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ischemic damage     
相关语句
  缺血损伤
     CONCLUSION:Na +-K +-ATPase is not sensitive to ischemic damage,but sensitive to reperfusion damage.
     结论 :脑组织Na+ -K+ -ATPase对缺血损伤不敏感 ,对再灌注损伤敏感 ;
短句来源
     ② After the last gastric perfusion for 1 hour the rats in the isoproterenol group and fenofibrate group were given 5 mg/kg isoproterenol with intraperitoneal injection to replicate acute myocardial ischemic damage models.
     ②末次灌胃后1h异丙肾上腺素组和非诺贝特组给予异丙肾上腺素5m g/kg腹腔注射复制急性心肌缺血损伤模型;
短句来源
     The expression of HSP70 could hardly be observed in hippocampal CA1 after HI in neonatal rats, the hippocampal CA1 district neuron showed severe ischemic damage.
     结果HI后新生大鼠海马CA1区仅能检出少量阳性HSP核蛋白(仅在核内表达的蛋白),海马CA1区神经元呈较严重的缺血损伤性改变。
短句来源
     Results The hippocampic MCa,CaM,MDA contents were significantly higher in NS group (P< 0.01 ),the hippocampic CA 1 district neuron showed severe ischemic damage. The changes of biochemistry and pathology had more remarkable improvement in GM 1 group than NS one.
     结果 NS组海马组织MCa、CaM、MDA含量显著升高(P< 0.01),海马CA1 区神经元呈较严重缺血损伤性改变,而GM1 组较NS组生化和病理变化明显改善。
短句来源
     To investigate the association between the plasma concentration of NT-ProBNP, ANP and the degree of ischemic damage after the first acute myocardial infarction (AMI).
     研究初发急性心肌梗死患者急性期血浆NT—ProBNP、ANP浓度与早期心脏缺血损伤程度的关系。
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  缺血性损伤
     13.1%~26.3% cases are complicated with pericarditis,uremic myocardiosis and myocardial ischemic damage after hemodialysis.
     13.1%~ 2 6 .3%并发透析后心包炎、尿毒症性心肌病和心肌缺血性损伤等心脏并发症
短句来源
     Conclusions FAP-1 in cerebal tissue of cerebral ischemic rats is over expression. It can show ischemic damage of brain.
     结论缺血性脑梗死大鼠脑组织的FAP-1表达明显增高,其表达增高可以提示脑组织有缺血性损伤
短句来源
     AIM: To observe the activation of peroxisome proliferate-activated receptor α(PPARα) by fenofibrate of agonists of PPARα after pre-application, and the effects on tumor necrosis factor alpha (TNF-α) in acute myocardial ischemic damage rats.
     目的观察预先应用过氧化物酶体增殖物激活受体α的激动剂非诺贝特活化过氧化物酶体增殖物激活受体α,对急性心肌缺血性损伤大鼠血清肿瘤坏死因子α的影响。
短句来源
     Blocking PS 1 expression could exaggerate the ischemic damage, destruct the neurons structure and cause them death. It indicates that PS 1 gene may play an important role in Alzhemer's disease (AD).
     结论 PS 1基因在脑缺血时起脑保护作用 ,如果抑制PS 1表达 ,则加重缺血性损伤 ,导致神经元结构改变 ,神经细胞内嗜银样变和突起迂曲以及神经细胞丢失 ,提示PS 1基因在阿尔茨海默病 (AD)中的作用机制值得进一步探讨
短句来源
     The results showed that ischemic ARF involved ischemic damage and reperfusion injury.
     结果,缺血性急性肾功能衰竭由缺血性损伤和再灌注损伤两部分组成。
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  缺血性损害
     Rabbits in GbE group had significantly decreased blood platelet aggrgation(P<0.01) and TXA2,ET(P<0.01), significantly increased 6-keto-PGF1a(P<0.01)and obviously alleviated ischemic damage of cardiac muscle,compared with rabbits in control group.
     GbE组与ARF组比较,血小板聚集率则明显降低(P<0.01),TXA2、ET明显降低(P均<0.01),6-Keto-PGF1a明显升高(P<0.01),心肌组织缺血性损害明显减轻。
短句来源
     Results:13 cases are identified as simple subarachnoid hemorrhage (SAH),5 cases as intracerebral hemorrhage (ICH),3 cases as intraventricular hemorrhage (IVH),4 cases as subependymal hemorrhage (SEH),5 cases as subdural hemorrhage (SDH),9 cases as hypoxic ischemic damage (HID),and 2 cases have normal CT findings.
     结果 :单纯蛛网膜下腔出血 13例 ,脑实质出血5例 ,脑室内出血 3例 ,室管膜下出血 4例 ,硬膜下出血 5例 ,单纯脑缺氧缺血性损害 9例 ,正常CT表现 2例。
短句来源
     Results 13 cases are identified as simple subarachnoid hemorrhage (SAH) ,2 cases as intracerebral hemorrhage (ICH) ,1 cases as intraventricular hemorrhage (IVH) , 5 cases as subdural hemorrhage (SDH) ,24 cases as hypoxic ischemic damage (HID) ,and 4 cases have normal CT findings.
     结果 单纯蛛网膜下腔出血 13例 ,脑实质出血 2例 ,脑室内出血 1例 ,硬膜下出血 5例 ,单纯脑缺氧缺血性损害 2 4例 ,新生儿败血症 1例 ,正常CT表现 4例。
短句来源
     It may play a beneficial role in salvaging neuronal cells from ischemic damage to intervene the mechanism of apoptosis as soon as possible up to 24 h after reperfusion.
     再灌流后24h内尽早干预细胞凋亡机制的发生仍可能更进一步挽救缺血性损害的神经细胞。
短句来源
     Conclusion:Most of high risk newborn infants have intracranial hemorrhage or hypoxic/ischemic damage. So it is sugested that CT examination should be of great benefit in the diagnosis.
     结论 :高危新生儿大部分存在颅内出血或脑缺氧缺血性损害 ,CT检查对这些病变有极高的诊断价值。
短句来源
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  缺血样损伤
     Myocardial ischemic damage after acute stroke
     卒中后的心肌缺血样损伤
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      ischemic damage
    In the myocardium without signs of ischemic damage, stimulation of the α1-receptors caused a slowly developing single-phase positive inotropic response.
          
