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cervical rigidity
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  颈抵抗
     Clinical manifestations included fever (100%), headache (96.6%), vomit (86.67%), unconsciousness (40%), cervical rigidity (100%), petechia and ecchymosis (66.67%), abnormal examination of cerebrospinal fluid (100%), and leukocytosis (100%). The main complications were toxic myocarditis and renal function damage.
     临床表现为发热(100%),头痛(96.67%),呕吐(86.67%),意识障碍(40%),伴颈抵抗(93.33%),淤点或淤斑(66.67%),CSF异常(100%),白细胞升高(100%),合并有中毒性心肌炎、肾损害为主。
短句来源
  “cervical rigidity”译为未确定词的双语例句
     Is Aging Associated with Cervical Rigidity (An Investigation on Mentosternal Distance in 1575 Normal Chinese)
     1575名正常国人颏胸距的调查
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  相似匹配句对
     Cervical Ganglioneuroma
     颈部神经节细胞瘤
短句来源
     Cervical radiculopathy
     颈神经根病
短句来源
     On Rigidity in Translation
     似信非信——翻译形式主义三戒
短句来源
     Is Aging Associated with Cervical Rigidity (An Investigation on Mentosternal Distance in 1575 Normal Chinese)
     1575名正常国人颏胸距的调查
短句来源
     Rigidity Theorems for Submanifolds
     子流形刚性定理
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查询“cervical rigidity”译词为用户自定义的双语例句

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Objective:To explore clinical characteristics of spontaneous subarachnoid hemorrhage (SAH) patients.Methods:We analyzed the etiologies, clinical manifestations and results of CT and DSA of 274 SAH patients of different ages in Xiangya Hospital from 1986 to 2000. Results: The etiologies we found included aneurysm (112 cases, 40.9%; mainly in the 45~59 years old group), artery malformation (33 cases, 12.1%; mainly in the under 45 years old group), and hypertensive arteriosclerosis (50 cases, 18.3%; mainly in the...

Objective:To explore clinical characteristics of spontaneous subarachnoid hemorrhage (SAH) patients.Methods:We analyzed the etiologies, clinical manifestations and results of CT and DSA of 274 SAH patients of different ages in Xiangya Hospital from 1986 to 2000. Results: The etiologies we found included aneurysm (112 cases, 40.9%; mainly in the 45~59 years old group), artery malformation (33 cases, 12.1%; mainly in the under 45 years old group), and hypertensive arteriosclerosis (50 cases, 18.3%; mainly in the above 60 years old group). The clinical manifestations consisted of headache, vomiting, disturbance of consciousness, cervical rigidity and limb paralysis, which were significant differences among different age groups and there were 138 healed cases (50.4%). 83 improved cases (30.3%), 25 deteriorated cases (9.1%) and 28 died cases (10.2%).Conclusion:The main causes of SAH of young patients are aneurysm and vascular malformations, and to the old is hypertensive arteriosclerosis. The clinical manifestations of the old are not as typical as the young. CT is the first choice to confirm SAH, while DSA is very important to find the etiology.

目的 :探讨自发性蛛网膜下腔出血 (SAH)患者的临床特征。方法 :回顾性分析了 1986~ 2 0 0 0年湘雅医院2 74例SAH住院患者的病因、临床表现、CT和血管造影检查结果。结果 :病因 :动脉瘤 112例 (40 .9% ) ,以 4 5~ 5 9岁组最多见 (P <0 .0 1) ;血管畸形 33例 (12 .1% ) ,小于 4 5岁组中多见 (P <0 .0 1) ;高血压动脉硬化 5 0例(18.3% ) ,主要集中在大于 6 0岁组 (P <0 .0 1)。临床表现 :头痛、呕吐、颈抗阳性在中青年SAH患者中的发生率显著高于老年患者 ;而意识障碍和肢体瘫痪症状在老年患者中发生率较高。头部CT检查阳性率79.6 6 % (14 1/ 177)。本组病人治愈 138例 (5 0 .4 % ) ,好转 83例 (30 .3% ) ,恶化 2 5例 (9.1% ) ,死亡 2 8例(10 .2 % )。结论 :青中年SAH患者的主要病因是动脉瘤和血管畸形 ,而老年患者为高血压动脉硬化 ;老年SAH患者的临床表现不如中青年患者典型 ;CT是首选检查方法 ,血管造影对明确病因意义重大。

Objective To investigate the clinical characteristics of epidemic cerebrospinal meningitis in Guangzhou in 2005, and to arose the awareness of physicians of the disease and strengthen the preventive measures. Methods The clinical data of 30 hospitalized cases with epidemic cerebrospinal meningitis in our hospitals in 2005 were retrospectively analyzed to study the epidemic features and clinical characteristics of epidemic cerebrospinal meningitis. Results Epidemic cerebrospinal meningitis had the character of...

