助手标题  
全文文献 工具书 数字 学术定义 翻译助手 学术趋势 更多
查询帮助
意见反馈
   cerebral damage 的翻译结果: 查询用时:0.191秒
图标索引 在分类学科中查询
所有学科
神经病学
外科学
儿科学
急救医学
中药学
临床医学
中医学
心血管系统疾病
药学
更多类别查询

图标索引 历史查询
 

cerebral damage
相关语句
  脑损伤
     There were no clinical signs of cerebral damage in both groups. The incidence of POMD was significantly lower (2/20) in group P than in group F (6/20) (P<0.01).
     术后P组POMD发生率(2/20)低于F组(6/20)(P<0.01),但是两组脑损伤症状均于术后10 d恢复正常;
短句来源
     Changes in SOD and LPO of Severe Cerebral Damage Patients after Hyperbaric Oxygen Therapy
     重型脑损伤患者高压氧治疗前后血清SOD,LPO含量变化
短句来源
     Clinical Study of Effects of Lidocaine on Cerebral Damage Secondary to Brain Trauma
     利多卡因抑制颅脑损伤后继发性脑损害的临床研究
短句来源
     Objective To discuss the effect of hyperbaric oxygen(HBO) on severe cerebral damage patients and on their lactoperoxidase(LPO) and superoxide dismutase(SOD) levels.
     目的探讨高压氧(HBO)对重型脑损伤患者疗效及对血清脂质过氧化物(LPO)、超氧化物歧化酶(SOD)的影响。
短句来源
     Objective To study the effects of hyperbaric oxygen therapy(HBOT) on hemorrheology,serum lipid peroxide(LPO) and superoxide dismutase(SOD) in patients with severe cerebral damage.
     目的研究高压氧(HBO)对重型脑损伤患者血液流变学及血清脂质过氧化物(LPO)、超氧化物歧化酶(SOD)的影响。
短句来源
更多       
  脑损害
     Objective To study the cause and methods of prevention and treatment of severe cerebral damage following cardiac operation with cardiopulmonary bypass (CPB) .
     目的 探讨体外循环(Cardiopulmonary bypass,CPB)心脏术后重度脑损害的原因及防治方法。
短句来源
     Methods To study the diagnosis, treatment and prognosis of severe cerebral damage during cardiac operation with CPB in 18 cases.
     方法 分析18例CPB术后并发重度脑损害的诊治过程及预后。
短句来源
     Study on Tone Expression of Pu Tong Hua and Cantonese in the Patients with Cerebral Damage
     脑损害患者的普通话和粤语声调表达研究
短句来源
     \ Results\ (1) 106 normotensives,498 hypertensives without target organ damage(TOD) and 721 hypertensives with TOD were included in the study,53% cardiac damage,16% cerebral damage,9% renal damage and 22% more than one \{organ\} damage were found in the TOD group.
     结果  (1)血压正常者 10 6例 ,单纯 EH 498例 ,合并 TOD72 1例 ,其中 5 3%心脏损害 ,16 %脑损害 ,9%肾损害 ,2 2 %多脏器损害。
短句来源
     According to the orthogonal design of L9 (3)4, the effects of VISA agent on defending hypoxic cerebral damage were randomly assessed by the parameters of cerebral conical somatosensory evoked potential (SEP) in the intact hypoxic rabbit model in comparison with the control.
     在兔脑低氧模型上,以大脑皮层体感诱发电位SEP为指标,按L9(3)4正交设计.随机对照研究VISA高效剂抗低氧/复氧脑损害的作用。
短句来源
更多       
  脑损伤的
     CONCLUSION:The upregulation of IL 6 expression may be one of the mechanisms of EA in treating ischemic cerebral damage.
     结论:电针提高血清IL-6的表达可能是电针治疗缺血性脑损伤的机制之一。
短句来源