    Structural ischemic damage was more pronounced in patients with severe hypertrophy and structural reconstitution was delayed.
          
    Verapamil, therefore, exerts a partial degree of protection of the ischemic myocardium but exerts some other effects which do not help prevent the spread of ischemic damage in the myocardium.
          
    Dexamethasone also reduced the extent of ischemic damage as assessed by a nitro-blue tetrazolium staining technique, providing anatomic verification of the reduced ischemic damage.
          
    The role of lipid peroxidation in pathogenesis of ischemic damage and the antioxidant protection of the heart
          
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    Pyeloureterocalycoplasties were performed in 12 patients with complicated renal stone and one case with concomitant ureteropelvic junction stricture. The results were satisfactory with successful removal of stones, patent anastomosis, adequate drainage of the urine, reduced hydronephrosis and improved renal function. We advocated that the matress sutures should be applied first over the planned line of parenchymal wedge resection, so that the occlusion of renal redical could be avoided; or the cut edges of the...

    Pyeloureterocalycoplasties were performed in 12 patients with complicated renal stone and one case with concomitant ureteropelvic junction stricture. The results were satisfactory with successful removal of stones, patent anastomosis, adequate drainage of the urine, reduced hydronephrosis and improved renal function. We advocated that the matress sutures should be applied first over the planned line of parenchymal wedge resection, so that the occlusion of renal redical could be avoided; or the cut edges of the renal parenchyma were sutured continously and the pedical clamp released as early as possible. Then, the stones could be removed comfortably and repairment carried on skillfully without further renal ischemic damage.

    本文报告采用肾盂输尿管下肾盏切开成形术治疗复杂的肾结石共12例。手术时不阻断肾血流者先褥式缝扎止血,再切开肾下极实质;阻断肾血流者宜先行实质创缘缝扎止血,恢复肾血流后再从容取石可缩短肾缺血时间。8例术后3~6个月排泄性尿路造影显示出良好低位引流,积水减轻,肾功能改善,无一例发生吻合部狭窄。复杂的肾结石,以及合并有肾盂输尿管联合部狭窄病例为本手术的适应证。

    In order to study the reperfusion injury of ischemic intestine, the SMA of rabbit was occluded for 1h, or perfused with 35 mmHg low pressure for 2h, then reperfused with blood from carotid artery via pressure pump at 90 mmHg for 2h. The results showed that severe tissue damage occurred progressively after reperfusion of the ischemic intestine, such as progressive increase of ACP, lactic acid and magnesium of plasma, hemorrhage of intestinal mucosa, progressive necrosis and edema of intestinal villi, decrease...

    In order to study the reperfusion injury of ischemic intestine, the SMA of rabbit was occluded for 1h, or perfused with 35 mmHg low pressure for 2h, then reperfused with blood from carotid artery via pressure pump at 90 mmHg for 2h. The results showed that severe tissue damage occurred progressively after reperfusion of the ischemic intestine, such as progressive increase of ACP, lactic acid and magnesium of plasma, hemorrhage of intestinal mucosa, progressive necrosis and edema of intestinal villi, decrease of arterial blood pressure and ischemic damage of myocardia, etc. The reperfusion injury was much severe than that occurred after 3h of sustained occlusion of SMA. The significance of reperfusion injury of ischemic intestine in the pathogenesis of shock was discussed.

    为研究缺血小肠的再灌注损伤,阻断家兔SMA血流1小时或35mmHg低压灌流SMA2小时,然后以90mm Hg恒压再灌注2小时。结果发现缺血小肠再灌注后出现逐渐加重的组织损伤,表现为血浆ACP活性、乳酸和镁进行性增加,小肠粘膜出血,小肠绒毛坏死和组织水肿进行性加重,以及体动脉压降低,心肌缺血损伤等。这种再灌注损伤,甚至比小肠持续缺血3小时者更严重。本文对缺血小肠再灌注损伤在休克发病学中的意义进行了讨论。

    Ischemic and post-ischemic injury was made by 45 minute reperfusion after 15 and 45 minute myocardial ischemia with blocking and reopening the left ventricular branch of coronary artery in vivo in 21 rabbits. Sections from both ischemic and non-ischemic areas were stained with Alizarin Red S (ARS) since calcium accumulation in cytoplasms and mitochondria was a mark of irreversible ischemic damage. Examinations of HE staining and electrocardiogram (ECG) were also performed.

    本文对缺血组织进行了水溶性莤素红(Alizarin Red S以下简称ARS)钙组织化学染色观察。此外还作了HE染色和心电图(ECG)检查。ARS染色阳性说明细胞内钙浓度异常增高。结果:正常对照组和缺血损伤组非缺血区对照ARS染色均为阴性;缺血区中,15分钟组9例动物中的1例和45分钟组12例动物中的7例为ARS阳性。上述各组的系列切片HE染色均未见坏死迹象。ECG检查发现8只动物有坏死性心电改变,出现异常Q波或QS波,恰与ARS(+)动物呈严格的对应关系。这些结果证明,ARS染色是一项敏感可靠的判定不可逆性缺血再灌注损伤的指标,它能够及时在细胞原位反映出钙增多这一致死性代谢改变。

     
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