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entered the analysis of results
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  进入结果分析
    RESULTS: According to intention-to-treat analysis, all the 135 patients with Alzheimer disease and 138 health controls entered the analysis of results.
    结果:按意向处理分析,135例阿尔茨海默病患者和138名正常健康人均进入结果分析
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    RESULTS: All the 7 patients entered the analysis of results.
    结果:7例患者均进入结果分析
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    RESULTS: All the 8 mice entered the analysis of results.
    结果:8只小鼠均进入结果分析
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    RESULTS: All the 29 ADHD children and 29 normal children entered the analysis of results.
    结果:纳入患者29例和正常儿童29例,均进入结果分析
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    RESULTS: No abnormal death occurred during the whole experiment, all the animals treated with injection of morphine were successfully induced to morphine-dependent models and entered the analysis of results.
    结果:整个实验过程未出现大鼠异常死亡,吗啡注射的所有动物均成功建立吗啡依赖动物模型,全部进入结果分析。 ①海马形态改变:吗啡依赖和戒断大鼠海马CA1区出现结构松散、神经元萎缩和坏死等改变。
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AIM: To investigate the relationship between cytochrome P450 1A1 gene polymorphism and Alzheimer disease.METHODS: The study was carried out in the central laboratory of Zhujiang Hospital, Southern Medical University from November 2004 to May 2005. Totally 135 patients with sporadic Alzheimer disease, who had no positive family history, and 138 normal healthy adults (normal control group), who were matched by age and gender, were selected in the study. 2 mL blood samples, drawn from each participant, were anticoagulated...

AIM: To investigate the relationship between cytochrome P450 1A1 gene polymorphism and Alzheimer disease.METHODS: The study was carried out in the central laboratory of Zhujiang Hospital, Southern Medical University from November 2004 to May 2005. Totally 135 patients with sporadic Alzheimer disease, who had no positive family history, and 138 normal healthy adults (normal control group), who were matched by age and gender, were selected in the study. 2 mL blood samples, drawn from each participant, were anticoagulated with sodium citrate, and then genome DNA was extracted from peripheral white blood cells with the phenol-chlorlform method. Polymerase chain reaction-restriction fragment length polymorphism (PCR-RFLP) method was used and mutant alleles of cytochrome P450 1A1 gene were studied in all the patients with Alzheimer disease and normal people. RESULTS: According to intention-to-treat analysis, all the 135 patients with Alzheimer disease and 138 health controls entered the analysis of results. ① The distribution of the gene frequency of cytochrome P450 1A1 alleles: The frequencies of alleles of m1 and m2 in the Alzheimer disease group were 63.0% and 37.0%, respectively, with were insignificantly different from those in the normal control group (68.1%, 31.9%, χ2=1.605, P > 0.05). ② The frequency of cytochrome P450 1A1 gene MSP 1 site genotypes distribution: The distributions of m1/m1, m1/m2, m2/m2 had no obvious differences between the Alzheimer disease group and normal control group (42.2%, 41.5%, 16.3%; 49.3%, 37.7%, 13.0%, χ2=1.483, P > 0.05). CONCLUSION: There is no correlation between the cytochrome P450 1A1 gene MSP 1 polymorphism and the genetic susceptibility to Alzheimer disease.

目的:探讨细胞色素P4501A1基因MSP1的多态性与阿尔茨海默病的关系。方法:实验于2004-11/2005-05在南方医科大学珠江医院中心实验室进行。选择135例无阳性家族史的散发性阿尔茨海默病患者,以年龄和性别相匹配的138名正常健康成人为正常对照组。所有被试者抽全血2mL,枸橼酸钠抗凝后采用苯酚-氯仿抽提法提取外周血白细胞基因组DNA。利用聚合酶链反应-限制性片段长度多态性技术分析其细胞色素P4501A1基因MSP1位点等位基因分布频率,比较基因型在阿尔茨海默病患者与正常人之间的分布差异。结果:按意向处理分析,135例阿尔茨海默病患者和138名正常健康人均进入结果分析。①细胞色素P4501A1等位基因频率分布:阿尔茨海默病组m1,m2分别为63.0%,37.0%,正常对照组的分别为68.1%,31.9%,两组比较差异不显著(χ2=1.605,P>0.05)。②细胞色素P4501A1基因MSP1位点基因型分布频率:阿尔茨海默病组m1/m1,m1/m2,m2/m2分布与正常对照组比较差异不明显(42.2%,41.5%,16.3%;49.3%,37.7%,13.0%,χ2=1.483P>0.05)。结论:细...

