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nimodipine group
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  尼莫地平组
     the content after reperfusion 2, 12 and 24 hours in the Nimodipine group were lower than the model group (45.99±8.13), (40.63±3.13), (36.72±4.38) μmol/L; (65.54±6.01), (57.08±4.79), (48.13±5.12) μmol/L;
     尼莫地平组再灌注2,12和24h均低于模型组[(45.99±8.13),(40.63±3.13),(36.72±4.38)μmol/L;(65.54±6.01),(57.08±4.79),(48.13±5.12)μmol/L;P均<0.05];
短句来源
     The nervous dysfunction score(I/R 6,12 d),the expression of TNF-α,ICAM-1(I/R 1d) and its mRNA(I/R 1,3 d) in Naomaitong group were higher than that of Nimodipine group.
     脑脉通组神经症状积分(I/R 6,12 d),TNF-α(I/R 1 d),ICAM-1(I/R 1 d)及其mRNA(I/R 1,3 d)表达低于尼莫地平组
短句来源
     In the Naomaitong Granules group the nerve dysfunction score (I/R 6 and 12 d), the expressions of nNOS (I/R 1 d) and iNOS (I/R 1 d) were lower than those in the nimodipine group, while the expression of eNOS (I/R 1 d) was higher.
     脑脉通组神经症状积分(I/R6、12d)和nNOS(I/R1d)、iNOS(I/R1d)的表达低于尼莫地平组,eNOS(I/R1d)高于尼莫地平组
短句来源
     RESULTS: In nimodipine group the scores of WMS, MMSE, ADL, CGI, EI were 59±22, 20±6, 30±12, 3.0±0.7, 1.9±0.7, as that were 70±18, 22±4, 26±9, 2.2± 0.9 , 2.5±0.8 in buflomedil group.
     结果 :治疗后WMS ,MMSE ,ADL ,CGI ,EI评分尼莫地平组为 (5 9± 2 2 ,2 0± 6 ,30± 12 ,3.0± 0 .7,1.9±0 .7)分 ;
短句来源
     Nimodipine group: 10 g/L nimodipine (1 mg/kg) was injected into intraperitone in10 minutes before ischemia.
     尼莫地平组:在缺血前10min腹腔注入10g/L尼莫地平1mg/kg;
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  “nimodipine group”译为未确定词的双语例句
     ① Administration: Rats in the trihexyphenidyl 1.5, 1 and 0.5 mg / kg groups and nimodipine group were treated with intraperitoneal injections of trihexyphenidyl (1.5, 1 and 0.5 mg / kg) and nimodipine (3 mg / kg) respectively, and no administration was given in the other two groups.
     ①给药:苯海索1.5,1,0.5mg/kg剂量组及尼莫地平3mg/kg组分别腹腔注射苯海索1.5,1,0.5mg/kg及尼莫地平3mg/kg,其他两组不给药。
短句来源
     The rabbits in the aspirin + nimodipine group were treated with 2.35 mg/kg aspirin and 2.82 mg/kg nimodipine every day.
     阿司匹林+尼莫地平合用组给予阿司匹林2.35mg/(kg·d)、尼莫地平2.82mg/(kg·d)。
短句来源
     ① Content of 5-HT: The contents of 5-HT in the cerebral cortex and striatum tissues were significantly lower in the ischemia reperfusion group than in the sham-operated group (P< 0.01), but significantly higher in the trihexyphenidyl 1.5, 1 and 0.5 mg / kg groups and nimodipine group than in the ischemia reperfusion group (P < 0.01 or 0.05).
     ①5-羟色胺含量:脑皮质和纹状体缺血再灌注组均显著低于假手术组(P<0.01),苯海索1.5,1,0.5mg/kg剂量组和尼莫地平3mg/kg组均显著高于缺血再灌注组(P<0.01或0.05)。
短句来源
     ② Level of norepinephrine: The levels of norepinephrine in the cerebral cortex and striatum tissues were significantly lower in the ischemia reperfusion group than in the sham-operated group (P < 0.01), but significantly higher in the trihexyphenidyl 1.5, 1 and 0.5 mg / kg groups and nimodipine group than in the ischemia reperfusion group (P < 0.01 or 0.05).
     ②去甲肾上腺素水平:脑皮质和纹状体缺血再灌注组均显著低于假手术组(P<0.01),苯海索1.5,1,0.5mg/kg剂量组和尼莫地平3mg/kg组均显著高于缺血再灌注组(P<0.01或0.05)。
短句来源
     ③ Content of dopamine: The contents of dopamine in the cerebral cortex and striatum tissues were significantly lower in the ischemia reperfusion group than in the sham-operated group (P < 0.01), but significantly higher in the trihexyphenidyl 1.5, 1 and 0.5 mg / kg groups and nimodipine group than in the ischemia reperfusion group (P < 0.01 or 0.05).
     ③多巴胺含量:脑皮质和纹状体缺血再灌注组均显著低于假手术组(P<0.01),苯海索1.5,1,0.5mg/kg剂量组和尼莫地平3mg/kg组均显著高于缺血再灌注组(P<0.01或0.05)。
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  相似匹配句对
     group d(nimodipine treatment) .
     尼莫地平治疗组。
短句来源
     Group D: treated with nimodipine;
     D组:尼莫通组;
短句来源
     The P&T Group
     巴马丹拿集团
短句来源
     group.
     静脉组未测得代谢物。
短句来源
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  nimodipine group
Six months after injury 93 (53%) of the nimodipine group and 86 (49%) of the control group had a favourable outcome (moderate/good recovery).
      
