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cirrhosis
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  肝硬化
     The Study on the Regulatory Mechanism of ET-1、NO、PGL_2 and C-Type Natriuretic Peptide on Hyperdynamic Circulation in Hepatic Cirrhosis and Portal Hypertension
     ET-1、NO、PGL_2、C型利钠肽对肝硬化门脉高压高动力循环调控机制的研究
短句来源
     Cloning and Expression of a Humanized M2 Triple Autoantigen and Its Application in Primary Biliary Cirrhosis
     人源M2三联体靶抗原的克隆表达及其在原发性胆汁性肝硬化中的应用
短句来源
     Study on Molecular Mechanism of the Effect of Liver Cirrhosis on Hepatocellular Carcinoma Carcinogenesis
     肝硬化在原发性肝癌的发生过程中所起作用的分子机制研究
短句来源
     Effects of ET-1 on Portal Hypertension in Hepatic Cirrhosis and the Mechanism of CNP、Radix Salviae Miltiorrhizae on ET-1-Mediated Contraction of HSCs
     ET—1对肝硬化门脉高压的作用及CNP、丹参对ET—1介导肝星状细胞收缩的调控机制
短句来源
     MRI and ~1H-MRS Study on Basal Ganglia Alterations in Patients with Liver Cirrhosis
     肝硬化患者颅脑MRI与~1H-MRS研究
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  肝硬变
     Cloning and Expression of PDC-E2 and PCD-E3BP in E.coli and Preliminary Study of Pathogenesis of Primary Biliary Cirrhosis
     PDC-E2和E3BP的克隆、表达、活性鉴定以及原发性胆汁性肝硬变发病机理的初步探讨
短句来源
     Study on the Relationship between Hepatitis C Virus and the Development of Chronic Hepatitis、Liver Cirrhosis、Primary Hepatic Carcinoma
     丙型肝炎病毒与慢性肝炎、肝硬变、原发性肝癌关系的研究
短句来源
     CLINICAL PRELIMINARY OBSERVATIONS ON THE RELATION BETWEEN LIVER-SPECIFIC MEMBRANE LIPOPROTEIN (LSP), CHRONIC HEPATITIS AND CIRRHOSIS
     临床初步观察特异性肝膜脂蛋白和慢性肝炎肝硬变的关系
短句来源
     Plasma insulin and C-peptide in cirrhosis of the liver
     肝硬变患者血浆胰岛素和C肽—肝脏摄取胰岛素减少在糖代谢障碍中的作用
短句来源
     ERYTHROCYTE INSULIN RECEPTORS IN HEPATIC CIRRHOSIS
     肝硬变病人的红细胞胰岛素受体
短句来源
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  肝硬化患者
     MRI and ~1H-MRS Study on Basal Ganglia Alterations in Patients with Liver Cirrhosis
     肝硬化患者颅脑MRI与~1H-MRS研究
短句来源
     Study on the Antigen-specific T Lymphocytes in Patients with Primary Biliary Cirrhosis
     原发性胆汁性肝硬化患者中抗原特异性T细胞的研究
短句来源
     Study on the CD4~+CD25~+ T Regulatory Cells in Patients with Primary Biliary Cirrhosis
     原发性胆汁性肝硬化患者中CD4~+CD25~+免疫调节性T细胞的研究
短句来源
     ORAL GLUCOSE TOLERANCE TEST AND PLASMA INSULIN LEVELS IN 20 CASES WITH CIRRHOSIS OF LIVER
     20例肝硬化患者的口服葡萄糖耐量试验和血浆胰岛素测定结果分析
短句来源
     The Change of Plasma C-AMP in Hepatitis B and Liver Cirrhosis Observed from Glucagon Tolerance Test
     乙型肝炎和肝硬化患者胰高血糖素负荷后血浆c—AMP的反应性
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  “cirrhosis”译为未确定词的双语例句
     Evaluation of Calcium-channel Blockers for Prevention of Recurrent Variceal Hemorrhage in Patients with Cirrhosis
     钙通道阻滞剂预防食管静脉曲张破裂出血的评价
短句来源
     RADIOIMMUNOASSAY OF GASTRIC MUCOSAL PROSTAGLANDIN E_2 IN PORTAL HYPERTENSIVE RATS WITH CIRRHOSIS
     门脉高压大鼠胃粘膜前列腺素E_2含量的放免法测定
短句来源
     CLINICAL FEATURES AND AUTOPSY FINDINGS OF KIDNEY INVOLVNENT IN 7 CASES OF CIRRHOSIS OF LIVER A RETROSPECTIVE STUDY
     肝硬化性肾小球肾炎7例临床与尸解
短句来源
     Changes of hyaluronic acid metabolism in rats of experimental liver injury and cirrhosis
     实验性肝纤维化发生发展过程中透明质酸代谢的变化
短句来源
     Morphometric Measurement of Collagen in Liver Tissue with Posthepatitis Liver Cirrhosis Portal Hypertension
     Morphometric Measurement of Collagen in Liver Tissue with Posthepatitis Liver Cirrhosis Portal Hypertension
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  cirrhosis
In treating hepatocellular carcinoma (HCC) patients with advanced cirrhosis, one of the most difficult problems is concomitant esophageal varices and hypersplenism.
      