目的:探讨细胞色素P4501A1基因MSP1的多态性与阿尔茨海默病的关系。方法:实验于2004-11/2005-05在南方医科大学珠江医院中心实验室进行。选择135例无阳性家族史的散发性阿尔茨海默病患者,以年龄和性别相匹配的138名正常健康成人为正常对照组。所有被试者抽全血2mL,枸橼酸钠抗凝后采用苯酚-氯仿抽提法提取外周血白细胞基因组DNA。利用聚合酶链反应-限制性片段长度多态性技术分析其细胞色素P4501A1基因MSP1位点等位基因分布频率,比较基因型在阿尔茨海默病患者与正常人之间的分布差异。结果:按意向处理分析,135例阿尔茨海默病患者和138名正常健康人均进入结果分析。①细胞色素P4501A1等位基因频率分布:阿尔茨海默病组m1,m2分别为63.0%,37.0%,正常对照组的分别为68.1%,31.9%,两组比较差异不显著(χ2=1.605,P>0.05)。②细胞色素P4501A1基因MSP1位点基因型分布频率:阿尔茨海默病组m1/m1,m1/m2,m2/m2分布与正常对照组比较差异不明显(42.2%,41.5%,16.3%;49.3%,37.7%,13.0%,χ2=1.483P>0.05)。结论:细胞色素P4501A1基因MSP1的多态性与阿尔茨海默病的遗传易感性无关。

AIM: To discuss the effect of visual feedback balance training for 20 times on the behavior program and balance functions of children with attention deficit hyperactivity disorder (ADHD). METHODS: Twenty-nine children 25 males and 4 females, mean aged (12±2) years were recruited from the child psychiatric clinics at Mental Health Institute of Peking University from September 2001 to September 2003 as the case group. Twenty-nine normal children (age and sex matched) from nearby primary school were recruited...

