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nimodipine group
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  尼莫地平组
    Method: One hundred and twenty rats were randomly divided into three CVB-D groups(2,1,0.5 mg·kg-1),Nimodipine group(2 mg·kg-1),model group and sham operated group,20 rats each group.
    方法:取大鼠120只,随机分为6组,即CVB-D高、中、低剂量组(2,1,0.5 mg.kg-1),尼莫地平组(2 mg.kg-1),模型对照组和假手术组,每组20只。
短句来源
    In the Naomaitong Granules group the nerve dysfunction score (I/R 6 and 12 d), the expressions of nNOS (I/R 1 d) and iNOS (I/R 1 d) were lower than those in the nimodipine group, while the expression of eNOS (I/R 1 d) was higher.
    脑脉通组神经症状积分(I/R6、12d)和nNOS(I/R1d)、iNOS(I/R1d)的表达低于尼莫地平组,eNOS(I/R1d)高于尼莫地平组
短句来源
    The nervous dysfunction score(I/R6d, 12d), the expression of TNF-a, ICAM-1(I/R1d)and its mRNA(I/R1d, 3d)in Naomaitong group were higher than that of Nimodipine group.
    脑脉通组神经症状积分(I/R6d、 12d)、TNF-α(I/R1d)、ICAM-1(I/R1d)及其mRNA(I/R1d、3d)表达低于尼莫地平组
    nimodipine group could prolong the time by 12.4r/o(0.01<P<0.05);
    尼莫地平组可延长小鼠断头后张口喘息时间12.41%(0.01<P<0.05)。
短句来源
    75mg/Kg group has a significant difference compared with nimodipine group(0.01<P<0.05).
    2.与尼莫地平组比:Bellidifolin 75mg·Kg~(-1)组延长小鼠断头后张口喘息时间0.93%(0.01<P<0.05)。
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  “nimodipine group”译为未确定词的双语例句
    Altogether 24 Sprague-Daleg rats were randomly divided into four groups: normal control group (NCG), ischemic injury group (BG), SSP group (SG) and nimodipine group (NG).
    造模大鼠随机分为 3组 :缺血再灌流组(BG) ,SSP治疗组 (SG) ,尼莫地平对照组 (NG) ;
短句来源
    The levels of TNFα,IL1β and the expression of VCAM1 in rats brain tissue in nimodipine group decreased,while the level of TGFβ increased.
    大黄苷元低剂量组TNFα和IL 1β水平及TNFα、VCAM 1表达均明显降低,TGFβ水平增高;
短句来源
    METHODS Ninety-six male Wistar rats were randomly divided into six groups(n=16): sham operated group,I-R group,nimodipine group and three baicalin groups,to which baicalin of 50,100 and 200 mg·kg~(1) was administered,respectively.
    方法Wistar大鼠随机分为假手术组、脑缺血再灌注模型组、黄芩苷组(50,100和200 mg.kg-1),以及尼莫地平0.4 mg. kg-1组。
短句来源
    2 Compared with the model group , the level of TUNEL,bax, NO, MDA, fas, Caspace-3 of HJG lowest group, HJG moderate group, OHJG maxim group and Nimodipine group decreased significantly (P<0. 05,P<0. 01), the level of bcl-2,SOD and bcl-2/bax increased significantly (P<0. 05 , P<0. 01).
    中药大剂量组大鼠脑组织凋亡阳性细胞数与西药组间无显著性差异(P>0.05)。 中药大剂量组大鼠脑组织凋亡阳性细胞数显著低于中药中、小剂量组(P<0.05,P<0.01)
短句来源
    1. Effect of bellidifolin on duration of gasping after the complete cerebral ischemia induced by decapitation in mice75 mice were arranged in 5 groups which were NS group,nimodipine group and 50mg/Kg 75mg/Kg, 100mg/Kg bellidifolin group.
    一、采用断头法制备小鼠永久性完全性全脑缺血模型,观察不同剂量Bellidifolin对小鼠断头后张口喘息时间的影响。
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  nimodipine group
Six months after injury 93 (53%) of the nimodipine group and 86 (49%) of the control group had a favourable outcome (moderate/good recovery).
      
The low blood flow in the nimodipine group might have been a consequence of brain edema caused by extravasates.
      
The underlying cause of death was major bleeding in 6 of 9 deaths, 5 in the nimodipine group and 1 in the placebo group.
      


The cerebral ischemic and hypotensive mice treated by re perfusion were used to study the effects of Kangdai mixture (KM) on LPO, SOD and CA1 cells. The results showed that on the 7th day after re perfusion, SOD activity was inhibited, LPO production was increased, and about 81% of CA1 cells was damaged in the mice. After being treated with KM and nimodipine, SOD activity was increased, LPO production was decreased, and importantly, pyramidal cells in hippocampal CA1 region were protected from delayed...

The cerebral ischemic and hypotensive mice treated by re perfusion were used to study the effects of Kangdai mixture (KM) on LPO, SOD and CA1 cells. The results showed that on the 7th day after re perfusion, SOD activity was inhibited, LPO production was increased, and about 81% of CA1 cells was damaged in the mice. After being treated with KM and nimodipine, SOD activity was increased, LPO production was decreased, and importantly, pyramidal cells in hippocampal CA1 region were protected from delayed neuronal death. About 87% of pyramidal cells survived in KM group and 49% in nimodipine group.