     Methods The model of focal cerebral ischemia was made by occluding middle cerebral artery(MCA) for 2 h and reperfusion for 0.5~48 h, HE Staining was used to investigate the histological features of ischemic cerebral damage,the immunohistochemical method was used to observe the distributive characteristics of GDNF in brain tissue in rats.
     方法 阻断大鼠大脑中动脉 (MCA)血流 2小时 ,再灌流 0 .5~ 48小时制成局灶性脑缺血模型 ,HE染色评价缺血性脑损伤的组织学特点 ,免疫组化法观察GDNF在脑组织的分布特点。
短句来源
     The method of treating serious cerebral damage patients and protecting cerebral tissues with Mg2+ still need to be study.
     但损伤脑组织中低镁与继发性脑损伤的因果关系以及镁制剂对脑损伤的治疗和脑保护作用具体机制尚待进一步研究。
短句来源
     Result:The CT date indicated that the incidence of cerebral damages were 54.84%,the relationship between asphyxia degree and cerebral damage showed that the incidence of cerebral damage was 44% in mild asphyxia group and 66% in serious group. There were no signficant differences in the two groups(P>0.05).
     结果:显示CT诊断脑损伤的阳性率为54.84% ,窒息程度与脑损伤的关系为: 轻度窒息组CT示脑损伤的发生率为44% , 重度窒息组为66% , 二者差异无显著性(P< 0.05)。
短句来源
     EA couldupregulate IL-6 expression and downregulate TNF- a expression, which might be one of the mechanisms of EA protecting against ischemic cerebral damage.
     脑缺血后IL-6、TNF-α的表达均呈上升趋势,电针上调IL-6的表达,下调TNF-α的表达,可能是电针治疗缺血性脑损伤的机制之一。
短句来源
更多       
  “cerebral damage”译为未确定词的双语例句
     Conclusion:CD54,CD106,CD62p are closely related to the development of MID and play an important role in pathlogical procedure of cerebral damage after MID. The levels of CD54,CD106,CD62p in patients with MID implies the degree of their neurological function deficit scores and might be an important indicator to observe the changes of disease.
     结论:CD54、CD106、CD62p参与了MID的病理变化过程,并与痴呆程度密切相关,在一定程度上反映了MID神经功能缺损的程度,可作为MID后监测病情变化的重要指标。
短句来源
     Results The operation patients of studing group were 26, and 22 patients in the control group, the death rates were 13 % and 22 % respectively ( P < U. 05). The contents of Mg2+ in cerebral damage tissues was (4.6 ±0. 2)#mol/g(normal ralual 5.0 -6. 0 #mol/g).
     结果 实验组和对照组施行开颅手术分别为26例和22例,两组死亡率分别为13%和22%(P<0.05),手术病人脑组织Mg2+含量测定平均为(4. 6±0.2)μmol/g,正常值(5~6)μmol/g。
短句来源
     Results Patients of Group A had ICAM 1 levels in CSF above normal range,presented marked cerebral damage and a disturbance of BBB (range 4.13~14.11ng/ml,n=8).
     结果 A组患者CSF可溶性ICAM 1水平为 4 .13~ 14 .11ng/ml,明显高于正常值。
短句来源
     Pharmacokinetics of sodium β-aescinate induced by ischemia-reperfusion in cerebral damage rats
     β-七叶皂苷钠在脑缺血再灌注大鼠体内药代动力学的研究
短句来源
     Relationship between the changes of β EP\,β\-2 MG's concentrations in serum after asphyxia neonate and hypoxia cerebral damage and renal damage
     窒息新生儿血浆β-EP、β2-MG值变化与脑、肾损伤的关系
短句来源
更多       
查询“cerebral damage”译词为用户自定义的双语例句