AIM: To discuss the effect of visual feedback balance training for 20 times on the behavior program and balance functions of children with attention deficit hyperactivity disorder (ADHD). METHODS: Twenty-nine children 25 males and 4 females, mean aged (12±2) years were recruited from the child psychiatric clinics at Mental Health Institute of Peking University from September 2001 to September 2003 as the case group. Twenty-nine normal children (age and sex matched) from nearby primary school were recruited as the control group. The ADHD children were treated with the Balance Master produced by Neurocom company. ①Primary training, 30 minutes: The ADHD children shifted their gravity during sitting, standing and walking for 30 minutes. ②Games training: The game was completed by shifting gravity for 20 minutes. The one-to-one training pattern was used in the treatment, 2-6 times in every week, and 20 times as a course for totally 2-4 months. Evaluation methods: ①Static posture stability was tested under 8 different conditions (firm surface with eyes open, firm surface with eyes closed, foam pad with eyes open, foam pad with eyes closed, unilateral stance with eyes open and closed), one condition was tested for three times, 20 s per time. The mean sway velocity was recorded. ② Conner's behavior rating scale-parent questionnaire was applied before and after therapy: The scaled consisted of six factors, including behavior problem, learning problem, body complaining, hyperactivity, anxiety and hyperactivity index. ③ Rutter children's behavior scale, it was scored by three grades, the highest total score was 62. When total score beyond 13, it meant the child had behavior problem, antisocial behavior or neurotic behavior. ④ ADHD scale-IV (parent version): The 18 items of Diagnostic and Statistical Manual of Mental Disorders-Fourth Edition (DSM-IV) were used as a rating scale for ADHD symptom. Each item was scored by 0-3 grades. ADHD children had these tests before and after therapy respectively, and those in the normal control only had balance function test. RESULTS: All the 29 ADHD children and 29 normal children entered the analysis of results. ① The sway velocities of gravity under the conditions of foam surface with eyes closed, unilateral stance with eyes open and close before therapy in the ADHD group were significantly higher than those in normal controls Before therapy: (1.6±0.5), (1.2±0.6), (1.2 ±0.5), (2.8±1.0), (2.8±1.0)°/s, normal control: (1.1±0.4), (0.9±0.2), (1.0 ±0.4), (1.9±0.7), (2.1±0.7)°/s, t=-3.287, -2.443, -2.529, -3.542, -3.032, P < 0.05. After therapy the sway velocity decreased under the condition of foam surface with eyes open Before and after therapy were (0.8±0.3), (0.6 ±0.2)°/s, t=3.808, P < 0.01. And also after therapy the sway velocity decreased under the condition of unilateral stance with eye close left was (2.8±1.0), (2.0±0.6) °/s right was (2.8±1.0), (2.2±0.7)°/s, t=3.957, 2.758, P < 0.05. ②After training for 20 times, the scores of hyperactivity factor and index of Conner's behavior rating scale decreased significantly hyperactivity factor: (5.5±2.8), (4.0±2.4), index: (12.8±4.5), (10.8±5.6), t =3.348, 2.299, P < 0.05. ③ The total score and score of breaking rule behavior were very significantly decreased total score before and after therapy: (17.0±6.4), (12.8±5.9) score of breaking rule behavior before and after therapy: (3.4±1.7), (2.2±1.9), t=3.832, 4.137, P < 0.01.④ The scores of inattention, hyperactivity-impulse and total scores of DSM-IV questionnaire were significantly decreased (15.9±3.5), (13.3±5.1) (10.2 ±4.8), (7.6±5.1) (26.1±6.1), (20.9±9.1), t=2.136, 3.260, 2.930, P < 0.05. ⑤ There was a positive correlation between the changing range of foam surface eye open and the score of breaking rule behavior in Rutter children's behavior scale (r=0.526, P=0.012). CONCLUSION: visual feedback balance training can improve the posture stability and behavior problems of ADHD children.

目的:探讨视反馈平衡功能训练20次对注意缺陷多动障碍患儿行为问题及平衡功能的影响。方法:选择2001-09/2003-09北京大学精神卫生研究所门诊就诊儿童29例,为病例组,男25例,女4例,平均年龄(12±2)岁。对照组29例,为附近小学按性别、年龄(相差不超过6个月)配对的正常儿童。采用Neurocom公司出产的BalanceMaster机型对注意缺陷多动障碍患者进行治疗。①基础训练:患儿在坐、站及行进中进行重心转移训练,共30min。②游戏训练:通过重心转移完成技术难点,共20min。治疗过程为一对一的训练模式。治疗2~6次/周,20次为1个疗程,共2~4个月。评估方法:①治疗前后评定固定平面睁眼和闭眼、海绵垫平面睁眼和闭眼、左/右脚睁眼和闭眼8种状态下被试的静态直立平衡功能,并与正常儿童比较。平衡功能测试不同感觉状态下重心摇摆度,识别参与控制姿态的视觉、前庭和本体等3种平衡功能,每一个状态测3次,20s/次。记录3次的平均摇摆速率。②治疗前后进行Conners儿童行为问卷调查:该量表可得出6个因子分,包括行为问题、学习问题、躯体问题、多动、焦虑和多动指数。③Rutter儿童行为问卷调查:问卷评分分为3...

