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damage of blood-brain barrier
相关语句
  血脑屏障的破坏
     Conclusion Early contrast- enhanced MRI is sensitive to reflect damage of blood-brain barrier, helping to predict ensuing hemorrhage.
     结论早期增强MRI检查能灵敏反映血脑屏障的破坏情况,预测继发出血的可能性。
短句来源
  “damage of blood-brain barrier”译为未确定词的双语例句
     Contrast-enhanced MRI in diagnosing damage of blood-brain barrier during reperfusion in rats suffering acute cerebral ischemia
     增强MRI在大鼠急性脑缺血再灌注血脑屏障损伤中的研究
短句来源
     Protective Effects of GInsenosindes and vitamin C on Damage of Blood-brain Barrier in Rats by Low Level Lead Using X-ray Enengy-dispersive Spectroscopy Microanalysis
     用EDX能谱分析研究人参皂甙和维生素C保护铅对血脑屏障损害的作用
短句来源
     These suggested that CSF-TNF may come both from mononuclear cells in CSF,and from blood which was through the damage of blood-brain barrier,also may come from neuroglial cells and vascular endothelial cells.
     这表明TNF一方面可能来源于鞘内的单个核细胞,另一方面由于血脑屏障受损可能来源于血液,另外还可能来源于神经胶质细胞及血管内皮细胞。
短句来源
     Conclusion: EB may protect the brain from ischemia-reperfusion injury by increasing the activity of antioxidase,improving the cerebral blood flow,and alleviating the damage of blood-brain barrier.
     结论:灯盏花素能增加脑缺血再灌注小鼠抗氧化酶的活性,改善脑血流,减轻血脑屏障的损伤,对脑缺血再灌注损伤具保护作用。
短句来源
     Objective To probe role of contrast-enhanced MRI in assessing damage of blood-brain barrier (BBB) during reperfusion after occlusion of right middle cerebral artery in rats.
     目的通过对大鼠局灶脑缺血再灌注的研究评价增强MRI在判定血脑屏障损伤中的作用。
短句来源
  相似匹配句对
     Statins and Blood-Brain Barrier
     他汀类药物与血脑屏障
短句来源
     P-Glycoprotein and Blood-Brain Barrier
     P-糖蛋白与血脑屏障
短句来源
     Blood brain barrier and epilepsy
     血-脑屏障与癫癎
短句来源
     The VBE can be caused by the damage of blood brain barrier (BBB).
     VBE的发生是血脑屏障(blood brain barrier, BBB)通透性增加的结果。
短句来源
     Effects of H7 on blood -brain barrier damage in brain injury
     H7在脑损伤后血脑屏障损害中的作用
短句来源
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The cytokines were determined in the patients of Guillain-Barre syndrome(GBS)and multiple sclerosis(MS).The tumor necrosis factor(TNF)levels in sera and CSF in active MS were significantly higher than those in both stable MS(P=0.022,P=0.047,separately)and control groups)P<0.001)and the TNF levels in GBS during exacerbation were significantly higher than those in both control group(P<0.001)and in treated-GBS(P=0.0015).Otherwise,the TNF levels in CSF of GBS and MS were higher than those in corresponding sera.We...

The cytokines were determined in the patients of Guillain-Barre syndrome(GBS)and multiple sclerosis(MS).The tumor necrosis factor(TNF)levels in sera and CSF in active MS were significantly higher than those in both stable MS(P=0.022,P=0.047,separately)and control groups)P<0.001)and the TNF levels in GBS during exacerbation were significantly higher than those in both control group(P<0.001)and in treated-GBS(P=0.0015).Otherwise,the TNF levels in CSF of GBS and MS were higher than those in corresponding sera.We also found the protein content in CSF of GBS and MS groups was evidently increased(P<0.01).In addition,white blood cell count in CSF of MS was correlated with both CSF-TNF level and CSF-protein content(P<0.01),and CSF-protein content was correlated with TNF level in both CSF(P=0.016)and serum(P=0.001).These suggested that CSF-TNF may come both from mononuclear cells in CSF,and from blood which was through the damage of blood-brain barrier,also may come from neuroglial cells and vascular endothelial cells.TNF might play an important role in early stage of inflammatory demylinat ing diseases.

