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hypoxic-ischemic     
相关语句
  缺氧缺血性
     Expression of Neuroglobin Gene in Hypoxic-ischemic Brain Injury of Newborn Rat
     脑红蛋白基因在新生大鼠缺氧缺血性脑损伤脑中的表达
短句来源
     Objective To observe the expression of growth-associated protein-43(GAP-43) in the hippocampus of neonatal rats with hypoxic-ischemic brain damage (HIBD).
     目的研究7日龄新生大鼠缺氧缺血性脑损伤(hypoxic-ischemic brain damage,HIBD)后海马神经组织生长相关蛋白(growth-associated protein,GAP-43)及其mRNA的表达变化。
短句来源
     Expression of Caspase-1 mRNA in the newborn rats with hypoxic-ischemic brain damage
     新生大鼠缺氧缺血性脑损伤Caspase-1mRNA的表达变化
短句来源
     Study on the Expression and significance of mRNA for gro、MIP1-βin Cerebral Tissue of Newborn Rat With Hypoxic-ischemic Brain Damage
     新生鼠缺氧缺血性脑损伤脑组织中趋化因子gro、MIP1-βmRNA的表达及意义
短句来源
     ER-α,IGF-1R Expressions and Co-expressions in Newborn Rats with Experimental Hypoxic-Ischemic Brain Damage
     新生鼠实验性缺氧缺血性脑损伤ER-α、IGF-1R的表达及双重表达
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  缺氧缺血
     Compared with the hypoxic-ischemic group, the SOD activity and GSH level were significantly high er[(2.73±0.21)mkat and(83.47±6.38) mg/g](P< 0.001,P< 0.005),and the MDA conten t was significantly lower in the treatment group[(6.07±0.68) pmol/g](P<0.001).
     而治疗组脑组织SOD、谷胱甘肽水平分别为(2.73±0.21)mkat、(83.47±6.38)mg/g,明显高于缺氧缺血组(P<0.001;P<0.005); 丙二醛含量为(6.07±0.68)pmol/g,较缺氧缺血组显著下降(P<0.001)。
短句来源
     The number of positive cells in 100 mg/kg sodium gamma-hydroxybutyrate group was significantly lower than that of hypoxic-ischemic injury group at 3,24,72 and 168 hours(P < 0.01),and low-er than that in 50 mg/kg sodium gamma-hydroxybutyrate group at 168 hours (t=4.45,P < 0.01), and there was no difference in other time points.
     羟丁酸钠100mg/kg组在缺氧缺血3,24,72,168h时明显低于缺氧缺血组(P<0.01),在缺氧缺血168h时明显低于羟丁酸钠50mg/kg组(t=4.45,P<0.01),其余各时间点无明显差异。
短句来源
     Positive cells of 50 mg/kg sodium gamma-hydroxybutyrate group was significantly lower than that of hypoxic-ischemic injury group at 3, 24 and 72 hours after hypoxia-ischemia(P < 0.05,0.01),and there was no signif-icant difference at 168 hours.
     羟丁酸钠50mg/kg组在缺氧缺血3,24,72h时明显低于缺氧缺血组(P<0.05,0.01),168h时与缺氧缺血组无明显差异。
短句来源
     body weight of 50 and 100 mg/kg sodium gamma-hydroxybutyrate groups was significantly higher than that of hypoxic-ischemic injury group at 72 and 168 hours after hypoxic-ischemia(P < 0.01), which was higher in 100 mg/kg sodium gamma-hydroxybutyrate group as compared with that in 50 mg/kg sodium gamma-hydroxybutyrate group.
     羟丁酸钠50,100mg/kg组新生大鼠在72,168h时明显高于缺氧缺血组(P<0.01),且羟丁酸钠100mg/kg组明显高于羟丁酸钠50mg/kg组(P<0.01)。
短句来源
     Effect of Calpain inhibitor-3 on Caspase-3 expression and neuronal apoptosis in hippocampal CA1 region of neonatal rats after hypoxic-ischemic brain damage
