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alloxan-induced diabetic rat
相关语句
  促糖尿病大鼠
     STUDY ON THE MOLECULAR MECHANISMS INVOLVED IN THE INCREASED COLLAGEN SYNTHESIS BY PLATELET-DERIVED WOUND HEALING FACTORS DURING WOUND HEALING IN ALLOXAN-INDUCED DIABETIC RAT
     血小板源伤口愈合因子促糖尿病大鼠伤口组织胶原合成机制的研究
短句来源
  “alloxan-induced diabetic rat”译为未确定词的双语例句
     METHODS: Contents of copper(Cu), manganese(Mn), zinc(Zn), iron(Fe),cadmium(Cd),chromium(Cr),cobalt(Co),molybdenum(Mo) and lithium(Li) in the alloxan-induced diabetic rat diaphragm muscles were measured by atomic absorption spectrophotometer, and their superoxide dismutase(SOD) activities and malondiadehyde(MDA) contents were detected.
     方法 :采用原子吸收光谱法 ,测定糖尿病 2 8d大鼠膈肌组织中铜 (Cu)、锰 (Mn)锌 (Zn)、铁 (Fe)、镉 (Cd)、铬 (Cr)、钴 (Co)、钼 (Mo)和锂 (Li)的含量 ,并同时测定超氧化物歧化酶 (SOD)活性和丙二醛 (MDA)含量。
短句来源
     Using the spectrum absorption, we have deternined the contents of the copper, manganese, zinc andiron in the alloxan-induced diabetic rat diaphragm muscles,ad detected their activities of superoxide dismutase (SOD) and contents of malondiadehyde(MDA).
     本研究采用原子吸收光谱法,测定糖尿病4周大鼠膈肌组织中铜(Cu)、锰(Mn)、锌(Zn)、铁(Fe)的含量。 并同时测定超氧化物歧化酶(SOD)和丙二醛(MDA)含量。
短句来源
  相似匹配句对
     \ The diabetic rat model was induced by alloxan.
     动物模型用四氧嘧啶塑造。
短句来源
     The rat model was induced by alloxan.
     动物模型用四氧嘧啶塑造。
短句来源
     The diabetic rat model was induced by STZ.
     糖尿病大鼠模型用链脲佐菌素(STZ)腹腔注射诱导。
短句来源
     Results: Diabetic rat model was successfully induced.
     结果:成功建立大鼠糖尿病模型。
短句来源
     METHODS: The diabetic rat model was made with alloxan.
     方法利用四氧嘧啶复制糖尿病大鼠模型。
短句来源
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  alloxan-induced diabetic rat
Restoration of ultrastructural and biochemical changes in alloxan-induced diabetic rat sciatic nerve on treatment with Na3VO4 an
      


The effects of PGE2 on glucose metabolism in alloxan-induced diabetic and normal rats were studied by subcutaneous injection of PGE2, with the serum levels of glucose, insulin and glucagon, and histopathological survey of pancreas as indices of assessment. The results revealed that the cytoprotective effect of PGE2 against the damage of pancreatic islets caused by alloxan was invisible, however, a slight reduction of serum glucose level, suppression of insulin secretion and stimulation of glucagon...

The effects of PGE2 on glucose metabolism in alloxan-induced diabetic and normal rats were studied by subcutaneous injection of PGE2, with the serum levels of glucose, insulin and glucagon, and histopathological survey of pancreas as indices of assessment. The results revealed that the cytoprotective effect of PGE2 against the damage of pancreatic islets caused by alloxan was invisible, however, a slight reduction of serum glucose level, suppression of insulin secretion and stimulation of glucagon secretion were observed. The hypoglycemic effect of PGE2 in alloxan-induced diabetic rats seemed to be dosage dependent and probably related to temporary hyperglycemia and inhibiton of glucagon on glycogenolysis in liver. Thus, it might be suggested that PGE2 has no cytoprotective effect on pancreatic islets against alloxan damage, but is rather an important factor in regulating insulin and glucagon secretion as well as serum glucose level both in normal and alloxan-induced diabetic rats.

