protective immunodeficiency after HCV infection.

目的探讨丙型肝炎患者保护性免疫缺陷的原因。方法根据HCV C区和NS4基因序列设计并人工合成3条合成肽,采用抗原捕捉酶联免疫吸附试验检测HCV感染者血清中抗.HCV IgG抗体轻链κ/λ比值。结果 抗-HCVSP42,CP10和CP9抗体轻链的表达呈明显的偏斜;84例抗-HCV阳性者中82例(97.62%)抗-HCV IgG抗体轻链κ/λ比值恒定不变。结论HCV感染者抗-HCV抗体的产生不均匀性并呈稳定的克隆性。B细胞克隆优势化可能是HCV感染后机体保护免疫缺陷的原因之一。

Objective To study the cause of protective immunodeficiency of patients with hepatitis C. Methods An antigen capture ELISA in which HCV synthetic peptides SP42, CP10 and CP9 derived from HCV NS4 and core gene region, respectively, were used as solid-phase antigens was used to detect the differences in light chain isotype expression of anti-HCV antibodies. Results Antibodies in 84 sera of HCV-infected patients against HCV SP42, CP10 and CP9 were characterized by a skewed light chain isotype expression. Eighty-two...

Objective To study the cause of protective immunodeficiency of patients with hepatitis C. Methods An antigen capture ELISA in which HCV synthetic peptides SP42, CP10 and CP9 derived from HCV NS4 and core gene region, respectively, were used as solid-phase antigens was used to detect the differences in light chain isotype expression of anti-HCV antibodies. Results Antibodies in 84 sera of HCV-infected patients against HCV SP42, CP10 and CP9 were characterized by a skewed light chain isotype expression. Eighty-two out of 84 sera of HCV infection (97 62%) showed at least one of the three anti-HCV antibodies skewed from the normal ratio of light chain isotype kappa/lambda. The kappa/lambda ratios of anti-HCV antibodies in all patients with hepatitis C were found to be unique and constant during one year follow-up, and 11 of them received two years follow-up. Conclusions Anti-HCV response was stable and clonally restricted in HCV infection. B-cell clonal dominance may be the cause of human protective immunodeficiency after HCV infection.

目的 探讨丙型肝炎病毒 (HCV)感染者保护性免疫缺陷的原因。方法 根据HCVC区和NS4区基因序列设计并人工合成 3条合成肽 ,采用抗原捕捉酶联免疫吸附试验检测HCV感染者血清中抗 -HCVIgG抗体轻链κ/λ比值。结果 抗 -HCVSP42 ,CP10和CP9抗体轻链的表达呈明显的偏斜 ;84例抗 -HCV阳性者中 82例 ( 97 62 %)抗 -HCVIgG抗体轻链κ/λ比值偏离。所有病例追踪观察一年 (其中 11例抗 -HCV阳性者随访二年 ) ,发现抗 -HCV抗体κ/λ比值恒定不变。结论 HCV感染者抗 -HCV抗体的产生不均匀性并呈稳定的克隆性。B细胞克隆优势化可能是HCV感染后机体保护性免疫缺陷的原因之一。

 
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