     Calpain抑制剂-3对新生大鼠脑缺氧缺血后海马CA1区Caspase-3表达和神经元凋亡的影响
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  缺血缺氧性
     ObjectiveTo investigate the migration and differentiation of human neural stem cells (NSC) in neonatal rat hippocampus following hypoxic-ischemic brain injury (HIBI) .
     将培养的人神经干细胞(neural stem cells,NSC)移植至新生鼠缺血缺氧性脑损伤(hypoxic-ischemic brain injure,HIBI)的海马区,观察植入细胞的迁移及分化。
短句来源
     Objective: to observe, in different period, the morphological changes, the production of HSP70 and the formation of cell death of brain cells of neonate rats after undergoing hypoxic-ischemic brain damage(HIBD), to probe into the correlation between the post-HIBD HSP70 production and apoptosis, in order to put forward experimental and theoredical foundation for HIBD.
     目的:利用新生大鼠缺血缺氧性脑损伤(hypoxic-ischemic brain damage HIBD)动物模型,观察缺血缺氧性脑损伤后不同时间脑细胞形态变化、HSP70的合成及细胞死亡形式,探讨HIBD后HSP70的合成与细胞凋亡的关系,为缺血缺氧性脑病提供实验和理论依据。
短句来源
     Objective: To detect the serum immunoglobin (Ig), complement C3, T lymphocyte subsets, interleukin-2 (IL-2) and serum soluble IL-2 receptor (SIL-2R) of peripheral blood in 38 cases with hypoxic-ischemic encephalopathy.
     目的 :检测38例缺血缺氧性脑病患儿外周血免疫球蛋白、C3、T淋巴细胞亚群 ,IL -2及SIL -2R。
短句来源
     This study was designed to explore the effective strategies for neonatal hypoxic-ischemic encephalopathy (HIE).
     探索缺血缺氧性脑病(hypoxic-ischemic encephalopathy,HIE)的治疗方法。
短句来源
     (2) Lactate level of cord blood of infants with hypoxic-ischemic encephalopathy( 9.24 ?1.63) was significantly higher than those without (5.43 ?1.35) .
     (2)窒息组中,发生缺血缺氧性脑病的新生儿脐动脉血乳酸水平增高,发生者(9.24±1.63)与未发生者(5.43±1.35)之间差异有显著性。
短句来源
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  低氧缺血性
     A DETECTION OF Th_2 LYMPHOCYTE FUNCTION IN NEONATES WITH HYPOXIC-ISCHEMIC ENCEPHALOPATHY
     新生儿低氧缺血性脑病Th_2细胞功能的检测
短句来源
     Opioid mechanism in the treatment of hypoxic-ischemic brain injury by intracerebral transplantation of genetically modified myoblasts producing brain-derived neurotrophic factor in neonatal rats
     脑源性神经营养因子载体细胞治疗新生大鼠低氧缺血性脑损伤的阿片机制
短句来源
     SHORT-TERM EFFECTS OF HIGH-DOSE INTRAVENOUS IMMUNOGLOBULIN THERAPY IN NEONATES WITH HYPOXIC-ISCHEMIC ENCEPHALOPATHY
     静脉注射大剂量免疫球蛋白治疗新生儿低氧缺血性脑病的近期效果
短句来源
     ObjectiveTo study the clinical effects and possible immunological mechanism of high-dose intravenous immunoglobulin(HIVIG) in neonates with hypoxic-ischemic encephalopathy(HIE).
     目的研究静脉注射大剂量免疫球蛋白(HIVIG)治疗新生儿低氧缺血性脑病(HIE)的临床疗效和免疫学机制。
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      hypoxic-ischemic
    We have summarized the results of psychophysiological studies in schoolchildren aged 10-12 years with cerebral functional disturbances of hypoxic-ischemic or traumatic origin with learning difficulties.
          