目的:探讨视反馈平衡功能训练20次对注意缺陷多动障碍患儿行为问题及平衡功能的影响。方法:选择2001-09/2003-09北京大学精神卫生研究所门诊就诊儿童29例,为病例组,男25例,女4例,平均年龄(12±2)岁。对照组29例,为附近小学按性别、年龄(相差不超过6个月)配对的正常儿童。采用Neurocom公司出产的BalanceMaster机型对注意缺陷多动障碍患者进行治疗。①基础训练:患儿在坐、站及行进中进行重心转移训练,共30min。②游戏训练:通过重心转移完成技术难点,共20min。治疗过程为一对一的训练模式。治疗2~6次/周,20次为1个疗程,共2~4个月。评估方法:①治疗前后评定固定平面睁眼和闭眼、海绵垫平面睁眼和闭眼、左/右脚睁眼和闭眼8种状态下被试的静态直立平衡功能,并与正常儿童比较。平衡功能测试不同感觉状态下重心摇摆度,识别参与控制姿态的视觉、前庭和本体等3种平衡功能,每一个状态测3次,20s/次。记录3次的平均摇摆速率。②治疗前后进行Conners儿童行为问卷调查:该量表可得出6个因子分,包括行为问题、学习问题、躯体问题、多动、焦虑和多动指数。③Rutter儿童行为问卷调查:问卷评分分为3级,总分的最高分为62分,凡≥13分者有行为问题,分为违纪行为或称反社会行为和神经症行为。④注意缺陷多动障碍评定量表-IV(父母版)调查:包括18个条目的量表,每个条目对应精神障碍诊断与统计手册第4版中注意缺陷多动障碍18个症状标准中的一条。每一条目按照0~3评分。总分为18个条目的评分总和。各项测试病例组患儿于治疗前后各评定1次,对照组仅评定1次平衡功能。结果:纳入患者29例和正常儿童29例,均进入结果分析。①注意缺陷多动障碍患儿治疗前海绵垫平面闭眼、左/右腿睁眼、闭眼条件下摇摆速率明显高于正常对照,差异显著[治疗前分别为(1.6±0.5),(1.2±0.6),(1.2±0.5),(2.8±1.0),(2.8±1.0)°/s,对照组分别为(1.1±0.4),(0.9±0.2),(1.0±0.4),(1.9±0.7),(2.1±0.7)°/s,t=-3.287,-2.443,-2.529,-3.542,-3.032,P<0.05]。治疗后海绵垫平面睁眼条件下摇摆速率明显低于治疗前,差异非常显著[治疗前后分别为(0.8±0.3),(0.6±0.2)°/s,t=3.808,P<0.01]。治疗后左、右腿单腿闭眼站立摇摆速率均明显低于治疗前,差异显著[左侧治疗前后分别为(2.8±1.0),(2.0±0.6)°/s,右侧治疗前后分别为(2.8±1.0),(2.2±0.7)°/s,t=3.957,2.758,P<0.05],达到正常水平。②训练20次后Conner测试中多动分和多动指数明显下降,差异显著[治疗前后多动分分别为(5.5±2.8),(4.0±2.4)分,治疗前后多动指数分别为(12.8±4.5),(10.8±5.6),t=3.348,2.299,P<0.05]。③Rutter测试中总分、违纪行为分明显下降,差异非常显著[治疗前后总分分别为(17.0±6.4),(12.8±5.9)分,治疗前后违纪行为分分别为(3.4±1.7),(2.2±1.9)分,t=3.832,4.137,P<0.01]。④注意缺陷多动障碍评定量表-IV问卷测试中注意缺陷分、多动、冲动分、总分均明显下降,差异显著[治疗前后注意缺陷分分别为(15.9±3.5),(13.3±5.1)分,治疗前后多动、冲动分分别为(10.2±4.8),(7.6±5.1)分,治疗前后总分分别为(26.1±6.1),(20.9±9.1)分,t=2.136,3.260,2.930,P<0.05]。⑤患者在海绵垫平面睁眼的变化幅度与Rutter行为量表中违纪行为变化幅度成正相关(r=0.526,P=0.012)。结论:视反馈平衡功能训练能提高患儿的姿势稳定性,同时能较全面的改善患儿的行为问题。

AIM: To investigate the changes of the expression of tumor necrosis factor-alpha (TNF-α) in the hippocampal CA1 region of morphine-dependent and morphine-withdrawal rats.METHODS: The experiment was carried out in the Laboratory of Histology and Embryology, Luzhou Medical College between April 2004 and March 2005. Twenty-four 2 or 3-month-old male Sprague-Dawley rats of common grade were divided randomly into four groups with 6 rats in each: the control group, morphine-dependent group, and naloxone-precipitated...

