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nimodipine groups
相关语句
  尼莫地平组
     METHODS: Forty SD rats were randomly divided into control, injury, cimetidine and nimodipine groups. Cimetidine 0.1 mg/kg, ip, tid, was given to cimetidine group and nimodipine 5 mg/kg, ip, tid, to nimodipine group. The gastric mucosal blood flow(GMBF), gastric fluid pH, and mucosal ulcer index were measured at 72 h after head concussion.
     方法:将40只SD大鼠随机分为对照组、创伤组、西咪替丁及尼莫地平组,共4组,用Dixon等方法致伤后西咪替丁组即予西咪替丁0.1mg/kg,尼莫地平组予尼莫地平5mg/kg,均ip,tid,72h后观察各组大鼠胃粘膜血流量(GMBF),胃液pH及胃粘膜损伤指数。
短句来源
     Methods Retinal ischemia was induced in rabbit by raising the intraocular pressure to 110mmHg(1kPa=7.5mmHg) for 60 minutes via an intrachameral catheter. The seventy two rabbits were randomly divided into control,model and nimodipine groups.
     方法  72只兔随机分为阴性对照组、模型组、尼莫地平组 ,经前房恒压 ( 110mmHg) ( 1kPa =7.5mmHg)灌注生理盐水 6 0min ,建立视网膜缺血损伤的动物模型。
短句来源
     Furthermore, the ECs in LPS group showed a strong immunoreactivity for the polyclonal MCP 1 antibody, whereas the ECs in control, L arginine and nimodipine groups revealed a weak postive immunostaining.
     免疫细胞化学显示 ,MCP 1为胞浆表达 ,LPS组EC对MCP 1多克隆抗体呈强阳性反应 ,而对照组、L 精氨酸组和尼莫地平组EC胞浆中的MCP 1表达呈弱阳性。
短句来源
     ①Bederson score: In the model and nimodipine groups, hemiplegia appeared instantly after the model animals awaked. The score showed stable after 24 hours, peeked at 24-72 hours, then declined, remained certain levels till 5 days.
     ①Bederson评分:模型组、尼莫地平组大鼠醒后迅速出现偏瘫,24h后评分趋于稳定,至72h之间评分最高,然后逐渐下降,5d时仍有体征。
短句来源
     Methods Duplicated cerebral ischemia reperfusion model rats with middle cerebral artery occlusion(MCAO) were divided into the sham-operated group and model groups,Naomaitong groups and Nimodipine groups,the later three groups with ischemia 3 h and I/R 6,12,24 h,3,6 d to observe the pathologic changes,the changes of the water content of cerebral constitution and the content of IgG.
     方法采用线栓法复制局灶性脑缺血模型,分为假手术组、模型组、脑脉通组、尼莫地平组,后3组又分为脑缺血(I)3 h和I/R 6、12、24 h、3、6 d时间点。 观察脑组织含水量、病理变化、免疫球蛋白(IgG)表达。
短句来源
  “nimodipine groups”译为未确定词的双语例句
     the ratio of platelet aggregation and adhesion in low dosage of rhubarb aglycone group decreased obviously as compared with that in aspirin and Nimodipine groups(P< 0.01).
     大黄苷元25.92mg/(kg·d)组大鼠的血小板黏附率和聚集率均较阿司匹林组和尼莫低平组显著降低(P<0.01)。
短句来源
     However,there was no statistical different between hydergine and nimodipine groups( P =0 2631).
     而海得静组与尼莫地平组间的比较的差异无显著性 (P=0 .2 6 31)。
短句来源
     Methods Rats was divided into three groups 1)sham operation group, 2) ischemia groups (middle cerebral artery occlusion) and 3) ischemia plus Nimodipine groups. We examined the phospholipase A 2 activity, cytosolic free Ca 2+ concentrations, brain water contents and the expression of sPLA 2 IIA mRNA and the cPLA2 IVmRNA in cerebral cortex 120 minutes after cerebral ischemia.
     方法 将局灶性脑缺血模型大鼠分为假手术组、缺血组及尼莫地平干预组 ,测定PLA2 活力、脑细胞游离Ca2 +浓度、脑含水量及脑组织PLA2 表达量的改变。
短句来源
  相似匹配句对
     (4)groups, Nimodipine were administered intraperitoneally.
     4 HIBD+尼莫地平治疗 B组。
短句来源
     FUZZY GROUPS I
     Fuzzy 群 1
短句来源
     The therapeutic effects of thethree groups were better than nimodipine group.
     各剂量组疗效均优于尼莫地平。
短句来源
     Compatibility of nimodipine
     尼莫地平输液的配伍实验
短句来源
     On P~s-Regular Groups
     关于P~S—正则群
短句来源
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Random comparative method was taken in patients with essential hypertensive diseases using nimodipine and nifedipine. Twenty five patients (M 24, F 1; age 64+5 a) in the nimodipine group received 30-60 mg, tid-qid, and 25 patients (M 23, F 2; age 65+4 a) in the nifedipine group received 10-20 mg, tid-qid, 4 wk as a course of treatment. The results showed that total response rate was 92% in nimodipine group and 88% in nifedipine group (P>0.05). In both groups the blood pressure...

