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focal brain ischemic
相关语句
  局灶脑缺血
     Experimental studies on the identification of focal brain ischemic penumbra with diffusion/perfusion weighted magnetic imaging
     磁共振弥散/灌注加权成像确认局灶脑缺血半暗带的实验研究
短句来源
     The influence of focal brain ischemic preconditioning in rat on expression of caspase-3
     大鼠局灶脑缺血预处理对caspase-3表达影响的研究
短句来源
     Based on rabbit MCAO focal brain ischemic model, we studied the effect of NO on ischemic brain by using L-Arginine, a precursor of NO and NOS inhibitor L-NNA.
     本文在兔MCAO局灶脑缺血模型基础上,利用外源性一氧化氮(NO)前体物质L-精氨酸和NOS抑制剂L-NNA研究NO对脑缺血后脑水肿和脑梗塞的影响。
短句来源
     Using quantitative method, we investigated neuroprotective efect of calcitonin gene related peptide (CGRP) by focal brain ischemic models in rats, which were made with thread embolism of MCA.
     线栓法建立大鼠局灶脑缺血动物模型,定量研究降钙素基因相关肽(CGRP)对大鼠局灶脑缺血体积的影响。
短句来源
  “focal brain ischemic”译为未确定词的双语例句
     CHANGES OF CYTOKINES IN RATS PLASMA AFTER FOCAL BRAIN ISCHEMIC REPERFUSION AND EFFECTS OF NIMODIPING
     局灶性脑缺血鼠血浆细胞因子变化及尼莫地平的治疗效果
短句来源
     The percentage of DNA fragmentation with apoptotic cell were observed by FCM on focal brain ischemic postreperfusion in Wistar rat.results showed the percentage of DNA fragmentation were gradully increased,peaked at 1d after reperfusion,then decreased at 3~7d after reperfusion.
     本研究采用流式细胞仪(FCM)检测大鼠局灶性脑缺血再灌流后凋亡细胞DNA断裂百分率。 结果表明:随再灌流时间的增加,DNA断裂百分率逐渐增加,再灌流1d时达高峰,再灌流3~7d逐渐下降。
短句来源
     Objective To investigate neuroprotective effect of ischemic hypoxia preconditioning by focal brain ischemic models in rats.
     目的观察低氧预处理对鼠脑局灶性脑缺血的保护作用。
短句来源
     Objective To study the effect of TNF-α pretreatment on cerebral ischemia-reperfusion injury and possible mechanism in rat. Methods Rat focal brain ischemic model of middle cerebral artery occlusion(MCAO) was made by using inserting thread method.
     目的 探讨肿瘤坏死因子-α(TNF-α)预处理对大鼠脑缺血再灌注损伤的影响及可能机制。 方法 采用线栓法制作大鼠局灶性脑缺血再灌注模型,并予以改良。
短句来源
  相似匹配句对
     brain
     脑
短句来源
     The brain of F.
     随发育脑体积增加,髓质占脑体积的比例增加。
短句来源
     Diagnosis of focal epilepsy in brain with CT
     癫痫患者脑部病变的CT诊断
短句来源
     Effects of agmatine on focal brain ischemia in rats
     胍丁胺对实验性大鼠局灶脑缺血的保护作用
短句来源
     Effect of Hemin on focal ischemic brain tissues of rats
     氯化血红素对大鼠局灶性脑缺血组织的作用探讨
短句来源
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Based on rabbit MCAO focal brain ischemic model,we studied the effect of NOon ischemic brain by using L-Arginine,a precursor of NO and NOS inhibitor-L-NNA, 42NZW rabbits was divided into 7 groups, namely control group,sham-operated group, MCAOischemic group,MCAO+NS group, MCAO+L-Arg group,MCAO+D-Arg group, MCAO+L-Arg +L-NNA group with 6 each. The results verified that the brain tissue H2O ,Na+,Ca2+content was decreased significantly (P<0.01) in L-Arg treated group compared with simpleischemic...