    我想查看译文中含有:的双语例句
例句
为了更好的帮助您理解掌握查询词或其译词在地道英语中的实际用法,我们为您准备了出自英文原文的大量英语例句,供您参考。
  cerebral damage
Cerebral damage and dysfunction in sexually aggressive men
      
Although it has been well established that hyperglycemia increases cerebral damage following transient cerebral ischemia, its effect on permanent focal ischemia is controversial.
      
Role of Nitric Oxide in Pathogenesis Underlying Ischemic Cerebral Damage
      
In order to assess the possible association of cytomegalovirus (CMV) with cerebral damage, 65 children aged between 2 and 10 years were tested for CMV isolation and antibody status.
      
Alteration of regional brain oxygen saturation (rSO2) in a patient with cerebral damage after aortic arch replacement: carbon di
      
更多          


The causes and short-tern porignosis of neonatal convulsions in 98 cases if bewbirb ubfabts from Jan,77 to Jan,1982 were studued,The propottion of neonaal convulsions cases vas 9.2% among 1071 newborn babies less han 4 weeks of age admiled in this hospital.The incidence of neonatal convulsions was 27.4% in both prematures and term infants of low birth weight compare to 7% in the fullterm newborns.The causes were various.cranio-cerebral damage (37.6%) was probably the main one,while hypocalcemia (27.7%)...

The causes and short-tern porignosis of neonatal convulsions in 98 cases if bewbirb ubfabts from Jan,77 to Jan,1982 were studued,The propottion of neonaal convulsions cases vas 9.2% among 1071 newborn babies less han 4 weeks of age admiled in this hospital.The incidence of neonatal convulsions was 27.4% in both prematures and term infants of low birth weight compare to 7% in the fullterm newborns.The causes were various.cranio-cerebral damage (37.6%) was probably the main one,while hypocalcemia (27.7%) was the next commonest and he others weredue to infections (7%),hypotlycemia (2%),etc The causes were unknown in 12.8% of he total cases.The most frequent seizure pattern was observed to be focal clonic seizures while generalized clonic and minimal seizures took the next.The toal mortality was 17.3%,of which 47.1% occurrod in low birth weight babues.Underlying diseases accouned for all he deaths.Among 39 cases being followed up from 8 months to 5% years,the incidence of sequelac was 28.2%(11/39).Prognosis was found to be the worst amoung the paients having craniocerebraldamage.

本院自1977年1月至1982年1月共收治新生儿(出生至28天)惊厥病人98例,占住院新生儿病人的9.2%,惊厥发生率较高。其中低体重儿惊厥发生率占住院低体重儿的27.2%,比住院足月新生儿惊厥发生率(7%)明显增高(P<0.001)。病因比较复杂,颅脑损伤(37.6%)和低血钙(27.7%)是新生儿惊厥的主要病因,其次为感染(7%)、低血糖(2%)等,原因不明者占(12.8%)。惊厥发作形式主要为限局性阵挛性发作,其次为全身性阵挛性发作和轻微发作。本组新生儿惊厥病死率为17.3%,低体重儿约占死亡病例的一半(47.1%)。均死于原发病。随访39例(8月~5 1/2岁),后遗症发生率为28.2%(11/39)。颅脑损伤是本组病人死亡和后遗症的重要原因。

In recent years the problem ofneonatal hypoglycemia with its sequ-ential cerebral damage has increasinglyclaimed the attention of service andresearch personnel involved in familyplanning, eugenics and perinatalhealth care. As a basis for clinical management,a study of the blood level changes innormal neonates within the first weekwas carried out in 583 normal birthsinvolving 706 blood specimens. Thesugar value was determined by modifiedortho-methylaniline method. Bloodsamples were taken from cord, andfrom...

In recent years the problem ofneonatal hypoglycemia with its sequ-ential cerebral damage has increasinglyclaimed the attention of service andresearch personnel involved in familyplanning, eugenics and perinatalhealth care. As a basis for clinical management,a study of the blood level changes innormal neonates within the first weekwas carried out in 583 normal birthsinvolving 706 blood specimens. Thesugar value was determined by modifiedortho-methylaniline method. Bloodsamples were taken from cord, andfrom the toe or heel at 1/2, 2, 4, 6,12and 18-24 hours and on full days 1,2, 3, 4, 5, 6, and 7 respectively.Each infant contributed one or twosamples. Statistical analysis, as shownin the accompanying curve, foundthe blood sugar level fell rapidly froman average of 50 mg % after deliveryto its lowest level of 43 mg % at 4-6hours and thence rose steadily withinthe week to its height of 63 mg % onthe 7th day. No significant differences as tosex, weeks of gestation, birth weight,ambient temperature and mode ofdelivery were found in the blood sugervalues. Analysis of 26 pairs of samplesobtained from both the cord and themother at the same time found apositive relationship between the two,i.e., the sugar value of the cordblood varies proportionally with thatof the mother's blood. The fall of blood sugar levelwithin the first 4 to 6 hours ofdelivery is presumably related to thelow glycogenolysis and insufficientglyconeogenesis inherent in the neonateat this period. The lowering ofcertain key rate-limiting enzymeactivities immediately before parturi-tion has been suggested as the causeof the latter condition. The fact that a few of the 4 and6 hour samples presented sugar valuesbelow 30 mg % indicated the existenceof realistic threat of cerebral damageto normally delivered neonates, soearly feeding glucose 4 hours afterbirth is suggested. The modified ortho-methylanilinemethod of determination of bloodsugar in the neonate is reliable andconvenient, involving a minimum ofblood and instrumentation and can beconveniently adopted by primary healthcare stations.