测定了多发性硬化(MS)和格林-巴利综合征(GBS)患者的细胞因子,发现活动的MS血清和脑脊液(CSF)TNF水平显著高于稳定的MS和对照组。GBS患者急性期CSF和血清TNF水平显著高于对照组及治疗后水平。另外,MS和GBS患者CSF的TNF水平均高于相应的血清水平。通过研究还发现MS和GBS组CSF的蛋白含量显著增高。此外,MS组CSF白细胞数与CSF的TNF水平及CSF蛋白含量相关,且CSF蛋白含量与血清、CSF的TNF水平相关。这表明TNF一方面可能来源于鞘内的单个核细胞,另一方面由于血脑屏障受损可能来源于血液,另外还可能来源于神经胶质细胞及血管内皮细胞。TNF可能在炎性脱鞘病发病初期起作用。

OBJECTIVE:To observe the effect of “Xiaoyuning” on cerebral bematoma.METHODS:The experimental cerebralhematoma models were produced by injecting autogenous clot in therats.The influences of “XYN” on contents of protein,contents of water,oxygen free radical in brain tissue,histomorphology at the third day,seventh day after cerebral hematoma were observed.RESULTS:In the pathological group brain contents of protein and water,malondialdehyde increased progressively,and the decline of activities of superoxide dismutase...

OBJECTIVE:To observe the effect of “Xiaoyuning” on cerebral bematoma.METHODS:The experimental cerebralhematoma models were produced by injecting autogenous clot in therats.The influences of “XYN” on contents of protein,contents of water,oxygen free radical in brain tissue,histomorphology at the third day,seventh day after cerebral hematoma were observed.RESULTS:In the pathological group brain contents of protein and water,malondialdehyde increased progressively,and the decline of activities of superoxide dismutase and glutathione peroxidase were obvious,especially in side of hematoma(right brain).CONCLUSION:“XYN” was advantageous in thetreatment of encephall edema induced by experiment cerebal hematoma,and it suggest that “XYN” had the evident effects in antagonizing the damage of blood brain barrier and encephalic cell caused by free radical in cerebral hematoma.

目的 :为探讨消瘀宁颗粒剂治疗脑出血中风的机制。方法 :以大鼠自体血凝块复制脑出血模型 ,用干湿比重法测脑组织水份 ,同时观察氧自由基 (O÷2 )有关参数与脑组织学的变化。结果 :脑血肿形成后 ,脑组织水份、脑蛋白、丙二醛含量明显增加 ;脑组织超氧化物歧化酶 谷胱甘肽过氧化物酶活性降低。结论 :消瘀宁颗粒剂有明显减轻脑血肿时O÷2 连锁反应对血脑屏障与脑细胞的损害作用 ,促进脑组织内血肿的吸收 ,对脑水肿有明显的防治作用

Objective:To establish a novel hypoxia ischemia model of animal.Methods:The entire uterus including the foetuses were obtained from female mice on the last day of gestation for experiment,and were incubated in at 37℃,divided into one control group and 2 asphyctic groups (15 min and 30 min).The time of asphyxia was measured from the blood circulation and the uterus was cut off till completion of the cesarean section when the uterus was opened and the pups were taken out.Results:(1)Pinocytotic vesicles of endothelia...

Objective:To establish a novel hypoxia ischemia model of animal.Methods:The entire uterus including the foetuses were obtained from female mice on the last day of gestation for experiment,and were incubated in at 37℃,divided into one control group and 2 asphyctic groups (15 min and 30 min).The time of asphyxia was measured from the blood circulation and the uterus was cut off till completion of the cesarean section when the uterus was opened and the pups were taken out.Results:(1)Pinocytotic vesicles of endothelia increased and astrocytic foot swelled after 15 min of hypoxia ischemia.(2)Mitochondria of endothelia swelled,basement membrane of microvessels disappered and edema around capillaries after 30 min of hypoxia ischemia appeared.Conculsion:It is suggested that the damage of blood brain barrier is originated from vessels,and is earlier than neuronal death,and a novel animal model of asphgxia is established,providing a new way for the study of asphyxia of new borns.

目的 :建立可靠的缺氧缺血动物模型。方法 :怀孕 2 1天小白鼠 ,剖腹后将含胎鼠的整个子宫置于 3 7℃水浴中 ,分别于水浴 1 5min、3 0min取胎鼠脑 (剖腹后立即取胎鼠脑作为空白对照 )。结果 :缺氧缺血 1 5min ,脑毛细血管内皮细胞吞饮小泡增多 ,部分星形胶质细胞脚板肿胀、血管周隙扩张 ,神经元无异常改变 ;缺氧缺血 3 0min ,毛细血管周围基膜样物质减少 ,血管周隙极度扩张 ,星形胶质细胞脚板大空泡样变 ,神经元显著肿胀 ;随着缺氧缺血时间的延长 ,脑含水量呈递增趋势 (P <0 .0 1 )。提示 :血脑屏障受损先于神经元 ,系血管源性脑水肿。对孕鼠实施“延迟剖宫产术” ,为新生儿缺氧缺血性脑病模型制作提供了一种新的可靠的实验方法

 
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