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interfering treatment
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  干预治疗
     Characteristics of glycometabolism in 58 corpulent children with acanthosis nigricans and interfering treatment with metformin
     肥胖儿童伴黑棘皮病糖代谢特征及二甲双胍干预治疗58例
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     (2)Two groups FINS, PINS, TC, and TG significantly decreased (P<0.01) and the insulin-sensitivily index (IAI) increased (P<0.01) after 8 wk interfering treatment with metformin. The adverse reactions of two groups were mild.
     (2 )二甲双胍干预治疗 8wk ,2组FINS ,PINS ,总胆固醇 (TC) ,三酰甘油 (TG) ,均有非常明显下降 (P <0 .0 1) ,胰岛素敏感指数 (IAI)上升 (P <0 .0 1) ,2组不良反应轻微。
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     Conclusions From the LPS induced ALI rat models,it is demonstrated that NO and iNOS play important roles in the onset of ALI:dexamethasone and rhubarb interfering treatment can ameliorate,the lung injury and decrease the concentrations of NO and iNOS,showing that these 2 agents have protective effect on LPS induced ALI and their therapeutic mecharnism is possibly through inhibiting the activities of NO and iNOS. [
     结论 在LPS诱导的ALI大鼠动物模型中证明NO和iNOS在ALI的发病过程中起到较为关键性的作用,用地塞米松、大黄进行干预治疗,可使损伤减轻相应地也使NO、iNOS浓度下降,表明这2 种药物对LPS诱导的ALI具有保护作用,其机制可能是通过抑制NO和iNOS活性实现。
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     Objectives To explore the effect of large dose methylprednisolone(MP) on the change of endogenous basic fibroblast growth factor (bFGF) in nerve cells of rats after interfering treatment of spinal cord injury(SCI).
     [目的]应用大剂量甲基强的松龙(MP)对脊髓损伤进行干预治疗,探讨其对脊髓神经细胞中内源性碱性成纤维细胞生长因子(bFGF)变化的影响。
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     Changes of tissue-type plasminogen activator and its inhibitor in blood and urine in children with Henoch-Schinlein purpura nephritis and effects of interfering treatment
     过敏性紫癜肾儿童血尿纤溶酶原激活物及其抑制物的变化及干预治疗
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  “interfering treatment”译为未确定词的双语例句
     The Protein Expressive Tendency of Plasminogen Activators(PAs) and 1-type Plasminogen Inhibitor(PAI-1) Remnant Renal Tissues of 5/6 Nephrectomy Young Rats and the Effects of Interfering Treatment
     5/6肾切除幼年大鼠PAs/PAI-1蛋白表达趋势及干预研究
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     Levels of adipocytokines in patients with type 2 diabetes and interfering treatment of rosiglitazone
     2型糖尿病多种脂肪细胞因子水平及罗格列酮干预治疗的影响
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     The relativity between AT1,AT2 and P65/Rel-A expression, and the effects of interfering treatment were evaluated.
     10.31士0.81%、10.44士1.55%、 10.15士1.49%); ATZ第1、2、3周分别为
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     L. P. treatment.
     L. P.
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     On Musical Treatment
     谈音乐治疗
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     Levels of adipocytokines in patients with type 2 diabetes and interfering treatment of rosiglitazone
     2型糖尿病多种脂肪细胞因子水平及罗格列酮干预治疗的影响
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     Objective To discuss the causes that interfering patients with acoustic neuroma from early seeking treatment.
     目的探讨阻碍听神经瘤患者早期就诊的原因。
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Objective To approach mainly the actions of nitric oxide(NO)and inducible nitric oxide synthase (iNOS) on endotoxin (lipopolysaccharidePaO 2<6 7kPa,LPS)induced rat acute lung injury (ALI) and the affeciton of rhubarb on them.Methods LPS was injected into the subingual vein of male Wistar rats to prepare ALI animal models.The rats were divided into the 4 groups:LPS group,control group,rhubarb group and dexamethasone group.Macroscopic and histopathological examinations were performed and biological indexes...