Objective To investigate the clinical characteristics of epidemic cerebrospinal meningitis in Guangzhou in 2005, and to arose the awareness of physicians of the disease and strengthen the preventive measures. Methods The clinical data of 30 hospitalized cases with epidemic cerebrospinal meningitis in our hospitals in 2005 were retrospectively analyzed to study the epidemic features and clinical characteristics of epidemic cerebrospinal meningitis. Results Epidemic cerebrospinal meningitis had the character of colony gathering. Clinical manifestations included fever (100%), headache (96.6%), vomit (86.67%), unconsciousness (40%), cervical rigidity (100%), petechia and ecchymosis (66.67%), abnormal examination of cerebrospinal fluid (100%), and leukocytosis (100%). The main complications were toxic myocarditis and renal function damage.Conclusions The outbreak of epidemic cerebrospinal meningitis in Guangzhou this year was rare in this region, and the cases mainly occurred in the group of labors. Clinical manifestations were typical without other special features. Epidemic cerebrospinal meningitis often caused multiple organ damage. To control the disease, we should mainly depend on prevention and medical treatment.

目的探讨2005年广州市流行性脑脊髓膜炎流行的临床特征,提高临床医师对流行性脑脊髓膜炎的全面认识和加强预防措施。方法将2005年春收治入院的所有30例流行性脑脊髓膜炎患者进行流行特点和临床特征分析。结果流行性脑脊髓膜炎发病有群体聚集性。临床表现为发热(100%),头痛(96.67%),呕吐(86.67%),意识障碍(40%),伴颈抵抗(93.33%),淤点或淤斑(66.67%),CSF异常(100%),白细胞升高(100%),合并有中毒性心肌炎、肾损害为主。结论本次流行性脑脊髓膜炎流行是近年少见的,以民工为主。临床表现典型但无特异性,合并有多脏器损害,强调以预防为主,防治结合。

BACKGROUND: Delayed vitamin K deficiency is characterized by acute onset, severe illness and high fatality rate. 33%-50% survivors accompany with other various nervous system sequelas. Therefore, diagnosis and treatment of intracranial hemorrhage in time become a key factor for improving healing rate and reducing fatality rate and incidence of sequela. OBJECTIVE: To investigate the clinical situation, experimental characteristics, CT examination and terminative characteristics of infants with intracranial hemorrhage...