AIM: To investigate the changes of the expression of tumor necrosis factor-alpha (TNF-α) in the hippocampal CA1 region of morphine-dependent and morphine-withdrawal rats.METHODS: The experiment was carried out in the Laboratory of Histology and Embryology, Luzhou Medical College between April 2004 and March 2005. Twenty-four 2 or 3-month-old male Sprague-Dawley rats of common grade were divided randomly into four groups with 6 rats in each: the control group, morphine-dependent group, and naloxone-precipitated withdrawal for 1 and 3 hours groups. Morphine-dependent models were established by the method of ascending doses of morphine, and the withdrawal was precipitated with naloxone. The rats were treated with subcutaneous injection of morphine on the back, twice a day, the dosage on the first day was 10 mg/kg, which was increased by 10 mg/kg once every other day, and the last dosage was 50 mg/kg on the sixth day. In the naloxone-precipitated withdrawal groups, the withdrawal symptoms were evoked by subcutaneous injection of naloxone (5 mg/kg) at 6 hours after the last injection of morphine, and the rats were given the same treatment as those in the morphine-dependent group after 1 and 3 hours. According to the parallel control principal, rats in the control group were injected with saline of the same volume with the same method. The activities of the rats under normal and experimental status were recorded. At the same time, the hippocampal CA1 regions of the rats were removed and treated with haematine-eosin staining, the expression of TNF-α was detected with immunohistochemistry, and the number and absorbance of immunological response positive cells were detected.RESULTS: No abnormal death occurred during the whole experiment, all the animals treated with injection of morphine were successfully induced to morphine-dependent models and entered the analysis of results. ① Hippocampal morphological changes: The changes of loose structure, neuronal atrophy and necrosis in hippocampal CA1 region occurred in the morphine-dependent and morphine-withdrawal rats. ② Expression of TNF-α protein: The expression of TNF-α protein was mild positive in the hippocampal CA1 region of rats in the control group, and the number of TNF-α positive cells was increased in the morphine-dependent and morphine-withdrawal groups, and the positive cells in the groups were mainly glial cells and neurons, and the average number of positive cells in the two groups were significantly different from those in the control group (34.83±3.54), (17.50±2.88), P < 0.01; (38.83±4.62), (17.50±2.88), P < 0.01; (58.17±6.62), (17.50±2.88), P < 0.01 , there was insignificant difference between the morphine-dependent group and naloxone-precipitated withdrawal for 3 hours group (34.83±3.54), (38.83±4.62), P > 0.05 . The absorbance of TNF-α positive cells was increased in the morphine-dependent and morphine-withdrawal groups, which were also significantly different from that in the control group (0.378 0 ±0.009 4), (0.310 8±0.001 0), P < 0.01; (0.399 9±0.012 0), (0.310 8 ±0.001 0), P < 0.01; (0.385 5±0.006 6), (0.310 8±0.001 0), P < 0.01 , there was insignificant difference between the morphine-dependent group and naloxone-precipitated withdrawal for 3 hours group (0.378 0±0.009 4), (0.385 5±0.006 6), P > 0.05 . The absorbance of TNF-α positive cells was significantly different between the naloxone-precipitated withdrawal for 1 and 3 hours groups (0.399 9±0.012 0), (0.385 5±0.006 6), P < 0.05 . CONCLUSION: TNF-α contributes to the pathophysiological process of morphine dependence and naloxone-precipitated withdrawal, in which TNF-α may have deleterious effects.