    Pretreatment with allopurinol in cardiac hypoxic-ischemic reperfusion injury in newborn lambs exerts its beneficial effect throu
          
    Hypoxic-ischemic encephalopathy (HIE) in neonates is often difficult to diagnose in "real time" at the bedside because of the variety of disorders that can cause neonatal seizures and other nonspecific signs of encephalopathy.
          
    Treatment of hypoxic-ischemic encephalopathy in newborns
          
    Hypoxic-ischemic (HI) brain injury is the most common cause of encephalopathy and seizures in term newborn infants.
          
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    Severe neonatal acidosis (pH 7 or less) occurred in 103 cases (1.9%)among 6,509 cases admitted from 1978 to 1985. Of these cases, 50 were term babies, 48 preterm, 3 post-term and 2 small-for-date. There were 20 primary diseases causing severe neonatal acidosis, They were respiratory tract diseases, including meconium aspiration, pneumonia. respiratory distress syndrome and pulmonary dysplasia (53 cases, 51.5%); septicemia (18 cases, 17.5%); gastrointestinal disorders, including diarrhea, necrotizing enterocolitis,...

    Severe neonatal acidosis (pH 7 or less) occurred in 103 cases (1.9%)among 6,509 cases admitted from 1978 to 1985. Of these cases, 50 were term babies, 48 preterm, 3 post-term and 2 small-for-date. There were 20 primary diseases causing severe neonatal acidosis, They were respiratory tract diseases, including meconium aspiration, pneumonia. respiratory distress syndrome and pulmonary dysplasia (53 cases, 51.5%); septicemia (18 cases, 17.5%); gastrointestinal disorders, including diarrhea, necrotizing enterocolitis, perforation of stomach and intestinal obstruction (14 cases. 13.5%) and others as hypoxic ischemic encephalopathy and/or intracranial hemorrhage, congenital cynotic heart disease, sclerema neonatorium and late acidosis in pretcrm babies (18 cases, 17.5%). The major symptoms were respiratory distress (90.3%), apnca (48.5%), cynosis (86.4%), grey face (78.6%) and hypothermia (77.7%). The mortality was high (73.8%), especially in those babies with respiratory tract diseases (84.9%) or in babies less than 7 days of age (87.5%). 16 of the lethal cases were complicated with massive pulmonary hemorrhage. The prognosis was grave for both term and preterm babies.

    1979~1985年住院新生儿患者中血pH≤7.0有103例,占同期住院新生儿1.9%。导致重度酸中毒的原发疾病有20种,以呼吸系统疾病占首位(51.5%),败血症第二位(17.5%),消化系疾病第三位(13.6%)。治愈好转率仅24.3%,呼吸系统疾病,败血症,缺血缺氧性脑病,颅内出血,先天性青紫型心脏病预后差。而消化系统疾病预后较好,早产儿晚期酸中毒预后好。生后一周内发病率与病死率均高,血PCO_2升高时预后亦差。对新生儿重度酸中毒的防治问题进行了讨论。

    Ultrasonographic scans were performed in 115 newborn babies with Apgar score≤7 at 1 minute after birth. The results were normal in 48 cases (41.7%); hypoxic-ischemic encephalopathy (HIE) in 36 (31.3%); intracranial hemorrhage (ICH) in 26 (22.6%) and ventriculomegaly in 5 (4.4%). The normal rate of ultrasonographic scans was 82.9% in those with low Apgar score but without clinical symptoms and signs. However the abnormal rate was 81.1% in those having clinical symptoms and signs, either irritable and hyperalert...

    Ultrasonographic scans were performed in 115 newborn babies with Apgar score≤7 at 1 minute after birth. The results were normal in 48 cases (41.7%); hypoxic-ischemic encephalopathy (HIE) in 36 (31.3%); intracranial hemorrhage (ICH) in 26 (22.6%) and ventriculomegaly in 5 (4.4%). The normal rate of ultrasonographic scans was 82.9% in those with low Apgar score but without clinical symptoms and signs. However the abnormal rate was 81.1% in those having clinical symptoms and signs, either irritable and hyperalert appearance or apathetic and hypotonic state. The ventricles were often enlarged following HIE and ICH. Among 26 cases, 14 had mild to moderate enlargement while 3 were progressively dilated. The babies with blood pH<7.25 and/or PCO_2>6.7 kPa were more common in ICH group. Therefore it is important to correct the acid-base imbalance, especially in respiratory acidosis.

