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ischemic damage
相关语句
  缺血性损伤
    13.1%~26.3% cases are complicated with pericarditis,uremic myocardiosis and myocardial ischemic damage after hemodialysis.
    13.1%~ 2 6 .3%并发透析后心包炎、尿毒症性心肌病和心肌缺血性损伤等心脏并发症
短句来源
    The results showed that ischemic ARF involved ischemic damage and reperfusion injury.
    结果,缺血性急性肾功能衰竭由缺血性损伤和再灌注损伤两部分组成。
短句来源
    Myocardial ischemic damage was found in 8 cases.
    3 10例 (2 6 .3% )符合尿毒症性心肌病 ,8例 (2 1.1% )疑有心肌缺血性损伤等。
短句来源
  “ischemic damage”译为未确定词的双语例句
    Role of Inositol 1,4,5-triphosphate during Acute Renal Ischemic Damage and Reperfusion Injury in the Rabbit Models
    三磷酸肌醇在兔急性肾缺血及再灌流损伤中的作用
短句来源
    Acute renal ischemic damage and reperfusion injury models in rabbits were established. Concentratioh of IP3,total calcium, and [Ca(2+)]i in renal histocytes were observed.
    用新西兰大白兔制作急性肾缺血及再灌流损伤模型,观察肾组织细胞内三磷酸肌醇(IP3)、细胞内总钙及胞浆游离钙([Ca(2+)]i)浓度变化。
短句来源
    Using cable tie to compress renal parenchyma makes it possible to reduce the time of renal artery clamping and thus to protect the kidney from ischemic damage.
    区域性肾实质血流阻断法,能缩短肾动脉阻断时间,减少患肾功能热缺血损害。
短句来源
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  ischemic damage
In the myocardium without signs of ischemic damage, stimulation of the α1-receptors caused a slowly developing single-phase positive inotropic response.
      
Structural ischemic damage was more pronounced in patients with severe hypertrophy and structural reconstitution was delayed.
      
Verapamil, therefore, exerts a partial degree of protection of the ischemic myocardium but exerts some other effects which do not help prevent the spread of ischemic damage in the myocardium.
      
Dexamethasone also reduced the extent of ischemic damage as assessed by a nitro-blue tetrazolium staining technique, providing anatomic verification of the reduced ischemic damage.
      
The role of lipid peroxidation in pathogenesis of ischemic damage and the antioxidant protection of the heart
      
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Pyeloureterocalycoplasties were performed in 12 patients with complicated renal stone and one case with concomitant ureteropelvic junction stricture. The results were satisfactory with successful removal of stones, patent anastomosis, adequate drainage of the urine, reduced hydronephrosis and improved renal function. We advocated that the matress sutures should be applied first over the planned line of parenchymal wedge resection, so that the occlusion of renal redical could be avoided; or the cut edges of the...

Pyeloureterocalycoplasties were performed in 12 patients with complicated renal stone and one case with concomitant ureteropelvic junction stricture. The results were satisfactory with successful removal of stones, patent anastomosis, adequate drainage of the urine, reduced hydronephrosis and improved renal function. We advocated that the matress sutures should be applied first over the planned line of parenchymal wedge resection, so that the occlusion of renal redical could be avoided; or the cut edges of the renal parenchyma were sutured continously and the pedical clamp released as early as possible. Then, the stones could be removed comfortably and repairment carried on skillfully without further renal ischemic damage.

本文报告采用肾盂输尿管下肾盏切开成形术治疗复杂的肾结石共12例。手术时不阻断肾血流者先褥式缝扎止血,再切开肾下极实质;阻断肾血流者宜先行实质创缘缝扎止血,恢复肾血流后再从容取石可缩短肾缺血时间。8例术后3~6个月排泄性尿路造影显示出良好低位引流,积水减轻,肾功能改善,无一例发生吻合部狭窄。复杂的肾结石,以及合并有肾盂输尿管联合部狭窄病例为本手术的适应证。

Ischemic acute renal failure(ARF) models in rabbits were established.the Changes of renal tubule were ultrapathomorphologically observed and serum lipid peroxide(LPO) concentration and superoxide disutase(SOD) activity in red cells were determined.The results showed that ischemic ARF involved ischemic damage and reperfusion injury.The structural change and functional failure of mitochondria in tubular epithelial cells were early pathomorphological features.The tubular injury was further aggravated during...