The surgical treatment methods for cirrhosis patients complicated with portal hypertension are complicated.
      
From 1979 to 2005, 274 cirrhosis patients with portal hypertension who underwent the new treatment strategy were followed up to observe different clinical indexes, which were then compared with those of the traditional surgery treatment.
      
Postoperative hepatic insulin-like growth factor-1 (IGF-1) production may be severely disturbed in patients with liver cirrhosis.
      
Twenty-four patients with HCC with cirrhosis who underwent hepatectomy were randomly divided into two groups: a PN group (n = 12) and an rhGH + PN group (n = 12).
      
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Fifty-one male rats (body weights:200—350gm) were raised with laboratory diet, 39 of them were injected once a week with CCl_4 (0.015cc/100gm body weight) in li- quid paraffin through the mesenteric veins and 12 rats without treatment as controls. Rats with 1,2,3,and 4 injections were killed 7 days afterwards.Pieces of liver tissue were taken from the left anterior lobe of the rats from both experimental and control groups.Paraffin sections were prepared from materials fixed in:(1)Susa or Stieve's for Heidenhain's...

Fifty-one male rats (body weights:200—350gm) were raised with laboratory diet, 39 of them were injected once a week with CCl_4 (0.015cc/100gm body weight) in li- quid paraffin through the mesenteric veins and 12 rats without treatment as controls. Rats with 1,2,3,and 4 injections were killed 7 days afterwards.Pieces of liver tissue were taken from the left anterior lobe of the rats from both experimental and control groups.Paraffin sections were prepared from materials fixed in:(1)Susa or Stieve's for Heidenhain's Azan staining;(2)Regaud's for Bensley's acid fuchsin-methyl green staining of mitochondria;(3)cold Carnoy's for methyl green-pyronin staining of ribo- nucleic acid (RNA),with control sections extracted with 20% perchloric acid or 0.17 M NaCl at 56℃ for 2 hrs.;(4)Carnoy's for Yakovlev's ((?)) and Barrnett- Seligman's methods for protein-bound SH-group,with control sections blocked with 0.1M monoiodoacetic acid;(5)Formol-alcohol for Hotchkiss's periodic acid-Schiff's (PAS) reaction of glycogen,with control sections treated with salivary digestion;(6)cold ace- tone for alkaline phosphatase (ALP);and (7) 10% neutral formalin for frozen sections and Sudan Ⅲ testing for fats. After CCl_4-intoxication,during the development of cirrhosis,most of the parenchymal cells degenerated and finally became necrotic.Free fat droplets and hyperplastic fibrous tissues accumulated in the necrotic regions.The liver lobules thus were destroyed and blood sinuses were engorged with blood cells.Surviving cells showed compensative hyperplasia and were isolated into nodules by the developing fibrous tissues.Cirrhosis developed in all the rats treated with CCl_4. The parenchymal cells showed a progressive changes during the development of cirrhosis.There was an increase of fat droplets and a decrease of the number of mito- chondria in the cytoplasm.The mitochondria fragmented into granules or fused in a mass.There was a progressive decline in the content of RNA and glycogen,but an increase of the ALP activity.Those cells showing compensative hyperplasia enlarged in size,and their number of mitochondria,RNA and glycogen content progressively in- creased. Small-sized cells appeared in the necrotic regions among the fat droplets,necrotic cells and fibrous tissues.They decreased in number as the fibrous tissue developed. These cells contained mitochondria and showed positive reactions of PAS,RNA and ALP.Their origin was discussed and their relation to the formation of collagen fibers needs further investigation.

