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   p 53 mrna 的翻译结果: 查询用时:0.01秒
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口腔科学
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p mrna
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  p53基因
     Expression of p53 mRNA was inhibited and expression of bcl 2 mRNA was enhanced in P group.
     P组p5 3基因mRNA表达显著低于NP组 ,bcl 2基因mRNA表达在P组显著高于NP组。
短句来源
     The expression level of DNA polβ mRNA was positively correlated with Fas mRNA (r=0.611,P<0.05), however the expression of p53 mRNA was not correlated with DNA polβ mRNA or Fas mRNA.
     脑肿瘤组织中DNApolβ与Fas基因mRNA的表达呈正相关((r=0.611,P<0.05)),与p53基因mRNA的表达无关; p53与Fas基因mRNA的表达亦无关。
短句来源
     Results: Higher expression of DNA polβ mRNA, Fas mRNA and lower expression of p53 mRNA in all of brain tumors were found compared with those in normal adjacent cancer tissue (P<0.01).
     结果:与相应瘤旁正常脑组织相比,脑胶质瘤及良性脑肿瘤组织中DNApolβ、Fas基因mRNA高表达,p53基因mRNA低表达(P均<0.01)。
短句来源
     p53 mRNA Expression in Non-Hodgkin's Lymphomas
     非霍奇金淋巴瘤中p53基因mRNA的表达
短句来源
     Conclusion: The high expression of DNA polβ mRNA, Fas mRNA and low expression of p53 mRNA may be related to the carcinogenesis and development of brain tumors and their malignant degree.
     结论:p53基因mRNA低表达及DNApolβ和Fas基因mRNA高表达与脑肿瘤的发生发展及恶性程度有关。
短句来源
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  p53基因mrna
     Expression of p53 mRNA was inhibited and expression of bcl 2 mRNA was enhanced in P group.
     P组p5 3基因mRNA表达显著低于NP组 ,bcl 2基因mRNA表达在P组显著高于NP组。
短句来源
     The expression level of DNA polβ mRNA was positively correlated with Fas mRNA (r=0.611,P<0.05), however the expression of p53 mRNA was not correlated with DNA polβ mRNA or Fas mRNA.
     脑肿瘤组织中DNApolβ与Fas基因mRNA的表达呈正相关((r=0.611,P<0.05)),与p53基因mRNA的表达无关; p53与Fas基因mRNA的表达亦无关。
短句来源
     Results: Higher expression of DNA polβ mRNA, Fas mRNA and lower expression of p53 mRNA in all of brain tumors were found compared with those in normal adjacent cancer tissue (P<0.01).
     结果:与相应瘤旁正常脑组织相比,脑胶质瘤及良性脑肿瘤组织中DNApolβ、Fas基因mRNA高表达,p53基因mRNA低表达(P均<0.01)。
短句来源
     p53 mRNA Expression in Non-Hodgkin's Lymphomas
     非霍奇金淋巴瘤中p53基因mRNA的表达
短句来源
     Conclusion: The high expression of DNA polβ mRNA, Fas mRNA and low expression of p53 mRNA may be related to the carcinogenesis and development of brain tumors and their malignant degree.
     结论:p53基因mRNA低表达及DNApolβ和Fas基因mRNA高表达与脑肿瘤的发生发展及恶性程度有关。
短句来源
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  p53mrna表达
     Besides, the expressions of Rb and p53 mRNA in osteosarcomas showed a positive correlation(r's = 0. 462, P < 0. 02).
     此外,骨肉瘤中Rb和p53mRNA表达呈正相关(r’s=0.462,P<0.02)。
短句来源
     (Bcl2 mRNA) expression of PBMCs in patients was significantly lower than that of healthy(volunteers)((0.11±0.09) vs.0.19±0.06,P<0.05),while p53 mRNA expression was higher in patients than that of(healthy)(volunteers) (0.45±0.09 vs.0.25±0.12,P<0.05).
     M ODS组PBM C s的B cl 2 mRNA表达量明显低于正常对照组(0.11±0.09比0.19±0.06,P<0.05); p53 mRNA表达量明显高于正常对照组(0.45±0.09比0.25±0.12,P<0.05)。
短句来源
     Effects of 1 (2,6 dimethylphenoxy) 2 (3,4 dimethoxyphenyl ethylamino)propane hydrochloride on expression of N ras and P53 mRNA in left ventricle of cardiac hypertrophy rats 1
     1-(2,6-二甲基苯氧基)-2-(3,4-二甲氧基苯乙氨基)丙烷盐酸盐对心肌肥厚大鼠左心室N-ras,P53mRNA表达的影响
短句来源
     The relationship between p53 mRNA and caspase 3 mRNA was positive correlation (r= 0.890) .
     p53mRNA表达与caspase3mRNA表达正相关(r=0.890);
短句来源
     PC prevented the PC12 cells nuclei from shrinkage, condensation and cleavage induced by Aβ 25-35. PC decreased the expressions of P53 mRNA and P53 protein, and increased the expression of bcl-2 mRNA and Bcl-2 protein.
     结果 不同剂量PC预处理PC12细胞 1h可剂量依赖性对抗Aβ25-35引起的凋亡,提高细胞的存活率,减少Aβ25-35引起的核固缩,凝聚和碎裂,降低P53mRNA表达以及P53蛋白表达,增加bcl 2mRNA表达以及Bcl 2蛋白表达。
短句来源
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  “p 53 mrna”译为未确定词的双语例句
     The p53 mRNA was significantly negatively correlated with bcl-2 mRNA ( r=-0.9648,P<0.01).
     p53 mRNA和bcl-2mRNA二者的表达之间存在明显的负相关性(r=-0.9648,P<0.01)。
短句来源
     There was obviously negative correlation between p53 mRNA expression and bcl-2 mRNA expression(r=-0.9648, P<0.001).
     p53mRNA和bcl-2mRNA二者的表达之间存在着明显的负相关性(r=-0.9648,P<0.001)。
短句来源
     Significantly higher expressions of p53 mRNA, caspase 3 mRNA and p53 protein at 3-9 μmol·L -1 Cr(Ⅵ) were observed(P< 0.05) .
     3~9μmol·L-1Cr(Ⅵ)处理组细胞p53mRNA,caspase3mRNA,p53蛋白表达水平明显高于对照组(P<0.05)。
短句来源
     Results: 1. The level of p53 mRNA is higher in NPC group than that in controls, the difference is statistically significant (t = 2.14, P < 0.05).
     结果:(1)NPC组p53mRNA的水平较高,差异有统计学意义(t=2.14,P<0.05);
短句来源
     Comparative study on P53 mRNA in colorectal tumor paraffin embedded samples by RT PCRIS
     大肠肿瘤石蜡标本P53 mRNA RT-PCRIS比较研究
短句来源
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  p mrna
However, pretreatment with FK506 and V10367 significantly prevented any increase in this ratio or p53 mRNA expression.
      
