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acute exacerbation period
相关语句
  急性加重期
     The level of PaO2 had a negative relation with serum myocardial enzymes(r = -0.67~-0.87,P<0.01) in the COPD patients during acute exacerbation period.
     COPD急性加重期患者PaO2水平与对应的血清心肌酶呈直线负相关(r=-0.67~-0.87,P<0.01)。
短句来源
     Clinical Study of Blood Sugar in Acute Exacerbation Period of Pneumocardial Disease
     肺心病急性加重期血糖的临床研究
短句来源
     METHODS Fifty-six patients with COPD in acute exacerbation period were divided into treatment group and control group.
     方法56例COPD急性加重期患者分为治疗组和对照组。
短句来源
     Objective To study the effects of NIPPV for patients with COPD and SAS in acute exacerbation period and stable period.
     目的探讨无创正压通气(NIPPV)在COPD并SAS急性加重期及稳定期治疗的效果评价。
短句来源
     Methods:The 68 COPD patients during acute exacerbation period were divided into three groups according partial pressure of oxygen in artery (PaO2).
     方法:68例COPD急性加重期患者按动脉血氧分压的水平分成3组。
短句来源
更多       
  急性加重期患者
     The level of PaO2 had a negative relation with serum myocardial enzymes(r = -0.67~-0.87,P<0.01) in the COPD patients during acute exacerbation period.
     COPD急性加重期患者PaO2水平与对应的血清心肌酶呈直线负相关(r=-0.67~-0.87,P<0.01)。
短句来源
     METHODS Fifty-six patients with COPD in acute exacerbation period were divided into treatment group and control group.
     方法56例COPD急性加重期患者分为治疗组和对照组。
短句来源
     Methods:Sixty-four cases with chronic bronchitis in acute exacerbation period were randomly divided into treatment group(moxifloxacin group)and control group(amoxycillin group),with32cases in each group.
     方法:64例慢性支气管炎急性加重期患者随机分为2组 ,治疗组用莫西沙星400mg每日1次口服 ,治疗5天 ;
短句来源
     Methods:The 68 COPD patients during acute exacerbation period were divided into three groups according partial pressure of oxygen in artery (PaO2).
     方法:68例COPD急性加重期患者按动脉血氧分压的水平分成3组。
短句来源
  加重期
     The level of PaO2 had a negative relation with serum myocardial enzymes(r = -0.67~-0.87,P<0.01) in the COPD patients during acute exacerbation period.
     COPD急性加重期患者PaO2水平与对应的血清心肌酶呈直线负相关(r=-0.67~-0.87,P<0.01)。
短句来源
     Clinical Study of Blood Sugar in Acute Exacerbation Period of Pneumocardial Disease
     肺心病急性加重期血糖的临床研究
短句来源
     METHODS Fifty-six patients with COPD in acute exacerbation period were divided into treatment group and control group.
     方法56例COPD急性加重期患者分为治疗组和对照组。
短句来源
     Objective To study the effects of NIPPV for patients with COPD and SAS in acute exacerbation period and stable period.
     目的探讨无创正压通气(NIPPV)在COPD并SAS急性加重期及稳定期治疗的效果评价。
短句来源
     38 Cases of Acute Exacerbation Period of Pulmonary Heart Disease with Combination of TCM and WM
     中西医结合治疗肺心病加重期38例
短句来源
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  “acute exacerbation period”译为未确定词的双语例句
     Using flow cytometry assay, Th1/Th2 ratio was estimated by quantification of cytokins (IL-4, r-IFN) inside the plasma of single CD4+ T cells. HBV DNA load in peripheral serum of chronic HBV infectants during and after acute exacerbation period was analyzed by polymerase chain reaction (PCR) assay combined with fluorescent probe (FQ-PCR).
     应用流式细胞仪测定外周血中淋巴细胞胞浆内含白细胞介素-4(IL-4)及干扰素-γ(IFN-γ)的细胞的百分比及比值,采用荧光定量聚合酶链反应技术(FQ-PCR),检测慢性乙型肝炎患者发作时及治疗后外周血清中HBV DNA的载量;
短句来源
     Objective To study the hemodynamic and oxygen dynamic effects and the mechanism of nitric oxide (NO) inhalation therapy in COPD induced chronic cor pulmonale at acute exacerbation period.
     目的观察吸入一氧化氮(NO)对慢性肺心病急性发作期血流动力学和氧动力学影响,并对其产生机理进行探讨。
短句来源
     Objective To investigate the effect of erythrocyte membrane lipid compositions on erythrocyte membrane fluidity and ultrastructure during the acute exacerbation period in patients with chronic cor pulmonale.
     目的 观察慢性肺心病患者急性发作期红细胞膜脂质成分对红细胞膜流动性和红细胞超微结构的影响。
短句来源
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  acute exacerbation period
When compared with the liver biopsies obtained about 2 months after the peak of serum transaminase, cytotoxic/suppressor cells were significantly increased in those obtained during the acute exacerbation period.
      