The low blood flow in the nimodipine group might have been a consequence of brain edema caused by extravasates.
      
The underlying cause of death was major bleeding in 6 of 9 deaths, 5 in the nimodipine group and 1 in the placebo group.
      


Random comparative method was taken in patients with essential hypertensive diseases using nimodipine and nifedipine. Twenty five patients (M 24, F 1; age 64+5 a) in the nimodipine group received 30-60 mg, tid-qid, and 25 patients (M 23, F 2; age 65+4 a) in the nifedipine group received 10-20 mg, tid-qid, 4 wk as a course of treatment. The results showed that total response rate was 92% in nimodipine group and 88% in nifedipine group (P>0.05). In both groups the blood pressure decreased...

Random comparative method was taken in patients with essential hypertensive diseases using nimodipine and nifedipine. Twenty five patients (M 24, F 1; age 64+5 a) in the nimodipine group received 30-60 mg, tid-qid, and 25 patients (M 23, F 2; age 65+4 a) in the nifedipine group received 10-20 mg, tid-qid, 4 wk as a course of treatment. The results showed that total response rate was 92% in nimodipine group and 88% in nifedipine group (P>0.05). In both groups the blood pressure decreased markedly (P<0.01), and adverse effects were mild.

原发性高血压病50例,其中采用尼莫地平治疗者25例(男24例,女1例,年龄64±5a)剂量30-60mg,tid-qid.另25例用硝苯啶对照治疗,其中男23例,女2例,年龄65±4a,剂量10-20mg,tid-qid。2组均以4wk为一个疗程。尼莫地平组总有效率92%,硝苯啶组为88%。2组自身对照t检验,P<0.01,组间比较,P>0.05。不良反应均轻微。

Sixty patients with acute ischemic cerebral infarction within 72 h were equally divided into 2groups.The nimodipine group 30 patients(M 21,F 9;age 59±s10 a) received nimodipine 2 mg/d intravenous infusion based on supported therapy(dehydrant,vit C)in 1-2 wk,oral nimodipine 60 mg every evening in 1-4 wk.Supported therapy was altered to improve hemodilution,microcirculation,and nervous cell activator in 3-4wk.Control group 30 patients(M23,F7,age 58±9 a) received only supported therapy.Irrespective...

