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inhibitory trend
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  抑制倾向
     Conclusion:SR could enhance the function of MΦ in normal mice and promote the proliferation of immunologic cells in the spleen,but there was the inhibitory trend on the function of activated MΦ.
     结论 ,槐定碱可增强正常小鼠 MΦ功能 ,并促进脾脏免疫细胞增殖 ,而对活化 MΦ的功能则表现抑制倾向
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  “inhibitory trend”译为未确定词的双语例句
     SR didn't affect the SOD activity,but there was the inhibitory trend on the NO production and the LSZ activity of the peritoneal MΦ activated by thioglycollate(TG)(no statistical significance).
     槐定碱不影响腹腔活化 MΦ内 SOD活性 ,但对腹腔活化 MΦ的 NO诱生量及胞内 L SZ活性有抑制趋势。
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     (2)5×10 8 to 1×10 7/L DC could have a trend of enhancing the killing activity of different effector/target(E/T)LAK cells. However,1×10 7/L to 5×10 7/L DC showed an inhibitory trend.
     (2)5×105~1×107/LDC有增强不同E/TLAK杀伤活性的趋势,而1×107~5×107/LDC对LAK活性有抑制趋势。
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     Conclusion:Ethanol has a direct inhibitory trend on small intestine smooth muscle.
     结论 :乙醇对小肠平滑肌活动有抑制性趋势。
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  相似匹配句对
     TREND
     趋势
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     Industrial Trend
     产业动态
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     inhibitory rate;
     抑制率;
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     Conclusion:Ethanol has a direct inhibitory trend on small intestine smooth muscle.
     结论 :乙醇对小肠平滑肌活动有抑制性趋势。
短句来源
     Inhibitory Glycine Receptor
     甘氨酸受体的研究进展
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  inhibitory trend
In this way, pleural neurons also introduce Pd13 neurons into the inhibitory trend when activated by intensive tactile stimulation.
      
Interestingly, the same inhibitory trend was found in the naming task of Experiment 2.
      
Within the 30-min experimental time frame, ouabain, a Na+/K+ pump inhibitor, had no significant effect on RVD (despite an inhibitory trend), cell swelling or on isotonic volume (n=6).
      
Results showed the significantly elevated activity of Ca2+-ATPase in the cells exposed to 1.0 and 5.0 mg/L fluoride (p >amp;lt; 0.05), and this enzyme activity indicated inhibitory trend in cells of the 7.5- and 12.5-mg/L fluoride-treated group.
      


Factorial design and orthogonal design were adopted to examine the function of dendritic cells(DC)in the system of lymphokin activated killer cells (LAK) anti HPBALL tumor celler.In the mean time,the interactive morphological character of DC,LAK and HPBALL cells was observed under light and transmission electron microscope,and programmed cell deaath assay was applied to research whether or not HPBALL cells had been triggered to apoptosis. The results were as follows:(1)DC could not kill HPBALL cells directly.(2)5×10...

Factorial design and orthogonal design were adopted to examine the function of dendritic cells(DC)in the system of lymphokin activated killer cells (LAK) anti HPBALL tumor celler.In the mean time,the interactive morphological character of DC,LAK and HPBALL cells was observed under light and transmission electron microscope,and programmed cell deaath assay was applied to research whether or not HPBALL cells had been triggered to apoptosis. The results were as follows:(1)DC could not kill HPBALL cells directly.(2)5×10 8 to 1×10 7/L DC could have a trend of enhancing the killing activity of different effector/target(E/T)LAK cells.However,1×10 7/L to 5×10 7/L DC showed an inhibitory trend.(3)The most optimal combined elements of DC,LAK for killing HPBALL cells was:DC cultured for 4 days concentration 5×10 6/L,E/T=10/1 LAK without IL 2.(4)It was also observed that DC could contact and cluster with LAK and HPBALL cells.(5)The positive reaction of programmed cell death apperaed in HPBALL cells. The results indicated that DC could regulate the cytotoxity of LAK killing HPBALL cells in some degree.

