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   p 16 genes 的翻译结果: 查询用时:0.202秒
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p genes
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  p16基因
     Study on expressions and clinical significance of C erbB 2 and P16 genes in ovarian neoplasms
     C-erbB_2、p16基因在卵巢肿瘤中的表达及其临床意义的研究
短句来源
     Study on the Growth Inhibition of Human Gastric Carcinoma Cell HGC27 by Combination of Wild-Type P53, P16 Genes
     野生型P53、P16基因联合抑制人胃癌细胞HGC27生长的实验研究
短句来源
     Deletion of p15 and p16 genes and overexpression of STK15 gene in primary hepatocellular carcinoma
     原发性肝癌中p15、p16基因缺失和STK15基因过表达的研究
短句来源
     ZLJ also decreased the expression of bcl 2 gene/protein,increased the level of bax and P16 genes/proteins.
     ZLJ可以下调bcl 2基因 /蛋白 ,上调bax和p16基因 /蛋白的表达。
短句来源
     Analysis of relationship between expression of C-erbB-2 and P16 genes and multidrug resistance in breast cancer
     乳腺癌C-erbB-2、P16基因蛋白表达与多药耐药的相关分析
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  p16基因的
     A Study On the Inactivation of p16 Genes and the Expressi on of P16 Protein in Primary Hepatocellular Carcinomas
     原发性肝细胞性肝癌中p16基因的失活与P16蛋白表达的研究
短句来源
     p16 genes were delivered to the Renca cells in vitro by adenovirus vectors. Gene transfer efficiency and changes of biological specificity of Renca cells were analyzed.
     [方法]将携带 p16基因的腺病毒体外转染小鼠肾癌Renca细胞 ,观察腺病毒对Renca细胞的转染效率以及 p16基因修饰的Renca细胞体外生物学性质的改变。
短句来源
     Methods The expressions of pRB and cyclin D1,p16 genes were screened in 23 gliomas (including 14 astrocytomas and 9 glioblastomas) and 6 normal brain tissues by immunohistochemical method.
     方法 用免疫组化的方法对 2 3例脑胶质瘤 (包括 14例星形细胞瘤和 9例胶质母细胞瘤 )以及 6例正常脑组织中 pRB以及cyclinD1、p16基因的表达进行检测。
短句来源
     Methods P16 genes were delivered to the T24 cells jn vitro by adenovirus vectors. Gene transfer efficiency and changes of biological specificity of T24 cells were analyzed.
     方法 将携带P16基因的腺病毒体外转染人膀胱癌T24细胞,观察腺病毒对T24细胞的转染效率以及P16基因修饰的T24细胞体外生物学特性的改变。
短句来源
     Methods:The expression of telomerase, p53 and p16 genes were examined in 40 NSCLC cases by SP and TRAP methods, respectively.
     方法 :应用SP法免疫组化技术和TRAP法分别检测 40例肺癌组织中端粒酶和p5 3、p16基因的表达。
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  “p 16 genes”译为未确定词的双语例句
     Study on Bcl-2 gene and its family genes Bax,Bcl Xl as well as Fas/Apo 1,P16 genes and their clinical significance in Acute leukemia
     Bcl-2基因及其家族Bax,Bcl-Xl和Fas/Apo-l,P16的检测在急性白血病中的临床意义
短句来源
     And we used Reverse Transcription-Polymerase Chain Reacton(RT-PCR) to detect the expression of Caspase-3, p53 and P16 genes in this apoptosis.
     并用逆转录一聚合酶链反应(RT—PCR)法检测与此次凋亡相关的基因Caspase-3、P53和P16的表达;
短句来源
     Objectives To detect the expressions of P53、 N-ras、TGFα、P16 genes usingPCR-SSCP and assess it’s value and clinical significance in multinodular hepatocellullar.
     目的 应用PCR-SSCP技术检测P53、 N-ras、TGFα、P16作为基因标记物在判断多结节性肝癌克隆起源中的价值及临床意义。
短句来源
     Homozygous deletion of p15. p16 genes in lung cancer
     肺癌患者组织中p15、p16纯合缺失
短句来源
     Further, overexpression of C-erbB-2 and P16 genes are probably the factor of multidrug resistance.
     C -erbB - 2、P16异常表达可能是乳腺癌产生多药耐药的因素之一。
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  p genes
Pancreatic cancers frequently carry mutations in the Kras, p53, and p16 genes, which regulate cell proliferation.
      