本文报告了PGE_2对Alloxan所致糖尿病大鼠及对正常大鼠血糖、胰岛素和胰升血糖素的影响。结果表明:PGE_2对正常大鼠和糖尿病大鼠均有降低血糖作用,对胰岛素的分泌有抑制作用,对胰升血糖素有刺激分泌的趋势。PGE_2对糖尿病大鼠的降血糖作用似乎有剂量依赖性。PGE_2降血糖作用可能与短暂性高血糖和抑制胰升血糖素的肝糖原分解作用有关。

Sorbitol pathway activity,myo-inositol content and ouabain-sensitive andouabain-resistant ATPase activities were measured in homogenates of the sciaticnerve from alloxan-induced diabetic rats for 4,8 and 12 weeks.Compared with theage-matched control,glucose,fructose and sorbitol contents of the sciatic nervefrom diabetic rats for 4,8 and 12 weeks were increased respectively to a level of3-4 fold,3-5 fold and 6-9 fold of those of the control rats.Myo-inositol wasreduced approximately 50%.There...

Sorbitol pathway activity,myo-inositol content and ouabain-sensitive andouabain-resistant ATPase activities were measured in homogenates of the sciaticnerve from alloxan-induced diabetic rats for 4,8 and 12 weeks.Compared with theage-matched control,glucose,fructose and sorbitol contents of the sciatic nervefrom diabetic rats for 4,8 and 12 weeks were increased respectively to a level of3-4 fold,3-5 fold and 6-9 fold of those of the control rats.Myo-inositol wasreduced approximately 50%.There was a significant decrease(P<0.01)in totalATPase,ouabain-sensitive and ouabain-resistant ATPase activities.These changescould be relevant to the development of diabetic neuropathy.

本文用四氧嘧啶诱导产生糖尿病动物模型,分别于糖尿病产生后第4,8,12周测定大鼠坐骨神经匀浆山梨醇通路活性,肌醇含量和哇巴因敏感的和不敏感的 ATP 酶活性。与同龄正常对照组比较,糖尿病发生4周后,坐骨神经葡萄糖含量增加3—4倍,果糖增加3—5倍,山梨醇增加6—9倍,肌醇含量降低到对照组的50%,总 ATP 酶和哇巴因敏感的Na~+-K~+-ATP 酶活性均极显著地低于同龄对照组(P<0.01)。结果提示这些代谢变化可能是糖尿病神经病变发病机制中的重要环节。

The ingredients of compound "Baxianjiangtang" are Radix Rehmanniae, Radix Polygoni Multiflori preparate, Rodix Astragali, Radix Ophiopogonis etc. Pharmacological tests showed that it can decrease LPO and increase the activity of SOD in blood and liver of Alloxan induced diabetic rats and mice.It also increased that phagocytosis of phagocytes of experimental anim als. It can increased tolerance to fatigue at low and high temperature conditions. According tothe above results, it can be considered...

The ingredients of compound "Baxianjiangtang" are Radix Rehmanniae, Radix Polygoni Multiflori preparate, Rodix Astragali, Radix Ophiopogonis etc. Pharmacological tests showed that it can decrease LPO and increase the activity of SOD in blood and liver of Alloxan induced diabetic rats and mice.It also increased that phagocytosis of phagocytes of experimental anim als. It can increased tolerance to fatigue at low and high temperature conditions. According tothe above results, it can be considered as safe and effective.

八仙降糖药由制首乌、黄芪、麦冬、生地等8味中药组成。药理研究表明:该制剂能使四氧嘧啶造型大、小鼠血、肝组织脂质过氧化物含量降低,超氧化物歧化酶活性增高;能显著提高实验动物腹腔巨噬细胞的吞噬活力;对小鼠有明显的抗疲劳、耐低温、耐高温的作用;毒理试验结果表明:本品安全无毒。

 
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