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nimodipine treated
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  尼莫地平
     In nimodipine treated group, at 30 minutes before cerebral ischemia, 0.2 g/L nimodipine (2 mg/kg) was injected intraperitoneally.
     尼莫地平干预组:脑缺血前30min,每只大鼠腹腔注射0.2g/L的尼莫地平2mg/kg。
短句来源
     Methods:40 SD rats were randomly divided into control group,injury group,cemetidine treated group and nimodipine treated group,GMBF,pH of gastric juice and mucosal ulcer index were measured on the 72th hour after head injury.
     方法:将40只SD大鼠随机分为对照组、创伤组、西米替丁防治组及尼莫地平防治组,致伤后72h观察各组大鼠GMBF、胃液pH及胃粘膜损伤指数。
短句来源
     Results:In comparison with injury group,both the GMBF and the pH of gastric juice raised significantly but the mucosal ulcer index decreased obviously(all P<0.01) in nimodipine treated group.
     结果:与创上组相比,尼莫地平组GMBF及胃液pH明显升高(P<0.01),胃粘膜损伤指数明显减少(P<0.01)。
短句来源
     Compare with the 3 days treated period,although it had declined in some extent that expression of BDNFmRNA content and positive neurons amount in 5 days NRg1 treated period,their expression levels was still better than that in 1 day or 3 days model group and the 1 day positive control group,which was similar to that of 1 day NRg1 treated period and 3 days nimodipine treated period.
     5 d后,虽然BDNFmRNA的含量及阳性神经元的数量有一定程度的下降,但仍与三七皂苷Rg1组1 d时的表达水平相当,且三七皂苷Rg1组5 d时的表达水平仍强于模型组1 d和3 d时表达的水平及尼莫地平组1 d时的水平。
短句来源
     Nimodipine (1 mg/kg) was injected intraperitoneally following brain ischemia, the calcium content in hippocampus had no significant difference between Nimodipine treated group and ischemic group.
     缺血-尼莫地平组的大鼠在夹闭颈总动脉造成鼠脑暂时性缺血的同时於腹腔内注射尼莫地平。 比较对照组及暂时性缺血再灌注72小时后海马组织钙含量,用原子吸收分光光度计测定发现缺血组海马组织钙含量增高,但缺血-尼莫地平组海马组织钙含量与缺血组差异无显著性。
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  “nimodipine treated”译为未确定词的双语例句
     75 rats were randomly assigned to control group,cerebral hemorrhage group and nimodipine treated group. After the injection,the rats were sacrificed at 6 h,1 d,3 d,5 d,7 d respectively.
     将75只大鼠随机分成对照组、脑出血组、尼膜同组,各实验组分6h、1d、3d、5d和7d5个时间点。
短句来源
     Result Both nitric oxide and endothelin levels in brain tissue increased from 1h to 24h after SAH respectively in pure SAH group (P<0.05~0.01), while the degree of increase of those in nimodipine treated group was statistically slighter compared with the former group(P<0.05~0.01).
     结果SAH组脑组织NO和ET含量均自SAH后1h开始显著增加(P<0.05~0.01) ,ND组于SAH后脑组织NO及ET含量增加的程度较SAH组为小(P<0.05~0.01)。
短句来源
     RESULTS: Compared with sham operated group, BA diameters of SAH group and nimodipine treated group notably decreased ( P <0.05) at 2 h, 24 h, 3 d and 7 d after double hemorrhage.
     结果 :与Sham组相比 ,2h ,2 4h ,3d ,7d时SAH组和ND组BA直径明显减小 (P <0 .0 5 ) ;
短句来源
     Compared with that in the SAH group at the same time points, c FOS expression of nimodipine treated group obviously decreased ( P <0.05) and the numbers of normal neurons of CA1 subfield relatively increased ( P <0.05).
     与SAH组相同时点比较 ,ND组海马CA1区内正常神经元计数明显增多 (P <0 .0 5 ) ,c FOS表达明显减少 (P <0 .0 5 ) .
短句来源
     The total effective rate for clinical symptoms in the Baisuifang treated group accounted to 76.25%, with statistical difference comparing to 58.75% of nimodipine treated group (P<0.05).
     两组中医症状疗效比较 ,治疗组总有效率为 76 .2 5 % ,对照组总有效率为 5 8.75 % ,治疗组明显优于对照组 (P <0 .0 5 ) ;
短句来源
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  相似匹配句对
     Group D: treated with nimodipine;
     D组:尼莫通组;
短句来源
     Group E: treated with mannitol and nimodipine.
     E组:甘露醇与尼莫通合用组。
短句来源
     treated group.
     缺血组动物常压下吸纯氧,每天进行一次。
短句来源
     Treated for 48h.
     DG水平下降64.7.处理48小时后.
短句来源
     Compatibility of nimodipine
     尼莫地平输液的配伍实验
短句来源
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  nimodipine treated
By 28 days post axotomy, lesioned neurons in nimodipine treated animals were covered by a mean of 2.6 (hypoglossal) and 2.9 (facial nucleus) astrocytic lamellae, compared with 1.7 lamellae in the placebo group.
      