    对115例出生时Apgar评分≤7分的新生儿进行B型超声检查。结果有48例超声检查正常(41.7%),缺氧缺血性脑病36例(31.3%),颅内出血26例(22.6%)。脑室扩大5例(4.4%)。Apgar评分低不伴有神经系统症状、体征者超声检查正常率为82.9%,而伴神经系统症状、体征者81.9%患儿超声检查发现异常。超声随访发现缺氧缺血性脑病、颅内出血者脑室扩大者多:轻到中度脑室扩大53.8%(14/26),进行性脑室扩大11.5%(3/16)。对72例临床及实验室资料回顾性分析发现,pH<7.25,PCO_2>6.7kPa,颅内出血组明显高于正常组,因此在抢救窒息新生儿时,加强呼吸管理及维持内环境稳定很重要。

    The animal model of postnatal cerebral hypoxia-ischemia was prepared in 7 days old rats by permanent ligation of right common carotid artery combined with a temporary systemic hypoxia at 37℃. The early effects of hypoxia-ischemia on the striatal cholinergic system were investigated 24 h after lesion by ACh RIA-determination and AChE-histochemical image analysis. The striatal ACh content and average optical density of the AChE-positive cell bodies declined by 28 % and 12% respectively in the hypoxic-ischemic...

    The animal model of postnatal cerebral hypoxia-ischemia was prepared in 7 days old rats by permanent ligation of right common carotid artery combined with a temporary systemic hypoxia at 37℃. The early effects of hypoxia-ischemia on the striatal cholinergic system were investigated 24 h after lesion by ACh RIA-determination and AChE-histochemical image analysis. The striatal ACh content and average optical density of the AChE-positive cell bodies declined by 28 % and 12% respectively in the hypoxic-ischemic group as compared with the normal control group (p<0.01, p<0.05). There was no difference in the values between the ligated and non-ligated sides in the hypoxic-ischemic group. The striatal ACh content in the animals of the hypoxic group, which was prepared by temporary exposure to hypoxia at 37℃ with the carotid arteries intact, was also found to be reduced to the same extent as in the hypoxic-ischemic group. The density of AChE-positive cell profiles in the caudate-putamen remained unchanged on both sides in the hypoxic-ischemic group. The results indicate that hypoxia-ischemia, at its early phase, brings about cell damage rather than cell death and, as a result, leads to the weakening of the neuron markers, ACh and AChE in this case of the striatal cholinergic system of the young rat. It is suggested that developing cholinergic system is susceptible to hypoxia-ischemia, probably with anoxia as the primary pathogeny.

    采用7日龄大鼠右侧颈总动脉结扎合并高温、低氧环境制作新生动物脑低氧缺血模型,观察了脑低氧缺血对新生动物纹状体胆碱能系统的影响。乙酰胆碱(ACh)放射免疫测定结果表明,低氧缺血损伤后24h,两侧纹状体ACh含量均比正常对照组明显下降。乙酰胆碱酯酶(AChE)组织化学图象定量提示,脑低氧缺血后24h,纹状体内拟胆碱能神经元数量未见减少,而胞体内AChE染色强度略有下降。胆碱能递质和该标志酶在新生鼠脑低氧缺血早期的一致改变,证明发育中纹状体胆碱能系统对低氧缺血敏感。鉴于动物不结扎动脉仅作低氧处理者双侧纹状体ACh含量出现与低氧缺血组相同的改变,故提示缺氧可能是造成胆碱能系统早期损伤的直接原因。

     
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