Methods: Left ventricular systolic and diastolic function in 74 patients with essential hypertension and 94 normal controls were studied by echocardiography using routine examination and Doppler tissue imaging (DTI).
Objective: This subject applies Doppler technology to evaluate the systolic and diastolic function of medium - light degree COPD patient's and thus investigates the relevance of medium-light COPD and change of right ventricular function.
②Regional cardiac systolic and diastolic function:compared with pre-PTCA,Vs,Ve and Ve/Va were increased,EDT and EAT were decreased respectively in myocardial ischemia group 1 month and myocardial infarction group 3 months after PTCA.
Materials and Methods: We elected control group 15 cases(G0),anterior myocardial infarction group 29 cases (G1),inferior myocardial infarction 39 cases(G2). We assessed the parameters of global and regional right ventricular systolic and diastolic function in the three groups.
Using radionuclide ventriculography technology,we assessed the influence on global and regional ventricular systolic and diastolic function with different location of left ventricular myocardial infarction.
Objective To investigate the value of quantitative tissue velocity imaging(QTVI) in assessing the left ventricular (LV) regional systolic and diastolic function in patients with coronary artery disease(CAD).
Systolic and diastolic function were assessed through developed pressure (LVDP) and end diastolic pressure (LVEDP), respectively.
In the later stage at 6 weeks, clinical RHF was associated with abnormalities of the right heart systolic and diastolic function along with a decrease in antioxidant reserve.
ATP, total adenine nucleotides, and NAD were similar in the cold-preserved donor hearts and in right ventricular endomyocardial biopsies from the 10 individuals with normal systolic and diastolic function.
Right ventricular endomyocardial biopsies were obtained from 43 patients with dilated cardiomyopathy, 6 with restrictive cardiomyopathy, 10 with normal systolic and diastolic function, and from 24 cold preserved human donor hearts.
These findings provide an explanation for mechanisms that may be partly responsible for the improvement in LV systolic and diastolic function seen in dogs with heart failure after long-term CSD therapy.