In addition, the comparison suggests that a gene conversion event is part of the evolution of the human p15 and p16 genes.
      
Ten xenografted tumors were characterized for alterations in p53, K-ras, and p16 genes, which were found in six, eight, and nine cases, respectively.
      
Replacement of tumour suppressor gene function using adenoviruses to transfer wild-type p53 and p16 genes can produce dramatic anti-tumour effects, both in vitro and in vivo.
      
Conclusion: Since the co-deletion of p15/p16 genes is significantly related to the prognosis of NSCLC patients, detecting co-deletion of both genes might be used as a potential marker for NSCLC prognosis.
      
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A survey of the distribution of ABO,P and Rh blood groups was made among960 primary and middle school students of Tujia Minority in Luota,Xiche andMiaoshi Communes in Longshan county.Slide method was used for the identificationof ABO and P blood groups and direct bromelin method,for Rh blood group.Thedistribution of ABO blood group system in The Minority of Tujia is shown inTable 1.The sequence of the frequencies of phenotypes is A>O>B,AB.As for thegene frequencies,the sequence is O>A>B.In view of...

A survey of the distribution of ABO,P and Rh blood groups was made among960 primary and middle school students of Tujia Minority in Luota,Xiche andMiaoshi Communes in Longshan county.Slide method was used for the identificationof ABO and P blood groups and direct bromelin method,for Rh blood group.Thedistribution of ABO blood group system in The Minority of Tujia is shown inTable 1.The sequence of the frequencies of phenotypes is A>O>B,AB.As for thegene frequencies,the sequence is O>A>B.In view of the frequencies of the pheno-types of P blood group system,P_1 is much more frequent than P_2(Table 2),on thecontrary,the gene frequency P(gene of P_2)is higher than P(gene of P_1)(Table 4).As for the Rh blood group,among 930 investigated individuals,we found only8 phenotypes.The sequence of the frequencies of these phenotypes is CCDee>CcDE>ccDE>CcDee>CCDE>ccDee>CCdE and ccdee(Table 3).The rate of Rh posi-tive is 99.78% and Rh negative,0.22%.The sequence of frequencies of Rh genecomplexes is R' R~e>r>R~o>R~z,while r,r'and r~y are zero(Table 4).Detailedcomparisons are made between our results and those reported by the Shanghai Inst-itute of Biological Products(Table 5 to 8).The probable reason why the frequencyof the phenotype P_1 is much higher than P_2 while the gene frequency is quite thecontrary is discussed in thits paper.

共调查了土家族青年学生960人的 ABO、P 及 Rh 等血型系统,结果表明在 ABO 血型系统中,表现型频率的次序为 A>O>B>AB 型,而基因频率的次序为 O 基因>A 基因>B 基因。P 血型的表现型频率,P_1远高于 P_2,但其基因频率却相反,P_2高于 P_1。在我们调查的930例 Rh 血型中各表现型频率的次序为 CCDee>CcDE>ccDE>CeDee>CCDE>ccDee>CCdE 及 ccdee·Rh阳性占99.78%,Rh 阴性仅占0.22%。基因频率的次序为 R~1>R~2>r>R~0>R~z·r′、r″及置 r~y 为O。我们将调查结果与上海生物制品研究所血型组的调查结果作了较详尽的比较,并指出土家族与各民族间某一血型的表现型分布上的差别以及基因频率的差别的显著性基本上一致,但也有个别不尽相同的情况。此外,本文还讨论了土家族 P 血型 P_1远多于 P_2而 P_1基因频率反较 P_2为小的可能原因。

In order to test the promoter function of two HBV DNA fragments, a soluble cell-free system extracted from Hela celis was used. In the in viiro transcriptional system using the 1.4kb DNA fragment as the templa-te, there were two RNA products whose transcriptional initiation sites were supposed to be at nucleotide 276±5% and 821±5% respectively on the HBV map. The first transcriptional initiation site is identical to the one that is directed by the HBV C gene promoter known before.The rela-tionship between...