Based on rabbit MCAO focal brain ischemic model,we studied the effect of NOon ischemic brain by using L-Arginine,a precursor of NO and NOS inhibitor-L-NNA, 42NZW rabbits was divided into 7 groups, namely control group,sham-operated group, MCAOischemic group,MCAO+NS group, MCAO+L-Arg group,MCAO+D-Arg group, MCAO+L-Arg +L-NNA group with 6 each. The results verified that the brain tissue H2O ,Na+,Ca2+content was decreased significantly (P<0.01) in L-Arg treated group compared with simpleischemic MCAO group. The brain infarction volume was also decreased remarkably (P<0.01 ). It is suggested that the effect of L-Arg was accomplished through the production of NO,NO has the function of relaxing blood vessels,hence Iessen ischemic brain edema and de-crease the infarction volume,which may improving the clinical prognosis of brain ischemia.

本文在兔MCAO局灶脑缺血模型基础上,利用外源性NO前体物质L-精氨酸和NO合成酶抑制剂L-NNA研究NO对脑缺血后脑水肿和脑缺血灶的影响、结果提示NO对脑缺血脑水肿具有保护作用,而L-NNA则会扩大脑缺血灶的范围。

Based on rabbit MCAO focal brain ischemic model, we studied the effect of NO on ischemic brain by using L-Arginine, a precursor of NO and NOS inhibitor L-NNA. 42 NZW rabhits were divided into 7 groups, namely control group, sham-operated group, MCAO ischemic group, MCAO + NS group, MCAO + L-Arg group, MCAO + D-Arg group, MCAO +L-Arg+L-NNA group with 6 each. The results verified that the brain tissue H2O, Na+, Ca2+ contents were decreased significantly (P<0. 01) in L-Arg treated...

Based on rabbit MCAO focal brain ischemic model, we studied the effect of NO on ischemic brain by using L-Arginine, a precursor of NO and NOS inhibitor L-NNA. 42 NZW rabhits were divided into 7 groups, namely control group, sham-operated group, MCAO ischemic group, MCAO + NS group, MCAO + L-Arg group, MCAO + D-Arg group, MCAO +L-Arg+L-NNA group with 6 each. The results verified that the brain tissue H2O, Na+, Ca2+ contents were decreased significantly (P<0. 01) in L-Arg treated group compared with simple ischemic MCAO group. The brain infarction volume was also decreased remarkably (P<0.01). It is suggested that the effect of L-Arg was accomplished through the production of NO. NO has the function of relaxing blood vessels, hence lessening ischemic brain edema and decreasing the infarct volume, which may improve the clinical management of brain ischemia.

本文在兔MCAO局灶脑缺血模型基础上,利用外源性一氧化氮(NO)前体物质L-精氨酸和NOS抑制剂L-NNA研究NO对脑缺血后脑水肿和脑梗塞的影响。NZW兔42只随机分为7组,每组6只,计对照组、假手术组、MCAO组、MCAO+NS组、MCAO+L-Arg组、MCAO+D-Arg组及MCAO+L-Arg+L-NNA组。结果提示:与单纯MCAO组比较,L-Arg组的脑组织H_2O、Na~+、Ca~(2+)含量明显下降(P<0.01),脑梗塞灶亦明显缩小(P<0.01)。从而显示L-Arg系通过产生NO促使血管舒张而减轻脑水肿和缩小梗塞灶,为临床改进脑缺血的治疗提供依据。

The percentage of DNA fragmentation with apoptotic cell were observed by FCM on focal brain ischemic postreperfusion in Wistar rat.results showed the percentage of DNA fragmentation were gradully increased,peaked at 1d after reperfusion,then decreased at 3~7d after reperfusion.The studies suggested that apoptosis may contribute to development of ischemic infarct and ischemic brain damage was a dynamic ongoing process.

本研究采用流式细胞仪(FCM)检测大鼠局灶性脑缺血再灌流后凋亡细胞DNA断裂百分率。结果表明:随再灌流时间的增加,DNA断裂百分率逐渐增加,再灌流1d时达高峰,再灌流3~7d逐渐下降。结果提示:细胞凋亡在局灶性脑缺血再灌流损伤中呈动态过程且系神经细胞死亡的重要形式。

 
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