Objective To approach mainly the actions of nitric oxide(NO)and inducible nitric oxide synthase (iNOS) on endotoxin (lipopolysaccharidePaO 2<6 7kPa,LPS)induced rat acute lung injury (ALI) and the affeciton of rhubarb on them.Methods LPS was injected into the subingual vein of male Wistar rats to prepare ALI animal models.The rats were divided into the 4 groups:LPS group,control group,rhubarb group and dexamethasone group.Macroscopic and histopathological examinations were performed and biological indexes were measured for the lung specimens.The indexes included lung wet weight/dry weight,the rats of neutrophils and protein content in the pulmonary alveolar lavage fluid,pulmonary vascular permeability and pulmonary alveolar permeability index.In the mean time,the activities of serum NO and lung tissue homogenate iNOS were measured.Results In the comparison of the lung histopathological examination between the LPS group and control group,the injury and cellular infiltration in the pulmonary stroma and alveoli were more prominent in the LPS group than in the control group.Lung wet weight/dry weight,rat of neutrophils,protein content,pulmonary alveolar permeability index,pulmonary vascular permeability were significantly increased ( P <0 01);NO and iNOS were also markedly elevated ( P <0 01).In the groups with dexamethasone and rhubarb intering treatment,the histopathological changes were singnificantly milder,and the above biological indexes of lung injury and the contents of NO and iNOS were correspondingly decreased ( P <0 05).Conclusions From the LPS induced ALI rat models,it is demonstrated that NO and iNOS play important roles in the onset of ALI:dexamethasone and rhubarb interfering treatment can ameliorate,the lung injury and decrease the concentrations of NO and iNOS,showing that these 2 agents have protective effect on LPS induced ALI and their therapeutic mecharnism is possibly through inhibiting the activities of NO and iNOS. [

目的 主要探讨一氧化氮(NO) 和诱导型一氧化氮合酶(iNOS)在内毒素(LPS) 诱导的大鼠急性肺损伤(ALI)的作用机制及大黄对其影响。方法 在雄性Wistar 大鼠利用舌下静脉注射LPS复制ALI动物模型,动物分为4 组:LPS组、对照组、大黄治疗组、地塞米松组。观察大体标本,组织病理以及生物学标志:肺湿/干重比、肺泡灌洗液中性粒细胞比、蛋白含量、肺血管通透性和肺泡通透性指数。同时测定血浆NO和肺组织匀浆iNOS活性。结果 LPS组与对照组相比,肺组织病理显示肺间质及肺泡明显损伤和细胞浸润。其生物学标志均显著升高(P<0-01),NO 和iNOS也显著升高( P<0-01) ,地塞米松和大黄干预治疗组则表现组织病理明显减轻。上述肺损伤生物标记物及NO、iNOS也相应下降( P< 0-05) 。结论 在LPS诱导的ALI大鼠动物模型中证明NO和iNOS在ALI的发病过程中起到较为关键性的作用,用地塞米松、大黄进行干预治疗,可使损伤减轻相应地也使NO、iNOS浓度下降,表明这2 种药物对LPS诱导的ALI具有保护作用,其机制可能是通过抑制NO和iNOS活性实现。

Objective: To explore the optimal vegimens for the eradication of helicobacter pylori(H.Pylori) Methods:56 patient with duodenal ulcer (Du) all were H.pylori positive joined trial and 50 completed cases were divided into 2 groups. Group A (n=27) were treated with triple therapy of ranitidine bismuth citrate (RBC) 350mg+amoxicillin 1000mg+furazolidone 100mg bid for 1 week .Group B (n=23) omeprazole 20mg+ clrarithromgcin 500mg+amoxicillin 1000mg bid for 1 week .Results of H.pylori eradication the thing of ulcer...

Objective: To explore the optimal vegimens for the eradication of helicobacter pylori(H.Pylori) Methods:56 patient with duodenal ulcer (Du) all were H.pylori positive joined trial and 50 completed cases were divided into 2 groups. Group A (n=27) were treated with triple therapy of ranitidine bismuth citrate (RBC) 350mg+amoxicillin 1000mg+furazolidone 100mg bid for 1 week .Group B (n=23) omeprazole 20mg+ clrarithromgcin 500mg+amoxicillin 1000mg bid for 1 week .Results of H.pylori eradication the thing of ulcer healing,clinical symptoms and side effects were observed and recorded . Results: The H.pylori eradication rates in groups A and B were 85.2%?82.5%. Ulcer healing rates were 81.5%?82.5%,percentage of disappearance of abdominal pain at third days were 70.4%?73.4%, and mean time were 4.23+2.5d?3.95+1.9d. The differences were not singificant statistically (P>0.05). Mild side effects noted in two groups. but they did not interfere treatment .The cost of anti-HP therapy every patient in group A?B were 102.2 and 355.9 (RMB.yuan). Conclusions:The short-term triple therapy of RBC?Amoxicillin ?Furazolidone are highly effective in active duodenal ulcer with H.pylori infection and the new regimens seems to have better tolance ,compliance and lower cost.