大白鼠肝在CCl_4中毒所引起肝硬化过程中,肝小叶结构被破坏。肝细胞中毒,坏死,脂肪游离增多。血窦因血流受阻而涨大。大部分肝细胞退变或坏死;部分残存而代偿性增生。结缔组织在坏变部分增生并包围肝小结而形成肝硬化。在肝硬化过程中,退变的肝细胞内脂滴增多。线粒体减少,成粒状或溶成一团。核糖核酸,糖元含量也随病变进展而减少。碱性磷酸酶反应增强。代偿性增生的肝细胞体积增大;肝细胞内线粒体增多,核糖核酸,糖元含量逐渐增多。在病变过程中出现冼多小型细胞。小型细胞出现在坏变的肝细胞区域,脂肪空泡附近及结缔组织内并随纤维的增加而减少。小型细胞含线粒体,核糖核酸,PAS反应及碱性磷酸酶反应均为阳性。文中讨论了肝细胞内细胞学和组织化学变化的意义;小型细胞的来源,与纤维形成的关系及纤维增生机制等问题。

The oligomycin-sensitive ATPase activity of mitochondria and submitochondrial particles isolated from human primary liver carcinoma (two patients) has been determined. For comparison, the ATPase activity of mitochondria from human cirrhosis liver, rat Walker sarcoma as well as from normal rat liver were also determined.It was found that the ATPase activity of mitochondria and submitochondrial particles of human primary liver carcinoma was very low (0.04~0.107μmoles·min~(-1)·mg protein~(-1)). The ATPase...

The oligomycin-sensitive ATPase activity of mitochondria and submitochondrial particles isolated from human primary liver carcinoma (two patients) has been determined. For comparison, the ATPase activity of mitochondria from human cirrhosis liver, rat Walker sarcoma as well as from normal rat liver were also determined.It was found that the ATPase activity of mitochondria and submitochondrial particles of human primary liver carcinoma was very low (0.04~0.107μmoles·min~(-1)·mg protein~(-1)). The ATPase activity of mitochondria from rat Walker sarcoma and from human cirrhosis liver was of the same order as that found in primary liver carcinoma, whereas in normal rat liver mitochondria the corresponding values were about ten times higher. Electron-microscopic observations, ANS binding estimations as well as experiments with 2, 4-dinitrophenol all showed that the mitochondria of human primary liver carcinoma is characterised by the loss of ATPase (F_1) from the inner mitochondrial membrane. By adding soluble F_1 isolated from rat liver mitochondria to the inner mitochondrial membrane of human primary liver carcinoma it was found that this soluble F_1 could bind to the inner mitochondrial membrane and that the resultant hybrid submitochondrial particles (SMP) had an ATPase activity much higher than that of the original SMP. The oligomycin-sensitive ATPase activity of hybrid SMP was also increased. Electron-microscopic examination of the negatively stained preparations showed that the hybrid SMP had the appearance of characteristic inner mitochondrial particles. The enhancement of ANS fluorescence intensity in the hybrid SMP over the original SMP was about 2.7 times. After binding of soluble F_1 of rat liver mitochondria to the SMP of human primary liver carcinoma the ATPase activity and the enhancement of ANS fluorescence of the hybrid SMP became resistant to cold inactivation. From these experimental results it may be concluded that the hybrid SMP had a number of properties similar to natural SMP. The significance of these findings in studies on the molecular evolution of biological membranes and on genetic control in carcinogenesis was discussed.

1974到1975年,我们用人原发性肝癌细胞的线粒体内膜进行ATP酶活力测定,结果证明人肝癌线粒体ATP酶活力极低(0.04~0.1微克分子/分/毫克蛋白)只相当于正常大鼠线粒体的1/10~1/25(0.49~1.07微克分子/分/毫克蛋白)。Walker肉瘤和人肝硬变组织的线粒体与人肝癌的酶活力相近。电镜负染标本观察证明肝癌线粒体内膜大部分失去特征性的直径为90(?)的ATP酶颗粒,表现为光滑膜。ANS萤光探针的发射萤光光谱测定和2,4-二硝基酚的激活试验均证明人肝癌细胞线粒体内膜的ATP酶大量消失是肝癌细胞的特征之一。用提取的大鼠肝线粒体ATP酶(F_1)与人肝癌线粒体内膜进行人工杂交重组,结果证明,重组后的杂交膜的ATP酶活力比人肝癌线粒体内膜高6~11倍;寡霉素敏感性也显著提高。电镜负染标本观察表明杂交膜出现了典型的直径为90(?)的ATP酶的颗粒形态;ANS萤光增强效应测定证明杂交膜的萤光强度比肝癌膜高276%(相对单位);0℃低温处理2小时,ANS萤光强度不变;酶活力在0℃2小时后,仍相当于原来活力的90%。此项试验结果证明杂交重组获得成功。鼠肝线粒体ATP酶与人肝癌线粒体内膜杂交后的特性表现了与...