研究采用小鼠反复脑缺血合并低血压模型 ,发现 7d时其脑内过氧化脂质(LPO)含量增高 ,SOD活力降低 ;1 0d时 ,海马CA1区神经元数量明显减少。抗呆合剂可对抗这种变化 ,减轻脑缺血再灌损伤 ,表明抗呆合剂的脑保护作用部分源于其自由基清除作用。

Senile rats are divided into model group,control group,Yi Yuan Huo Xue Dan dose group,rhubard group,Xue Shuan Xin Mai Ning group,and compared with the Nimodipine group.Observed are the pathologic changes of the brain,heart,liver,kidney,stomach,and small intestines as well as the content of E,NE and DA 30min after cerebral ischemia occurs in those rats followed by 60min of perfusion.It’s shown that rhubarb,Yi ...

Senile rats are divided into model group,control group,Yi Yuan Huo Xue Dan dose group,rhubard group,Xue Shuan Xin Mai Ning group,and compared with the Nimodipine group.Observed are the pathologic changes of the brain,heart,liver,kidney,stomach,and small intestines as well as the content of E,NE and DA 30min after cerebral ischemia occurs in those rats followed by 60min of perfusion.It’s shown that rhubarb,Yi Yuan Huo Xue Dan,and Xue Shuan Xin Mai Ning have a protective action on the multi organic injury as the result of perfusion after cerebral ischemia.The mechanism has something to do with the inhibition of sympathoadreanal system excitation.

将老龄大鼠分为模型组、对照组、益元活血丹大小剂量组、大黄组、血栓心脉宁组 ,并与尼莫地平组对照 ,观察各组老龄大鼠全脑缺血 30min再灌注 6 0min药物对脑、心、肝、肾、胃、小肠病理改变和E、NE、DA含量的作用。结果显示 ,大黄和益元活血丹及血栓心脉宁对老龄大鼠脑缺血再灌注引起多个器官损伤有不同程度的保护作用 ,其机制与抑制交感—肾上腺系统兴奋增强有关

The mechanisms of the protective effects of Yiyuanhuoxue Pellets (YHP), rhubarb and Xueshuanxinmaining (XXN) against myocardial injury caused by cerebral ischemia reperfusion in aged rats were studied. The aged rats were divided into the control group, model group, nimodipine group, large dose YHP group, small dose YHP group, rhubarb group and XXN group; and the changes in the levels of lactate dehydrogenase (LDH), creatine phosphokinase (CPK), noradrenalin (NE), dopamine (DA),...

The mechanisms of the protective effects of Yiyuanhuoxue Pellets (YHP), rhubarb and Xueshuanxinmaining (XXN) against myocardial injury caused by cerebral ischemia reperfusion in aged rats were studied. The aged rats were divided into the control group, model group, nimodipine group, large dose YHP group, small dose YHP group, rhubarb group and XXN group; and the changes in the levels of lactate dehydrogenase (LDH), creatine phosphokinase (CPK), noradrenalin (NE), dopamine (DA), epinephrine (E), endotheliolysin (ET), calcitonin gene related peptide (CGRP) and neuropeptide Y (NPY) were detected. The results showed that in the rats in the model group, levels of serum CPK and LDH, plasmatic ET and cerebral NPY were increased, and the excitability of sympathetic adrenal system was reinforced; compared with the model group, the levels of serum LDH and CPK were decreased in the rhubarb group, the level of plasmatic ET was decreased in the large dose YHP group, XXN group and the rhubarb group; the level of cerebral NPY was decreased in the rhubarb group and large dose YHP group; the level of plasmatic CGRP was increased in the small dose YHP group; and the excitability of sympathetic adrenal system was decreased in all the groups treated with the above mentioned drugs. The results suggest that the protective effects of rhubarb, XXN and YHP against myocardial injury may relate with their inhibiting the excitability of sympathetic adrenal system and regulating the imbalance between CGRP and ET.

为研究益元活血丹和大黄及血栓心脉宁对老龄大鼠脑缺血再灌注心肌损伤防护作用的机制。将老龄大鼠分为对照组、模型组、尼莫地平组、益元活血丹大小剂量组、大黄组、血栓心脉宁组 ,观察LDH、CPK活性和NE、DA、E、ET、CGRP、NPY的含量的变化。模型组血清中CPK、LDH活性和血浆ET及脑组织NPY含量增高 ,交感 -肾上腺系统兴奋增强。与模型组比较 ,大黄组血清LDH和CPK活性减低 ,益元活血丹大剂量组和血栓心脉宁组及大黄组血浆ET含量、大黄组和益元活血丹大剂量组脑组织NPY含量降低 ,益元活血丹小剂量组血浆中CGRP水平增高 ;用药各组交感 -肾上腺系统兴奋减低。提示大黄和血栓心脉宁及益元活血丹对心肌损伤的保护作用可能与其抑制交感 -肾上腺系统兴奋增强和调节CGRP与ET间的平衡失调有关。

 
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