本文报告用改良邻甲苯胺法对583例初生七天内正常新生儿进行了706人次毛细血管全血血糖测定。同时测定26对脐血与母血血糖水平,脐血与母血呈正相关。本组测得4~6小时血糖少数低于30毫克%。为提高生后4~6小时血糖水平,避免颅脑损伤,指出可将开始喂糖水时间提前到生后4小时。

The effect of cardiopulmonary bypass in different hypothermia on brain tissue was studied. Lactate dehydrogenase (LDH), creatine phosphokinase (CPK) in cerebral spinal fluid and histochemical analysis of LDH, CPK, ATP-ase in brain tissue were used as indicators in 18 mongrel dogs underwent cardiopulmonary bypass with ischemic arrest for 1 hour produced by infusing 4℃ potassium cardioplegic solution at aortic root every 20 minutes to protect the myocardium. The animals were randomly designed into four different...

The effect of cardiopulmonary bypass in different hypothermia on brain tissue was studied. Lactate dehydrogenase (LDH), creatine phosphokinase (CPK) in cerebral spinal fluid and histochemical analysis of LDH, CPK, ATP-ase in brain tissue were used as indicators in 18 mongrel dogs underwent cardiopulmonary bypass with ischemic arrest for 1 hour produced by infusing 4℃ potassium cardioplegic solution at aortic root every 20 minutes to protect the myocardium. The animals were randomly designed into four different systemic temperature groups i.e.Group Ⅰ (n=7), 18-24℃; Group Ⅱ (n=6), 25-28.4=0; Group in (n=5) 35-37℃, and Group Ⅳ, served as blank control without cardiopulmonary bypass, for which only anesthesia and thoracotomy was induced.It was found that LDH and CPK in CSF showed no. significant change in Group Ⅰ, but increased moderately in gronp n, and considerably in Group Ⅲ. Results in the histochemical study of cerebral tissues were similar to that of the CSF. The authors proposed that determination of CSF-LDH and/or-CPK were useful clinically in assessing cerebral damage. The profound hypothemia per se did not cause brain damage, instead it was a protective measure during cardiopulmonary bypass.

以脑脊液LDH、CPK及组化LDH、CPK、ATP-ase反应作为观察指标,探讨不同程度低温体外循环对脑组织的影响。结果发现,深低温组脊 LDH液及CPK 无明显变化;中度低温组略有增高;而常温组则显著增高(P<0.01)。脑细胞组化酶学反应结果显示情况与脑脊液酶学测定结果基本一致,表明脑脊液LDH 及/或CPK 测定在评价脑细胞损害方面有其临床应用价值。据此,作者认为,深低温本身不足以造成脑部损害,反而对脑组织有一定的保护作用。

 
<< 更多相关文摘    
图标索引 相关查询

 


 
CNKI小工具
在英文学术搜索中查有关cerebral damage的内容
在知识搜索中查有关cerebral damage的内容
在数字搜索中查有关cerebral damage的内容
在概念知识元中查有关cerebral damage的内容
在学术趋势中查有关cerebral damage的内容
 
 

CNKI主页设CNKI翻译助手为主页 | 收藏CNKI翻译助手 | 广告服务 | 英文学术搜索
版权图标  2008 CNKI-中国知网
京ICP证040431号 互联网出版许可证 新出网证(京)字008号
北京市公安局海淀分局 备案号:110 1081725
版权图标 2008中国知网(cnki) 中国学术期刊(光盘版)电子杂志社