Dysfunction of calcium and natrium pump activities in the acute exacerbation period of chronic cor pulmonale were closely related to NO level.
      


Objective To study the hemodynamic and oxygen dynamic effects and the mechanism of nitric oxide (NO) inhalation therapy in COPD induced chronic cor pulmonale at acute exacerbation period. Method Right heart catheterization was performed in 11 cases of chronic cor pulmonale. The hemodynamic parameters and plasma endothelium I(ET I) level were examined and recorded before and after inhalation of 40 ppm NO for 20 minutes. The plasma level of NO and lipid peroxide Ca 2+ Mg 2+ ATPase(calcium...

Objective To study the hemodynamic and oxygen dynamic effects and the mechanism of nitric oxide (NO) inhalation therapy in COPD induced chronic cor pulmonale at acute exacerbation period. Method Right heart catheterization was performed in 11 cases of chronic cor pulmonale. The hemodynamic parameters and plasma endothelium I(ET I) level were examined and recorded before and after inhalation of 40 ppm NO for 20 minutes. The plasma level of NO and lipid peroxide Ca 2+ Mg 2+ ATPase(calcium pump) and Na + K + ATPase(natrium pump) activities of RBC membrane in 30 cases of chronic cor pulmonale and 30 healthy controls were determined synchronously. Results After NO therapy, the average pulmonary pressure (PAPM) and pulmonary vascular resistance index (PVRI) were lowered by 16 6% and 25 9% respectively ( P <0.05). Intrapulmonary shunt was lowered by 16.3% ( P <0.05). ET I also lowered and showed a positive correlation with PAPM ( r =0.59, P <0.05). The activities of calcium and natrium pump were lowered when NO level declined, showing a positive correlation with NO concentration and negative correlation with lipid peroxides. Conclusion NO inhalation therapy exerted favorable hemodynamic and oxygen dynamic effects on COPD induced chronic cor pulmonale in its exacerbation period. Suppression of ET Ⅰ release might be one of the important factors for pulmonary vessel relaxation. Dysfunction of calcium and natrium pump activities in the acute exacerbation period of chronic cor pulmonale were closely related to NO level.

目的观察吸入一氧化氮(NO)对慢性肺心病急性发作期血流动力学和氧动力学影响,并对其产生机理进行探讨。方法对11例肺心病急性发作期患者给予鼻面罩吸入NO40ppm20分钟。治疗前后测定其血流动力学参数和血浆内皮素1(ET1)含量。并测定血浆NO含量,红细胞膜Ca2+Mg2+ATP酶(钙泵);Na+K+ATP酶(钠泵)活性及过氧化脂质含量。结果治疗前后肺动脉平均压(PAPM)和肺血管阻力指数分别下降166%及259%(P<005);肺血分流率下降163%(P<005),ET1含量下降并与PAPM呈正相关,慢性肺心病急性发作期NO含量下降,钙泵与钠泵活性降低,与NO含量呈正相关与过氧化脂质呈负相关。结论吸入NO对肺心病急性发作期患者有良好的血流动力学和氧动力学效应,抑制ET1的释放可能是舒张肺血管的重要机理之一。

Objective to evaluate the efficacy and safety of low dose,topical mitomycin C eyedrops (0.01g·L -1 MMC)in patients with severe vernal keratoconjunctivitis(VKC).Methods Twenty eyes of ten consecutive patients with severe VKC refractory to steroid treatment were treated with topical 0.01g·L -1 MMC eyedrops three times daily for 2 weeks.Results There were manifestly improved symptoms in patients with VKC at the end of the first week.There was significant decrease in ropy mucous discharge,photophobia,conjunctival...