为探讨树突状细胞(DC)在LAK抗HPBALL细胞中的作用,采用多因素、多水平的杀伤试验,同时以光镜、电镜观察DC、LAK、HPBALL相互作用的形态特征及DNA断端标记法检测瘤细胞是否凋亡。结果表明:(1)DC无直接杀伤HPBALL作用。(2)5×105~1×107/LDC有增强不同E/TLAK杀伤活性的趋势,而1×107~5×107/LDC对LAK活性有抑制趋势。(3)DC、LAK杀伤HPBALL的最佳组合条件为:DC培养4d、浓度5×106/L,LAKE/T=10/1,rIL-2=0。(4)光镜、电镜下均可见DC的突起与LAK、HPBALL细胞紧密接触形成细胞簇。(5)DNA断端标记法显示瘤细胞呈末端脱氧核糖核酸转移酶阳性反应。结论:DC对LAK杀伤HPBALL活性具有双向调节作用

Objective To compare the strain differences of tumor necrosis factor alpha(TNF\|α) and nitric oxide (NO)production of alveolar macrophages(AMs)originated from Brown Norway(BN) and Lewis(LW)rats. Methods The AMs were collected by the bronchoalveolar lavage method,and exposed to concentrations of HgCl\-2(0\^1~ 30 μmol/L ) for fifferent times(12~ 96 h ).The relative LDH activities in the medium were measured to assess the cytotoxicity.The TNF\|α concentration was measured by an ELISA kit and the nitrite...

Objective To compare the strain differences of tumor necrosis factor alpha(TNF\|α) and nitric oxide (NO)production of alveolar macrophages(AMs)originated from Brown Norway(BN) and Lewis(LW)rats. Methods The AMs were collected by the bronchoalveolar lavage method,and exposed to concentrations of HgCl\-2(0\^1~ 30 μmol/L ) for fifferent times(12~ 96 h ).The relative LDH activities in the medium were measured to assess the cytotoxicity.The TNF\|α concentration was measured by an ELISA kit and the nitrite was detected with Griess regent. Results The HgCl\-2 was not cytotoxic to the AMs at the concentration of \{3 μmol/L\} within 72 h of incubation.In the control groups the concentrations of TNF\|α and nitrite were (609\^45±111\^94)pg/m and (1\^868±0\^165)μmol/ml,respectively,in BN,which were significantly higher than those in LW strain[(340\^62±44\^7)pg/ml and (1\^313±0\^084)μmol/ml].The production of TNF\|α and NO in both strains were inhibited to some extent by mercuric chloride. Conclusion There are some strain differences in the production of TNF\|α and NO between BN and LW strain of rats.Mercuric chloride inhibited the production of TNF\|α and NO.The inhibitory trends were more serious in BN compared with those in LW.

【目的】 比较BrownNorway(BN)和Levis(LE)两种品系大鼠肺泡巨噬细胞产生肿瘤坏死因子 (TNF α)和一氧化氮 (NO)的能力及对氯化汞的反应是否存在差别。【方法】 用肺泡灌洗法收集肺泡巨噬细胞进行培养 ,加入不同浓度的氯化汞 (0 1~ 30 μmol/L) ,处理不同的时间 (12~ 96h)后 ,测定培养基中LDH的相对活性 ,以反映细胞毒性 ,并测定TNF α和亚硝酸盐的含量。【结果】 氯化汞在 3μmol/L以下 ,培养 48h对肺泡巨噬细胞无明显细胞毒性。对照组BN和LW品系TNF α的浓度分别为 (6 0 9 45± 111 94) pg/ml和 (340 6 2± 44 74)pg/ml,两者相比 ,差异有显著性。亚硝酸盐的浓度分别为 (1 86 8± 0 16 5 ) μmol/ml和 (1 313± 0 0 84) μmol/ml,差异也有显著性。氯化汞对TNF α和NO的生成都有一定程度的抑制。【结论】 两种品系大鼠肺泡巨噬细胞体外生成TNF α和NO的量有所不同 ,氯化汞能抑制两种品系大鼠的肺泡巨噬细胞产生TNF α和NO ,抑制作用以BN品系为明显 ,但...