Sixty patients with acute ischemic cerebral infarction within 72 h were equally divided into 2groups.The nimodipine group 30 patients(M 21,F 9;age 59±s10 a) received nimodipine 2 mg/d intravenous infusion based on supported therapy(dehydrant,vit C)in 1-2 wk,oral nimodipine 60 mg every evening in 1-4 wk.Supported therapy was altered to improve hemodilution,microcirculation,and nervous cell activator in 3-4wk.Control group 30 patients(M23,F7,age 58±9 a) received only supported therapy.Irrespective of condition of patients or size of infarction, nervous functional defect scores of the nimodipine group decreased more than those of the control group(P<0.01).The effects of nimodipine showed no difference between those within 30h after onset and those within 31-72h.

发病72h内的急性缺血性脑梗死60例,分为2组。尼莫地平组30例(男性21例,女性9例;年龄59±s10a).wk1,2给支持疗法(脱水剂、维生素C等)和尼莫地平2mg/d于5%葡萄糖液500mL内静脉滴注,wk3,4改用扩容、改善微循环、细胞活性药,wk1-4口服尼莫地平60mg,qn。对照组30例(男性23例,女性7例;年龄58±9a),不给尼莫地平,其余同上。4wk后,前组神经功能缺损积分值较后组下降显著(P<0.01),与病情轻重、病灶大小、治疗早晚无关。*P<0.01。讨论作者采用临床对照研究,静滴尼莫地平治疗发病72h内的缺血性脑梗死,无论病情分级或总体疗效,治疗前后的神经功能缺损的积分差,与对照组比较,差别均有非常显著意义(P<0.01),提示尼莫地平对急性缺血性脑梗死的近期疗效肯定。但是深部小梗死灶引起的中、重型患者,2组疗效差别无显著意义,可能与小梗死灶病理变化轻,不能充分发挥尼莫地平的阻止钙内流和细胞膜崩解、保护半暗带等作用有关[4]。动物模型局灶性脑缺血30min后开始用尼莫地平治疗即不能增加局部脑血流量[5]。用不同剂量的尼莫地平治疗发病48h内的急性脑梗死1064例,发现只有120?

Abstract Severe brain ischemia caused by occlusion of bilateral carotid and vertebral arteries were performed on expermental rabbits.Microcirculation of cerebral piamater were studied through the fenestration of cranial bone by closed-circuit TV microscope system the active changes of microcirculation after the brainresuscitation by Nimodipine were watched directly.In the ischemia group the arteriole began contract 40 min after its distinct dilation.Blood flow velocity decity decreased and part of...

Abstract Severe brain ischemia caused by occlusion of bilateral carotid and vertebral arteries were performed on expermental rabbits.Microcirculation of cerebral piamater were studied through the fenestration of cranial bone by closed-circuit TV microscope system the active changes of microcirculation after the brainresuscitation by Nimodipine were watched directly.In the ischemia group the arteriole began contract 40 min after its distinct dilation.Blood flow velocity decity decreased and part of venule blood stagnated.In Nimodipine group there were no vasospasm found,at the saitie time the arterioles became dilated and lasted more than 100 min.From above detas the authors think Nimodipine could be ameliorate abnormal microcirculation after the brain ischemia.Increase the blood supply of the brain tissue and mitigate reperfusion damage.Therefore,it is suggested that Nimodipine may be used clinically for brain resuscitation .

在夹闭双侧颈总动脉和椎动脉造成严重脑缺血20分钟的基础上,应用微循环闭路电视显微系统行颅骨开窗法对兔脑软脑膜微循环录像,直接动态观察钙拮抗剂尼莫地平对脑复苏后微循环的影响。再灌注期,尼莫地平能显著地扩张微动脉,并持续100分钟以上,明显地延缓延迟性低灌注时相。结果提示:尼莫地平具有不同程度地改善脑缺血后微循环异常,增加脑组织血液供给,减轻再灌注损伤。

 
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