目的 :探索根除率较高、价廉、安全实用的HP根除方案。方法 :5 6例HP阳性的十二指肠球部溃疡患者进入试验 ,可评价病例 5 0例 ,随机分组 ,A组 2 7例 :枸椽酸铋雷尼替丁 35 0mg +阿莫西林 10 0 0mg +呋喃唑酮 10 0mg每天 2次 ,疗程 7d。B组 2 3例 :奥美拉唑 2 0mg +克拉霉素 5 0 0mg +阿莫西林 10 0 0mg每天 2次 ,疗程 7d。观察记录HP根除情况、溃疡愈合情况、临床症状改善情况及不良反应。结果 :A、B二组HP根除率分别为 85 .2 %、82 .5 % ,溃疡愈合率分别为 81.5 %、82 .5 % ,三日腹痛缓解率分别为 70 .4 %、73.4 % ,平均腹痛缓解时间分别为 4 .2 3± 2 .5d、3.95± 1.9d ,各组间差异无显著性 ,P >0 .0 5 ,二组均有轻微不良反应 ,但不影响治疗。A、B二组每例抗HP所需费用分别为 10 2 .2元、35 5 .9元。结论 :以枸椽酸铋雷尼替丁、阿莫西林、呋喃唑酮三联一周方案是一种新的、根除率较高、价廉、安全实用的HP根除方案。

Objectives To explore the effect of large dose methylprednisolone(MP) on the change of endogenous basic fibroblast growth factor (bFGF) in nerve cells of rats after interfering treatment of spinal cord injury(SCI). Methods) Forty Wister rats were randomly divided into normal (n=8), control (n=16) and experiment group (n=16). In the normal group, all rats were not operated upon before sacrifice, but in both control and experiment groups, all rats were subjected to a 25gx 10 cm weight-drop SCI (Allen's method)...

Objectives To explore the effect of large dose methylprednisolone(MP) on the change of endogenous basic fibroblast growth factor (bFGF) in nerve cells of rats after interfering treatment of spinal cord injury(SCI). Methods) Forty Wister rats were randomly divided into normal (n=8), control (n=16) and experiment group (n=16). In the normal group, all rats were not operated upon before sacrifice, but in both control and experiment groups, all rats were subjected to a 25gx 10 cm weight-drop SCI (Allen's method) at the T8 level respectively. For the experiment group, the rats were treated by intravenous injection of MP 30 mg/kg through the caudal vein in the first hour and 5.4 mg/(kg-h) infusion for 23 hours. The equal volume of normal saline solution was given to the rats of control group for the same duration.. Three groups rats were sacrificed at 2 d, 5d, 12d. 28d after injury ,and spinal cord tissue in the injured area was separated and made into sections. The bFGF protein levels were estimated by immunohistochemical method. Results In normal group, bFGF located within nuclei of astrocytes in grey matter and in cytoplasm of some neurons, its expression was much increased after SCI in rats. Intravenous injection of large dose methylprednisolone could prominently promote bFGF expression at all intervals. Conclusion Our findings suggest that bFGF may play an important role in selfrepairing and regeneration after SCI, and large dose methylprednisolone can improve greatly neurotrophic restoration of injured spinal nerve cells by enhancing the expression of endogenous bFGF .

[目的]应用大剂量甲基强的松龙(MP)对脊髓损伤进行干预治疗,探讨其对脊髓神经细胞中内源性碱性成纤维细胞生长因子(bFGF)变化的影响。[方法]40只wister大鼠分为三组:未损伤未给药组(A),损伤后注射生理盐水组(B),损伤后注射MP组(C)。利用Allen氏重物坠击法(weight drop,WD)技术,以2.5g×10cm致伤力造成T8脊髓损伤模型,C组分别于术后即刻经尾静脉按首次剂量30mg/kg注射MP,以后按5.4 mg/(kg·h),共23h.分4次静推;B组在同时间点注入等量生理盐水。术后于2d、5d、12d、28d取A、B、C各组损伤部位脊髓组织作bFGF的免疫组化检测。[结果]在各个时间点上,大剂量MP可以显著地提高bFGF的表达。[结论]大剂量MP可能通过提高损伤脊髓组织中bFGF的表达,来发挥受损脊髓神经细胞的营养作用。

 
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