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cardiac contractile proteins
相关语句
  心肌收缩蛋白
     The Advances of Research for Cardiac Contractile Proteins Structure and Function
     心肌收缩蛋白的结构与功能研究进展
短句来源
     Cardiac contractile proteins are the material basis of cardiac contractility.
     心肌收缩蛋白是心肌产生收缩的物质基础。
短句来源
     This paper introduces theStructures of two major cardiac contractile proteins: myosin and actin, especially the details of the two proteins'structure relevant to their function. At the same time, it describes the myosin and actin proteins' changes in normal and abnormal hearts so as to enrich our mind with knowlege of cardiac hypertrophy and heart failure at themolecular level.
     本文介绍了两种主要心肌收缩蛋白:肌凝蛋白(myosin)和肌动蛋白(actin)的结构,尤为详细地讨论了与其功能相关的部分,并就正常与异常心脏中两者的改变作一阐述,以期深化心肌肥大和心力衰竭分子水平上的变化认识。
短句来源
  相似匹配句对
     The Advances of Research for Cardiac Contractile Proteins Structure and Function
     心肌收缩蛋白的结构与功能研究进展
短句来源
     Cardiac contractile proteins are the material basis of cardiac contractility.
     心肌收缩蛋白是心肌产生收缩的物质基础。
短句来源
     proteins.
     另外3个蛋白没有得到鉴定,可能是未知蛋白。
短句来源
     ALTERATION OF CARDIAC G PROTEINS DURING SEPSIS
     脓毒血症时大鼠心肌G蛋白的变化
短句来源
     The Contractile Proteins and RegulatoryMechanism of the Crustacean Striated Muscle.
     甲壳动物横纹肌的收缩蛋白质与调节机制
短句来源
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  cardiac contractile proteins
To identify direct actions of E-1020 on cardiac contractile proteins, effects of E-1020 on myofibrillar Ca2+ dependent MgATPase and force generation in chemically skinned fiber bundles were measured.
      
Concerning cardiac contractile proteins, antigenicity and myocarditogenicity were discussed.
      
Cardiac contractile proteins and autoimmune myocarditis
      
A new in vitro motility assay technique to evaluate calcium sensitivity of the cardiac contractile proteins
      


Cardiac contractile proteins are the material basis of cardiac contractility. This paper introduces theStructures of two major cardiac contractile proteins: myosin and actin, especially the details of the two proteins'structure relevant to their function. At the same time, it describes the myosin and actin proteins' changes in normal and abnormal hearts so as to enrich our mind with knowlege of cardiac hypertrophy and heart failure at themolecular level.

心肌收缩蛋白是心肌产生收缩的物质基础。本文介绍了两种主要心肌收缩蛋白:肌凝蛋白(myosin)和肌动蛋白(actin)的结构,尤为详细地讨论了与其功能相关的部分,并就正常与异常心脏中两者的改变作一阐述,以期深化心肌肥大和心力衰竭分子水平上的变化认识。

Sports cardiac hypertrophy is a response of cardiac myocytes to various exercise induced stimuli such as mechanical overload, humoral factors which lead to genetic expression changes in cardiac contractile protein genes. Apart from increase in cell size and alteration in cell function, changes of gene expression are its essential mechanism and cellular signal conduction is the important link of external stimuli to nucleus reaction. The structure and function of cardiac myocytes are also...

Sports cardiac hypertrophy is a response of cardiac myocytes to various exercise induced stimuli such as mechanical overload, humoral factors which lead to genetic expression changes in cardiac contractile protein genes. Apart from increase in cell size and alteration in cell function, changes of gene expression are its essential mechanism and cellular signal conduction is the important link of external stimuli to nucleus reaction. The structure and function of cardiac myocytes are also closely related to extracellular matrix. At present pathways of cellular signal conduction and extracellular matrix in cardiac hypertrophy are intensively studied and much progress has been made. But studies are limited on the mechanism of sports cardiac hypertrophy. Advances in studies on signal conduction and extracellular matrix of sports cardiac hypertrophy are reviewed.

运动性心肌肥大是心肌细胞对运动诱发的各种刺激因素如机械负荷、体液因子以及由这些因素导致的心肌收缩蛋白基因表达改变的功能反应。它除了表现为细胞形态及功能改变外 ,基因表达变化是其本质特征 ,而细胞的信号传导则是将胞外刺激转变为核内反应的重要连接。心肌细胞本身的结构或功能改变与心肌间质组织尤其是心肌胶原有密切关系。目前 ,心肌肥大的信号传导通路及细胞外基质的作用已有深入的研究并取得重要进展 ,但在运动性心肌肥大发生中的机制仍了解有限 ,本文就运动性心肌肥大的研究进展及展望进行了综述

MCI-154,a calcium sensitizer,was found to have obviously beneficial effect for endo-toxic and hemorrhagic shock, especially for the decompensatory period of shock. The superiority of MCI-154 to traditional positive inotropic agents is that it did not increase the cellular calcium overload of shocked animal. MCI-154 improved the cardiac function of shocked animal mainly by increasing the sensitivity of cardiac contractile protein to calcium. However, the effects of MCI-154 increasing [Ca2+]i and cAMP...

MCI-154,a calcium sensitizer,was found to have obviously beneficial effect for endo-toxic and hemorrhagic shock, especially for the decompensatory period of shock. The superiority of MCI-154 to traditional positive inotropic agents is that it did not increase the cellular calcium overload of shocked animal. MCI-154 improved the cardiac function of shocked animal mainly by increasing the sensitivity of cardiac contractile protein to calcium. However, the effects of MCI-154 increasing [Ca2+]i and cAMP in myocardial cell was much weaker than that of milrinone. MCI-154 could improve the constriction and dilation function of blood vessels by decreasing cytoplasmic free calcium concentration of vascular smooth muscle cell (VSMC) ,and restoring the activity of Calmodulin and the ex- pression of c protein kinase C. Meanwhile MCI-154 could dilate blood vessel by enhancing the expression of calponin-2 in VSMC.

钙增敏剂MCI-154有明显的拮抗失血性休克及内毒素休克的作用,特别是对休克失代偿期有良好的疗效。与传统的加强心肌收缩的药物不同,本品不会明显增加休克动物心肌细胞内的钙超载,而主要通过增加心肌收缩蛋白对Ca2+的敏感性而改善休克动物的心功能,其增加心肌细胞内Ca2+水平及cAMP含量的作用明显比米力农弱。本品降低休克动物血管平滑肌细胞内游离Ca2+浓度,恢复钙调蛋白活性及c蛋白激酶C的表达,以及相对增加Calponin-α的表达,从而改善休克舒张功能。

 
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