Random comparative method was taken in patients with essential hypertensive diseases using nimodipine and nifedipine. Twenty five patients (M 24, F 1; age 64+5 a) in the nimodipine group received 30-60 mg, tid-qid, and 25 patients (M 23, F 2; age 65+4 a) in the nifedipine group received 10-20 mg, tid-qid, 4 wk as a course of treatment. The results showed that total response rate was 92% in nimodipine group and 88% in nifedipine group (P>0.05). In both groups the blood pressure decreased markedly (P<0.01), and adverse effects were mild.

原发性高血压病50例,其中采用尼莫地平治疗者25例(男24例,女1例,年龄64±5a)剂量30-60mg,tid-qid.另25例用硝苯啶对照治疗,其中男23例,女2例,年龄65±4a,剂量10-20mg,tid-qid。2组均以4wk为一个疗程。尼莫地平组总有效率92%,硝苯啶组为88%。2组自身对照t检验,P<0.01,组间比较,P>0.05。不良反应均轻微。

Sixty patients with acute ischemic cerebral infarction within 72 h were equally divided into 2groups.The nimodipine group 30 patients(M 21,F 9;age 59±s10 a) received nimodipine 2 mg/d intravenous infusion based on supported therapy(dehydrant,vit C)in 1-2 wk,oral nimodipine 60 mg every evening in 1-4 wk.Supported therapy was altered to improve hemodilution,microcirculation,and nervous cell activator in 3-4wk.Control group 30 patients(M23,F7,age 58±9 a) received only supported therapy.Irrespective...

Sixty patients with acute ischemic cerebral infarction within 72 h were equally divided into 2groups.The nimodipine group 30 patients(M 21,F 9;age 59±s10 a) received nimodipine 2 mg/d intravenous infusion based on supported therapy(dehydrant,vit C)in 1-2 wk,oral nimodipine 60 mg every evening in 1-4 wk.Supported therapy was altered to improve hemodilution,microcirculation,and nervous cell activator in 3-4wk.Control group 30 patients(M23,F7,age 58±9 a) received only supported therapy.Irrespective of condition of patients or size of infarction, nervous functional defect scores of the nimodipine group decreased more than those of the control group(P<0.01).The effects of nimodipine showed no difference between those within 30h after onset and those within 31-72h.

发病72h内的急性缺血性脑梗死60例,分为2组。尼莫地平组30例(男性21例,女性9例;年龄59±s10a).wk1,2给支持疗法(脱水剂、维生素C等)和尼莫地平2mg/d于5%葡萄糖液500mL内静脉滴注,wk3,4改用扩容、改善微循环、细胞活性药,wk1-4口服尼莫地平60mg,qn。对照组30例(男性23例,女性7例;年龄58±9a),不给尼莫地平,其余同上。4wk后,前组神经功能缺损积分值较后组下降显著(P<0.01),与病情轻重、病灶大小、治疗早晚无关。*P<0.01。讨论作者采用临床对照研究,静滴尼莫地平治疗发病72h内的缺血性脑梗死,无论病情分级或总体疗效,治疗前后的神经功能缺损的积分差,与对照组比较,差别均有非常显著意义(P<0.01),提示尼莫地平对急性缺血性脑梗死的近期疗效肯定。但是深部小梗死灶引起的中、重型患者,2组疗效差别无显著意义,可能与小梗死灶病理变化轻,不能充分发挥尼莫地平的阻止钙内流和细胞膜崩解、保护半暗带等作用有关[4]。动物模型局灶性脑缺血30min后开始用尼莫地平治疗即不能增加局部脑血流量[5]。用不同剂量的尼莫地平治疗发病48h内的急性脑梗死1064例,发现只有120?

Abstract Severe brain ischemia caused by occlusion of bilateral carotid and vertebral arteries were performed on expermental rabbits.Microcirculation of cerebral piamater were studied through the fenestration of cranial bone by closed-circuit TV microscope system the active changes of microcirculation after the brainresuscitation by Nimodipine were watched directly.In the ischemia group the arteriole began contract 40 min after its distinct dilation.Blood flow velocity decity decreased and part of...

Abstract Severe brain ischemia caused by occlusion of bilateral carotid and vertebral arteries were performed on expermental rabbits.Microcirculation of cerebral piamater were studied through the fenestration of cranial bone by closed-circuit TV microscope system the active changes of microcirculation after the brainresuscitation by Nimodipine were watched directly.In the ischemia group the arteriole began contract 40 min after its distinct dilation.Blood flow velocity decity decreased and part of venule blood stagnated.In Nimodipine group there were no vasospasm found,at the saitie time the arterioles became dilated and lasted more than 100 min.From above detas the authors think Nimodipine could be ameliorate abnormal microcirculation after the brain ischemia.Increase the blood supply of the brain tissue and mitigate reperfusion damage.Therefore,it is suggested that Nimodipine may be used clinically for brain resuscitation .

在夹闭双侧颈总动脉和椎动脉造成严重脑缺血20分钟的基础上,应用微循环闭路电视显微系统行颅骨开窗法对兔脑软脑膜微循环录像,直接动态观察钙拮抗剂尼莫地平对脑复苏后微循环的影响。再灌注期,尼莫地平能显著地扩张微动脉,并持续100分钟以上,明显地延缓延迟性低灌注时相。结果提示:尼莫地平具有不同程度地改善脑缺血后微循环异常,增加脑组织血液供给,减轻再灌注损伤。

 
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