Objective to evaluate the efficacy and safety of low dose,topical mitomycin C eyedrops (0.01g·L -1 MMC)in patients with severe vernal keratoconjunctivitis(VKC).Methods Twenty eyes of ten consecutive patients with severe VKC refractory to steroid treatment were treated with topical 0.01g·L -1 MMC eyedrops three times daily for 2 weeks.Results There were manifestly improved symptoms in patients with VKC at the end of the first week.There was significant decrease in ropy mucous discharge,photophobia,conjunctival hyperemia at the end of the second week treatment period.No adverse effects of treatment with MMC were observed.Conclusions Short term,low dose,topical MMC may be considered to be used in the acute exacerbation periods of patients with severe VKC refractory difficult to be managed by conventional treatment.

目的 评价应用低浓度丝裂霉素 C液 (0 .0 1g·L- 1 MMC)局部治疗重症春季角结膜炎 (VKC)患者的疗效与安全性。方法  10例 2 0眼用皮质类固醇难治的重症 VKC局部应用 0 .0 1g· L- 1 MMC液 ,每日 3次滴眼治疗 2周。结果 在治疗第 1周末患者的自觉症状即有好转 ,至第 2周末 ,粘丝状分泌物、怕光和结膜充血均有明显地减轻 ,未见有任何 MMC治疗的副反应发生。结论 对用传统疗法难于控制的重症 VKC急性恶化期 ,可考虑短期应用低浓度 MMC液作局部治疗

Objective To investigate the effect of erythrocyte membrane lipid compositions on erythrocyte membrane fluidity and ultrastructure during the acute exacerbation period in patients with chronic cor pulmonale. Methods Cholesterol/phospholipide contents of erythrocyte membrane were determined. Erythrocyte membrane fluidity was measured with DPH probe (1,6 Diphenyl 1,3,5 hexatrine). Morphology of erythrocyte was studied with scan electron microscope. Results Membrane cholesterol content, and cholesterol/phospholipide...

Objective To investigate the effect of erythrocyte membrane lipid compositions on erythrocyte membrane fluidity and ultrastructure during the acute exacerbation period in patients with chronic cor pulmonale. Methods Cholesterol/phospholipide contents of erythrocyte membrane were determined. Erythrocyte membrane fluidity was measured with DPH probe (1,6 Diphenyl 1,3,5 hexatrine). Morphology of erythrocyte was studied with scan electron microscope. Results Membrane cholesterol content, and cholesterol/phospholipide ratio were increased significantly. Membrane phospholipide content and membrane fluidity were decreased. Obvious more erythrocyte with morphological abnormality was seen in the patients with chronic cor pulmonale. Linear correlation analysis showed that the reduce of erythrocyte membrane fluidity had correlation with the change of lipid compositions of erythrocyte membrane. Conclusion The change of erythrocyte membrane lipid compositions is a basic factor causing the decrease of erythrocyte membrane fluidity and morphology abnormality in patients with chronic cor pulmonale.

目的 观察慢性肺心病患者急性发作期红细胞膜脂质成分对红细胞膜流动性和红细胞超微结构的影响。方法 慢性肺心病患者急性发作期患者 45例 ,测定膜胆固醇、磷脂含量、胆固醇 磷脂比值 ,同时用DPH探针测定红细胞膜流动性 ,扫描电镜观察红细胞形态。结果 肺心病患者膜胆固醇含量、胆固醇 磷脂比值明显升高 ,膜磷脂含量和膜流动性明显降低 ,异常形态的红细胞明显增多 ;直线相关分析显示 ,红细胞膜流动性降低与红细胞膜脂质成分改变有明显相关性。结论 红细胞膜胆固醇、磷脂含量改变是引起红细胞膜流动性降低和红细胞形态异常的基本因素。

 
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