BACKGROUND: Delayed vitamin K deficiency is characterized by acute onset, severe illness and high fatality rate. 33%-50% survivors accompany with other various nervous system sequelas. Therefore, diagnosis and treatment of intracranial hemorrhage in time become a key factor for improving healing rate and reducing fatality rate and incidence of sequela. OBJECTIVE: To investigate the clinical situation, experimental characteristics, CT examination and terminative characteristics of infants with intracranial hemorrhage induced by delayed vitamin K deficiency. DESIGN: Case analysis. SETTING: Department of Pediatrics, Zhongshan Hospital Affiliated to Xiamen University. PARTICIPANTS: A total of 17 infants with intracranial hemorrhage induced by delayed vitamin K deficiency aged 1-3 months including 11 boys and 6 girls were selected from Department of Pediatrics, Zhongshan Hospital Affiliated to Xiamen University from January 1994 to December 2005. All infants had drowsiness, rejective milk, spiting milk, gaze of both eyes, tic, coma, full anterior fontanelle, high muscular tension and cerebral hernia, etc. Experimental examination demonstrated that infants had anemia at various degrees; prothrombin time and partial thromboplastin time were prolonged; platelet count was normal. CT examination indicated that screenages of subarachnoid hemorrhage, subdural hematoma, cerebral parenchyma hemorrhage and intraventricular hemorrage were changed. Hemorrhage was stopped by the application of vitamin K. All patients provided informed consent. METHODS: ① Clinical situation and physical sign of infants were observed after hospitalization and scanned with rapid spiral CT scanning system. The thickness and average space of layers were 8-10 mm and the scanning time was 5 s with window width of 30-80 Hu and window position of 28-35 Hu. ② After hospitalization, four items of blood coagulation was measured with Futura meter and biochemical indexes of blood, such as serum calcium, serum alkaline phosphatase, total bilirubin, direct bilirubin and indirect bilirubin, were detected with Roche Modular PPI automatic biochemistry analyzer. ③ After hospitalization, infants were given 5-10 mg vitamin K1 for 3-5 days, and then, they were transfused with 10-15 mL/kg fresh plasma or whole blood for 1-3 times and received other relative therapies. Therapeutic effects were evaluated based on clinical diagnosis and criteria. MAIN OUTCOME MEASURES: ① Clinical situation, physical sign and CT examination; ② results of experimental examination; ③ treatment and termination. RESULTS: A total of 17 infants with intracranial hemorrhage were involved in the final analysis. ① Clinical situation, physical sign and CT examination: All infants had pale facial expression and full or bossing anterior fontanelle. Among them, 13 infants had drowsiness or dysphoria, 12 rejective milk or emesis, 11 tic, 13 injection site hemorrhage, 2 gastrointestinal hemorrhage, 3 cerebral hernia, 11 high muscular tension and 6 cervical rigidity. CT examination demonstrated that most infants (88%, 15/17) had subarachnoid hemorrhage; 10 (59%, 10/17) had subdural hematoma; 8 (47%, 8/17) had cerebral parenchyma hemorrhage; few had intraventricular hemorrhage. In addition, results of CT examination also indicated that 17 infants had intracranial hemorrhage. Hemorrhage sites of 12 infants were equal to or more than 2, which was accounted for 70% (12/17); meanwhile, partial cases accompanied with a large area of focus of cerebral infarction or cerebral hypoxia-ischemia lesion. ② Results of experimental examination: There were 6 infants with elongation of prothrombin time, 5 with partial elongation of prothrombin time, 4 with decrease of serum calcium (1.69-2.25 mmol/L), 3 with increase of serum alkaline phosphatase, 3 with increase of total bilirubin, 3 with increase of direct bilirubin, and 3 with increase of indirect bilirubin. ③ Treatment and termination: After treatment, 12 infants were cured well, 3 improved, 1 given up and 1 died. Later, ten infants received CT re-examination at 3 months after treatment. The results indicated that 3 infants had simple subarachnoid hemorrhage and 4 had subarachnoid hemorrhage accompanying with subdural hematoma. Their focuses were absorbed well and not show as obvious sequela. One infant had subdural hematoma accompanying with subarachnoid hemorrhage, cerebral parenchyma hemorrhage, intraventricular hemorrage and cerebral hypoxia ischemia, and then, after hematom absorption, obvious cerebral malacia focus, hydrocephalus, brain atrophy and inferior accumulating fluid of dura mater were observed; 2 had subdural hematoma accompanying with subarachnoid hemorrhage and cerebral hypoxia ischemia, and then, after bleeding absorption, brain atrophy was changed remarkably; changes of hydrocephalus were observed in one infant. CONCLUSION: ① Symptoms of pale facial expression, full or bossing anterior fontanelle, drowsiness and dysphoria are observed in infants with delayed vitamin K deficiency . ② Experimental indexes demonstrate that prothrombin time and partial prothrombin time are prolonged, and numbers of infants having decrease of serum calcium are in the third place. ③ Poly-intracranial hemorrhage is a notable characteristic of CT examination. Partial infants who have poly-intracranial hemorrhage always accompany with cerebral hypoxia-ischemia lesion or cerebral infarction. Clinical situation and prognosis of infants who have a large area of intracranial hemorrhage and cerebral hypoxia-ischemia changes are poor; however, those of infants who have simple subarachnoid hemorrhage or combination of subarachnoid hemorrhage with subdural hematoma are well. ④ Effect of vitamin K on this kind of disease is well.

BACKGROUND: Delayed vitamin K deficiency is characterized by acute onset, severe illness and high fatality rate. 33%-50% survivors accompany with other various nervous system sequelas. Therefore, diagnosis and treatment of intracranial hemorrhage in time become a key factor for improving healing rate and reducing fatality rate and incidence of sequela. OBJECTIVE: To investigate the clinical situation, experimental characteristics, CT examination and terminative characteristics of infants with intracranial h...