H2O2 increased both the ratio of bax/bcl-2 and the p53 mRNA expression.
      
No correlation was found between LOH status and the levels of p53 mRNA and protein.
      
Normal sized p53 mRNA and protein were present in all the tumor samples; however, their levels were 1.5- to 4-fold higher compared to the control suggesting deregulated p53 pathway in these tumors.
      
The level of p53 mRNA, p53 protein and Ser 392 phosphorylated p53 protein were also analyzed in all tumor samples.
      
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The expression of glutathione S transferase placental (GST P) mRNA was observed in hepatic preneoplastic lesions induced with diethylnitrosamine (DEN) with in situ hybridization in rats. It was found that GST P mRNA mainly expressed in altered foci and some of oval cells in hepatic preneoplastic lesions and the extent of its expression was different among foci or/and positive cells in the same foci whereas no expression was observed in normal and regenerating hepatic tissue. Our findings...

The expression of glutathione S transferase placental (GST P) mRNA was observed in hepatic preneoplastic lesions induced with diethylnitrosamine (DEN) with in situ hybridization in rats. It was found that GST P mRNA mainly expressed in altered foci and some of oval cells in hepatic preneoplastic lesions and the extent of its expression was different among foci or/and positive cells in the same foci whereas no expression was observed in normal and regenerating hepatic tissue. Our findings indicate that cells in altered foci and oval cells may be preneoplastic cells in the experimental hepatocellular carcinoma at the molecular level.

应用原位杂交技术,观察了二乙基亚硝胺(DEN)诱发大鼠肝癌前病变组织中胎盘型谷胱甘肽S转移酶(GST-P)mRNA的表达。结果显示,GST-PmRNA主要在癌前病变肝组织中的变异灶及灶外卵圆形细胞内表达,且在变异灶间或和同一灶内阳性细胞间表达程度不尽一致,而正常肝、再生肝组织中未见其表达。提示在分子水平上变异灶细胞及卵圆型细胞可能成为实验性肝癌的癌前期细胞

Digoxigenin-labelled P53and c-myc cDNA probes were used to study gallbladder carcinoma by in situhybridization on the paraffin sections for mRNA expression in 21 cases.The resillts were as follows:c-myc and P53mRNAexpressiOn were both located in the nuclei.The positive incidences of expression of both c-myc and P53mRNA in gallbladderwere 66.6%and 5 7.1%respectivelyNo expression in the normal tissue surrounding the carcinoma.The strength of theexpression of c-myc and P53mRNA...