In order to test the promoter function of two HBV DNA fragments, a soluble cell-free system extracted from Hela celis was used. In the in viiro transcriptional system using the 1.4kb DNA fragment as the templa-te, there were two RNA products whose transcriptional initiation sites were supposed to be at nucleotide 276±5% and 821±5% respectively on the HBV map. The first transcriptional initiation site is identical to the one that is directed by the HBV C gene promoter known before.The rela-tionship between the location of the second initiation site and the gene open reading frame suggests that the promoter may direct the synthesis of P gene mRNA.The 0.8kb DNA fragment starts from the core structure gene, not in-cluding the regulating sequence. Deducing from the 708±5% nt-long RNA product, the transcriptional initiation site is 588 + 5% on the HBV DNA map. Associated with this RNA start site, there is an ATG codon at po-sition 677 downstream, suggsting that the ATG codon may be a start site of a new open reading frame.

自adr亚型乙型肝炎病毒DNA重组质粒中获得两个DNA片段,用体外转录方法研究启动子的位点。其中1.4kb片段有两个转录产物,其转录起始点分别位于乙型肝炎病毒DNA序列的276±5%位和821±5%位,第一个转录起始点与已报道的乙型肝炎病毒核心抗原基因上游启动子位置一致,第二个转录起始点在888位P基因的起始密码子上游。0.8kb片段自校心抗原结构基因起始密码子ATG以下的序列开始,不含有已知的调控序列,其708±5%核苷酸长的RNA产物,根据其长度计算共转录起始点位于乙型肝炎病毒DNA序列588±5%位,与此位置相关的下游ATG密码子位于677位。

The autho rs found that rhizomania resistant charac tor was controlled by two pairs of dominant genes by our observing on the resistance of differ rent original materials, their cross progenies and test cross progenies to the disease in the breeding of rhizomania resistance. The gene types and genetic niodel of sugarbeet (Beta vulgaris) rhizomania resistance were infered acco rding to the result. Let us assume that"P" and "R"represent the two pairs domnant genes, so the RRPP for immune...

The autho rs found that rhizomania resistant charac tor was controlled by two pairs of dominant genes by our observing on the resistance of differ rent original materials, their cross progenies and test cross progenies to the disease in the breeding of rhizomania resistance. The gene types and genetic niodel of sugarbeet (Beta vulgaris) rhizomania resistance were infered acco rding to the result. Let us assume that"P" and "R"represent the two pairs domnant genes, so the RRPP for immune type; RrPP, RRPp for disease resistanc e typel RrPp, RRpp, rrPP for disease tolerant type Ⅰ; Rrpp, rrp for disease tolerant typeⅡ and rrpp for sensitive type.The cultivated sugarbeet has lost a pair of the dominant gene in their cultivating process. If the sugarbeet lost the "p", we obtain only the stock of disease tolerant type, but it is not possible get the stocks of disease resistant and immune types in our selecting works.However, we wi1l get the disease resistant or immune type, after the sugarbeet will pick up the lost"P"gene by crossing with their ancestors.

在从事甜菜抗丛根病育种工作中,通过对各类育种材料的抗病性观察,及对其互交和测交后代的调查,发现甜菜的抗丛根病性是由两对显性基因控制的。经过一步分析,提出了甜菜抗丛根病的基因型和遗传模式。假定以“R”和“P”代表这两对显性基因,免疫型为RPRP;抗病型为RPRp、RPrP耐病Ⅰ型为RPrp、RpRp、rPrP;耐病Ⅱ型为Rprp、rPrP;敏感型为rprp。推测:糖用甜菜在驯化过程中,丢失了其中的一对显性基因,假设丢失的“P”,那么,只有耐病Ⅰ型(RpRp)、耐病Ⅱ型(Rprp)和敏感型(rprp)。这样,采用选择的方法,只能选育出耐病型品种,不可能选育出抗病型和免疫型的品种。只有通过与其祖先杂交,将丢失的“P”基因找回来,才能育成抗病型和免疫型品种。

 
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