Ischemic acute renal failure(ARF) models in rabbits were established.the Changes of renal tubule were ultrapathomorphologically observed and serum lipid peroxide(LPO) concentration and superoxide disutase(SOD) activity in red cells were determined.The results showed that ischemic ARF involved ischemic damage and reperfusion injury.The structural change and functional failure of mitochondria in tubular epithelial cells were early pathomorphological features.The tubular injury was further aggravated during the reperfusion,which bore a close relationship to oxygen free radical injury.The detachment of whole epithelial cells from tubular basement-membrane was the manifestation of the renal tubular injury in ischemic ARF.

本文用新西兰大白兔制作缺血性急性肾功能衰竭模型。动态观察肾小管的超微病理变化并测定血浆脂质过氧化物(LPO)含量,红细胞内超氧化物歧化酶(SOD)活性。结果,缺血性急性肾功能衰竭由缺血性损伤和再灌注损伤两部分组成。肾小管上皮细胞内线粒体的结构变化和功能障碍是缺血损伤的早期病理特征。再灌注时肾小管损伤进一步加重且与氧自由基损伤密切相关。完整的上皮细胞从肾小管基底膜上的脱离是缺血性急性肾功衰时肾小管损伤的表现之一。

Acute renal ischemic damage and reperfusion injury models in rabbits were established. Concentratioh of IP3,total calcium, and [Ca(2+)]i in renal histocytes were observed.The results showed that concentrations of intracellular IP3 [(430.3±46.8),(297.9±54.3) min(-1),P<0.01] and [Ca(2+)]i [(212.5±43.5),(106.1±15.5) nmol/L,P<0.01] were increased in ischemic groups compared with the contrasted group,concentration of intracellular total calcium [(398.6±32.3),(385.0±32.4)μmol/L,P>0.05] was not increased...

Acute renal ischemic damage and reperfusion injury models in rabbits were established. Concentratioh of IP3,total calcium, and [Ca(2+)]i in renal histocytes were observed.The results showed that concentrations of intracellular IP3 [(430.3±46.8),(297.9±54.3) min(-1),P<0.01] and [Ca(2+)]i [(212.5±43.5),(106.1±15.5) nmol/L,P<0.01] were increased in ischemic groups compared with the contrasted group,concentration of intracellular total calcium [(398.6±32.3),(385.0±32.4)μmol/L,P>0.05] was not increased obviously,whereas concentrations of intra cellular IP3 [(776.0±68.0) min(-1)]、[Ca(2+)]i [(412.1±47.3)μmol/L] and total calcium [(447.1±42.5)μmol/L, P<0.01]in reperfusion group (for 60 min) were increased remarkably as compared with the contrasted group and ischemic group.These data suggest that IP3 plays an important role in Ca(2+)-overloading forming of acute renal ischemic reperfusion injury.

用新西兰大白兔制作急性肾缺血及再灌流损伤模型,观察肾组织细胞内三磷酸肌醇(IP3)、细胞内总钙及胞浆游离钙([Ca(2+)]i)浓度变化。结果显示,缺血组与对照组的IP3浓度(±s)分别为(430.3±46.8)、(297.9±54.3)min(-1),[ca(2+)]i浓度分别为(212.5±43.5)、(106.1±15.5)nmol/L,前者较后者均显著性升高(均为P<0.01),总钙浓度分别为(398.6±32.3)、(385.0±32.4)μmol/L,两组无显著性差异(P>0.05)。再灌流60min后IP3浓度为(776.0±68.0)min(-1)、[Ca(2+)]i浓度为(412.1±47.3)nmol/L、总钙浓度为(447.1±42.5)μmol/L,较对照组和缺血组均显著性增加(P<0.01)。提示,细胞内IP3对急性肾缺血及再灌流损伤中肾组织细胞内钙超负荷的形成具有重要作用。

 
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