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nimodipine group
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  尼莫地平组
    METHODS: The experiment was conducted in the laboratory of biochemistry and pharmacology, Institute of Geriatric Cardiovascular Disease, General Hospital of Chinese PLA from March to December 2005. ①Ninety-six healthy Wistar rats were selected and randomly divided into sham-operated group, model group, Nimodipine group and Imi group with 24 rats in each group.
    方法:实验于2005-03/12在解放军总医院老年心血管病研究所生化药理实验室进行。 ①选用健康Wistar大鼠96只,随机分成假手术组、模型组、尼莫地平组和米帕明组4组,每组24只。
短句来源
    ②The rats in the Imipramine group were intraperitoneally (ip) injected 10 mg/kg Imipramine; Nimodipine group was ip injected 2 mg/kg Nimodipine; the sham-operated group and model group were given matching normal saline;
    ②米帕明组腹腔注射米帕明10mg/kg,尼莫地平组腹腔注射尼莫地平2mg/kg,假手术组和模型组腹腔注射等容积生理盐水;
短句来源
    ①NO content: The levels of NO content after reperfusion 2, 12 and 24 hours in the model group, Nimodipine group and Imipramine group were higher than the sham-operated group (P < 0.05);
    ①一氧化氮浓度:模型组、尼莫地平组和米帕明组再灌注2,12和24h均高于假手术组(P<0.05);
短句来源
    the content after reperfusion 2, 12 and 24 hours in the Nimodipine group were lower than the model group (45.99±8.13), (40.63±3.13), (36.72±4.38) μmol/L; (65.54±6.01), (57.08±4.79), (48.13±5.12) μmol/L;
    尼莫地平组再灌注2,12和24h均低于模型组[(45.99±8.13),(40.63±3.13),(36.72±4.38)μmol/L;(65.54±6.01),(57.08±4.79),(48.13±5.12)μmol/L;P均<0.05];
短句来源
    P < 0.05. ②NOS activity: The levels of NOS activity at focal cerebral ischemia 2 hours and reperfusion 2, 12 and 24 hours in the model group, Nimodipine group and Imipramine group were all higher than the sham-operated group (P < 0.05);
    ②一氧化氮合酶活性:模型组、尼莫地平组和米帕明组再灌注2,12和24h均高于假手术组(P<0.05);
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  “nimodipine group”译为未确定词的双语例句
    Results:The total response rate and significant response rate in the nimodipine group were 73.7% and 55.3% respectively, that was absolutely better than those in the control group (41.2%and 14.7% respectively). There was statistically significant difference between the two groups ( P <0.01, P <0.005).
    结果 :尼莫地平治疗组临床疗效评定总有效率为 73.7% ,显效率为 5 5 .3% ,明显优于常规治疗组 (41.2 %、14 .7% ) ,两组比较有显著差异 (P <0 .0 1,P <0 .0 0 5 ) ;
短句来源
    40 Wistar female rats within 1 month were divided into 5 groups randomly with 8 rats each: model group (no use of medicine), nimodipine group(0.2mg/kg), mannitol group(0.5g/kg), nimodipine and mannitol group, sham-operated group (no use of medicine and no clipping process).
    40只 1个月龄雌性Wistar大鼠 ,随机分成 5组 ,每组 8只。 A组 :缺血再灌注尼莫地平 ( 0 2mg/kg)治疗组 ;
短句来源
    Results (1) Compared with control group, rCBFs in CVS group, Nimodipine group and GM 1 therapy group were apparently decreased after second intracisternal blood injection (all P<0.01), but was not apparently decreased in GM 1 prevent group.
    结果  (1)rCBF :二次注血后CVS组、Nim组、GM1 T组的rCBF明显下降 (均P <0 0 1) ,6h时Nim组、GM1 T组的rCBF回升至注血前水平 ,CVS组的rCBF无明显回升 ;
短句来源
    They were apparently increased in Nimodipine group, GM 1 therapy group and GM 1 prevent group (all P<0.01).
    GM1 T组的微血管数目、面积及平均面积较对照组、CVS组显著增加 (均P <0 0 1) ;
短句来源
    Results: On day 31, the patients who developed CVS, DCI and rebleeding in the magnesium sulphate group and the nimodipine group were 15 and 25 cases (-P-<0.05), respectively, including 6 and 8 CVSs (-P->0.05), 3 and 11 DCIs (-P-<0.05), 6 and 7 rebleedings (-P->0.05), respectively.
    结果:第31天时硫酸镁治疗组和尼莫地平治疗组发生CVS、DCI或再出血者分别为15例和26例(P<0.05),其中CVS分别为6例和8例(P>0.05),DCI分别为3例和11例(P<0.05),再出血分别为6例和7例(P>0.05)。
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  nimodipine group
Six months after injury 93 (53%) of the nimodipine group and 86 (49%) of the control group had a favourable outcome (moderate/good recovery).
      
The low blood flow in the nimodipine group might have been a consequence of brain edema caused by extravasates.
      
The underlying cause of death was major bleeding in 6 of 9 deaths, 5 in the nimodipine group and 1 in the placebo group.
      


Sixty patients with acute ischemic cerebral infarction within 72 h were equally divided into 2groups.The nimodipine group 30 patients(M 21,F 9;age 59±s10 a) received nimodipine 2 mg/d intravenous infusion based on supported therapy(dehydrant,vit C)in 1-2 wk,oral nimodipine 60 mg every evening in 1-4 wk.Supported therapy was altered to improve hemodilution,microcirculation,and nervous cell activator in 3-4wk.Control group 30 patients(M23,F7,age 58±9 a) received only supported therapy.Irrespective...