【目的】 比较BrownNorway(BN)和Levis(LE)两种品系大鼠肺泡巨噬细胞产生肿瘤坏死因子 (TNF α)和一氧化氮 (NO)的能力及对氯化汞的反应是否存在差别。【方法】 用肺泡灌洗法收集肺泡巨噬细胞进行培养 ,加入不同浓度的氯化汞 (0 1~ 30 μmol/L) ,处理不同的时间 (12~ 96h)后 ,测定培养基中LDH的相对活性 ,以反映细胞毒性 ,并测定TNF α和亚硝酸盐的含量。【结果】 氯化汞在 3μmol/L以下 ,培养 48h对肺泡巨噬细胞无明显细胞毒性。对照组BN和LW品系TNF α的浓度分别为 (6 0 9 45± 111 94) pg/ml和 (340 6 2± 44 74)pg/ml,两者相比 ,差异有显著性。亚硝酸盐的浓度分别为 (1 86 8± 0 16 5 ) μmol/ml和 (1 313± 0 0 84) μmol/ml,差异也有显著性。氯化汞对TNF α和NO的生成都有一定程度的抑制。【结论】 两种品系大鼠肺泡巨噬细胞体外生成TNF α和NO的量有所不同 ,氯化汞能抑制两种品系大鼠的肺泡巨噬细胞产生TNF α和NO ,抑制作用以BN品系为明显 ,但总的趋势相似

Objective:To observe the effects of Sophoridine (SR) on the function of macrophages (MΦ).Methods:YC-rosettes forming test,nitric oxide(NO) production of activated MΦ,activities of lysozyme(LSZ) and superoxide dismutase(SOD) in activated MΦ,histologic changes of spleen and thymus of mice were used as the parameters of observation.Results:The number of C 3b receptors on the peritoneal macrophages from SR-treated mice were higher than that of the control.Compared with the control,the malpighian corpuscles of spleen...

Objective:To observe the effects of Sophoridine (SR) on the function of macrophages (MΦ).Methods:YC-rosettes forming test,nitric oxide(NO) production of activated MΦ,activities of lysozyme(LSZ) and superoxide dismutase(SOD) in activated MΦ,histologic changes of spleen and thymus of mice were used as the parameters of observation.Results:The number of C 3b receptors on the peritoneal macrophages from SR-treated mice were higher than that of the control.Compared with the control,the malpighian corpuscles of spleen were enlarged and the number of MΦ were increased in the splenic tissue.SR didn't affect the SOD activity,but there was the inhibitory trend on the NO production and the LSZ activity of the peritoneal MΦ activated by thioglycollate(TG)(no statistical significance).Conclusion:SR could enhance the function of MΦ in normal mice and promote the proliferation of immunologic cells in the spleen,but there was the inhibitory trend on the function of activated MΦ.

观察槐定碱对巨噬细胞功能的影响。以 YC花环试验、活化 MΦ的 NO诱生量及胞内溶菌酶与超氧化物岐化酶的活性、小鼠脾脏和胸腺的组织学形态变化为观察指标。结果 ,灌胃槐定碱小鼠的腹腔 MΦC3 b受体数量较对照组显著升高 ;与对照组比较 ,脾组织中脾小体融合增大 ,MΦ增多。槐定碱不影响腹腔活化 MΦ内 SOD活性 ,但对腹腔活化 MΦ的 NO诱生量及胞内 L SZ活性有抑制趋势。结论 ,槐定碱可增强正常小鼠 MΦ功能 ,并促进脾脏免疫细胞增殖 ,而对活化 MΦ的功能则表现抑制倾向。

 
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