1974到1975年,我们用人原发性肝癌细胞的线粒体内膜进行ATP酶活力测定,结果证明人肝癌线粒体ATP酶活力极低(0.04~0.1微克分子/分/毫克蛋白)只相当于正常大鼠线粒体的1/10~1/25(0.49~1.07微克分子/分/毫克蛋白)。Walker肉瘤和人肝硬变组织的线粒体与人肝癌的酶活力相近。电镜负染标本观察证明肝癌线粒体内膜大部分失去特征性的直径为90(?)的ATP酶颗粒,表现为光滑膜。ANS萤光探针的发射萤光光谱测定和2,4-二硝基酚的激活试验均证明人肝癌细胞线粒体内膜的ATP酶大量消失是肝癌细胞的特征之一。用提取的大鼠肝线粒体ATP酶(F_1)与人肝癌线粒体内膜进行人工杂交重组,结果证明,重组后的杂交膜的ATP酶活力比人肝癌线粒体内膜高6~11倍;寡霉素敏感性也显著提高。电镜负染标本观察表明杂交膜出现了典型的直径为90(?)的ATP酶的颗粒形态;ANS萤光增强效应测定证明杂交膜的萤光强度比肝癌膜高276%(相对单位);0℃低温处理2小时,ANS萤光强度不变;酶活力在0℃2小时后,仍相当于原来活力的90%。此项试验结果证明杂交重组获得成功。鼠肝线粒体ATP酶与人肝癌线粒体内膜杂交后的特性表现了与天然线粒体内膜的ATP酶的一系列相似的特性。讨论了ATP酶复合体杂交重组试验在探索肝癌发生与细胞中两个遗传系统控制的可能关系问题。

The normal values of the serum γ-Gt activity in 110 healthy adultdonors were less than 50 units and those of the 9 newborns ranged from107 to 334 units. The γ-Gt of 48 patients suffermg from carcinoma of the liver increasedby 100%, those with γ-Gt over 200 units made up 79.5%. The α-FPdetermination and radioisotopic scanning simultaneously performed in 37cases were positive in 70.6% and 92.5% respectively. In comparison withγ-Gt determination it appeared that the latter was more sensitive thanthe formers. γ-Gt...

The normal values of the serum γ-Gt activity in 110 healthy adultdonors were less than 50 units and those of the 9 newborns ranged from107 to 334 units. The γ-Gt of 48 patients suffermg from carcinoma of the liver increasedby 100%, those with γ-Gt over 200 units made up 79.5%. The α-FPdetermination and radioisotopic scanning simultaneously performed in 37cases were positive in 70.6% and 92.5% respectively. In comparison withγ-Gt determination it appeared that the latter was more sensitive thanthe formers. γ-Gt of 1 patient of carcinoma of the pancreas was 1, 098 units,3 casesof carcinoma of the billiary tract ranged from 76.9 to 663 units. Therise of γ-Gt of these patients was closely related to the obstruction ofbilliary tract and/or metastasis into the liver. There were 13 cases of hepatitis with jaundice, of which 12 caseshad a rise of γ-Gt (66.5-446 units), but one case of severe hepatitisshowed only 50 units. Among 15 patients of cirrhosis of the liver,11 had a rise of γ-Gt, 4 of them returned to normal level in the latestage. One patient of acute schistosomiasis showed high levels of γ-Gt, butupon treatment it gradually decreased to normal. Among 15 cases of billiary tract diseases 13 showed rise in γ-Gt(101.5-516 units), while 2 were normal. In 2 patients with acute billiarydiseases γ-Gt was originally highly raised, later decreased gradually after the drainage. of common bile ducts. It therefore seemed that the rise ofγ-Gt was related to the obstruction of the billiary tract. To sum up, the determination of γ-Gt in serum was definitely valu-able for the diagnosis of diseases of the liver and billiary tract, espe-cially for primary as well as secondary carcinoma of the liver.

本文作者报告用自行合成的γ—L—谷氨酸—α萘胺为基质用改良法测定110名健康献血员、9名新生儿和167例患者血清γ—谷氨酰转肽酶(γ—GT)的结果。作者认为血清γ—GT的测定对肝胆系统疾病,特别是肝癌有一定的诊断价值。

 
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