Sixty patients with acute ischemic cerebral infarction within 72 h were equally divided into 2groups.The nimodipine group 30 patients(M 21,F 9;age 59±s10 a) received nimodipine 2 mg/d intravenous infusion based on supported therapy(dehydrant,vit C)in 1-2 wk,oral nimodipine 60 mg every evening in 1-4 wk.Supported therapy was altered to improve hemodilution,microcirculation,and nervous cell activator in 3-4wk.Control group 30 patients(M23,F7,age 58±9 a) received only supported therapy.Irrespective of condition of patients or size of infarction, nervous functional defect scores of the nimodipine group decreased more than those of the control group(P<0.01).The effects of nimodipine showed no difference between those within 30h after onset and those within 31-72h.

发病72h内的急性缺血性脑梗死60例,分为2组。尼莫地平组30例(男性21例,女性9例;年龄59±s10a).wk1,2给支持疗法(脱水剂、维生素C等)和尼莫地平2mg/d于5%葡萄糖液500mL内静脉滴注,wk3,4改用扩容、改善微循环、细胞活性药,wk1-4口服尼莫地平60mg,qn。对照组30例(男性23例,女性7例;年龄58±9a),不给尼莫地平,其余同上。4wk后,前组神经功能缺损积分值较后组下降显著(P<0.01),与病情轻重、病灶大小、治疗早晚无关。*P<0.01。讨论作者采用临床对照研究,静滴尼莫地平治疗发病72h内的缺血性脑梗死,无论病情分级或总体疗效,治疗前后的神经功能缺损的积分差,与对照组比较,差别均有非常显著意义(P<0.01),提示尼莫地平对急性缺血性脑梗死的近期疗效肯定。但是深部小梗死灶引起的中、重型患者,2组疗效差别无显著意义,可能与小梗死灶病理变化轻,不能充分发挥尼莫地平的阻止钙内流和细胞膜崩解、保护半暗带等作用有关[4]。动物模型局灶性脑缺血30min后开始用尼莫地平治疗即不能增加局部脑血流量[5]。用不同剂量的尼莫地平治疗发病48h内的急性脑梗死1064例,发现只有120?

Abstract Severe brain ischemia caused by occlusion of bilateral carotid and vertebral arteries were performed on expermental rabbits.Microcirculation of cerebral piamater were studied through the fenestration of cranial bone by closed-circuit TV microscope system the active changes of microcirculation after the brainresuscitation by Nimodipine were watched directly.In the ischemia group the arteriole began contract 40 min after its distinct dilation.Blood flow velocity decity decreased and part of...

Abstract Severe brain ischemia caused by occlusion of bilateral carotid and vertebral arteries were performed on expermental rabbits.Microcirculation of cerebral piamater were studied through the fenestration of cranial bone by closed-circuit TV microscope system the active changes of microcirculation after the brainresuscitation by Nimodipine were watched directly.In the ischemia group the arteriole began contract 40 min after its distinct dilation.Blood flow velocity decity decreased and part of venule blood stagnated.In Nimodipine group there were no vasospasm found,at the saitie time the arterioles became dilated and lasted more than 100 min.From above detas the authors think Nimodipine could be ameliorate abnormal microcirculation after the brain ischemia.Increase the blood supply of the brain tissue and mitigate reperfusion damage.Therefore,it is suggested that Nimodipine may be used clinically for brain resuscitation .

在夹闭双侧颈总动脉和椎动脉造成严重脑缺血20分钟的基础上,应用微循环闭路电视显微系统行颅骨开窗法对兔脑软脑膜微循环录像,直接动态观察钙拮抗剂尼莫地平对脑复苏后微循环的影响。再灌注期,尼莫地平能显著地扩张微动脉,并持续100分钟以上,明显地延缓延迟性低灌注时相。结果提示:尼莫地平具有不同程度地改善脑缺血后微循环异常,增加脑组织血液供给,减轻再灌注损伤。

The authors enrolled 105 patients with acute ischemic stroke in a clinical study of intravenous nimodipine. The therapeutic effects well evaluated with five-degree scale. During the two weeks treatment period, the results showed that intravenous nimodipine group has a significantly better neurologic outcome than control group. In intravenous nimodipine group, subgroup analysis showed those treated within 36 hours has statistically significantly better neurologic outcome than those without 36...

The authors enrolled 105 patients with acute ischemic stroke in a clinical study of intravenous nimodipine. The therapeutic effects well evaluated with five-degree scale. During the two weeks treatment period, the results showed that intravenous nimodipine group has a significantly better neurologic outcome than control group. In intravenous nimodipine group, subgroup analysis showed those treated within 36 hours has statistically significantly better neurologic outcome than those without 36 hours. Blood pressure in the intravenous nimodipine treated patients decressed, and the effect on blood pressure may be related to concentration or dose. There was no major side effect except for two cases of fever and two cases of hiccup may have been related to treatment with nimodipine.

用国产尼莫地平注射液治疗急性缺血性脑血管病(AICVD)105例,疗效采用量表和5级标准分别评定。结果表明,尼莫地平组2周疗效显著优于对照组,36h内用药疗效显著优于36h后用药,尼莫地平有降压作用,此作用与剂量和滴速有关,无明显副作用。

 
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