Digoxigenin-labelled P53and c-myc cDNA probes were used to study gallbladder carcinoma by in situhybridization on the paraffin sections for mRNA expression in 21 cases.The resillts were as follows:c-myc and P53mRNAexpressiOn were both located in the nuclei.The positive incidences of expression of both c-myc and P53mRNA in gallbladderwere 66.6%and 5 7.1%respectivelyNo expression in the normal tissue surrounding the carcinoma.The strength of theexpression of c-myc and P53mRNA in gallbladder were related to the size of primary tumors,metastasis of lymph node anddistance metastasis,However,there was no relationship between the expression of p"mRNA and the degree of carcinoma celldifferentiation but c-myc mRNA.

应用地高辛标记的P ̄(53)、c-myccDNA探针对21例胆囊癌石腊切片上的mRNA进行原泣杂交。结果表明c-myc和P ̄(53)mRNA的表达均位于细胞核内。c-myc和P ̄(53)mRNA在胆囊癌的表达率分别为66.6%和57.1%,在癌周正常组织中没有c-myc、P ̄(53)mRNA的表达。c-myc、P ̄(53)mRNA表达的强度和胆囊癌的原发肿瘤大小,有无淋巴结转移及远处转移有关。分期越晚,表达率越高。有淋巴结转移者高于无转移者。P ̄(53)mRNA表达的阳性率和胆囊癌的分化程度无关;而c-mycmRNA的表达和分化程度有关,分化越差,表达率越高。

CD34 + celis contain hemopoietic stem progenitor celis, vvhich can be induced to differentiate and proli-ferate by interleukine-3 (IL-3), granulocvte-macrophage colonj'-stimulating factor (GM-CSF) and granulocyte-CSF (G-CSF) etc. Whether normai CD34+ celis participate in the production of any cytokines remains uncertain. We inves-tigated IL-lp, IL-3, IL-6, IL-4, GM-CSF, TNF-a, TNF-P and c-kit gene expression by using reverse transcription-polvmerase chain reaction {RT-PCR) in flow cvtometric sorted normai...

CD34 + celis contain hemopoietic stem progenitor celis, vvhich can be induced to differentiate and proli-ferate by interleukine-3 (IL-3), granulocvte-macrophage colonj'-stimulating factor (GM-CSF) and granulocyte-CSF (G-CSF) etc. Whether normai CD34+ celis participate in the production of any cytokines remains uncertain. We inves-tigated IL-lp, IL-3, IL-6, IL-4, GM-CSF, TNF-a, TNF-P and c-kit gene expression by using reverse transcription-polvmerase chain reaction {RT-PCR) in flow cvtometric sorted normai bone marrow CD34 + celis. We also analyzed the effects of recombinant IL-1P, IL-3, IL-7, GM-CSF, PIXY-321 (fusion protein IL-3/GM-CSF) and stem celi factor (SCF) on the regulation of these cvtokines. It was found that freshly sorted CD34 + celis have high Ievels of IL-1P, c-kit mRNA and low Ievels of IL-3, TNF-a and TNF-P mRNA. After stimnlating with exogenous cvtokines, the Ievels of IL-1P, TNF-a and TNF-p mRNA were upregulated by all stimuli to various extent. GM-CSF mRNA became positive only under the effect of IL-7 stimulation. IL-7 also significantly enhanced IL-6 gene expression. None of these factors have a significant effect on c-kit or IL-3 transcription.

CD34~+细胞包含有造血干/祖细胞,而造血干/祖细胞能被IL-3,GM-SCF,G-CSF等诱导分化与增殖。正常的CD34~+细胞是否参与一些细胞因子的产生尚未被阐明。我们采用逆转录-聚合酶链反应分析了经流式细胞仪分选出的正常骨髓CD34~+细胞IL-1β,IL-3,IL-4,IL-6,GM-CSF,TNF-α,TNF-β及c-kit基因的表达,并且分析了重组IL-1β,IL-3,IL-7,GM-CSF,PIXY321(IL-3/GM-CSF融合蛋白)及干细胞因子(SCF)对这些细胞因子的调节作用。结果发现新分选出的CD34~+细胞表达较高水平的IL-1β,c-kit mRNA及低水平的IL-3,TNF-α,TNF-βmRNA。经外源性的细胞因子刺激后,IL-1β,TNF-α及TNF-βmRNA均有不同程度的上调,唯IL-7能使GM-CSF mRNA的表达变为阳性,IL-7亦能显著增强IL-6基因的表达。所有这些细胞因子对c-kit及IL-3基因表达均无明显作用。

 
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