目的:探讨吗啡依赖和戒断大鼠海马CA1区肿瘤坏死因子α的表达变化。方法:实验于2004-04/2005-03在泸州医学院组织胚胎学实验室完成。实验动物选择普通级两三月龄雄性SD大鼠24只。按随机数字法将实验动物分为4组:对照组、吗啡依赖组和盐酸纳洛酮催促戒断1h组和戒断3h组(后两组合称戒断组),每组6只。采用剂量递增法建立吗啡依赖动物模型,用纳洛酮催促吗啡依赖动物戒断反应。具体方法为每天两次在大鼠背部皮下注射盐酸吗啡,首日10mg/kg,隔日每次增加10mg/kg,至第6天末次注射50mg/kg。吗啡依赖组末次注射后6h麻醉下灌注固定。盐酸纳洛酮催促戒断组大鼠于末次注射吗啡6h后,以盐酸纳洛酮5mg/kg皮下注射激发戒断症状,1h和3h后,同吗啡依赖组处理实验动物。对照组大鼠按照同期平行对照的原则,以相同方式注射同体积的生理盐水。记录大鼠在正常及实验状态下的活动状况。同时取大鼠海马CA1区分别作苏木精-伊红和肿瘤坏死因子α免疫组织化学,测免疫反应阳性细胞的数量和吸光度。结果:整个实验过程未出现大鼠异常死亡,吗啡注射的所有动物均成功建立吗啡依赖动物模型,全部进入结果分析。①海马形态改变:吗啡依赖和戒断大鼠海马...

目的:探讨吗啡依赖和戒断大鼠海马CA1区肿瘤坏死因子α的表达变化。方法:实验于2004-04/2005-03在泸州医学院组织胚胎学实验室完成。实验动物选择普通级两三月龄雄性SD大鼠24只。按随机数字法将实验动物分为4组:对照组、吗啡依赖组和盐酸纳洛酮催促戒断1h组和戒断3h组(后两组合称戒断组),每组6只。采用剂量递增法建立吗啡依赖动物模型,用纳洛酮催促吗啡依赖动物戒断反应。具体方法为每天两次在大鼠背部皮下注射盐酸吗啡,首日10mg/kg,隔日每次增加10mg/kg,至第6天末次注射50mg/kg。吗啡依赖组末次注射后6h麻醉下灌注固定。盐酸纳洛酮催促戒断组大鼠于末次注射吗啡6h后,以盐酸纳洛酮5mg/kg皮下注射激发戒断症状,1h和3h后,同吗啡依赖组处理实验动物。对照组大鼠按照同期平行对照的原则,以相同方式注射同体积的生理盐水。记录大鼠在正常及实验状态下的活动状况。同时取大鼠海马CA1区分别作苏木精-伊红和肿瘤坏死因子α免疫组织化学,测免疫反应阳性细胞的数量和吸光度。结果:整个实验过程未出现大鼠异常死亡,吗啡注射的所有动物均成功建立吗啡依赖动物模型,全部进入结果分析。①海马形态改变:吗啡依赖和戒断大鼠海马CA1区出现结构松散、神经元萎缩和坏死等改变。②肿瘤坏死因子α蛋白表达:对照组大鼠海马CA1区肿瘤坏死因子α蛋白表达轻度阳性,吗啡依赖组和戒断组肿瘤坏死因子α阳性细胞数均增加,各组阳性细胞主要是胶质细胞和神经元。各组阳性细胞均数与对照组比较,差异均有统计学意义[(34.83±3.54),(17.50±2.88),P<0.01];[(38.83±4.62),(17.50±2.88),P<0.01];[(58.17±6.62),(17.50±2.88),P<0.01]。而吗啡依赖组与盐酸纳洛酮催促戒断3h组差异无统计学意义[(34.83±3.54),(38.83±4.62),P>0.05]。吗啡依赖组和戒断组肿瘤坏死因子α阳性细胞吸光度增加,各组阳性细胞吸光度均数与对照组比较差异也均有统计学意义[(0.3780±0.0094),(0.3108±0.0010),P<0.01];[(0.3999±0.0120),(0.3108±0.0010),P<0.01];[(0.3855±0.0066),(0.3108±0.0010),P<0.01],吗啡依赖组与盐酸纳洛酮催促戒断3h组比较差异无统计学意义[(0.3780±0.0094),(0.3855±0.0066),P>0.05]。盐酸纳洛酮催促戒断1h组与戒断3h组肿瘤坏死因子α阳性细胞吸光度差异有显著性[(0.3999±0.0120),(0.3855±0.0066),P<0.05]。结论:肿瘤坏死因子α参与了吗啡依赖和戒断的病理生理过程,可能是药物依赖形成中的脑损伤初始因素之一。

 
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