BACKGROUND: Delayed vitamin K deficiency is characterized by acute onset, severe illness and high fatality rate. 33%-50% survivors accompany with other various nervous system sequelas. Therefore, diagnosis and treatment of intracranial hemorrhage in time become a key factor for improving healing rate and reducing fatality rate and incidence of sequela. OBJECTIVE: To investigate the clinical situation, experimental characteristics, CT examination and terminative characteristics of infants with intracranial hemorrhage induced by delayed vitamin K deficiency. DESIGN: Case analysis. SETTING: Department of Pediatrics, Zhongshan Hospital Affiliated to Xiamen University. PARTICIPANTS: A total of 17 infants with intracranial hemorrhage induced by delayed vitamin K deficiency aged 1-3 months including 11 boys and 6 girls were selected from Department of Pediatrics, Zhongshan Hospital Affiliated to Xiamen University from January 1994 to December 2005. All infants had drowsiness, rejective milk, spiting milk, gaze of both eyes, tic, coma, full anterior fontanelle, high muscular tension and cerebral hernia, etc. Experimental examination demonstrated that infants had anemia at various degrees; prothrombin time and partial thromboplastin time were prolonged; platelet count was normal. CT examination indicated that screenages of subarachnoid hemorrhage, subdural hematoma, cerebral parenchyma hemorrhage and intraventricular hemorrage were changed. Hemorrhage was stopped by the application of vitamin K. All patients provided informed consent. METHODS: ① Clinical situation and physical sign of infants were observed after hospitalization and scanned with rapid spiral CT scanning system. The thickness and average space of layers were 8-10 mm and the scanning time was 5 s with window width of 30-80 Hu and window position of 28-35 Hu. ② After hospitalization, four items of blood coagulation was measured with Futura meter and biochemical indexes of blood, such as serum calcium, serum alkaline phosphatase, total bilirubin, direct bilirubin and indirect bilirubin, were detected with Roche Modular PPI automatic biochemistry analyzer. ③ After hospitalization, infants were given 5-10 mg vitamin K1 for 3-5 days, and then, they were transfused with 10-15 mL/kg fresh plasma or whole blood for 1-3 times and received other relative therapies. Therapeutic effects were evaluated based on clinical diagnosis and criteria. MAIN OUTCOME MEASURES: ① Clinical situation, physical sign and CT examination; ② results of experimental examination; ③ treatment and termination. RESULTS: A total of 17 infants with intracranial hemorrhage were involved in the final analysis. ① Clinical situation, physical sign and CT examination: All infants had pale facial expression and full or bossing anterior fontanelle. Among them, 13 infants had drowsiness or dysphoria, 12 rejective milk or emesis, 11 tic, 13 injection site hemorrhage, 2 gastrointestinal hemorrhage, 3 cerebral hernia, 11 high muscular tension and 6 cervical rigidity. CT examination demonstrated that most infants (88%, 15/17) had subarachnoid hemorrhage; 10 (59%, 10/17) had subdural hematoma; 8 (47%, 8/17) had cerebral parenchyma hemorrhage; few had intraventricular hemorrhage. In addition, results of CT examination also indicated that 17 infants had intracranial hemorrhage. Hemorrhage sites of 12 infants were equal to or more than 2, which was accounted for 70% (12/17); meanwhile, partial cases accompanied with a large area of focus of cerebral infarction or cerebral hypoxia-ischemia lesion. ② Results of experimental examination: There were 6 infants with elongation of prothrombin time, 5 with partial elongation of prothrombin time, 4 with decrease of serum calcium (1.69-2.25 mmol/L), 3 with increase of serum alkaline phosphatase, 3 with increase of total bilirubin, 3 with increase of direct bilirubin, and 3 with increase of indirect bilirubin. ③ Treatment and termination: After treatment, 12 infants were cured well, 3 improved, 1 given up and 1 died. Later, ten infants received CT re-examination at 3 months after treatment. The results indicated that 3 infants had simple subarachnoid hemorrhage and 4 had subarachnoid hemorrhage accompanying with subdural hematoma. Their focuses were absorbed well and not show as obvious sequela. One infant had subdural hematoma accompanying with subarachnoid hemorrhage, cerebral parenchyma hemorrhage, intraventricular hemorrage and cerebral hypoxia ischemia, and then, after hematom absorption, obvious cerebral malacia focus, hydrocephalus, brain atrophy and inferior accumulating fluid of dura mater were observed; 2 had subdural hematoma accompanying with subarachnoid hemorrhage and cerebral hypoxia ischemia, and then, after bleeding absorption, brain atrophy was changed remarkably; changes of hydrocephalus were observed in one infant. CONCLUSION: ① Symptoms of pale facial expression, full or bossing anterior fontanelle, drowsiness and dysphoria are observed in infants with delayed vitamin K deficiency . ② Experimental indexes demonstrate that prothrombin time and partial prothrombin time are prolonged, and numbers of infants having decrease of serum calcium are in the third place. ③ Poly-intracranial hemorrhage is a notable characteristic of CT examination. Partial infants who have poly-intracranial hemorrhage always accompany with cerebral hypoxia-ischemia lesion or cerebral infarction. Clinical situation and prognosis of infants who have a large area of intracranial hemorrhage and cerebral hypoxia-ischemia changes are poor; however, those of infants who have simple subarachnoid hemorrhage or combination of subarachnoid hemorrhage with subdural hematoma are well. ④ Effect of vitamin K on this kind of disease is well.

 
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