By 42-days, glial fibrillary acid protein-immunoreactivity was stronger in the nimodipine treated animals and by 112 days, glial fibrillary acid protein-immunoreactive astrocytes occured only in nimodipine-treated animals.
      
Lower pressure nimodipine treated animals exhibited reduction of both fast and slow activity post-occlusion.
      


Using a modified method of Pulsinelli's induction of brain ischemic damage in wistar rat by 4-vessel occlusion, calcium content in hippocampus was found significantly higher in ischemic group. Nimodipine (1 mg/kg) was injected intraperitoneally following brain ischemia, the calcium content in hippocampus had no significant difference between Nimodipine treated group and ischemic group.

本文将51只雄性Wistar大鼠随机分成假手术对照组、缺血组、缺血-尼莫地平组。采用改良Pulsinelli法建立鼠脑缺血模型,并经脑电图监测及组织病理学检查证实缺血模型是成功的。缺血-尼莫地平组的大鼠在夹闭颈总动脉造成鼠脑暂时性缺血的同时於腹腔内注射尼莫地平。比较对照组及暂时性缺血再灌注72小时后海马组织钙含量,用原子吸收分光光度计测定发现缺血组海马组织钙含量增高,但缺血-尼莫地平组海马组织钙含量与缺血组差异无显著性。

The authors enrolled 105 patients with acute ischemic stroke in a clinical study of intravenous nimodipine. The therapeutic effects well evaluated with five-degree scale. During the two weeks treatment period, the results showed that intravenous nimodipine group has a significantly better neurologic outcome than control group. In intravenous nimodipine group, subgroup analysis showed those treated within 36 hours has statistically significantly better neurologic outcome than those without 36 hours. Blood pressure...

The authors enrolled 105 patients with acute ischemic stroke in a clinical study of intravenous nimodipine. The therapeutic effects well evaluated with five-degree scale. During the two weeks treatment period, the results showed that intravenous nimodipine group has a significantly better neurologic outcome than control group. In intravenous nimodipine group, subgroup analysis showed those treated within 36 hours has statistically significantly better neurologic outcome than those without 36 hours. Blood pressure in the intravenous nimodipine treated patients decressed, and the effect on blood pressure may be related to concentration or dose. There was no major side effect except for two cases of fever and two cases of hiccup may have been related to treatment with nimodipine.

用国产尼莫地平注射液治疗急性缺血性脑血管病(AICVD)105例,疗效采用量表和5级标准分别评定。结果表明,尼莫地平组2周疗效显著优于对照组,36h内用药疗效显著优于36h后用药,尼莫地平有降压作用,此作用与剂量和滴速有关,无明显副作用。

The young rabbits were divided into three groups:control,asphyxial and asphyxia treated with nimodipine.Serum LPO,H 2O content of cerebral tissue,auricular microcirculation and pathological changes of cerebral tissue were observed.As compared asphyxial group with control,it was found that in asphyxial guoup serum LPO,H 2O of cerebral tissue and multiple scores(MS) of auricular microcirculation were increased significantly.Pathologic changes in...

The young rabbits were divided into three groups:control,asphyxial and asphyxia treated with nimodipine.Serum LPO,H 2O content of cerebral tissue,auricular microcirculation and pathological changes of cerebral tissue were observed.As compared asphyxial group with control,it was found that in asphyxial guoup serum LPO,H 2O of cerebral tissue and multiple scores(MS) of auricular microcirculation were increased significantly.Pathologic changes in the brain were obvious.Serum LPO with H 2O of cerebral tissue,MS with H 2O of cerebral tissue and serum LPO with MS were all in positive correlations. Compared nimodipine treated group with asphyxial group,serum LPO,cerebral H 2O and MS of the treated group decreased significantly,pathologic changes in brain were relieved.It is concluded that OFR and microcirculation dysfunction induce injury in brain subjected to acute asphyxia.Nimodipine may effectively prevent injury of the brain tissue of young rabbits.

将幼兔分为对照、窒息、窒息+尼莫地平3组,分别测定各组动物血清、脑组织水分含量并观察耳廓微循环和脑病理改变。结果表明:与对照组相比,窒息组血清LPO、脑水分含量、微循环加权积分值均明显增高,脑病理改变明显。窒息组LPO与脑水分含量,微循环加权积分值与脑水分含量,LPO与微循环加权积分值间均呈正相关关系。与窒息组相比,窒息+尼莫地平组血清LPO、脑水分含量、脑病理改变及微循环指标均有明显改善。提示自由基及微循环障碍参与了窒息所致的脑损伤,而尼莫地平可能具有保护作用

 
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